Search results for "Nucleus"

showing 10 items of 1803 documents

Studies on protein kinases involved in regulation of nucleocytoplasmic mRNA transport

1988

The rate of energy-dependent nucleoside triphosphatase (NTPase)-mediated nucleocytoplasmic translocation of poly(A)-containing mRNA [poly(A)+mRNA] across the nuclear envelope is thought to be regulated by poly(A)-sensitive phosphorylation and dephosphorylation of nuclear-envelope protein. Studying the phosphorylation-related inhibition of the NTPase, we found that phosphorylation of one polypeptide of rat liver envelopes by endogenous NI- and NII-like protein kinase was particularly sensitive to poly(A). This polypeptide (106 kDa) was also phosphorylated by nuclear-envelope-bound Ca2+-activated and phospholipid-dependent protein kinase (protein kinase C). Activation of kinase C by tumour-pr…

MaleCytoplasmNuclear EnvelopeMitogen-activated protein kinase kinasePhosphatidylinositolsBiochemistryMAP2K7AnimalsRNA Messengerc-RafProtein kinase AMolecular BiologyProtein Kinase CProtein kinase CCell NucleusMembrane GlycoproteinsMAP kinase kinase kinasebiologyCyclin-dependent kinase 2Membrane ProteinsNuclear ProteinsBiological TransportRats Inbred StrainsCell BiologyMolecular biologyRatsNuclear Pore Complex ProteinsMicroscopy ElectronLiverBiochemistrybiology.proteinCyclin-dependent kinase 9PeptidesPoly AResearch ArticleBiochemical Journal
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Downregulation and Nuclear Relocation of MLP During the Progression of Right Ventricular Hypertrophy Induced by Chronic Pressure Overload

2000

Abstract The cardiac LIM domain protein MLP plays a crucial role in the architecture and mechanical function of cardiac myocytes. Mice lacking the MLP gene develop cardiac hypertrophy, dilated cardiopathy and heart failure. We investigated whether downregulation of MLP is induced by pressure overload and contributes to the physiopathology of cardiac hypertrophy and failure. We studied this mechanism in rat right ventricles submitted to pulmonary arterial hypertension, because it is known that this ventricle is very vulnerable to the deleterious effects of pressure overload. During the progression of cardiac hypertrophy to failure over a 31 days period there was a dramatic decrease by 50% of…

MaleCytoplasmmedicine.medical_specialtyTime FactorsTranscription GeneticHeart VentriclesDown-RegulationMuscle ProteinsCardiomegalyCytosolMyofibrilsDownregulation and upregulationRight ventricular hypertrophyInternal medicinePressureAnimalsVentricular FunctionMedicineMyocyteRNA MessengerRats WistarLungMolecular BiologyCell NucleusHomeodomain ProteinsPressure overloadReverse Transcriptase Polymerase Chain Reactionbusiness.industryMyocardiumLIM Domain Proteinsmedicine.diseaseImmunohistochemistryPulmonary hypertensionRatsMicroscopy Electronmedicine.anatomical_structureVentricleHeart failureCardiologyCardiology and Cardiovascular MedicinebusinessMyofibrilJournal of Molecular and Cellular Cardiology
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Electron microscopic observations on primary hepatocyte cultures infected with herpes simplex virus Types I and II

1984

The replication cycle of the Herpes simplex virus (HSV) strains I and II has so far been described mainly in established proliferative cell cultures. Most of the biochemical data and ultrastructural cell changes regarding the virus-cell interaction have been obtained from ‘permissive’ cells which allow almost unrestricted viral multiplication. It seems obvious, however, that the in vivo viral infections are not represented adequately by these experiments. In order to achieve a more realistic view of the ultrastructural events during HSV infection of adult tissue, cell cultures were prepared from adult mouse and rat livers and infected with several HSV strains. Established ‘permissive’ cell …

MaleCytoplasmvirusesCellBiologyVirus Replicationmedicine.disease_causeHerpesviridaeVirusMicesymbols.namesakemedicineAnimalsCells CulturedCell NucleusEndoplasmic reticulumHerpes SimplexDesmosomesGolgi apparatusVirologyRatsMicroscopy Electronmedicine.anatomical_structureHerpes simplex virusLiverCell cultureHepatocytesymbolsFemaleVirchows Archiv B Cell Pathology Including Molecular Pathology
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Directional Deep Brain Stimulation for Parkinson's Disease: Results of an International Crossover Study With Randomized, Double-Blind Primary Endpoin…

