Search results for "Nociception"

showing 10 items of 110 documents

Different Brain Circuitries Mediating Controllable and Uncontrollable Pain.

2015

Uncontrollable, compared with controllable, painful stimulation can lead to increased pain perception and activation in pain-processing brain regions, but it is currently unknown which brain areas mediate this effect. When pain is controllable, the lateral prefrontal cortex (PFC) seems to inhibit pain processing, although it is unclear how this is achieved. Using fMRI in healthy volunteers, we examined brain activation during controllable and uncontrollable stimulation to answer these questions. In the controllable task, participants self-adjusted temperatures applied to their hand of pain or warm intensities to provoke a constant sensation. In the uncontrollable task, the temperature time …

0301 basic medicineAdultMaleNociceptionAdolescentPainPrefrontal CortexStimulus (physiology)AnxietyBrain mappingbehavioral disciplines and activities03 medical and health sciencesYoung Adult0302 clinical medicineSensationmedicineHumansThermosensingPrefrontal cortexAnterior cingulate cortexInternal-External ControlPain MeasurementCerebral CortexBrain MappingGeneral NeuroscienceBrainArticlesMagnetic Resonance ImagingHealthy VolunteersDorsolateral prefrontal cortex030104 developmental biologymedicine.anatomical_structureNociceptionFemaleNerve NetPsychologyInsulaNeuroscience030217 neurology & neurosurgerypsychological phenomena and processesThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Acute Exercise Modulates Pain-induced Response on Sensorimotor Cortex ∼20 Hz Oscillation.

2019

Exercise affects positively on self-reported pain in musculoskeletal pain conditions possibly via top-down pain inhibitory networks. However, the role of cortical activity in these networks is unclear. The aim of the current exploratory study was to investigate the effects of acute exercise on cortical nociceptive processing and specifically the excitability in the human sensorimotor cortex. Five healthy adults (mean age 32.8 years) were recorded with a whole-head 306-channel magnetoencephalography (MEG, Elekta Neuromag® Triux™). Participant’s right hand third fingertip was stimulated electrically with an intracutaneous non-magnetic copper tip electrode before and immediately after an exerc…

0301 basic medicineAdultbrain oscillationsPainStimulationEvoked fieldIsometric exerciseliikuntaStimulus (physiology)Inhibitory postsynaptic potentialSomatosensory systemkivunhoito03 medical and health sciences0302 clinical medicineEvoked Potentials SomatosensorymedicineHumanselectrical stimulationsensorimotor cortexExerciseMEGexercisemedicine.diagnostic_testbusiness.industryGeneral NeuroscienceMagnetoencephalographyMagnetoencephalographySomatosensory Cortexaivokuori030104 developmental biologyNociceptionmagnetoencephalographstimulointiSensorimotor CortexbusinessNeuroscience030217 neurology & neurosurgeryliikuntahoitoNeuroscience
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The role of (E)-6-chloro-3-(3-methyl-1-phenyl-1H-pyrazol-5-yl)-2-styrylquinazolin-4(3H)-one in the modulation of cannabinoidergic system. A pilot stu…

2018

Abstract Background Compounds acting on endocannabinoid system regulate different neuronal processes through the cannabinoid receptors activation. The main aim of this study was determining whether the 2-styrylquinazolin-4(3H)-one 5, a structural analogue of rimonabant, was able to counteract the behavioural signs of the activation of the endocannabinoidergic system induced by CP 55.940. Methods Behavioural assessment was carried out using the tetrad task and the novel object recognition test. The endocannabinoidergic system activation was possible by the administration of CP 55.940 and 30 min after rats were tested in the tetrad task for the evaluation of the antinociceptive-, cataleptic-,…

0301 basic medicineAgonistCannabinoid receptormedicine.drug_classmedicine.medical_treatmentPilot ProjectsPharmacologyCannabinoidergicStyrenes03 medical and health sciences0302 clinical medicineRimonabantmedicineAnimalsRats WistarLatency (engineering)PharmacologyDose-Response Relationship DrugCannabinoid CB1 receptor antagonist Quinazolinone derivate Tetrad task Declarative memoryCannabinoidsChemistryRecognition PsychologyGeneral MedicineCyclohexanolsEndocannabinoid systemSettore CHIM/08 - Chimica FarmaceuticaRats030104 developmental biologyNociceptionQuinazolinesSettore BIO/14 - FarmacologiaCannabinoidLocomotion030217 neurology & neurosurgerymedicine.drug
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Progranulin overexpression in sensory neurons attenuates neuropathic pain in mice: Role of autophagy

2016

Peripheral or central nerve injury is a frequent cause of chronic pain and the mechanisms are not fully understood. Using newly generated transgenic mice we show that progranulin overexpression in sensory neurons attenuates neuropathic pain after sciatic nerve injury and accelerates nerve healing. A yeast-2-hybrid screen revealed putative interactions of progranulin with autophagy-related proteins, ATG12 and ATG4b. This was supported by colocalization and proteomic studies showing regulations of ATG13 and ATG4b and other members of the autophagy network, lysosomal proteins and proteins involved in endocytosis. The association of progranulin with the autophagic pathway was functionally confi…

