Search results for "Nucleases"

showing 10 items of 147 documents

DNA polymorphism of the human complement C8 beta gene: formal genetics and intragenic localization.

1989

The eighth component of human complement consists of three subunits of different molecular mass, which are coded for by three separate genetic loci. Polymorphisms have been described at the protein level for the alpha and beta subunits by means of sodium dodecyl sulfate gel electrophoresis and isoelectric focusing. Using a full-length human C8 beta cDNA probe, we have studied more than 100 individuals by Southern blot analysis to detect DNA polymorphisms. We have found two restriction fragment length polymorphisms (RFLPs) with the enzymes Taq I and Bam HI. The Taq I polymorphism is defined by two alleles, i.e., a single 4.9 kb fragment or two 2.8/2.1 kb fragments. The allele frequencies are…

GeneticsGel electrophoresisDeoxyribonuclease BamHIImmunologyBiologyMolecular biologyComplement C8Restriction fragmentBlotting SouthernGene mappingComplementary DNAGeneticsbiology.proteinHumansRestriction fragment length polymorphismDeoxyribonucleases Type II Site-SpecificGeneAllele frequencyAllelesPolymorphism Restriction Fragment LengthSouthern blotImmunogenetics
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Intragenomic recombination between homologous regions of genes II and IV promotes formation of bacteriophage f1 miniphages.

1987

GeneticsRecombination GeneticBase SequenceGenes ViralDNA Restriction EnzymesBiologybiology.organism_classificationColiphagesVirusHomology (biology)BacteriophageSequence Homology Nucleic AcidGeneticsHomologous chromosomeBase sequenceDeoxyribonucleases Type II Site-SpecificGeneRecombinationNucleic acids research
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DNA double-strand breaks trigger apoptosis in p53-deficient fibroblasts

2001

DNA double-strand breaks (DSBs) are induced by ionizing radiation (IR) and various radiomimetic agents directly, or indirectly as a consequence of DNA repair, recombination and replication of damaged DNA. They are ultimately involved in the generation of chromosomal aberrations and were reported to cause genomic instability, gene amplification and reproductive cell death. To address the question of whether DSBs act as a trigger of apoptosis, we induced DSBs by means of restriction enzyme electroporation and compared the effect with IR in mouse fibroblasts that differ in p53 status [wild-type (+/+) versus p53-deficient (-/-) cells]. We show that (i) electroporation of PVU:II is highly effici…

Genome instabilityCancer ResearchProgrammed cell deathTime FactorsDNA RepairDNA repairBlotting WesternApoptosisBiologymedicine.disease_causeCell LineMiceNecrosischemistry.chemical_compoundProto-Oncogene ProteinsRadiation IonizingmedicineAnimalsDeoxyribonucleases Type II Site-SpecificCells Culturedbcl-2-Associated X ProteinMice KnockoutRecombination GeneticMutationElectroporationDose-Response Relationship RadiationDNAGeneral MedicineTransfectionFibroblastsGenes p53Molecular biologyElectroporationProto-Oncogene Proteins c-bcl-2chemistryGamma RaysApoptosisComet AssayTumor Suppressor Protein p53DNADNA DamageCarcinogenesis
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PSD3 downregulation confers protection against fatty liver disease

2022

Fatty liver disease (FLD) is a growing health issue with burdening unmet clinical needs. FLD has a genetic component but, despite the common variants already identified, there is still a missing heritability component. Using a candidate gene approach, we identify a locus (rs71519934) at the Pleckstrin and Sec7 domain-containing 3 (PSD3) gene resulting in a leucine to threonine substitution at position 186 of the protein (L186T) that reduces susceptibility to the entire spectrum of FLD in individuals at risk. PSD3 downregulation by short interfering RNA reduces intracellular lipid content in primary human hepatocytes cultured in two and three dimensions, and in human and rodent hepatoma cell…

