Search results for "Nitroprusside"

showing 10 items of 68 documents

NANC inhibitory neurotransmission in mouse isolated stomach: Involvement of nitric oxide, ATP and vasoactive intestinal polypeptide

2003

1. The neurotransmitters involved in NANC relaxation and their possible interactions were investigated in mouse isolated stomach, recording the motor responses as changes of endoluminal pressure from whole organ. 2. Field stimulation produced tetrodotoxin-sensitive, frequency-dependent, biphasic responses: rapid transient relaxation followed by a delayed inhibitory component. 3. The inhibitor of the synthesis of nitric oxide (NO), L-NAME, abolished the rapid relaxation and significantly reduced the slow relaxation. Apamin, blocker of Ca 2+-dependent K + channels, or ADPβS, which desensitises P 2y purinoceptors, reduced the slow relaxation to 2-8 Hz, without affecting that to 16-32 Hz or the…

NitroprussideMuscle RelaxationNANC inhibitory neurotransmitterNitric OxideSynaptic TransmissionSettore BIO/09 - FisiologiaGastric relaxationMiceAdenosine TriphosphateAdrenergic FiberChymotrypsinEnzyme InhibitorThionucleotideCholinergic FiberPharmacologyDose-Response Relationship DrugAnimalIn Vitro TechniqueMouse stomachStomachNitric Oxide DonorElectric StimulationATPVIPAdenosine DiphosphateMice Inbred C57BLNG-Nitroarginine Methyl EsterApaminReceptors Vasoactive Intestinal PeptideNitric Oxide SynthaseVasoactive Intestinal Peptide
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Effects of fenspiride on human bronchial cyclic nucleotide phosphodiesterase isoenzymes: functional and biochemical study.

1998

We have investigated the role of human bronchial cyclic nucleotide phosphodiesterases in the effects of fenspiride, a drug endowed with bronchodilator and anti-inflammatory properties. Functional studies on human isolated bronchi showed that fenspiride (10(-6)-3 x 10(-3) M, 30 min) induced a shift to the left of the concentration-response curves for isoprenaline and sodium nitroprusside with -logEC50 values of 4.1+/-0.1 (n = 7) and 3.5+/-0.2 (n = 8), respectively. Biochemical studies were carried out on three human bronchi in which separation of cyclic nucleotide phosphodiesterase isoenzymes was performed by ion exchange chromatography followed by determination of phosphodiesterase activity…

NitroprussideMuscle RelaxationVasodilator AgentsPhosphodiesterase 3FenspirideBronchimedicineHumansSpiro CompoundsPharmacologyCyclic nucleotide phosphodiesterasebiologyDose-Response Relationship DrugChemistryIsoproterenolPhosphodiesteraseBronchodilator AgentsIsoenzymesBiochemistryEnzyme inhibitor3'5'-Cyclic-AMP PhosphodiesterasescGMP-specific phosphodiesterase type 5biology.proteinPhosphodiesterase 2Sodium nitroprussidemedicine.drugMuscle ContractionEuropean journal of pharmacology
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Halothane inhibits endothelium-dependent relaxation elicited by acetylcholine in human isolated pulmonary arteries.

1997

This study examined whether a clinically relevant concentration of the volatile anaesthetic halothane modifies the endothelium-dependent relaxation produced by acetylcholine (3 nM-10 microM), histamine (1 pM-0.1 microM) and anti-human immunoglobulin E (1:1000) in human isolated pulmonary arteries submaximally precontracted with noradrenaline. An inhibitor of nitric oxide formation, N(G)-nitro-L-arginine (100 microM), attenuated acetylcholine-induced relaxation but failed to inhibit histamine- and anti-human immunoglobulin E-induced relaxation. Indomethacin (2.8 microM, a cyclooxygenase inhibitor) preferentially reduced the relaxation to histamine and anti-human IgE. Halothane (2%) significa…

Nitroprussidemedicine.medical_specialtyCromakalimEndotheliumArginineVasodilator AgentsDrug Evaluation PreclinicalProstaglandinVasodilationIn Vitro TechniquesPulmonary ArteryNitric oxidechemistry.chemical_compoundInternal medicinemedicineHumansBenzopyransPyrrolesPharmacologyColforsinImmunoglobulin EAcetylcholineEnzyme ActivationEndocrinologymedicine.anatomical_structurechemistryGuanylate CyclaseAnesthetics InhalationEndothelium VascularHalothaneHalothaneAcetylcholineHistaminemedicine.drugAdenylyl CyclasesEuropean journal of pharmacology
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Pharmacological and biochemical study on the effects of selective phosphodiesterase inhibitors on human term myometrium

