Search results for "OTR"

showing 10 items of 6791 documents

Modulation of GABAA receptors by neurosteroids. A new concept to improve cognitive and motor alterations in hepatic encephalopathy

2016

Hepatic encephalopathy (HE) is a complex neuropsychiatric syndrome affecting patients with liver diseases, mainly those with liver cirrhosis. The mildest form of HE is minimal HE (MHE), with mild cognitive impairment, attention deficit, psychomotor slowing and impaired visuo-motor and bimanual coordination. MHE may progress to clinical HE with worsening of the neurological alterations which may lead to reduced consciousness and, in the worse cases, may progress to coma and death. HE affects several million people in the world and is a serious health, social and economic problem. There are no specific treatments for the neurological alterations in HE. The mechanisms underlying the cognitive …

0301 basic medicinemedicine.medical_specialtyNeuroactive steroidCirrhosisEndocrinology Diabetes and MetabolismClinical BiochemistryBiochemistry03 medical and health sciences0302 clinical medicineEndocrinologyCognitionMedicineAnimalsHumansHyperammonemiaPsychiatryMolecular BiologyHepatic encephalopathyHepatic encephalopathyPsychomotor learningComaNeurotransmitter Agentsbusiness.industryGABAA receptorsBrainCognitionHyperammonemiaCell Biologymedicine.diseaseReceptors GABA-AMotor coordination030104 developmental biologyHepatic EncephalopathyMolecular MedicineNeuroteroidsMotor coordinationCognitive functionmedicine.symptombusinessNeuroscience030217 neurology & neurosurgeryLocomotionPsychomotor Performance
researchProduct

Neurological aspects of medical use of cannabidiol

2017

Background: Cannabidiol (CBD) is among the major secondary metabolites of Cannabis devoid of the delta-9-tetra-hydrocannabinol psychoactive effects. It is a resorcinol-based compound with a broad spectrum of potential therapeutic properties, including neuroprotective effects in numerous pathological conditions. CBD neuroprotection is due to its antioxidant and antiinflammatory activities and the modulation of a large number of brain biological targets (receptors, channels) involved in the development and maintenance of neurodegenerative diseases. Objective: The aim of the present review was to describe the state of art about the pre-clinical research, the potential use and, when existing, t…

0301 basic medicinemedicine.medical_specialtyNeurologyCannabidiol; Neurodegenerative diseases; Neurological; Neurology; Neuroprotection; Neuroscience (all); PharmacologyDiseaseBioinformaticsNeurodegenerative diseaseNeuroprotection03 medical and health sciences0302 clinical medicineSettore MED/43 - Medicina LegaleCentral Nervous System DiseasesMedicineAnimalsCannabidiolHumansneurodegenerative diseasesYoung adultAmyotrophic lateral sclerosisPharmacologyNeuroscience (all)biologybusiness.industryGeneral NeuroscienceMultiple sclerosismedicine.diseasebiology.organism_classificationNeuroprotection030104 developmental biologyNeuroprotective AgentsNeurologyNeurologicalcannabidiol; neurodegenerative diseases; neurological; neurology; neuroprotectionCannabisbusinessCannabidiol030217 neurology & neurosurgerymedicine.drug
researchProduct

Current disease modifying approaches to treat Parkinson's disease

2015

Parkinson's disease (PD is a progressive neurological disorder characterized by the degeneration and death of midbrain dopamine and non-dopamine neurons in the brain leading to motor dysfunctions and other symptoms, which seriously influence the quality of life of PD patients. The drug L-dopa can alleviate the motor symptoms in PD, but so far there are no rational therapies targeting the underlying neurodegenerative processes. Despite intensive research, the molecular mechanisms causing neuronal loss are not fully understood which has hampered the development of new drugs and disease-modifying therapies. Neurotrophic factors are by virtue of their survival promoting activities attract candi…

0301 basic medicinemedicine.medical_specialtyParkinson's diseaseNeurturinNeurotrophic factorBiologySettore BIO/09 - Fisiologia03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeuroinflammationDopamineNeurotrophic factorsInternal medicineα-SynucleinmedicineGlial cell line-derived neurotrophic factorMolecular BiologyCerebral dopamine neurotrophic factorDopamine neuronPharmacologyDopaminergicCell Biologymedicine.diseaseDopamine neurons; ER stress; Mitochondria; Neuroinflammation; Neuropeptides; Neurotrophic factors; Protein aggregation; α-Synuclein; Molecular Medicine; Molecular Biology; Pharmacology; Cellular and Molecular Neuroscience; Cell Biology3. Good healthMitochondriaNeuropeptide030104 developmental biologyNerve growth factorEndocrinologybiology.proteinER streMolecular MedicineProtein aggregationNeuroscience030217 neurology & neurosurgerymedicine.drug
researchProduct

