Search results for "Nitroprusside"

showing 10 items of 68 documents

Altered electrical activity in colonic smooth muscle cells from dystrophic (mdx) mice

2001

Because the colon from dystrophic (mdx) mice shows an altered motor pattern, probably due to neural disorders, our aim was to examine the electrophysiological properties of muscle cells and the functionality of nitrergic transmission in circular muscle from normal and mdx colon. Normal colonic cells (resting membrane potential [RMP] about -50 mV) showed spontaneous hyperpolarizations (inhibitory junction potentials; IJPs) and cyclic slow depolarizations were sometimes recorded. Mdx colon had a depolarized RMP (about -36 mV) and spontaneous IJPs, but the cyclic activity was never observed. In the normal colon, Nomega-nitro-L-arginine methyl ester (L-NAME) induced depolarization and abolished…

MaleDuchenne muscular dystrophymedicine.medical_specialtyInhibitory junction potentialColonPhysiologyDuchenne muscular dystrophyInhibitory postsynaptic potentialSynaptic TransmissionSettore BIO/09 - FisiologiaProximal colonMembrane PotentialsMiceSmooth muscleInternal medicinemedicineAnimalsMyocyteEnzyme InhibitorsMembrane potentialNeuroscience (all)Endocrine and Autonomic SystemsChemistryGastroenterologyMuscle SmoothNitric oxideDepolarizationMuscular Dystrophy AnimalHyperpolarization (biology)medicine.diseaseElectric StimulationElectrophysiologyMice Inbred C57BLMuscular Dystrophy DuchenneMdx miceElectrophysiologyNG-Nitroarginine Methyl EsterEndocrinologyMice Inbred mdxSodium nitroprussideNeurosciencemedicine.drugNeurogastroenterology and Motility
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Sulfhydryl G Proteins and Phospholipase A2-Associated G Proteins Are Involved in Adrenergic Signal Transduction in the Rat Pineal Gland

2001

The rat pineal gland with its circadian noradrenaline-regulated melatonin rhythm is an excellent model for studying adrenergic signal transduction with respect to cAMP and cGMP formation. The stimulatory G(s) proteins play a well-established role in this process. In contrast, the potential roles of the inhibitory G(i) proteins, the functionally unclear other G(o) proteins, and a number of G protein subtypes are not known. The present study examines the effects on beta(1)- and beta(1)-plus-alpha(1)-stimulated cAMP and cGMP formation of a number of G protein modulators in rat pinealocyte suspension cultures. The effects of the nitric oxide donor sodium nitroprusside on cGMP were also examined…

MaleNitroprussideArylamine N-AcetyltransferaseG proteinAdrenergicWasp VenomsPhospholipaseBiologyNitric OxidePertussis toxinBenzylisoquinolinesPineal GlandPhospholipases APinealocyteRats Sprague-DawleyPhenylephrineAlkaloidsEndocrinologyPhospholipase A2GTP-Binding ProteinsCyclic AMPAnimalsp-Methoxy-N-methylphenethylamineVirulence Factors BordetellaCyclic GMPSulfhydryl ReagentsIsoproterenolAdrenergic beta-AgonistsRatsReceptors AdrenergicPhospholipases A2Pertussis ToxinBiochemistryEthylmaleimideMastoparanbiology.proteinIntercellular Signaling Peptides and ProteinsAnimal Science and ZoologySignal transductionPeptidesAdrenergic alpha-AgonistsSignal TransductionGeneral and Comparative Endocrinology
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No improvement in perfusion and oxygenation of experimental tumors upon application of vasodilator drugs

2001

The oxygen deficiency seen in solid tumors is predominantly caused by an insufficient O2 supply as a result of inadequate tumor perfusion. The aim of this study was to analyze whether a number of vasodilator drugs might be suitable to increase tumor perfusion and consequently improve the oxygenation status of experimental tumors. Rats with s.c. DS-sarcomas were treated with either Na+-nitroprusside (7-25 microg x min(-1) x kg(-1) BW) or nifedipine (10 microg x min(-1) x kg(-1) BW). Red blood cell (RBC) flux was assessed continuously using laser-Doppler flowmetry and mean tumor pO2 was measured polarographically using O2-sensitive catheter electrodes. Systemic application of the vasodilator …

