Search results for "Notch"

showing 10 items of 93 documents

Neurovascular EGFL7 regulates adult neurogenesis in the subventricular zone and thereby affects olfactory perception

2016

Adult neural stem cells reside in a specialized niche in the subventricular zone (SVZ). Throughout life they give rise to adult-born neurons in the olfactory bulb (OB), thus contributing to neural plasticity and pattern discrimination. Here, we show that the neurovascular protein EGFL7 is secreted by endothelial cells and neural stem cells (NSCs) of the SVZ to shape the vascular stem-cell niche. Loss of EGFL7 causes an accumulation of activated NSCs, which display enhanced activity and re-entry into the cell cycle. EGFL7 pushes activated NSCs towards quiescence and neuronal progeny towards differentiation. This is achieved by promoting Dll4-induced Notch signalling at the blood vessel-stem …

Male0301 basic medicineGeneral Physics and AstronomyNEURAL STEM-CELLSMOUSEMiceSUBEPENDYMAL ZONENeural Stem CellsLateral VentriclesLINEAGE PROGRESSIONBRAININ-VIVOMice KnockoutNeuronal PlasticityMultidisciplinaryCell CycleQNeurogenesisNICHEAnatomyNeural stem cellCell biologyAdult Stem Cellsmedicine.anatomical_structureSignal TransductionSTIMULATES NEUROGENESISEGF Family of ProteinsNeurogenesisScienceNotch signaling pathwaySubventricular zoneBiologyInhibitory postsynaptic potentialArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesNeuroplasticitymedicineBiological neural networkAnimalsCalcium-Binding ProteinsProteinsGeneral ChemistryOlfactory PerceptionENDOTHELIAL-CELLSnervous system diseasesOlfactory bulbMice Inbred C57BLSELF-RENEWAL030104 developmental biologynervous system
researchProduct

The

2016

ABSTRACT Members of the Junctophilin (JPH) protein family have emerged as key actors in all excitable cells, with crucial implications for human pathophysiology. In mammals, this family consists of four members (JPH1-JPH4) that are differentially expressed throughout excitable cells. The analysis of knockout mice lacking JPH subtypes has demonstrated their essential contribution to physiological functions in skeletal and cardiac muscles and in neurons. Moreover, mutations in the human JPH2 gene are associated with hypertrophic and dilated cardiomyopathies; mutations in JPH3 are responsible for the neurodegenerative Huntington's disease-like-2 (HDL2), whereas JPH1 acts as a genetic modifier …

NotchGenotypeCardiomyopathyGenes InsectAnimals Genetically ModifiedAnimalsDrosophila ProteinsAllelesMammalsNeuronsHuntingtin ProteinReceptors NotchMusclesMyocardiumMembrane ProteinsReproducibility of ResultsDrosHuntington's diseaseDisease Models AnimalDrosophila melanogasterPhenotypeGene Knockdown TechniquesMutationNerve DegenerationPhotoreceptor Cells InvertebrateRNA InterferenceJunctophilinDrosophilaTrinucleotide Repeat ExpansionSignal TransductionResearch ArticleDisease modelsmechanisms
researchProduct

Truncating mutations in the last exon of NOTCH2 cause a rare skeletal disorder with osteoporosis.

2010

Hajdu-Cheney syndrome is a rare autosomal dominant skeletal disorder with facial anomalies, osteoporosis and acro-osteolysis. We sequenced the exomes of six unrelated individuals with this syndrome and identified heterozygous nonsense and frameshift mutations in NOTCH2 in five of them. All mutations cluster to the last coding exon of the gene, suggesting that the mutant mRNA products escape nonsense-mediated decay and that the resulting truncated NOTCH2 proteins act in a gain-of-function manner.

AdultMaleHeterozygoteHajdu–Cheney syndromeAdolescentmedia_common.quotation_subjectNonsenseMolecular Sequence DataBiologymedicine.disease_causeHajdu-Cheney SyndromeFrameshift mutationExonYoung AdultRare DiseasesSkeletal disorderGeneticsmedicineHumansAmino Acid SequenceReceptor Notch2Frameshift MutationGeneExome sequencingmedia_commonGeneticsMutationBase SequenceDNAExonsMiddle Agedmedicine.diseasePedigreeCodon NonsenseChild PreschoolMutationFemaleSignal TransductionNature genetics
researchProduct

Assessment of multiaxial fatigue behaviour of welded joints under combined bending and torsion by application of a fictitious notch radius

2004

Abstract A major problem in the assessment of the multiaxial fatigue behaviour in welded joints is the determination of local notch stresses, since the real local radii in most cases are not known. This can be circumvented by introducing a fictitious notch radius, according to Neuber and Radaj. The calculations of local equivalent stress amplitudes for welded joints (tube–tube and flange–tube) under pure bending, pure torsion and combined in- and out-of-phase bending with torsion can then be carried out by stress-based and strain energy density-based criteria. Among the investigated methods, the most reliable results are obtained with the effective equivalent stress hypothesis, an integral …

