Search results for "Nod"

showing 10 items of 4007 documents

Enhanced production of CCL18 by tolerogenic dendritic cells is associated with inhibition of allergic airway reactivity

2012

Background IL-10–treated dendritic cells (DCs) have been shown to inhibit T-cell responses through induction of anergy and regulatory T cells in various model systems, including allergic inflammation, but the factors being involved in this inhibition are still unclear. Objective This study set out to analyze such factors produced or induced by IL-10–treated DCs by using gene expression profiling and to explore their function. Methods CD4 + T cells from allergic donors were stimulated with autologous monocyte-derived allergen-pulsed mature DCs or IL-10–treated DCs. After 24 hours, the transcriptional profile was analyzed by using Affymetrix technology. Results were validated by using quantit…

CD4-Positive T-LymphocytesChemokinemedicine.medical_treatmentImmunologyT-Lymphocytes RegulatoryAllergic inflammationMiceMice Inbred NODImmune ToleranceRespiratory HypersensitivitymedicineAnimalsHumansImmunology and AllergyCCL17dendritic cellsCells CulturedT(H)1/T(H)2 cellsMice KnockoutbiologyCCL18FOXP3regulationDendritic cellMicroarray AnalysisallergyCoculture TechniquesInterleukin-10Disease Models Animalhumanized miceCytokineChemokines CCImmunologyHumanized mousebiology.proteinChemokinesTranscriptomeJournal of Allergy and Clinical Immunology
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Reconstitution of CD8 T cells is essential for the prevention of multiple-organ cytomegalovirus histopathology after bone marrow transplantation.

1998

Cytomegalovirus (CMV) infection in the period of temporary immunodeficiency after haematoablative treatment and bone marrow transplantation (BMT) is associated with a risk of graft failure and multiple-organ CMV disease. The efficacy of immune system reconstitution is decisive for the prevention of CMV pathogenesis after BMT. Previous data in murine model systems have documented a redundancy in the immune effector mechanisms controlling CMV. CD8 T cells proved to be relevant but not irreplaceable as antiviral effectors. Specifically, in a state of long-term in vivo depletion of the CD8 T-cell subset, CD4 T cells were educed to become deputy effectors controlling CMV by a mechanism involving…

CD4-Positive T-LymphocytesCongenital cytomegalovirus infectionCytomegalovirusGraft vs Host DiseaseCD8-Positive T-LymphocytesBiologyVirus ReplicationLymphocyte DepletionPathogenesisMiceImmune systemRisk FactorsIn vivoVirologymedicineAnimalsHumansCytotoxic T cellImmunodeficiencyBone Marrow TransplantationMice Inbred BALB CEffectorvirus diseasesmedicine.diseaseVirologyDisease Models AnimalTransplantation IsogeneicCytomegalovirus InfectionsImmunologyCD8Journal of General Virology
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Adoptive transfer of protective immunity from Cryptosporidium parvum-infected interferon-gamma and interleukin-12-deficient mice to naive recipients.

2008

We investigated the possibility of transfer immunity from Cryptosporidium parvum-infected interferon-gamma (GKO) and interleukin-12p40 (IL-12KO) deficient C57BL/6 mice to naive mice by transfer of intraepithelial lymphocytes (IELs) and CD4(+) T cells from spleen and mesenteric lymph nodes (MLNs). Three days after the transfer recipients were infected with C. parvum. IELs isolated from GKO donor mice after resolution of infection (day 15) but not at the peak of infection (day 8) significantly reduced the parasite load in recipient mice. In IL-12KO mice, IELs and also CD4(+) T cells isolated from the spleen and MLNs of donor mice at the peak of infection (day 5) and after resolution (day 15) …

CD4-Positive T-LymphocytesMaleAdoptive cell transferCryptosporidiosisSpleenHost-Parasite InteractionsInterferon-gammaMiceImmunityCell Movementparasitic diseasesmedicineMesenteric lymph nodesAnimalsImmunity MucosalCryptosporidium parvumMice KnockoutGeneral VeterinaryGeneral Immunology and MicrobiologybiologyImmunomagnetic SeparationPublic Health Environmental and Occupational Healthbiology.organism_classificationAdoptive TransferInterleukin-12Mice Inbred C57BLInfectious Diseasesmedicine.anatomical_structureCryptosporidium parvumAdoptive immunityImmunologyInterleukin 12Molecular MedicineIntraepithelial lymphocyteFemaleLymph NodesSpleenVaccine
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Angioimmunoblastic T-cell lymphoma

2008

Angioimmunoblastic T-cell lymphoma (AITL) is a rare and aggressive neoplasm clinically characterized by sudden onset of constitutional symptoms, lymphadenopathy, hepatosplenomegaly, frequent autoimmune phenomena, particularly hemolytic anemia and thrombocytopenia, and polyclonal hypergammaglobulinemia. The lymph node histological picture is also distinctive, constituted by a polymorphic infiltrate, a marked proliferation of high endothelial venules, and a dense meshwork of dentritic cells. The neoplastic CD4+ T-cells represent a minority of the lymph node cell population; its detection is facilitated by the aberrant expression of CD10. Almost all cases arbor an EBV infected B-cell populatio…