0301 basic medicineAutophagy-Related ProteinsMiceProgranulinsGanglia SpinalDorsal root gangliaGranulinsPain MeasurementCD11b AntigenMicrofilament ProteinsChronic painSciatic nerve injuryCysteine Endopeptidasesmedicine.anatomical_structureNociceptionNeurologyNeuropathic painIntercellular Signaling Peptides and Proteinsmedicine.symptomMicrotubule-Associated ProteinsNerve injuryProgranulinSensory Receptor CellsGreen Fluorescent ProteinsPainMice Transgeniclcsh:RC321-571ATG1203 medical and health sciencesLysosomal-Associated Membrane Protein 1mental disordersmedicineAutophagyAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryActivating Transcription Factor 3Sensory neuronbusiness.industryAutophagyCalcium-Binding ProteinsNerve injurymedicine.diseaseSensory neuronMice Inbred C57BLDisease Models Animal030104 developmental biologyGene OntologyNeuralgiabusinessApoptosis Regulatory ProteinsNeuroscienceNeurobiology of Disease
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Treatment with albumin-hydroxyoleic acid complex restores sensorimotor function in rats with spinal cord injury: Efficacy and gene expression regulat…

2017

Sensorimotor dysfunction following incomplete spinal cord injury (SCI) is often characterized by paralysis, spasticity and pain. Previously, we showed that intrathecal (i.t.) administration of the albumin-oleic acid (A-OA) complex in rats with SCI produced partial improvement of these symptoms and that oral 2-hydroxyoleic acid (HOA, a non-hydrolyzable OA analogue), was efficacious in the modulation and treatment of nociception and pain-related anxiety, respectively. Here we observed that intrathecal treatment with the complex albumin-HOA (A-HOA) every 3 days following T9 spinal contusion injury improved locomotor function assessed with the Rotarod and inhibited TA noxious reflex activity in…

0301 basic medicineMaleNociceptionCritical Care and Emergency Medicinelcsh:MedicineGene ExpressionOleic AcidsPharmacologyProstaglandin E synthaseImmune ReceptorsBiochemistry0302 clinical medicineCell SignalingMedicine and Health SciencesMembrane Receptor Signalinglcsh:ScienceSpinal Cord InjurySpinal cord injuryToll-like ReceptorsTrauma MedicineInjections SpinalProstaglandin-E SynthasesExtracellular Matrix ProteinsMultidisciplinaryImmune System ProteinsbiologyTenascin CTenascinComplement ReceptorsImmune Receptor SignalingNociceptionTreatment OutcomeNeurologySpinal CordPhospholipasesmedicine.symptomTraumatic InjuryLocomotionResearch ArticleSignal TransductionTransmembrane ReceptorsImmunologyPainInflammationNerve Tissue ProteinsGrowth Differentiation Factor 10Drug Administration Schedule03 medical and health sciencesAlbuminsmedicineGeneticsAnimalsParalysisSpasticityRats WistarSpinal Cord Injuriesbusiness.industrylcsh:RBiology and Life SciencesProteinsCell BiologyRecovery of Functionmedicine.diseaseNeuroregenerationRats030104 developmental biologyGene Expression RegulationGDF10Rotarod Performance Testbiology.proteinlcsh:QbusinessNeurotrauma030217 neurology & neurosurgeryPLoS ONE
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Loss of synaptic zinc transport in progranulin deficient mice may contribute to progranulin-associated psychopathology and chronic pain

2017

Affective and cognitive processing of nociception contributes to the development of chronic pain and vice versa, pain may precipitate psychopathologic symptoms. We hypothesized a higher risk for the latter with immanent neurologic diseases and studied this potential interrelationship in progranulin-deficient mice, which are a model for frontotemporal dementia, a disease dominated by behavioral abnormalities in humans. Young naïve progranulin deficient mice behaved normal in tests of short-term memory, anxiety, depression and nociception, but after peripheral nerve injury, they showed attention-deficit and depression-like behavior, over-activity, loss of shelter-seeking, reduced impulse cont…

0301 basic medicineNeurotransmitter transportermedicine.medical_specialtyMice03 medical and health sciencesProgranulins0302 clinical medicinePeripheral Nerve InjuriesInternal medicinemental disordersmedicineAnimalsPrefrontal cortexMolecular BiologyGranulinsMice KnockoutIon Transportbusiness.industryChronic painmedicine.diseaseZinc030104 developmental biologyNociceptionEndocrinologyCompulsive behaviorNeuropathic painPeripheral nerve injuryIntercellular Signaling Peptides and ProteinsNeuralgiaMolecular MedicineChronic Painmedicine.symptomCarrier Proteinsbusiness030217 neurology & neurosurgeryFrontotemporal dementiaBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Complex regional pain syndrome:intradermal injection of phenylephrine evokes pain and hyperalgesia in a subgroup of patients with upregulated α1-adre…