MaleNitroprussideCancer Researchmedicine.medical_specialtyNifedipineVasodilator AgentsRats Sprague-DawleyNifedipineInternal medicineLaser-Doppler FlowmetrymedicineAnimalsInfusions IntravenousOncogenebusiness.industryOxygenationLaser Doppler velocimetryRatsOxygenRed blood cellmedicine.anatomical_structureEndocrinologyOncologyRegional Blood FlowCardiologyVascular resistanceSarcoma ExperimentalSodium nitroprussidebusinessPerfusionmedicine.drugInternational Journal of Oncology
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Nitric oxide induces muscular relaxation via cyclic GMP-dependent and -independent mechanisms in the longitudinal muscle of the mouse duodenum

2003

The aim of this study was to investigate, in mouse duodenum, the role of nitric oxide (NO) in the relaxation of longitudinal muscle evoked by nerve activation and the coupled action mechanism. Electrical field stimulation (EFS; 0.5ms, 10-s train duration, supramaximal voltage, at various frequencies) under nonadrenergic noncholinergic conditions evoked muscular relaxation occasionally followed, at the higher stimulus frequencies, by rebound contractions. Inhibition of the synthesis of NO by Nω-nitro-L-arginine methyl ester (L-NAME; 100μM) virtually abolished the evoked relaxation. The relaxation was reduced also by apamin (0.1μM) and by 1H-[1,2,4]oxadiazolo[4,3-a]quinoxalin-1-one (ODQ; 1μM)…

MaleNitroprussideCancer Researchmedicine.medical_specialtyPotassium ChannelsDuodenumPhysiologyMuscle RelaxationClinical BiochemistryNonadrenergic noncholinergic relaxationStimulationStimulus (physiology)Inhibitory postsynaptic potentialApaminSettore BIO/09 - FisiologiaBiochemistryNitric oxideMicechemistry.chemical_compoundInternal medicineK+ -channelmedicineAnimalsCyclic GMPMolecular BiologyDose-Response Relationship DrugMuscle SmoothNitric oxideElectric StimulationDose–response relationshipEndocrinologychemistryTetrodotoxinSodium nitroprussideMouse duodenummedicine.drugNitric Oxide
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Spontaneous mechanical activity and evoked responses in isolated gastric preparations from normal and dystrophic (mdx) mice

2002

This study examined whether alterations of the spontaneous and evoked mechanical activity are present in the stomach of the mdx mouse, the animal model for Duchenne muscular dystrophy. The gastric mechanical activity from whole-organ of normal and mdx mice was recorded in vitro as changes of intraluminal pressure. All gastric preparations developed spontaneous tone and phasic contractions, although the tone of the mdx preparations was significantly greater. Atropine reduced the tone of the two preparations by the same degree. Nomega-nitro-l-arginine methyl ester (l-NAME) significantly increased the tone and spontaneous contractions only in the stomach from normal animals, but did not affect…

MaleNitroprussideDuchenne muscular dystrophymedicine.medical_specialtymdx mouseContraction (grammar)PhysiologyDuchenne muscular dystrophyTetrodotoxinCholinergic AgonistsSettore BIO/09 - FisiologiaContractilityMicechemistry.chemical_compoundOrgan Culture TechniquesInternal medicinemedicineAnimalsNitric Oxide Donorsmdx mouseAnesthetics LocalEnzyme InhibitorsNeuroscience (all)Endocrine and Autonomic SystemsChemistryStomachStomachGastroenterologyMuscle SmoothNitric oxideAnatomyMuscular Dystrophy AnimalGastric smooth musclemedicine.diseaseElectric StimulationMuscular Dystrophy DuchenneGastric mechanical activityAtropineNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologyMice Inbred mdxTetrodotoxinCholinergicCarbacholMuscle Contractionmedicine.drug
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The Effects of Sodium Nitroprusside-Induced Hypotension on Splanchnic Perfusion and Hepatocellular Integrity

