Search results for "Normal"

showing 10 items of 2571 documents

Normal Values for Microperimetry with the MAIA Microperimeter: Sensitivity and Fixation Analysis in Healthy Adults and Children

2017

Purpose To establish normative values of retinal sensitivity and parameters describing the fixation pattern using macular analyzer integrity assessment (MAIA) microperimetry (Centervue, Padova, Italy) in adults and children. Methods A sample of 237 eyes of 237 healthy subjects aged between 10 and 70 years (mean age 30.63 ± 16.23 years) was evaluated using the MAIA microperimeter. The following parameters provided by the MAIA device were evaluated: average threshold (AT), macular integrity, fixation indexes (P1% and P2%), bivariate contour ellipse area (BCEA) for 95% and 63% of points, and horizontal (H) and vertical (V) axes of the ellipse of fixation. Differences between different age-rela…

MAIAAdultMalemedicine.medical_specialtyVisual acuityAdolescentVisual AcuityFixation OcularNormal valuesMicroperimetryMicroperimeterYoung Adult03 medical and health sciencesOcular physiology0302 clinical medicineReference ValuesOphthalmologyHumansMedicineRetinal sensitivityMacula LuteaChildÓpticaAgedbusiness.industryReproducibility of ResultsGeneral MedicineMiddle AgedIntegrity assessmentVisual fieldOphthalmologyEye fixationReference valuesFixation (visual)030221 ophthalmology & optometryVisual Field TestsFemaleVisual Fieldsmedicine.symptombusinessMicroperimetry030217 neurology & neurosurgeryEuropean Journal of Ophthalmology
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Global Functional Analyses of Cellular Responses to Pore-Forming Toxins

2011

Here we present the first global functional analysis of cellular responses to pore-forming toxins (PFTs). PFTs are uniquely important bacterial virulence factors, comprising the single largest class of bacterial protein toxins and being important for the pathogenesis in humans of many Gram positive and Gram negative bacteria. Their mode of action is deceptively simple, poking holes in the plasma membrane of cells. The scattered studies to date of PFT-host cell interactions indicate a handful of genes are involved in cellular defenses to PFTs. How many genes are involved in cellular defenses against PFTs and how cellular defenses are coordinated are unknown. To address these questions, we pe…

MAPK/ERK pathwayTranscription GeneticImmunology/Innate ImmunityMessengerInteractomeInfectious Diseases/Bacterial InfectionsRNA interference2.1 Biological and endogenous factorsAetiologyBiology (General)Genes HelminthCaenorhabditis elegansOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesGenomebiologyReverse Transcriptase Polymerase Chain ReactionGenetics and Genomics/Functional Genomics030302 biochemistry & molecular biologyrespiratory systemCell biologyInfectious DiseasesMedical MicrobiologyRNA InterferenceSignal transductionDNA microarrayTranscriptionBiotechnologyResearch ArticleSignal TransductionPore Forming Cytotoxic ProteinsQH301-705.5Virulence FactorsMAP Kinase Signaling System1.1 Normal biological development and functioningBacterial ToxinsImmunologyMicrobiologyDNA-binding proteinCell Line03 medical and health sciencesBacterial ProteinsGeneticUnderpinning researchVirologyEscherichia coliHelminthGeneticsAnimalsHumansRNA MessengerCaenorhabditis elegansCaenorhabditis elegans ProteinsMolecular BiologyGene030304 developmental biologyGenome HelminthCell MembraneGenetics and GenomicsRC581-607biology.organism_classificationrespiratory tract diseasesTranscription Factor AP-1Emerging Infectious DiseasesGenesRNAParasitologyGeneric health relevanceRNA HelminthImmunologic diseases. AllergyPLoS Pathogens
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Abstract LB-085: A new role for LKB1 to regulate Heat Shock Protein 90 activity