2022

Objective Published reports on directional deep brain stimulation (DBS) have been limited to small, single-center investigations. Therapeutic window (TW) is used to describe the range of stimulation amplitudes achieving symptom relief without side effects. This crossover study performed a randomized double-blind assessment of TW for directional and omnidirectional DBS in a large cohort of patients implanted with a DBS system in the subthalamic nucleus for Parkinson's disease. Materials and methods Participants received omnidirectional stimulation for the first three months after initial study programming, followed by directional DBS for the following three months. The primary endpoint was a…

MaleDeep brain stimulationParkinson's diseasemedicine.medical_treatmentParkinson's diseaseDeep Brain StimulationStimulation03 medical and health sciences0302 clinical medicinemedicineClinical endpointHumanstherapeutic windowNeurostimulationTherapeutic windowCross-Over Studiesbusiness.industrydirectional programmingParkinson DiseaseGeneral Medicinemedicine.diseaseCrossover studySubthalamic nucleusAnesthesiology and Pain MedicineTreatment OutcomeNeurologyAnesthesiaQuality of LifeFemaleNeurology (clinical)business030217 neurology & neurosurgery
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Cross-frequency coupling between gamma oscillations and deep brain stimulation frequency in Parkinson's disease.

2020

Abstract The disruption of pathologically enhanced beta oscillations is considered one of the key mechanisms mediating the clinical effects of deep brain stimulation on motor symptoms in Parkinson’s disease. However, a specific modulation of other distinct physiological or pathological oscillatory activities could also play an important role in symptom control and motor function recovery during deep brain stimulation. Finely tuned gamma oscillations have been suggested to be prokinetic in nature, facilitating the preferential processing of physiological neural activity. In this study, we postulate that clinically effective high-frequency stimulation of the subthalamic nucleus imposes cross-…

MaleDeep brain stimulationmedicine.medical_treatmentDeep Brain StimulationStimulationcross-frequency couplingsource analysis610 Medicine & healthArticlePremotor cortexvolume of tissue activatedSubthalamic NucleusCerebellumGamma RhythmNeural PathwaysmedicineGamma RhythmHumans610 Medicine & healthAgedMovement DisordersSupplementary motor areaResting state fMRIChemistryMotor CortexElectroencephalographyParkinson DiseaseMiddle AgedSubthalamic nucleusmedicine.anatomical_structureFemaleNeurology (clinical)gamma oscillationsBeta RhythmNeuroscienceAlgorithmsMotor cortex
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Hampered long-term depression and thin spine loss in the nucleus accumbens of ethanol-dependent rats.

2014

Alcoholism involves long-term cognitive deficits, including memory impairment, resulting in substantial cost to society. Neuronal refinement and stabilization are hypothesized to confer resilience to poor decision making and addictive-like behaviors, such as excessive ethanol drinking and dependence. Accordingly, structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing the use of alcohol after chronic ingestion. Here we show that ethanol-dependent rats display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, accompanied by a reduction of tyrosine hydroxylase immunostaining and postsynaptic density 95…

MaleDendritic spineDendritic SpinesGlutamic AcidNucleus accumbensNeurotransmissionMedium spiny neuronSynaptic TransmissionNucleus AccumbensOrgan Culture TechniquesAnimalsRats WistarLong-term depressionLong-Term Synaptic Depressiondopamine synaptic plasticity Golgi glutamateMultidisciplinaryNeuronal PlasticityEthanolDopaminergic NeuronsLong-Term Synaptic DepressionCentral Nervous System DepressantsRatsAlcoholismPNAS PlusSynaptic plasticitySettore BIO/14 - FarmacologiaPsychologyNeurosciencePostsynaptic densityProceedings of the National Academy of Sciences of the United States of America
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Modifications of head turning and circling movement following sulpiride microinjections into nucleus accumbens in the rat

1995

The aim of this study was to determine whether a relationship exists between nucleus accumbens D2 receptors, circling behavior, and its first stage, the head turning. Rats were unilaterally lesioned in the substantia nigra with 6-hydroxydopamine and afterward treated with d-amphetamine IP following bilateral intraaccumbens microinjections (1, 5, 10 micrograms/0.5 microliters) of sulpiride, a D2 receptor antagonist. Computer-assisted video analysis allowed the study of some parameters (number of turns, type of turn, head turning duration, degree and speed) characterizing rotatory activity. Sulpiride microinfusion resulted in a dose-dependent decrease of the number of turns and head rotation …

MaleDextroamphetamineMicroinjectionsRotationDopamine AgentsSubstantia nigraNucleus accumbensNucleus Accumbenschemistry.chemical_compoundDopamineBasal gangliamedicineAnimalsRats WistarOxidopamineMicroinjectionDose-Response Relationship DrugGeneral NeuroscienceSympathectomy ChemicalRatsDopamine D2 Receptor AntagonistschemistryMicroinjectionsDopamine AntagonistsStereotyped BehaviorSulpirideSulpiridePsychologyHeadNeuroscienceOxidopaminemedicine.drugBrain Research Bulletin
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[18F]PR04.MZ PET/CT Imaging for Evaluation of Nigrostriatal Neuron Integrity in Patients With Parkinson Disease.