CD4-Positive T-LymphocytesMaleEpstein-Barr Virus InfectionsPathologyAutologous transplantHerpesvirus 4 HumanHepatosplenomegalyImmunosuppressive AgentEpstein-Barr Virus InfectionHypergammaglobulinemiaLymph nodeNon-Hodgkin lymphomaAngioimmunoblastic lymphomaB-Lymphocyteseducation.field_of_studyB-LymphocyteLymph NodeHematologyThalidomideSurvival RateTransplantation Autologoumedicine.anatomical_structureOncologyCD4-Positive T-LymphocyteFemaleNeprilysinmedicine.symptomImmunosuppressive AgentsHumanmedicine.medical_specialtyAngioimmunoblastic T-cell lymphomaPopulationHigh endothelial venulesDendritic CellLymphoma T-CellTransplantation AutologousmedicineHumanseducationCell Proliferationbusiness.industryPeripheral T-cell lymphomaDendritic Cellsmedicine.diseasePeripheral T-cell lymphomaLymphomaTransplantationImmunologyLymph NodesGeriatrics and GerontologybusinessStem Cell Transplantation
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Development of hapten-induced IL-4-producing CD4+ T lymphocytes requires early IL-4 production by alphabeta T lymphocytes carrying invariant V(alpha)…

1998

This paper investigates the mechanisms responsible for the generation of IL-4-producing CD4+ T cells during contact sensitization with the hapten trinitrochlorobenzene (TNCB). Lymph node cells taken 1 day after immunization spontaneously released IL-4 while lymph node cells taken 2 and 3 days after immunization did not produce IL-4. A second wave of IL-4 production that was both antigen-specific and MHC class II (I-A)-restricted was observed 4 days after immunization. The spontaneous release of IL-4 at day 1 was due to the alphabeta+ double-negative (CD4- CD8-) T lymphocytes that also expressed NK1.1 and showed V(alpha)14 rearrangement, while alphabeta+ CD4+ T lymphocytes were the source of…

CD4-Positive T-LymphocytesMaleReceptors Antigen T-Cell alpha-betaT cellImmunologyPicryl ChlorideCD8-Positive T-LymphocytesBiologyMiceInterleukin 21AntigenmedicineAnimalsImmunology and AllergyCytotoxic T cellMice Inbred BALB CT-cell receptorAntibodies MonoclonalGeneral MedicineT lymphocyteMolecular biologyInterleukin-10Mice Inbred C57BLmedicine.anatomical_structureImmunizationInterleukin-4Lymph NodesHaptensCD8Alpha chainInternational Immunology
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WHOLE BODY IRRADIATION INDUCES IFN-γ PRODUCTION IN BALB/c MICE BY PREVENTING THE APPEARANCE OF A Vα14+NK T DOWNREGULATORY POPULATION

2000

Lymph node cells from TNCB-immune BALB/c mice fail to produce IFN-gamma when exposed to antigen in vitro. Conversely, lymph node cells of irradiated (550 rads) BALB/c mice produce IFN-gamma. Transfer experiments show that normal BALB/c mice contain cells which suppress IFN-gamma production. These downregulatory cells are CD4(+)alpha beta(+)and rearrange the invariant V alpha 14-J alpha 281 T cell receptor alpha chain, thus belonging to the NK T cell subset. Downregulatory cells probably act by producing IL-4 as their effect is blocked by mAb to IL-4.

CD4-Positive T-LymphocytesMalemedicine.drug_classCD8 AntigensReceptors Antigen T-Cell alpha-betaImmunologyPopulationWhole body irradiationAntigen-Presenting CellsDown-RegulationAntigens ProtozoanEnzyme-Linked Immunosorbent AssayCell SeparationMonoclonal antibodyBiochemistryBALB/cInterferon-gammaMiceAntigenmedicineAnimalsImmunology and AllergyeducationMolecular BiologyLymph nodeLeishmania majorMice Inbred BALB Ceducation.field_of_studybiologyChemistryT-cell receptorAntibodies MonoclonalHematologyFlow Cytometrybiology.organism_classificationMolecular biologyIn vitroKiller Cells NaturalPhenotypemedicine.anatomical_structureCD4 AntigensImmunologyInterleukin-4Lymph NodesSpleenCytokine
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Activated glycoprotein A repetitions predominant (GARP)-expressing regulatory T cells inhibit allergen-induced intestinal inflammation in humanized m…

2015

Background Recently, we developed a humanized mouse model of allergen-induced IgE-dependent gut inflammation in PBMC-engrafted immunodeficient mice. Objective In the present study, we wanted to investigate the role of regulatory T (Treg) cells and their activation status in this model. Methods Nonobese diabetic-severe combined immunodeficiency-γc −/− mice were injected intraperitoneally with human PBMCs from allergic donors together with the respective allergen or NaCl as control in the presence or absence of different concentrations of CD4 + CD25 + Treg cells of the same donor. After an additional allergen boost 1 week later, mice were challenged with the allergen rectally on day 21 and gu…