2018

Abstract Approximately 30% of human non-small cell lung cancer (NSCLC) patients harbor a somatic KRAS mutation resulting, in aberrant activation of downstream signaling pathways that control cell proliferation, cell growth, and cell survival. Importantly, alleles of LKB1, a serine/threonine kinase that functions as a tumor suppressor, are somatically inactivated in ~30% of NSCLCs within KRAS-mutant NSCLC. The loss of LKB1 gives rise to aggressive, highly metastatic, and highly drug resistant tumors. We have previously demonstrated that the inactivation of the tumor suppressor lkb1 rendered mutant kras murine NSCLC resistant to targeted agents including BET bromodomain and kinase inhibitors.…

MAPK/ERK pathwaycongenital hereditary and neonatal diseases and abnormalitiesCancer ResearchCell signalingChemistryKinaseCell growthIsogenic human disease modelsOncologyHeat shock proteinCancer researchKinase activityskin and connective tissue diseasesPI3K/AKT/mTOR pathwayCancer Research
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Triplet 12B-12C-12N : Search for States with Halo

2020

Previously in [1] neutron halo was confirmed for the 2¯, 1.67 MeV and 1¯, 2.62 MeV states in 12B on base of Asymptotic Normalization Coefficients (ANC) method analysis of the obtained experimental data. An unexpected result was received for the unbound 3¯, 3.39 MeV state. Its halo radius was found to be increased and equal to ~5.9 fm. This result can be considered as an evidence of the halo-like structure in this 12B state. It should be noted that last neutron in this state has a non-zero orbital momentum (l = 2). So question arises about possible existence of states with halo in other members of the isobaric triplet 12B–12C–12N. We can expect the formation of a proton halo in the 2¯, 1.19 …

MDM modelNuclear Theoryasymptotic normalization coefficients neutron and proton haloradii of excited statesNuclear Experiment
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The nuclear receptor PPARγ selectively inhibits Th17 differentiation in a T cell–intrinsic fashion and suppresses CNS autoimmunity

2009

T helper cells secreting interleukin (IL)-17 (Th17 cells) play a crucial role in autoimmune diseases like multiple sclerosis (MS). Th17 differentiation, which is induced by a combination of transforming growth factor (TGF)-beta/IL-6 or IL-21, requires expression of the transcription factor retinoic acid receptor-related orphan receptor gamma t (ROR gamma t). We identify the nuclear receptor peroxisome proliferator-activated receptor gamma (PPAR gamma) as a key negative regulator of human and mouse Th17 differentiation. PPAR gamma activation in CD4(+) T cells selectively suppressed Th17 differentiation, but not differentiation into Th1, Th2, or regulatory T cells. Control of Th17 differentia…

MESH: Nuclear Receptor Subfamily 1 Group F Member 3Helper-InducerReceptors Retinoic AcidT-LymphocytesMESH: Interleukin-17Cellular differentiationRetinoic AcidPeroxisome proliferator-activated receptorNeurodegenerativeInbred C57BLMedical and Health SciencesMiceInterleukin 210302 clinical medicineGroup FRAR-related orphan receptor gammaMESH: Nuclear Receptor Co-Repressor 2Receptors2.1 Biological and endogenous factorsThyroid HormoneImmunology and AllergyMESH: AnimalsAetiologyEncephalomyelitisPromoter Regions Geneticchemistry.chemical_classificationOrphan receptor0303 health sciencesReceptors Thyroid HormoneInterleukin-17Cell DifferentiationT-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 33. Good healthCell biologyDNA-Binding Proteinsmedicine.anatomical_structureMESH: Repressor Proteins[SDV.IMM]Life Sciences [q-bio]/ImmunologyInterleukin 17MESH: Cell Differentiationmedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisNuclear Receptor Subfamily 1Member 31.1 Normal biological development and functioningT cellImmunologyBiologyAutoimmune DiseasePromoter RegionsExperimental03 medical and health sciencesGeneticUnderpinning researchMESH: Mice Inbred C57BLInternal medicineMESH: Promoter Regions GeneticGeneticsmedicineAnimalsHumansNuclear Receptor Co-Repressor 2MESH: Receptors Thyroid HormoneMESH: T-Lymphocytes Helper-InducerMESH: Encephalomyelitis Autoimmune ExperimentalMESH: Mice030304 developmental biologyMESH: Receptors Retinoic AcidMESH: HumansInflammatory and immune systemNeurosciencesBrief Definitive ReportCorrectionMESH: Multiple SclerosisBrain DisordersMice Inbred C57BLPPAR gammaRepressor ProteinsEndocrinologyMESH: PPAR gammaNuclear receptorchemistryMESH: DNA-Binding Proteins030217 neurology & neurosurgeryAutoimmuneJournal of Experimental Medicine
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Introducing coherent time control to cavity magnon-polariton modes