2018

The aim of this study was to determine whether upregulated cutaneous expression of α1-adrenoceptors (α1-AR) is a source of pain in patients with complex regional pain syndrome (CRPS). Immunohistochemistry was used to identify α1-AR on nerve fibres and other targets in the affected and contralateral skin of 90 patients, and in skin samples from 38 pain-free controls. The distribution of α1-AR was compared between patients and controls, and among subgroups of patients defined by CRPS duration, limb temperature asymmetry, and diagnostic subtype (CRPS I vs CRPS II). In addition, α1-AR expression was investigated in relation to pain and pinprick hyperalgesia evoked by intradermal injection of th…

AdultMale0301 basic medicineAgonistInjections Intradermalmedicine.drug_classPainAdrenergicClonidinePhenylephrineYoung Adult03 medical and health sciences0302 clinical medicineReceptors Adrenergic alpha-1Adrenergic alpha-2 Receptor AgonistsmedicineHumansIntradermal injectionPhenylephrineAgedbusiness.industryMiddle Agedmedicine.diseaseUp-Regulation030104 developmental biologyAnesthesiology and Pain MedicineComplex regional pain syndromeNociceptionNeurologyHyperalgesiaAnesthesiaHyperalgesiaImmunohistochemistryFemaleAdrenergic alpha-1 Receptor AgonistsNeurology (clinical)medicine.symptombusinessComplex Regional Pain SyndromesReceptors Purinergic P2X3030217 neurology & neurosurgerymedicine.drug
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Secondary tactile hypoesthesia: a novel type of pain-induced somatosensory plasticity in human subjects

2004

Quantitative sensory testing revealed that pain induced by intracutaneous capsaicin injection elicited secondary hyperalgesia coexisting with secondary tactile hypoesthesia. Mapping the areas of altered mechanical sensations adjacent to the capsaicin injection disclosed that the area of secondary hyperalgesia was always nested in a larger area of secondary hypoesthesia easily detected as numbness by most subjects. Psychometric functions revealed a twofold rightward shift of tactile detection (hypoesthesia), which coexisted with a more than fourfold leftward shift of pricking pain detection (hyperalgesia) in the same skin area. As a mechanism we propose a functional switch at the spinal leve…

AdultMaleAdolescentPresynaptic TerminalsPainNeurological disorderSomatosensory systemSynaptic TransmissionHypesthesiachemistry.chemical_compoundmedicineHumansNeurons AfferentSkinAfferent PathwaysNerve Fibers UnmyelinatedNeuronal PlasticityGeneral NeuroscienceNociceptorsPeripheral Nervous System DiseasesNeural InhibitionHypoesthesiaMiddle Agedmedicine.diseaseMechanoreceptorNociceptionmedicine.anatomical_structurechemistryTouchCapsaicinAnesthesiaHyperalgesiaNociceptorFemaleCapsaicinmedicine.symptomPsychologyMechanoreceptorsNeuroscienceNeuroscience Letters
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Heat-evoked vasodilatation in human hairy skin: axon reflexes due to low-level activity of nociceptive afferents.

1996

1. Spreading vasodilatation of the axon reflex type was evoked by contact heat stimulation of the hairy skin in the human forearm (13.3 cm2 stimulus area) and was detected by laser Doppler flowmetry at 8, 19 and 30 mm distance. 2. From a base temperature of 35 degrees C, rapidly rising short heat stimuli (4 degrees C s-1, 2 s plateau) elicited vasodilatation at an average threshold of 39.4 degrees C. For slowly rising sustained heat stimuli (64 s duration) the average threshold was 39.6 degrees C (n.s.) Laser Doppler flowmetry revealed a rapid onset within about 4 s, a long duration of several minutes beyond the end of the stimulus, and a rapid spread of vasodilatation to remote skin areas.…

AdultMaleAgingHot TemperatureAdolescentPhysiologyPainStimulationStimulus (physiology)Laser-Doppler FlowmetrymedicineHumansNeurons AfferentAxonSkinChemistryNociceptorsMiddle AgedLaser Doppler velocimetryAxonsVasodilationmedicine.anatomical_structureNociceptionAnesthesiaReflexNociceptorFemaleAxon reflexNeuroscienceHairResearch ArticleThe Journal of Physiology
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Psychophysical evidence for long-term potentiation of C-fiber and Adelta-fiber pathways in humans by analysis of pain descriptors.

2007

Long-term potentiation of human pain perception (nociceptive LTP) to single electrical test stimuli was induced by high-frequency stimulation (HFS) of cutaneous nociceptive afferents. Numerical pain ratings and a list of sensory pain descriptors disclosed the same magnitude of nociceptive LTP (23% increase for >60 min, P < 0.001), whereas affective pain descriptors were not significantly enhanced. Factor analysis of the sensory pain descriptors showed that facilitation was restricted to two factors characterized by hot and burning (+41%) and piercing and stinging (+21%, both P < 0.01), whereas a factor represented by throbbing and beating was not significantly increased (+9%, P = …

AdultMaleAnalysis of VarianceNerve Fibers UnmyelinatedPhysiologyFiber (mathematics)General NeuroscienceLong-Term PotentiationPainStimulationLong-term potentiationMiddle AgedElectric StimulationNociceptionPsychophysicsPain perceptionHumansFemalePsychologyNeurosciencePain MeasurementJournal of neurophysiology
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