GenotypeEndocrinology Diabetes and MetabolismVARIANTSUSCEPTIBILITYPolymorphism Single NucleotideArticleCell LineMiceRibonucleasesPhysiology (medical)Internal MedicineAnimalsGuanine Nucleotide Exchange FactorsHumansRNA-SeqAllelesNon-alcoholic steatohepatitisNONALCOHOLIC STEATOHEPATITISHERITABILITYGene Expression ProfilingfungiNASHGenetic VariationCell BiologyMetabolic syndromeFatty LiverMetabolismGene Expression RegulationLiverEXOME-WIDE ASSOCIATION3121 General medicine internal medicine and other clinical medicineACIDHepatocytesSECRETIONDisease SusceptibilityVLDLBiomarkersTRIGLYCERIDESNon-alcoholic fatty liver disease
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High-resolution genotyping of Campylobacter strains isolated from poultry and humans with amplified fragment length polymorphism fingerprinting.

1999

ABSTRACT For epidemiological studies of Campylobacter infections, molecular typing methods that can differentiate campylobacters at the strain level are needed. In this study we used a recently developed genotyping method, amplified fragment length polymorphism (AFLP), which is based on selective amplification of restriction fragments of chromosomal DNA, for genetic typing of Campylobacter jejuni and Campylobacter coli strains derived from humans and poultry. We developed an automated AFLP fingerprinting method in which restriction endonucleases Hin dIII and Hha I were used in combination with one set of selective PCR primers. This method resulted in evenly distributed band patterns for amp…

GenotypeGenetics and Molecular BiologyCampylobacter coliDeoxyribonuclease HindIIImedicine.disease_causeApplied Microbiology and BiotechnologyCampylobacter jejuniPolymerase Chain ReactionPoultryMicrobiologyRestriction fragmentCampylobacter jejuniGenotypeCampylobacter InfectionsmedicineAnimalsDeoxyribonucleases Type II Site-SpecificGenotypingDNA PrimersGeneticsEcologybiologyCampylobacterfood and beveragesReproducibility of ResultsCampylobacterbiology.organism_classificationDNA FingerprintingBacterial Typing TechniquesElectrophoresis Gel Pulsed-FieldDNA profilingCampylobacter colibiology.proteinAmplified fragment length polymorphismFood ScienceBiotechnologyApplied and environmental microbiology
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Isolation and Characterization of Epidermal DNA and RNA from Guinea Pig Skin

1971

DNA and RNA were isolated from mammalian epidermis in a relatively small scale procedure. The high purity and native state of the DNA isolated is reflected by its molar absorptivity E (P), its thermal hyperchromicity and its hyperchromicity upon DNase treatment and by its sedimentation profile as well as by its profile in a cesium chloride density gradient. The very low content of protein and RNA, as well as the data of DNA determination, indicate that this method permits the isolation of a highly purified product. This is further substantiated by the determination of UV absorption spectra and by analysis of the base composition.The mammalian skin DNA showed the following properties: Mol. w…

GuanineChemical PhenomenaGuanineRNase PGuinea PigsColorDermatologyBiologyBiochemistry03 medical and health scienceschemistry.chemical_compoundCytosine0302 clinical medicineRibonucleasesCentrifugation Density GradientAnimalsChemical PrecipitationMolecular Biology030304 developmental biologySkin0303 health sciencesDeoxyribonucleasesChemistry PhysicalAdenineHydrolysisSpectrum AnalysisHyperchromicityRNAPhosphorusDNACell BiologyMolecular biologyThymineSedimentation coefficientMolecular Weightchemistry030220 oncology & carcinogenesisRNACytosineDNAThymineDensitometryJournal of Investigative Dermatology
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Oxidative DNA base damage induced by singlet oxygen and photosensitization: recognition by repair endonucleases and mutagenicity.