2020

By connecting light to magnetism, cavity-magnon-polaritons (CMPs) can build links from quantum computation to spintronics. As a consequence, CMP-based information processing devices have thrived over the last five years, but almost exclusively been investigated with single-tone spectroscopy. However, universal computing applications will require a dynamic control of the CMP on demand and within nanoseconds. In this work, we perform fast manipulations of the different CMP modes with independent but coherent pulses to the cavity and magnon system. We change the state of the CMP from the energy exchanging beat mode to its normal modes and further demonstrate two fundamental examples of coheren…

Magnetism530 PhysicsGeneral Physics and AstronomyFOS: Physical sciencesPhysics::Opticslcsh:AstrophysicsTopology01 natural sciences010309 opticsNormal mode0103 physical sciencesMesoscale and Nanoscale Physics (cond-mat.mes-hall)lcsh:QB460-466Polaritonddc:530010306 general physicsQuantum computerPhysicsQuantum networkSpintronicsCondensed Matter - Mesoscale and Nanoscale PhysicsCondensed Matter::OtherPhysicsMagnonNanosecond530 Physiklcsh:QC1-999lcsh:PhysicsCommunications Physics
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Overrating Classifier Performance in ROC Analysis in the Absence of a Test Set: Evidence from Simulation and Italian CARATkids Validation

2019

Background The use of receiver operating characteristic curves, or “ROC analysis,” has become quite common in biomedical research to support decisions. However, sensitivity, specificity, and misclassification rates are still often estimated using the training sample, overlooking the risk of overrating the test performance. Methods A simulation study was performed to highlight the inferential implications of splitting (or not) the dataset into training and test set. The normality assumption was made for the classifier given the disease status, and the Youden's criterion considered for the detection of the optimal cutoff. Then, an ROC analysis with sample split was applied to assess the disc…

Male020205 medical informaticsperformance estimatorsmedia_common.quotation_subjectHealth Informatics02 engineering and technology03 medical and health sciences0302 clinical medicineHealth Information ManagementSurveys and QuestionnairesStatisticstrue predictive performanceRinite Alérgica0202 electrical engineering electronic engineering information engineeringmedicineHumanssample splitComputer Simulation030212 general & internal medicineChildAsmaNormalityAsthmaMathematicsmedia_commonAdvanced and Specialized NursingReceiver operating characteristicasthma control testasthma control test sample split performance estimators optimal cutoff simulation study true predictive performanceDiscriminant validityReproducibility of ResultsEstimatormedicine.diseasesimulation studyRhinitis AllergicAsthmaConfidence intervalROC CurveTest setoptimal cutoffFemaleClassifier (UML)
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TBL1XR1 mutations in Pierpont syndrome are not restricted to the recurrent p.Tyr446Cys mutation

2018

IF 2.264; International audience; Pierpont syndrome is a rare and sporadic syndrome, including developmental delay, facial characteristics, and abnormal extremities. Recently, a recurrent de novo TBL1XR1 variant (c.1337A > G; p.Tyr446Cys) has been identified in eight patients by whole‐exome sequencing. A dominant‐negative effect of this mutation is strongly suspected, since patients with TBL1XR1 deletion and other variants predicting loss of function do not share the same phenotype. We report two patients with typical Pierpont‐like syndrome features. Exome sequencing allowed identifying a de novo heterozygous missense TBL1XR1 variant in both patients, different from those already reported: …