1999

This study was aimed at evaluating the in vitro effects of phosphodiesterase inhibitors and beta2-adrenoceptor agonists on spontaneous contractions of human term myometrium. Rolipram, RP 73401 (3-cyclopentyloxy-N-(3,5(-dichloro-4-pyridil)-4-methoxybenzamide) and Ro 20-1724 (1-4-(3-butoxy-4-methoxybenzyl)-2-imidozolidinone) (phosphodiesterase 4 inhibitors) inhibited spontaneous myometrial contractions (Emax approximately 100%; pD2 of 6.80+/-0.28, 6.84+/-0.32 and 6.31+/-0.03, respectively). Salbutamol and formoterol were less effective (Emax=40+/-6% and 35+/-12%, respectively) than phosphodiesterase 4 inhibitors to reduce myometrial contractility. Inhibitors of phosphodiesterase 3 (milrinone …

Nitroprussidemedicine.medical_specialtyTocolytic agentPhosphodiesterase InhibitorsSiguazodanPhosphodiesterase 3In Vitro Techniqueschemistry.chemical_compoundPregnancyInternal medicinemedicineHumansDrug InteractionsRolipramPharmacologyChromatographyDose-Response Relationship DrugPhosphoric Diester HydrolasesPhosphodiesteraseGeneral MedicineAdrenergic beta-AgonistsIsoenzymesEndocrinologychemistryMyometriumMilrinoneFemaleSodium nitroprussideZaprinastmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Involvement of PKC and NF-κB in Nitric Oxide Induced Apoptosis in Human Coronary Artery Smooth Muscle Cells

2001

Apoptosis of vascular smooth muscle cells is critically involved in progression of atherosclerosis and may prevent intimal hyperplasia in restenosis and vascular remodeling. Nitric oxide (NO) is known to induce apoptosis, but the signaling pathways still remain unclear. We investigated p53 accumulation, protein kinase C (PKC) activation and nuclear transcription factor (NF-kappaB) binding activity as possible signaling mechanisms of NO-induced apoptosis. Apoptosis was induced dose-dependently with the NO-donors sodiumnitroprusside (SNP: 232+/-48%) and SIN-1 (241+/-90% of actinomycin D induced apoptosis; means +/- SEM, *por =0.05 vs. control) in HSMC. Inhibition of PKC significantly attenuat…

Nitroprussidemedicine.medical_specialtyVascular smooth muscleIntimal hyperplasiaPhysiologyApoptosisDNA FragmentationNaphthalenesNitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundNF-KappaB Inhibitor alphaRestenosisInternal medicinemedicineHumansNitric Oxide DonorsEnzyme InhibitorsCells CulturedProtein Kinase CProtein kinase CCell Nucleusbusiness.industryNF-kappa BNF-κBStaurosporinemedicine.diseaseCoronary VesselsDNA-Binding Proteinsmedicine.anatomical_structurechemistryApoptosisMolsidomineCancer researchCardiologyI-kappa B ProteinsTumor Suppressor Protein p53businessArteryCellular Physiology and Biochemistry
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Nitric oxide and excitatory postsynaptic currents in immature rat sympathetic preganglionic neurons in vitro.

1997

Neuronal nitric oxide synthase immunoreactivity was localized to sympathetic preganglionic neurons of the intermediolateral cell column and cyclic GMP immunoreactivity to nerve fibers projecting into the intermediolateral cell column of 20-25-day-old rats. Whole-cell patch-clamp recordings were made from sympathetic preganglionic neurons in spinal cord slices of immature rats and the role of nitric oxide and cyclic GMP on excitatory postsynaptic currents was studied. Superfusing the slices with the nitric oxide precursor L-arginine (300 microM) increased the amplitude of evoked excitatory postsynaptic currents as well as the frequency of spontaneous miniature excitatory postsynaptic current…

Patch-Clamp TechniquesPostsynaptic CurrentArginineNitroarginineSynaptic TransmissionNitric oxideRats Sprague-Dawleychemistry.chemical_compoundHemoglobinsNerve Fibers1-Methyl-3-isobutylxanthinemedicineAnimalsPhosphodiesterase inhibitorCyclic GMPEvoked PotentialsNeuronsGanglia SympatheticbiologyGeneral NeuroscienceGlutamate receptorImmunohistochemistryElectric StimulationRatsNitric oxide synthasechemistryBiochemistrySpinal Cordbiology.proteinExcitatory postsynaptic potentialBiophysicsNMDA receptorSodium nitroprussideNitric Oxide Synthasemedicine.drugNeuroscience
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Neurogenic contraction and relaxation of human penile deep dorsal vein