Materials sciencebusiness.industryMechanical EngineeringTorsion (mechanics)Strain energy density functionStructural engineeringWeldingIndustrial and Manufacturing Engineeringlaw.inventionStrain energyEquivalent stressAmplitudeNotch radiusMechanics of MaterialslawModeling and SimulationPure bendingGeneral Materials SciencebusinessInternational Journal of Fatigue
researchProduct

TOX3 regulates neural progenitor identity

2016

The human genomic locus for the transcription factor TOX3 has been implicated in susceptibility to restless legs syndrome and breast cancer in genome-wide association studies, but the physiological role of TOX3 remains largely unknown. We found Tox3 to be predominantly expressed in the developing mouse brain with a peak at embryonic day E14 where it co-localizes with the neural stem and progenitor markers Nestin and Sox2 in radial glia of the ventricular zone and intermediate progenitors of the subventricular zone. Tox3 is also expressed in neural progenitor cells obtained from the ganglionic eminence of E15 mice that express Nestin, and it specifically binds the Nestin promoter in chromati…

0301 basic medicineNeurogenesisBiophysicsNotch signaling pathwaySubventricular zoneMice TransgenicBiologyBiochemistryMice03 medical and health sciences0302 clinical medicineNeural Stem CellsSOX2PregnancyStructural BiologyGeneticsmedicineAnimalsRNA Small InterferingProgenitor cellMolecular BiologyCells Culturedreproductive and urinary physiologyNeuronsNeurogenesisGene Expression Regulation DevelopmentalNestinEmbryo MammalianMolecular biologyNeural stem cellMice Inbred C57BL030104 developmental biologymedicine.anatomical_structurenervous systemembryonic structuresTrans-ActivatorsFemaleStem cellApoptosis Regulatory ProteinsReceptors Progesterone030217 neurology & neurosurgeryBiochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms
researchProduct

Notch-1 decreased expression contributes to leptin receptor downregulation in nasal epithelium from allergic turbinates

2019

Abstract BACKGROUND: Allergic rhinitis is characterized by a remodeling of nasal epithelium. Since the Notch and TGF-β signaling pathways are known to be involved in cell differentiation and remodeling processes and leptin adipokine has already been identified as a marker for homeostasis in human bronchial and nasal epithelial cells of asthmatics, roles played by these pathways have been investigated for chronic allergic rhinitis. METHODS: The leptin/leptin receptor expression has been investigated in a study with 40 biopsies from allergic (AR, n = 18) and non-allergic (C, n = 22) inferior turbinates, using immunohistochemistry, immunofluorescence staining and RT-PCR. In addition, extracts …

0301 basic medicineAdultLeptinMalemedicine.medical_specialtyBiopsyPrimary Cell CultureAdipokineTurbinatesCell LineTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineDownregulation and upregulationInternal medicinemedicineHomeostasisHumansRNA MessengerReceptor Notch1610 Medicine & healthReceptorMolecular BiologyNotch 1Leptin receptorChemistryLeptindigestive oral and skin physiologyEpithelial CellsMiddle AgedRhinitis AllergicAllergic rhinitis Epithelium Leptin NotchEpitheliumNasal Mucosa030104 developmental biologyEndocrinologymedicine.anatomical_structureGene Expression Regulation030220 oncology & carcinogenesisCase-Control StudiesMolecular MedicineReceptors LeptinFemaleSignal transductionSignal Transduction
researchProduct

Duchenne Muscular Dystrophy (DMD): Should it be Considered a Systemic Disease?

2016

Duchenne muscular dystrophy (DMD) is an X-linked muscle disease characterized by progressive skeletal muscle loss and development of respiratory failure due to involvement of respiratory muscles. Similar to human DMD, the mdx mouse model lacks dystrophin but is characterized by relatively mild muscle injury, allowing testing the effects of mild endurance exercise training on dystrophic skeletal muscle. We were interested to study the effects of exercise training on airway cells in trained mdx mice by applying the same protocol previously tested in Swiss mice. We found that mdx mice showed little airway inflammation associated with training, but developed increasing apoptosis of airway cells…

musculoskeletal diseasescongenital hereditary and neonatal diseases and abnormalitiesmdx mousePathologymedicine.medical_specialtyAirway epitheliumDuchenne muscular dystrophyNotch pathwaySkeletal muscleSettore MED/10 - Malattie Dell'Apparato RespiratorioBiologymedicine.diseaseChaperonin Hsp60Settore BIO/09 - FisiologiaDystrophinmedicine.anatomical_structureRespiratory failureEndurance trainingmedicinebiology.proteinRespiratory epitheliumRespiratory systemDystrophinGoblet cellSingle Cell Biology
researchProduct

Presenilin is the molecular target of acidic γ-secretase modulators in living cells.

