Search results for "Normal"

showing 10 items of 2571 documents

Heritability Estimates of Differences in Arbitrary Embryonic Mortality Traits in Turkeys

1971

Abstract INTRODUCTION SPECIFIC embryonic abnormalities in Broad Breasted Bronze turkeys which contributed to low hatchability were classified into eight groups which are referred to as traits in this paper. References to these traits or similar abnormalities in chickens and turkeys are shown in Table 1. The objective of this study was to determine the differences in the rates, relative to unhatched fertile eggs, at which these specific traits could be changed by artificial selection which was accompanied by inbreeding. Rapid changes would indicate the traits more responsive to selection. MATERIALS AND METHODS Four different strains of Broad Breasted Bronze turkeys were introduced as eggs to…

MaleGeneticsTurkeysmedia_common.quotation_subjectZoologyFertilityChick EmbryoGeneral MedicineHeritabilityBiologyBody weightCongenital AbnormalitiesFertilityAnimalsFemaleInbreedingAnimal Science and ZoologySelection GeneticInbreedingPoultry DiseasesSelection (genetic algorithm)media_commonPoultry Science
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Interstitial deletion of chromosome 2p15-16.1: Report of two patients and critical review of current genotype–phenotype correlation

2011

Abstract We report two individuals with developmental delay and dysmorphic features, in whom array-based comparative genomic hybridization (array CGH) led to the identification of a 2p15p16.1 de novo deletion. In the first patient (Patient 1) a familial deletion of 6q12, inherited from her father, was also detected. In the second patient (Patient 2) in addition to the 2p15p16.1 microdeletion a de novo deletion in Xq28 was detected. Both individuals shared dysmorphic features and developmental delay with the six reported patients with a 2p15p16.1 microdeletion described in medical literature. Conclusion: in the first patient a 642 kb 2p16.1 deletion (from 60.604 to 61.246 Mb), and a 930 kb 6…

MaleGenotypeDevelopmental delayDevelopmental DisabilitiesBioinformaticsContiguous gene syndromeGenotype phenotypeCorrelationGeneticsHumansChromosomal delectionMedicineAbnormalities MultipleClinical phenotypeGenetic Association StudiesIn Situ Hybridization FluorescenceSex Chromosome AberrationsGenetics (clinical)Sequence DeletionGeneticsChromosomes Human XComparative Genomic Hybridizationbusiness.industryInfantChromosomeSyndromeGeneral MedicineMicrodeletion syndromemedicine.diseaseXq28PhenotypeChild PreschoolChromosomes Human Pair 2FemaleChromosome DeletionbusinessComparative genomic hybridizationEuropean Journal of Medical Genetics
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Preserving the abductor mechanism in robotic THR: the influence of stem design and cutterpath.

2004

The tip of the greater trochanter is the attachment site for the abductor muscles of the hip joint. Its preservation in robotic and conventional THR is important for normal gait. The effect of different stem designs and robotic cutterpaths on the preservation of the trochanter tip is examined.One anatomical stem, one straight stem, and one stem specifically designed for robotic THR were implanted virtually in CT scans of osteoarthrotic hip joints using the Torch preoperative planning unit (URS-ortho). In transverse sections of the trochanter tip, dimensions of the trochanter area removed by the milling tool were recorded for each stem design and different cutterpaths (3-axis versus 5-axis m…

MaleGreater trochanterMaterials scienceAttachment siteArthroplasty Replacement HipUser-Computer InterfaceProsthesis FittingOsteoarthritismedicineHumansFemurMuscle SkeletalGaitPreoperative planningTrochantertechnology industry and agricultureStraight stemAnatomyRoboticsComputer Science ApplicationsMechanism (engineering)Normal gaitmedicine.anatomical_structureSurgery Computer-AssistedSurgeryMuscles of the hipFemaleHip JointFamily PracticeTomography X-Ray ComputedBiomedical engineeringComputer aided surgery : official journal of the International Society for Computer Aided Surgery
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Cross-species transcriptomic analysis elucidates constitutive aryl hydrocarbon receptor activity

2014

Background Research on the aryl hydrocarbon receptor (AHR) has largely focused on variations in toxic outcomes resulting from its activation by halogenated aromatic hydrocarbons. But the AHR also plays key roles in regulating pathways critical for development, and after decades of research the mechanisms underlying physiological regulation by the AHR remain poorly characterized. Previous studies identified several core genes that respond to xenobiotic AHR ligands across a broad range of species and tissues. However, only limited inferences have been made regarding its role in regulating constitutive gene activity, i.e. in the absence of exogenous ligands. To address this, we profiled transc…