NT-3 protein levels are enhanced in the hippocampus of PRG1-deficient mice but remain unchanged in PRG1/LPA2 double mutants

2015

The plasticity-related gene 1 (PRG1) modulates bioactive lipids at the postsynaptic density and is a novel player in neuronal plasticity and regulation of glutamatergic transmission at principal neurons. PRG1, a neuronal molecule, is highly expressed during development and regeneration processes at the postsynaptic density, modulates synaptic lysophosphatidic acid (LPA) levels and is related to epilepsy and brain injury. In the present study, we analyzed the interaction between the synaptic molecules PRG1 and LPA2R with other plasticity-related molecules the neurotrophins. The protein levels of NGF, BDNF and NT-3 were measured using ELISA in hippocampal tissue of homozygous (PRG(-/-)) and h…

0301 basic medicinemedicine.medical_specialtyPhosphatidate PhosphataseHippocampusHippocampal formationHippocampusMice03 medical and health sciences0302 clinical medicineNeurotrophic factorsInternal medicineNerve Growth FactormedicineAnimalsNerve Growth FactorsReceptors Lysophosphatidic AcidMice KnockoutBrain-derived neurotrophic factorbiologyBrain-Derived Neurotrophic FactorGeneral NeuroscienceWild typeMice Mutant Strains030104 developmental biologyNerve growth factorEndocrinologynervous systemBiochemistrySynapsesbiology.proteinPostsynaptic density030217 neurology & neurosurgeryNeurotrophinNeuroscience Letters
researchProduct

Optogenetically enhanced pituitary corticotroph cell activity post-stress onset causes rapid organizing effects on behaviour

2016

The anterior pituitary is the major link between nervous and hormonal systems, which allow the brain to generate adequate and flexible behaviour. Here, we address its role in mediating behavioural adjustments that aid in coping with acutely threatening environments. For this we combine optogenetic manipulation of pituitary corticotroph cells in larval zebrafish with newly developed assays for measuring goal-directed actions in very short timescales. Our results reveal modulatory actions of corticotroph cell activity on locomotion, avoidance behaviours and stimulus responsiveness directly after the onset of stress. Altogether, the findings uncover the significance of endocrine pituitary cell…

0301 basic medicinemedicine.medical_specialtyScienceGeneral Physics and AstronomyBiologyStimulus (physiology)OptogeneticsGeneral Biochemistry Genetics and Molecular BiologyArticleAnimals Genetically Modified03 medical and health sciencesAnterior pituitaryInternal medicinemedicineZebrafish larvaeAvoidance LearningEndocrine systemAnimalsCorticotrophsZebrafishQLMultidisciplinaryQGeneral ChemistryCorticotroph CellOptogenetics030104 developmental biologyEndocrinologymedicine.anatomical_structureCorticotropic cellNeuroscienceLocomotionStress PsychologicalHormone
researchProduct

Reducing Peripheral Inflammation with Infliximab Reduces Neuroinflammation and Improves Cognition in Rats with Hepatic Encephalopathy

2016

Inflammation contributes to cognitive impairment in patients with hepatic encephalopathy (HE). However, the process by which peripheral inflammation results in cognitive impairment remains unclear. In animal models, neuroinflammation and altered neurotransmission mediate cognitive impairment. Taking into account these data, we hypothesized that in rats with HE: (1) peripheral inflammation is a main contributor to neuroinflammation; (2) neuroinflammation in hippocampus impairs spatial learning by altering AMPA and/or NMDA receptors membrane expression; (3) reducing peripheral inflammation with infliximab (anti-TNF-a) would improve spatial learning; (4) this would be associated with reduced n…

0301 basic medicinemedicine.medical_specialtyTNF-ahepatic encephalopathyHippocampusMorris water navigation taskInflammationAMPA receptorNeurotransmissionlcsh:RC321-571neuroinflammation03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineInternal medicinemedicineneurotransmissionlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyNeuroinflammationOriginal Researchcognitive impairmentbusiness.industryGlutamate receptor030104 developmental biologyEndocrinologynervous systemNMDA receptormedicine.symptombusinessNeuroscience030217 neurology & neurosurgeryNeuroscience
researchProduct

Type IV Laryngotracheoesophageal Cleft Associated with Type III Esophageal Atresia in 1p36 Deletions Containing the RERE Gene: Is There a Causal Role…