1998

The aim of the present study was to characterize neurogenic and pharmacological responses of human penile deep dorsal vein and to determine whether the responses are mediated by nitric oxide from neural or endothelial origin. Ring segments of human penile deep dorsal vein were obtained from 22 multiorgan donors during procurement of organs for transplantation. The rings were suspended in organ bath chambers for isometric recording of tension. We then studied the contractile and relaxant responses to electrical field stimulation and to vasoactive agents. Electrical field stimulation (0.5–2 Hz) and noradrenaline (3×10−10–3×10−5M) caused frequency- and concentration-dependent contractions that…

PharmacologyEndotheliumbusiness.industryAdrenergicVasodilationPharmacologyNitric oxideTransplantationchemistry.chemical_compoundmedicine.anatomical_structurechemistryAnesthesiamedicineSodium nitroprussidebusinessGuanethidineBlood vesselmedicine.drugBritish Journal of Pharmacology
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NANC inhibitory neurotransmission in mouse isolated stomach: involvement of nitric oxide, ATP and vasoactive intestinal polypeptide

2003

1. The neurotransmitters involved in NANC relaxation and their possible interactions were investigated in mouse isolated stomach, recording the motor responses as changes of endoluminal pressure from whole organ. 2. Field stimulation produced tetrodotoxin-sensitive, frequency-dependent, biphasic responses: rapid transient relaxation followed by a delayed inhibitory component. 3. The inhibitor of the synthesis of nitric oxide (NO), l-NAME, abolished the rapid relaxation and significantly reduced the slow relaxation. Apamin, blocker of Ca2+-dependent K+ channels, or ADPbetaS, which desensitises P2y purinoceptors, reduced the slow relaxation to 2-8 Hz, without affecting that to 16-32 Hz or the…

Pharmacologymedicine.medical_specialtyRelaxation (psychology)Vasoactive intestinal peptideStimulationNeurotransmissionApaminchemistry.chemical_compoundMuscle relaxationEndocrinologychemistryInternal medicinemedicineBiophysicsSodium nitroprussideNeurotransmittermedicine.drugBritish Journal of Pharmacology
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Tachykinergic neurotransmission is enhanced in duodenum from dystrophic (mdx ) mice

2005

1 Duodenal longitudinal muscle of mdx mice, an animal model for Duchenne muscular dystrophy, showed a decrease in the electrically evoked nonadrenergic, noncholinergic (NANC) inhibitory responses associated with a reduction of the participation of nitric oxide (NO). In this study, we investigated whether the impairment of NO could also lead to alterations in the NANC excitatory transmission. 2 Nerve-evoked responses consisted of an inhibitory phase followed, at the end of stimulation, by an excitatory response characterised by an increase in amplitude of the spontaneous contractions. In mdx mice, the amplitude of the nerve-evoked contractions was significantly higher than in normals. 3 N(om…

Pharmacologymedicine.medical_specialtySubstance PStimulationBiologyNeurotransmissionApaminchemistry.chemical_compoundEndocrinologychemistryInternal medicineExcitatory postsynaptic potentialmedicineSodium nitroprussideNeurokinin ASoluble guanylyl cyclasemedicine.drugBritish Journal of Pharmacology
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Inhibition of neuroeffector transmission in human vas deferens by sildenafil

2000

Sildenafil (0.1 - 30 microM), a cyclic GMP phosphodiesterase 5 (PDE 5) inhibitor, induced inhibition of electrically evoked contractions of ring segments of human vas deferens from 34 vasectomies. Zaprinast (0.1 - 100 microM), another PDE 5 inhibitor, and the nitric oxide (NO) donor sodium nitroprusside (SNP) (0.1 - 100 microM) had no effect on neurogenic contractions. The inhibition induced by sildenafil was not modified by the inhibitor of guanylate cyclase 1H-[1,2,4]oxadiazolo[4,3-a]quinoxaline-1-one (ODQ) (1 - 30 microM) but it was abolished by the K(+) channel blockers tetraethylammonium (TEA, 1 mM), iberiotoxin (0.1 microM) and charybdotoxin (0.1 microM). Sildenafil, zaprinast and SNP…

Pharmacologymedicine.medical_specialtyTetraethylammoniumVas deferensIberiotoxinPotassium channelPDE5 drug designchemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryInternal medicinemedicineChannel blockerSodium nitroprussideZaprinastmedicine.drugBritish Journal of Pharmacology
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