CD4-Positive T-LymphocytesMalemedicine.medical_treatmentImmunologyInflammationNodMice SCIDBiologyImmunoglobulin ET-Lymphocytes RegulatoryMicemedicineHypersensitivityImmunology and AllergyAnimalsHumansIL-2 receptorAntibodies BlockingCells CulturedCell ProliferationImmunosuppression TherapyInflammationSevere combined immunodeficiencyInterleukin-2 Receptor alpha SubunitMembrane Proteinshemic and immune systemsForkhead Transcription FactorsDendritic cellAllergensImmunoglobulin Emedicine.diseaseIntestinesDisease Models AnimalCytokineImmunologyHumanized mouseAntibody FormationCD4 Antigensbiology.proteinLeukocytes MononuclearFemalemedicine.symptomThe Journal of allergy and clinical immunology
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Chemokines enhance immunity by guiding naive CD8+ T cells to sites of CD4+ T cell-dendritic cell interaction.

2005

CD8+ T cells have a crucial role in resistance to pathogens and can kill malignant cells; however, some critical functions of these lymphocytes depend on helper activity provided by a distinct population of CD4+ T cells. Cooperation between these lymphocyte subsets involves recognition of antigens co-presented by the same dendritic cell, but the frequencies of such antigen-bearing cells early in an infection and of the relevant naive T cells are both low. This suggests that an active mechanism facilitates the necessary cell-cell associations. Here we demonstrate that after immunization but before antigen recognition, naive CD8+ T cells in immunogen-draining lymph nodes upregulate the chemok…

CD4-Positive T-LymphocytesReceptors CCR5T cellAntigen presentationCell CommunicationBiologyCD8-Positive T-LymphocytesLymphocyte ActivationInterleukin 21MiceCell MovementmedicineCell AdhesionCytotoxic T cellAnimalsAntigen-presenting cellChemokine CCL4Chemokine CCL3MultidisciplinaryCD28Dendritic cellDendritic CellsMacrophage Inflammatory ProteinsNatural killer T cellmedicine.anatomical_structureChemokines CCImmunologyLymph NodesChemokinesImmunologic MemoryNature
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A new model of chronic colitis in SCID mice induced by adoptive transfer of CD62L+ CD4+ T cells: insights into the regulatory role of interleukin-6 o…

2003

<i>Objective:</i> The proinflammatory cytokine interleukin (IL)-6 is involved in various chronic inflammatory processes. IL-6 is a predominant cytokine produced by lamina propria T cells in Crohn’s disease and experimental colitis. This study was designed to examine the effect of a neutralizing IL-6-receptor (IL-6R) antibody on the programmed cell death of mucosal T cells in the CD62L+ CD4+ SCID transfer model of chronic experimental colitis in mice and to gain more insight into the pathogenesis of this transfer colitis model. <i>Methods:</i> For adoptive transfer, we isolated CD62L+ CD4+ double-positive T cells from wild-type BALB/c mice followed by intraperitoneal …

CD4-Positive T-LymphocytesSTAT3 Transcription FactorAdoptive cell transferCell TransplantationColonApoptosisMice SCIDPathology and Forensic MedicineProinflammatory cytokineInterleukin 21MiceInterleukin 25In Situ Nick-End LabelingAnimalsIL-2 receptorIntestinal MucosaL-SelectinInterleukin 6Antibodies BlockingMolecular BiologyMice Inbred BALB CbiologyInterleukin-6InterleukinCell BiologyGeneral MedicineFlow CytometryAdoptive TransferReceptors Interleukin-6DNA-Binding ProteinsDisease Models AnimalImmunologybiology.proteinTrans-ActivatorsInterleukin 18Colitis UlcerativeSevere Combined ImmunodeficiencySpleenPathobiology : journal of immunopathology, molecular and cellular biology
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Cytokine profile, HLA restriction and TCR sequence analysis of human CD4+ T clones specific for an immunodominant epitope of Mycobacterium tuberculos…

2003

SUMMARY The identification of immunodominant and universal mycobacterial peptides could be applied to vaccine design and have an employment as diagnostic reagents. In this paper we have investigated the fine specificity, clonal composition and HLA class II restriction of CD4+ T cell clones specific for an immunodominant epitope spanning amino acids 91–110 of the 16-kDa protein of Mycobacterium tuberculosis. Twenty-one of the tested 28 clones had a Th1 profile, while seven clones had a Th0 profile. None of the clones had a Th2 profile. While the TCR AV gene usage of the clones was heterogeneous, a dominant TCR BV2 gene family was used by 18 of the 28 clones. The CDR3 regions of BV2+ T cell c…

CD4-Positive T-LymphocytesSequence analysisT cellImmunologyReceptors Antigen T-CellEpitopeInterferon-gammaAntigenClinical StudiesmedicineHumansTuberculosisImmunology and AllergyGene familyGeneCells CulturedGeneticsAntigens BacterialbiologyImmunodominant EpitopesT-cell receptorHLA-DR AntigensMycobacterium tuberculosisComplementarity Determining RegionsPeptide Fragmentsmedicine.anatomical_structureImmunologybiology.proteinCytokinesAntibodyClinical and Experimental Immunology
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