MaleHEPATIC GENE-EXPRESSION413 Veterinary scienceMedical and Health SciencesTranscriptomeDIOXIN RECEPTORMice0302 clinical medicineTCDD-induced toxicityReceptorsTranscriptional regulationABNORMAL LIVER DEVELOPMENT2.1 Biological and endogenous factorsCluster AnalysisAetiologyReceptorAH RECEPTORIN-VIVOAryl hydrocarbon receptorGeneticsRegulation of gene expression0303 health sciencesBiological Sciencesrespiratory systemCore-gene batteryAryl HydrocarbonOrgan Specificity030220 oncology & carcinogenesisAHR endogenous ligands2378-TETRACHLORODIBENZO-P-DIOXIN TCDDSignal transductionResearch ArticleBiotechnologySignal TransductionProtein BindingBioinformatics1.1 Normal biological development and functioningeducationRAT-LIVERConstitutive gene expressionBiologyMICE LACKING03 medical and health sciencesSpecies SpecificityUnderpinning researchInformation and Computing SciencesGeneticsAnimals030304 developmental biologyAryl hydrocarbon receptor activityGene Expression ProfilingComputational BiologyAryl hydrocarbon receptorCELL-CYCLE CONTROLRatsrespiratory tract diseasesGene expression profilingReceptors Aryl HydrocarbonGene Expression RegulationSUBCHRONIC EXPOSUREbiology.proteinDigestive DiseasesTranscriptome
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Enzyme polymorphisms and haemoglobin variants in Greeks

1975

Several enzyme polymorphisms and hemoglobin variants were typed in a sample of n = 219 non-related Greek blood-donors. The following gene frequencies were observed: pa = 0.201, pb = 0.701, pc = 0.098;PGDA = 0.985, PGDc = 0.015; AK1 = 0.942, AK2 = 0.058; HbA = 0.988, HbS = 0.012. No polymorphic variation was seen in LDH, s-MDH, PHI, or SOD. The population genetical aspects of these results are discussed.

MaleHemoglobins AbnormalAcid PhosphatasePopulationBlood DonorsBiologyHaemoglobin variantsGene FrequencyMalate DehydrogenaseGeneticsHumansMetabolic diseaseeducationGeneAllele frequencyAllelesGenetics (clinical)Geneticschemistry.chemical_classificationeducation.field_of_studyPolymorphism GeneticGreeceL-Lactate DehydrogenaseSuperoxide DismutasePhosphogluconate DehydrogenasePhosphotransferasesGlucose-6-Phosphate IsomeraseGenetic VariationHemoglobin variantsMolecular biologyAK2IsoenzymesPhenotypeEnzymechemistryFemaleHuman Genetics
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Association between the HFE mutations and unsuccessful ageing: a study in Alzheimer's disease patients from Northern Italy

2003

Mutations in the class I-like Major Histocompatibility Complex gene HFE are associated with hereditary hemochromatosis (HH), a disorder caused by excessive iron uptake. Three common mutations have been found: C282Y, H63D, and S65C. Moreover, several studies have suggested that HFE mutations may be involved in several age-related chronic diseases such as Alzheimer's disease (AD) and coronary heart disease, but apparently paradoxically also with longevity. In particular, in AD, patients carrying the H63D allele have been suggested to have a mean age at onset of 72 vs. 77 years for those who were homozygous for the wild-type allele. Thus, it seems that H63D mutations may anticipate sporadic AD…

MaleHeterozygotecongenital hereditary and neonatal diseases and abnormalitiesAgingDiseasemedicine.disease_causeDegenerative diseaseGene FrequencyAlzheimer DiseaseGenotypeHumansPoint MutationMedicineAlleleHemochromatosis ProteinHemochromatosisAgedGeneticsMutationbusiness.industryHistocompatibility Antigens Class IHomozygoteMembrane Proteinsnutritional and metabolic diseasesMiddle Agedmedicine.diseaseItalyHereditary hemochromatosisFemaleAlzheimer's diseasebusinessDevelopmental BiologyMechanisms of Ageing and Development
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A significant p value is not equivalent to the superiority of one test index over another

2019

Background In patients with septic shock, the skin is often chosen for the evaluation of peripheral perfusion and oxygenation. Changes in skin microcirculatory vessel oxygen saturation and relative hemoglobin concentration can be described using a mottling score or captured with hyperspectral imaging. However, the effectiveness of the mottling score in assessing microcirculation remains to be shown. We hypothesize that the mottling score in patients with septic shock is related to skin microcirculatory perfusion indices quantified by hyperspectral imaging, biomarkers that reflect endothelium activation and damage, and clinical outcome. Methods Hyperspectral imaging of the knee area was perf…