2020

Introduction Degeneration of dopaminergic, nigrostriatal neurons is the hallmark of Parkinson disease (PD), and PET quantification of dopamine transporters is a widely accepted method for differential diagnosis between idiopathic PD and essential tremor. [18F]PR04.MZ is a new PET tracer with excellent imaging properties allowing for precise quantification of striatal and extrastriatal dopamine transporter. Here we describe our initial experience with [18F]PR04.MZ PET/CT in a larger cohort of healthy controls and PD patients as a proof-of-concept study for this tracer. Methods Eighteen healthy subjects, 19 early PD patients (Hoehn-Yahr I–II), and 13 moderate-advanced PD patients (Hoehn-Yahr …

MaleDopamineCaudate nucleusPET imagingSubstantia nigra[18F]PR04.MZ030218 nuclear medicine & medical imagingCohort Studies03 medical and health sciences0302 clinical medicineDopaminePositron Emission Tomography Computed TomographyMedicineHumansRadiology Nuclear Medicine and imagingdopamine transporterDopamine transporterAgedNeuronsDopamine Plasma Membrane Transport ProteinsbiologyEssential tremorbusiness.industryPars compactaPutamenDopaminergicParkinson DiseaseGeneral MedicineOriginal ArticlesMiddle Agedmedicine.diseaseSubstantia Nigranervous system030220 oncology & carcinogenesisbiology.proteinmovement disordersFemalebusinessNuclear medicinemedicine.drugClinical nuclear medicine
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Effects of scopolamine on dopamine neurons in the substantia nigra : role of the pedunculopontine tegmental nucleus

2009

Previous neurochemical and behavioral studies suggest that muscarinic receptor antagonism has an excitatory effect on the nigrostriatal dopamine (DA) system. Using in vivo extracellular single unit recording, this study examined whether blockade of the muscarinic receptor by scopolamine alters the firing properties of DA neurons in the substantia nigra (SN). Scopolamine was administered either systemically or locally to DA neurons using microiontophoresis. Surprisingly, scopolamine did not cause any significant change in either the firing rate or pattern of the spontaneously active DA neurons. However, systemic injection of scopolamine significantly increased the number of active DA neurons…

MaleDopamineParkinson's diseaseScopolamineAction PotentialsSubstantia nigraMuscarinic AntagonistsStriatumelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaRats Sprague-DawleyCellular and Molecular NeuroscienceDopamineBasal gangliaMuscarinic acetylcholine receptorPedunculopontine Tegmental NucleusmedicineSubstantia nigraAnimalsPedunculopontine Tegmental NucleusNeuronsAnalysis of VarianceDose-Response Relationship DrugChemistryDrug Administration RoutesIontophoresisAcetylcholineRatsSubstantia Nigranervous systemAutoreceptorBasal gangliaNeuroscienceAcetylcholinemedicine.drug
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PSA-NCAM expression in the piriform cortex of the adult rat. Modulation by NMDA receptor antagonist administration.

2002

Administration of NMDA receptor antagonists upregulates the expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) in the adult hippocampus. Since the piriform cortex is also populated by PSA-NCAM immunoreactive neurons during adulthood, we sought to characterize them in detail and to test whether NMDA receptor antagonists also modulate PSA-NCAM in this cortical region. PSA-NCAM immunoreactivity is located mainly in layer II, where many neurogliaform and some pyramidal-semilunar transitional neurons are labeled. Many large neurons in layer III and endopiriform nucleus also express PSA-NCAM. Interestingly, some small labeled cells resembling migratory neuroblas…

MaleDoublecortin ProteinSynaptogenesisHippocampusNeural Cell Adhesion Molecule L1Receptors N-Methyl-D-AspartateRats Sprague-DawleyNeuroblastCell MovementPiriform cortexmedicineAnimalsMolecular BiologyNeural Cell Adhesion MoleculesNeuronsbiologyGeneral NeuroscienceOlfactory PathwaysDoublecortinRatsmedicine.anatomical_structurenervous systembiology.proteinSialic AcidsNeural cell adhesion moleculeNeurology (clinical)NeuNNeuroscienceNucleusExcitatory Amino Acid AntagonistsInjections IntraperitonealDevelopmental BiologyBrain research
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