1999

UNLABELLED The purpose of our study was to investigate the effects of sodium nitroprusside-induced hypotension on splanchnic perfusion and hepatocellular integrity. Thirty patients undergoing radical prostatectomy were allocated randomly to a sodium nitroprusside (SNP) or control group (control). Regional pco2 was measured using gastric tonometry, and the regional to arterial difference in partial pressure of CO2 and intramucosal pH were calculated. The cytosolic liver enzyme alpha-glutathione S-transferase and standard liver enzyme markers (alanine aminotransferase, aspartate aminotransferase, and gamma-glutamyltransferase) were also measured. Mean arterial pressure in the SNP group was 50…

MaleNitroprussideMean arterial pressuremedicine.medical_specialtyPartial PressureVasodilator AgentsUrologyHemodynamicsBlood PressurepCO2medicineHumansSplanchnic CirculationGastric tonometryAntihypertensive AgentsGlutathione TransferaseProstatectomybusiness.industryLiver DiseasesCarbon DioxideMiddle AgedIsoenzymesOxygenAnesthesiology and Pain MedicineLiverAnesthesiaSodium nitroprussideLiver functionChemical and Drug Induced Liver InjuryHypotensionSplanchnicbusinessPerfusionBiomarkersmedicine.drugAnesthesia & Analgesia
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Effects Of COOH-terminal tripeptide alpha-MSH (11-13) on corneal epithelial wound healing:role of nitric oxide

2006

It is known that alpha-melanocyte stimulating hormone (alpha-MSH) may exert anti-inflammatory effects and facilitate reparative processes in different tissues. The effective message sequence of alpha-MSH resides in the COOH-terminal tripeptide alpha-MSH(11-13). This study was undertaken to investigate the effects of topical administration of the COOH-terminal tripeptide sequence of alpha-MSH (alpha-MSH(11-13), KPV) on corneal epithelial wound healing in rabbits and the possible role of nitric oxide (NO) in these effects. The whole corneal epithelium was denuded in both eyes by mechanical abrasion. The area of the corneal epithelial defect was stained with fluorescein, photographed, and then…

MaleNitroprussideMelanocyte-Stimulating Hormonemedicine.medical_treatmentCorneal abrasionRabbitPharmacologyKPVNitric OxideNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundPeptide Fragmentα-MSHmedicineAnimalsEnzyme InhibitorNitric Oxide DonorsMelanocyte-Stimulating HormonesFluoresceinEnzyme InhibitorsSalineCells CulturedCorneal epitheliumCell ProliferationEpithelial CellWound HealingbiologyDose-Response Relationship DrugAnimalcorneal wound healingEpithelium CornealEpithelial CellsNitric Oxide Donormedicine.diseaseSensory SystemsPeptide FragmentsNitric oxide synthaseOphthalmologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterchemistryBiochemistrybiology.proteinSodium nitroprussideRabbitsWound healingmedicine.drug
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In rat pinealocytes the cyclic GMP response to NO is regulated by Ca2+ and protein kinase C

1995

There is ample evidence that beta-adrenergic stimulation of cyclic GMP formation is potentiated by alpha1-adrenergic mechanisms, the latter leading to elevation of intracellular Ca2+ concentration ([Ca2+]i) and protein kinase C (PKC) activation. Recent studies have shown that nitric oxide synthase (NOS) and nitric oxide (NO) are a component of the adrenoceptor-cyclic GMP signalling pathway. The aim of the present investigation was to study the roles of alpha1-adrenergic mechanisms, Ca2+ and PKC on NO-stimulated cyclic GMP formation. To this end suspension cultures of rat pinealocytes were treated with the NO donor sodium nitroprusside (SNP) in the presence of alpha1-adrenergic agonists, [Ca…