2000

We have analyzed the recognition by various repair endonucleases of DNA base modifications induced by three oxidants, viz. [4-(tert-butyldioxycarbonyl)benzyl]triethylammonium chloride (BCBT), a photochemical source of tert-butoxyl radicals, disodium salt of 1,4-etheno-2,3-benzodioxin-1,4-dipropanoic acid (NDPO(2)), a chemical source of singlet oxygen, and riboflavin, a type-I photosensitizer. The base modifications induced by BCBT, which were previously shown to be mostly 7,8-dihydro-8-oxoguanine (8-oxoGua) residues, were recognized by Fpg and Ogg1 proteins, but not by endonuclease IIII, Ntg1 and Ntg2 proteins. In the case of singlet oxygen induced damage, 8-oxoGua accounted for only 35% of…

GuanineDNA LigasesLightGuanineDNA damageRiboflavinMolecular Sequence DataToxicologySubstrate Specificitychemistry.chemical_compoundEndonucleaseBacterial ProteinsGeneticsPhotosensitizerPentosyltransferasesMolecular BiologybiologyBase SequenceSinglet oxygenEscherichia coli ProteinsMutagenesisCorticoviridaeProteinsEndonucleasesDNA-(apurinic or apyrimidinic site) lyaseOxygenBiochemistrychemistryDNA ViralMutationbiology.proteinOxidation-ReductionDNADNA DamageMutation research
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Association Between Exercise and Pubertal BMD Is Modulated by Estrogen Receptor α Genotype

2003

Genetic and environmental factors contribute to bone mass, but the ways they interact remain poorly understood. This study of 245 pre- and early pubertal girls found that the PvuII polymorphism in the ER- gene modulates the effect of exercise on BMD at loaded bone sites. Introduction: Impaired achievement of bone mass at puberty is an important risk factor for the development of osteoporosis in later life. Genetic, as well as environmental, factors contribute to bone mass, but the ways they interact with each other remain poorly understood. Materials and Methods: We investigated the interaction between a PvuII polymorphism at the ER- gene and physical activity (PA) on the modulation of bone…

Heterozygotemedicine.medical_specialtyAdolescentGenotypemedicine.drug_classEndocrinology Diabetes and MetabolismOsteoporosisEstrogen receptorSingle-nucleotide polymorphismPhysical exerciseBiologyBone and BonesBody Mass IndexRisk FactorsInternal medicineGenotypemedicineBody SizeHumansOrthopedics and Sports MedicineChildDeoxyribonucleases Type II Site-SpecificExercisePolymorphism GeneticHomozygotePubertyEstrogen Receptor alphaHeterozygote advantagemedicine.diseaseEndocrinologyEstrogenBody CompositionFemaleBody mass indexJournal of Bone and Mineral Research
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C4 DNA RFLP reference typing report.

1990

One hundred and three individual DNA samples (including 23 families) were studied at the gene level during the reference typing of the fourth component of human complement at the VIth Complement Genetics Workshop in Mainz (1989). All samples were analyzed with the restriction enzyme Taq I and with two DNA probes recognizing the 5' ends of both C4 genes and the two adjacent 21-hydroxylase genes. This RFLP is informative for the number of C4 genes as well as for their respective gene size. We found a high degree of variation regarding the number of C4 genes, i.e. haplotypes with 1-3 structural C4 genes of 16 or 22 kb size. By correlating these haplotypes to the complotypes obtained by protein…

ImmunologyBiologyMajor Histocompatibility Complexchemistry.chemical_compoundHumansTypingDeoxyribonucleases Type II Site-SpecificGeneAllelesGeneticsModels GeneticHybridization probeHaplotypeGenetic VariationComplement C4HematologyDNARestriction enzymeBlotting SouthernchemistryHaplotypesMultilocus sequence typingSteroid 21-HydroxylaseRestriction fragment length polymorphismDNAPolymorphism Restriction Fragment LengthComplement and inflammation
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Bgl II restriction fragment length polymorphism of human complement C4A gene coincides with BF*F allele of factor B.

1988

ImmunologyImmunogeneticsBiologyComplement factor Bchemistry.chemical_compoundRestriction mapBacterial ProteinsGeneticsHumansAlleleDeoxyribonucleases Type II Site-SpecificGeneAllelesSouthern blotGeneticsRecombination GeneticEnzyme PrecursorsPolymorphism GeneticComplement C4aNucleic Acid HybridizationComplement C4DNA Restriction EnzymesMolecular biologychemistryHaplotypesRestriction fragment length polymorphismDNAPolymorphism Restriction Fragment LengthComplement Factor BImmunogenetics
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