2012

The intramembrane-cleaving protease γ-secretase catalyzes the last step in the generation of toxic amyloid-β (Aβ) peptides and is a principal therapeutic target in Alzheimer's disease. Both preclinical and clinical studies have demonstrated that inhibition of γ-secretase is associated with prohibitive side effects due to suppression of Notch processing and signaling. Potentially safer are γ-secretase modulators (GSMs), which are small molecules that selectively lower generation of the highly amyloidogenic Aβ42 peptides but spare Notch processing. GSMs with nanomolar potency and favorable pharmacological properties have been described, but the molecular mechanism of GSMs remains uncertain an…

CellsProtein subunitDrug Evaluation PreclinicalNotch signaling pathwaylcsh:MedicineCHO CellsBiochemistryModels BiologicalPresenilinInhibitory Concentration 50CricetulusCricetinaeAmyloid precursor proteinAnimalsHumansMolecular Targeted TherapyEnzyme InhibitorsMode of actionlcsh:ScienceBiologyCells CulturedMultidisciplinarybiologyEnzyme ClassesChemistryAnti-Inflammatory Agents Non-SteroidalHEK 293 cellslcsh:RChemical ReactionsPresenilinsProteinsSmall moleculeEnzymesChemistryHEK293 CellsNeurologyBiochemistrybiology.proteinMedicineDementialcsh:QAmyloid Precursor Protein SecretasesAmyloid precursor protein secretaseResearch ArticlePLoS ONE
researchProduct

A central role for Notch in effector CD8(+) T cell differentiation.

2014

Activated CD8(+) T cells choose between terminal effector cell (TEC) or memory precursor cell (MPC) fates. We found that the signaling receptor Notch controls this 'choice'. Notch promoted the differentiation of immediately protective TECs and was correspondingly required for the clearance of acute infection with influenza virus. Notch activated a major portion of the TEC-specific gene-expression program and suppressed the MPC-specific program. Expression of Notch was induced on naive CD8(+) T cells by inflammatory mediators and interleukin 2 (IL-2) via pathways dependent on the metabolic checkpoint kinase mTOR and the transcription factor T-bet. These pathways were subsequently amplified d…

ImmunologyNotch signaling pathwayMice TransgenicCell SeparationBiologyAdaptive ImmunityCD8-Positive T-LymphocytesEffector cellLymphocyte ActivationReal-Time Polymerase Chain ReactionArticlememoryMiceOrthomyxoviridae InfectionsCell surface receptorT-Lymphocyte SubsetsTransduction GeneticPrecursor cellImmunology and AllergyCytotoxic T cellAnimalsGeneticsReceptors NotchEffectorCell DifferentiationFlow CytometryAdoptive TransferTEC3. Good healthCell biologyMice Inbred C57BLeffectorCD8 T cellMPCInfluenza A virusinflammationTranscriptomeCD8Nature immunology
researchProduct

Fauna Europaea: Hymenoptera - Apocrita (excl. Ichneumonoidea)

2015

Fauna Europaea provides a public web-service with an index of scientific names (including important synonyms) of all living European land and freshwater animals, their geographical distribution at country level (up to the Urals, excluding the Caucasus region), and some additional information. The Fauna Europaea project covers about 230,000 taxonomic names, including 130,000 accepted species and 14,000 accepted subspecies. This represents a huge effort by more than 400 contributing specialists throughout Europe and is a unique (standard) reference suitable for many users in science, government, industry, nature conservation and education. Hymenoptera is one of the four largest orders of inse…

Fauna EuropaeaBiologisk systematikInsectaKulbastaviaMymarommatoideaBiodiversityStephanoideaHymenopteraBiological SystematicsCarbotripluridaBilateriaApocritataxonomic indexingBiology (General)lcsh:QH301-705.5ProctotrupoideaData ManagementChrysidoideaPterygotaEcologybiologyEcologyCenozoicCephalornisCircumscriptional namesEvanioideaEuropeBoltonocostidaeVespoideaIchneumonoideaTiphiinaeBiogeographyCircumscriptional nameCeraphronoideaNeogeneApoideaData PaperCoelenterataFauna EuropaeaChalcidoideaArthropodaBioinformaticsQH301-705.5HymenopteridaNephrozoaZoologyProtostomiaBasalPlatygastroideaZoologiCircumscriptional names of the taxon underBiodiversity informaticsTrigonalyoideaSystematicsAnimaliaEumetabolaEcology Evolution Behavior and Systematicsbiology.organism_classificationStrashila incredibilisHymenopteralcsh:Biology (General)NotchiaEcdysozoaCynipoideaApocritaZoologyBiodiversity Data Journal
researchProduct