MaleNitroprussidePhosphodiesterase InhibitorsNitric OxidePineal GlandPinealocyteNitric oxideRats Sprague-DawleyPhenylephrinechemistry.chemical_compoundCalmodulinmedicineAnimalsOuabainCyclic GMPProtein Kinase InhibitorsMolecular BiologyProtein Kinase CProtein kinase CbiologyKinaseGeneral NeuroscienceIsoproterenolPhosphodiesterasePhosphoric Monoester HydrolasesRatsNitric oxide synthaseCytosolBiochemistrychemistrybiology.proteinCalciumNeurology (clinical)Sodium nitroprussideDevelopmental Biologymedicine.drugBrain Research
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Inhaled Prostacyclin, Nitric Oxide, and Nitroprusside in Pulmonary Hypertension After Mitral Valve Replacement

2005

Abstract Objective: Pulmonary hypertension increases morbidity and mortality in patients undergoing heart surgery. Mitral valve stenosis is frequently associated with an increase in pulmonary vascular resistance (PVR). Cardiopulmonary bypass exacerbates pulmonary hypertension in patients undergoing cardiac surgery. The aim of this study was to compare the hemodynamic effects of inhaled prostacyclin and nitric oxide and the administration of i.v. nitroprusside during cardiac surgery with a clinical, pharmacodynamic dose-response, prospective, randomized, and double-blind study (Group A: inhaled prostacyclin; Group B: inhaled nitric oxide; Group C: nitroprusside). Materials and Methods: Fifty…

MaleNitroprussidePulmonary and Respiratory Medicinemedicine.medical_specialtyCardiac outputHypertension Pulmonarymedicine.medical_treatmentProstacyclinNitric OxideNitric oxidechemistry.chemical_compoundMitral valve stenosisDouble-Blind MethodInternal medicineAdministration InhalationmedicineHumansMitral Valve StenosisAntihypertensive AgentsEndothelium-Dependent Relaxing FactorsHeart Valve Prosthesis ImplantationCardiopulmonary Bypassbusiness.industryMitral valve replacementMiddle Agedmedicine.diseaseEpoprostenolPulmonary hypertensionCardiac surgerymedicine.anatomical_structurechemistryAnesthesiacardiovascular systemCardiologyVascular resistanceMitral ValveFemaleSurgeryCardiology and Cardiovascular Medicinebusinessmedicine.drugJournal of Cardiac Surgery
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Mechanisms underlying the nitric oxide inhibitory effects in mouse ileal longitudinal muscle

2005

We investigated the mechanisms involved in the nitric oxide (NO)-induced inhibitory effects on longitudinal smooth muscle of mouse ileum, using organ bath technique. Exogenously applied NO, delivered as sodium nitroprusside (SNP; 0.1–100 µmol/L) induced a concentration-dependent reduction of the ileal spontaneous contractions. 1H-[1,2,4]oxadiazolol[4,3,a]quinoxalin-1-one (ODQ; 1 µmol/L), a guanilyl cyclase inhibitor, reduced the SNP-induced effects. Tetraethylammonium chloride (20 mmol/L), a non-selective K+ channel blocker, and charybdotoxin (0.1 µmol/L), blocker of large conductance Ca2+-dependent K+ channels, significantly reduced SNP-induced inhibitory effects. In contrast, apamin (0.1…

MaleNitroprussideThapsigarginCharybdotoxinPhysiologyMouse ileumIn Vitro TechniquesPharmacologyApaminSettore BIO/09 - FisiologiaPotassium channelsMicePotassium Channels Calcium-Activatedchemistry.chemical_compoundIleumPhysiology (medical)Cyclic GMP-Dependent Protein KinasesPotassium Channel BlockersmedicineAnimalsNitric Oxide DonorsChannel blockerCyclic GMPPharmacologyRyanodineRyanodine receptorCalcium storeMuscle SmoothPotassium channel blockerNitric oxideGeneral MedicineTetraethylammonium chlorideMice Inbred C57BLchemistryCalciumSodium nitroprussideMuscle ContractionSignal Transductionmedicine.drug
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