Male0301 basic medicineAdolescentGenotypeReceptors Cytoplasmic and NuclearBiology03 medical and health sciences0302 clinical medicinePIERPONT SYNDROMEGeneticsHumansTBL1XR1Missense mutationAbnormalities MultipleRecurrent mutationGenetic TestingAllelesGenetics (clinical)Exome sequencingLoss functionUltrasonographyGeneticsComparative Genomic Hybridization[SDV.GEN]Life Sciences [q-bio]/GeneticsBrainFaciesNuclear ProteinsSyndromeMagnetic Resonance ImagingPhenotype3. Good healthRepressor ProteinsPhenotype030104 developmental biologyAmino Acid Substitution030220 oncology & carcinogenesisMutationMutation (genetic algorithm)Pierpont syndromeAmerican Journal of Medical Genetics Part A
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G6PD protects from oxidative damage and improves healthspan in mice

2016

S.N.-P. and P.J.F.-M. have been funded by the Spanish Association Against Cancer(aecc). Work in the laboratory of M.S. is funded by the CNIO and by grants from the Spanish Ministry of Economy co-funded by the European Regional Development Fund(SAF project), the European Research Council (ERC Advanced Grant), the Regional Government of Madrid co-funded by the European Social Fund (ReCaRe project), the European Union (RISK-IR project), the Botin Foundation and Banco Santander(Santander Universities Global Division), the Ramon Areces Foundation, and the AXA Foundation. Work in the laboratory of J.V. was supported by grants SAF2013-44663-R,from the Spanish Ministry of Education and Science (MEC…

Male0301 basic medicineAgingCellGeneral Physics and AstronomyDehydrogenaseEndogenymedicine.disease_causestressMicehemic and lymphatic diseasesratmécanismegenesreactive oxygen specieschemistry.chemical_classificationMultidisciplinary[SDV.BA]Life Sciences [q-bio]/Animal biologyQvieillissementCell biologymedicine.anatomical_structureanimal transgéniqueFemaleGenetically modified mouse[SDV.OT]Life Sciences [q-bio]/Other [q-bio.OT]congenital hereditary and neonatal diseases and abnormalitiesScienceTransgeneLongevityMice TransgenicGlucosephosphate DehydrogenaseBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesparasitic diseasesmedicineAnimalsHumansReactive oxygen speciesgènenutritional and metabolic diseasesGeneral ChemistrycellMolecular biologytransgenic mouseOxidative Stress030104 developmental biologymechanistic theorychemistryageingenzyme antioxydanteAgeingespèce reactive de l'oxygènecelluleReactive Oxygen SpeciesNADPOxidative stressNature Communications
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Impact of obesity and aging on crestal alveolar bone height in mice

2018

Obesity and aging are associated with periodontitis, which represents a chronic inflammatory disease of the tooth-supporting tissues, i.e. the periodontium. However, if both risk factors also have a negative impact on crestal alveolar bone in a clinically healthy periodontium, has yet to be elucidated and was analyzed in this in-vivo study. Eight C57BL/6 mice were fed a normal diet during the entire study. Half of these mice were sacrificed at week 19 (group 1: younger lean mice), whereas the other half of the animals were sacrificed at week 25 (group 2: older lean mice). In addition, four mice were fed a high-fat diet until their sacrifice at week 19 (group 3: younger obese mice). Mandible…

Male0301 basic medicineAgingmedicine.medical_specialtyNormal dietAlveolar Bone LossGingivaGene ExpressionMandibleDiet High-FatMice03 medical and health sciences0302 clinical medicineInternal medicineAlveolar ProcessMaxillamedicineAnimalsObesityNicotinamide PhosphoribosyltransferaseDental alveolusPeriodontitisAdiponectinbusiness.industryX-Ray Microtomography030206 dentistryGeneral MedicinePeriodontiummedicine.diseaseMolarMice Inbred C57BLCementoenamel junction030104 developmental biologyEndocrinologyCyclooxygenase 2MaxillaCytokinesCrestInflammation MediatorsAnatomybusinessDevelopmental BiologyAnnals of Anatomy - Anatomischer Anzeiger
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