MaleIndex (economics)LetterHyperspectral imagingCritical Care and Intensive Care MedicineStatistics NonparametricSepsisStatisticsMedicineHumansp-valueEndotheliumAgedModels Statisticalbusiness.industryResearchMicrocirculationlcsh:Medical emergencies. Critical care. Intensive care. First aidlcsh:RC86-88.9Middle AgedShock SepticTest (assessment)PerfusionResearch DesignData Interpretation StatisticalSkin AbnormalitiesFemaleTissue oxygenationbusinessBiomarkersCritical Care
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Tales of healthy men: Male reproductive bodies in biomedicine from ‘Lebensborn’ to sperm banks

2012

Using the example of ‘sperm tales’, borne out of the biomedical technologies that went hand in hand with the establishment of the ‘science of man’ (andrology), the article engages with the epistemic evolution of interrelated biomedical theories and concepts of what constitutes a ‘healthy’ reproductive male body. The article asks: how has the normative ideal male body been either perpetuated or interrogated through these tales of male reproduction at the interface between scientific and medical technologies? And how were changes to the normalization of male bodies central to clinical practices and cultural understandings of health and illness? With many aspects of the medical history of male…

MaleInfertilityScience of manHealth (social science)ConceptualizationSperm Banksbusiness.industryHistory 19th CenturyGender studiesHistory 20th CenturyModels Theoreticalmedicine.diseaseSperm bankSpermatozoaMale infertilityReproductive HealthReproductive TechniquesGermanySociology of health and illnessmedicineHumansNormativeNormalization (sociology)businessInfertility MaleHealth: An Interdisciplinary Journal for the Social Study of Health, Illness and Medicine
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Giant axonal neuropathy and leukodystrophy

1991

Abstract An 11-year-old Persian boy, born to consanguineous parents, manifested a progressive gait abnormality beginning at 5 years of age. A severe cerebellar disorder developed with associated dysfunction of the peripheral nervous system, but no sign of mental impairment. The sensory and motor nerve conduction velocities were greatly reduced, especially in the lower extremities. Cerebrospinal fluid protein was normal. Computed tomography and magnetic resonance imaging revealed leukoencephalopathy, especially in the cerebellum, but also in periventricular areas. The diagnosis of giant axonal neuropathy was established by biopsy of the sural nerve. The few previous histologic examinations h…

MaleIntermediate FilamentsMotor nerveGenes RecessiveSural nerveCerebral VentriclesLeukoencephalopathyConsanguinityDevelopmental NeuroscienceCerebellummedicineHumansCerebellar disorderGliosisPeripheral NervesChildMyelin SheathSpinocerebellar DegenerationsGiant axonal neuropathybusiness.industryLeukodystrophyAnatomymedicine.diseaseMagnetic Resonance ImagingAxonsMicroscopy Electronmedicine.anatomical_structurenervous systemNeurologyPeripheral nervous systemPediatrics Perinatology and Child HealthGait abnormalityNeurology (clinical)medicine.symptomHereditary Sensory and Motor NeuropathybusinessPediatric Neurology
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Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

1996

International audience; Friedreich's ataxia (FRDA) is an autosomal recessive, degenerative disease that involves the central and peripheral nervous systems and the heart. A gene, X25, was identified in the critical region for the FRDA locus on chromosome 9q13. This gene encodes a 210-amino acid protein, frataxin, that has homologs in distant species such as Caenorhabditis elegans and yeast. A few FRDA patients were found to have point mutations in X25, but the majority were homozygous for an unstable GAA trinucleotide expansion in the first X25 intron.

MaleIron-sulfur cluster assemblyPolymerase Chain Reaction0302 clinical medicineTrinucleotide RepeatsIron-Binding ProteinsGenetics0303 health sciencesMultidisciplinaryAutosomal recessive cerebellar ataxiaPedigree3. Good healthFemalemedicine.symptomChromosomes Human Pair 9HumanPair 9Heterozygotecongenital hereditary and neonatal diseases and abnormalitiesAtaxiaMolecular Sequence DataGenes RecessiveLocus (genetics)BiologyChromosomes03 medical and health sciencesGene mappingAlleles; Amino Acid Sequence; Base Sequence; Chromosomes Human Pair 9; DNA Primers; Female; Friedreich Ataxia; Genes Recessive; Heterozygote; Humans; Male; Molecular Sequence Data; Pedigree; Point Mutation; Polymerase Chain Reaction; Proteins; Sequence Alignment; Introns; Iron-Binding Proteins; Trinucleotide RepeatsmedicineRecessiveHumansPoint MutationAmino Acid SequenceAlleleAllelesDNA Primers030304 developmental biologyBase SequencePoint mutationProteins[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologymedicine.diseaseMolecular biologyIntronsGenes[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsFriedreich AtaxiaFrataxinbiology.proteinSequence Alignment030217 neurology & neurosurgeryScience
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