Search results for "Normal tissue"

showing 2 items of 22 documents

Analysis of cardiac and pulmonary complication probabilities after radiation therapy for patients with early-stage breast cancer

2009

Objective. The purpose of this study was to evaluate the radiobiological implications of clinical use of respiratory-gated techniques for postoperative radiation therapy of early-stage left-sided breast cancer after breast-conserving surgery. Material and methods. Radiation therapy treatment plans of 80 patients with early-stage breast cancer (stage I–II), receiving whole breast irradiation after breast-conserving therapy, were analyzed. The control group consisting of 47 patients received standard radiation therapy, and the respiratory-gated group consisting of 33 patients received deep inspiration-gated radiation therapy. Normal tissue complication probabilities (NTCP) for cardiac mortali…

medicine.medical_specialtyRespiratory-Gated Imaging TechniquesHeart Diseasesearly-stage breast cancermedicine.medical_treatmentBreast NeoplasmsMastectomy SegmentalStatistics NonparametricCohort StudiesBreast cancerWhole Breast Irradiationnormal tissue complication probabilitymedicineHumansBreastStage (cooking)Radiation Injuriesrespiratory-gated radiotherapyPneumonitisNeoplasm StagingProbabilitybusiness.industryrespiratory-gated radiotherapy; early-stage breast cancer; breast-conserving therapy; normal tissue complication probabilityPatient SelectionRespirationPulmonary ComplicationRadiotherapy DosageGeneral Medicinemedicine.diseaseLatviaSurgeryRadiation therapyRadiation Pneumonitisbreast-conserving therapyFemaleRadiotherapy AdjuvantRadiotherapy ConformalComplicationbusinessTomography X-Ray ComputedMastectomyMedicina
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Inhibition of Rac1 signaling by lovastatin protects against anthracycline-induced cardiac toxicity

2011

Normal tissue damage limits the efficacy of anticancer therapy. For anthracyclines, the clinically most relevant adverse effect is cardiotoxicity. The mechanisms involved are poorly understood and putative cardioprotectants are controversially discussed. Here, we show that the lipid-lowering drug lovastatin protects rat H9c2 cardiomyoblasts from doxorubicin in vitro. Protection by lovastatin is related to inhibition of the Ras-homologous GTPase Rac1. It rests on a reduced formation of DNA double-strand breaks, resulting from the inhibition of topoisomerase II by doxorubicin. Doxorubicin transport and reactive oxygen species are not involved. Protection by lovastatin was confirmed in vivo. I…

rac1 GTP-Binding ProteinCancer ResearchAnthracyclineDoxorubicin transportCardiac fibrosismedicine.medical_treatmentImmunologyPharmacologyBiologyDNA damage responsestatinsMiceCellular and Molecular NeuroscienceRho GTPasespolycyclic compoundsmedicineAnimalsDNA Breaks Double-StrandedMyocytes CardiacDoxorubicinLovastatinanthracyclinesCardiotoxicityAntibiotics AntineoplasticTroponin IConnective Tissue Growth FactorCell Biologymedicine.diseaseRatsCTGFDNA Topoisomerases Type IICytokinenormal tissue damageDoxorubicinOriginal Articlelipids (amino acids peptides and proteins)LovastatinAtrial Natriuretic FactorSignal Transductionmedicine.drugCell Death & Disease
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