2018

The causes of embryological developmental anomalies leading to laryngotracheoesophageal clefts (LTECs) are not known, but are proposed to be multifactorial, including genetic and environmental factors. Haploinsufficiency of the RERE gene might contribute to different phenotypes seen in individuals with 1p36 deletions. We describe a neonate of an obese mother, diagnosed with type IV LTEC and type III esophageal atresia (EA), in which a 1p36 deletion including the RERE gene was detected. On the second day of life, a right thoracotomy and extrapleural esophagus atresia repair were attempted. One week later, a right cervical approach was performed to separate the cervical esophagus from the tra…

0301 basic medicinemedicine.medical_specialtyType IV Laryngotracheoesophageal Cleft Type III Esophageal Atresia 1p36 Deletions RERE Genemedicine.medical_treatmentAnastomosisGastroenterology03 medical and health sciences0302 clinical medicineInternal medicineMedicineThoracotomyEsophagus030223 otorhinolaryngologyEpigenomicsbusiness.industrylcsh:RJ1-570lcsh:PediatricsGeneral Medicinemedicine.diseasePhenotype030104 developmental biologymedicine.anatomical_structureAtresiaFailure to thrivemedicine.symptombusinessHaploinsufficiencyCase Reports in Pediatrics
researchProduct

The genomic sequence of Exiguobacterium chiriqhucha str. N139 reveals a species that thrives in cold waters and extreme environmental conditions

2017

We report the genome sequence of Exiguobacterium chiriqhucha str. N139, isolated from a high-altitude Andean lake. Comparative genomic analyses of the Exiguobacterium genomes available suggest that our strain belongs to the same species as the previously reported E. pavilionensis str. RW-2 and Exiguobacterium str. GIC 31. We describe this species and propose the chiriqhucha name to group them. ‘Chiri qhucha’ in Quechua means ‘cold lake’, which is a common origin of these three cosmopolitan Exiguobacteria. The 2,952,588-bp E. chiriqhucha str. N139 genome contains one chromosome and three megaplasmids. The genome analysis of the Andean strain suggests the presence of enzymes that confer E. ch…

0301 basic medicinemegaplasmidBioinformaticsOtras Ciencias Biológicas[SDV]Life Sciences [q-bio]Microbial metabolismBiodiversitylcsh:MedicineGenomicsTryptophan biosynthesisMicrobiology High altitude Andean lakesBiologySubjects BiochemistryGenomeBiochemistryMicrobiologyGeneral Biochemistry Genetics and Molecular BiologyCiencias Biológicas//purl.org/becyt/ford/1 [https]03 medical and health sciencesExtremophilesArsenic resistanceExiguobacteriumBotanyExtremophile//purl.org/becyt/ford/1.6 [https]genome2. Zero hungerWhole genome sequencingGeneticsGeneral Neurosciencelcsh:RGeneral MedicineBiodiversityGenomicsMetals or metalloidsExiguobacteriumbiology.organism_classificationHigh altitude Andean lakes030104 developmental biologyMicrobial population biology13. Climate actionUV resistanceBacterial metabolismGeneral Agricultural and Biological SciencesCIENCIAS NATURALES Y EXACTAS
researchProduct

Odorant Metabolism Analysis by an Automated Ex Vivo Headspace Gas-Chromatography Method

2015

International audience; In the olfactory epithelium (OE), odorant metabolizing enzymes have the dual function of volatile component detoxification and active clearance of odorants from the perireceptor environment to respectively maintain the integrity of the tissues and the sensitivity of the detection. Although emphasized by recent studies, this enzymatic mechanism is poorly documented in mammals. Thus, olfactory metabolism has been characterized mainly in vitro and for a limited number of odorants. The automated ex vivo headspace gas-chromatography method that was developed here was validated to account for odorant olfactory metabolism. This method easily permits the measurement of the f…

0301 basic medicineodorant metabolizing enzymesPhysiology[SDV]Life Sciences [q-bio][ SDV.BA ] Life Sciences [q-bio]/Animal biologyheadspace gas-chromatographylocalizationAutomationBehavioral Neurosciencerabbit (Oryctolagus cuniculus)rat olfactory mucosaComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationnewborn rabbit[SDV.BA]Life Sciences [q-bio]/Animal biologyperireceptor eventsmammary pheromoneSensory Systemsmedicine.anatomical_structureBiochemistryPheromonepartition-coefficientsRabbitsbiotransformationpsychological phenomena and processesolfactionChromatography GasOlfactionequilibrium03 medical and health sciencesOlfactory mucosaOlfactory MucosaPhysiology (medical)medicineAnimals[CHIM]Chemical Sciences[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyChromatographyMetabolismIn vitro030104 developmental biologyEnzymechemistry13. Climate actionOdorantsolfactory epitheliumacetateepitheliumOlfactory epitheliumEx vivonasal-mucosa
researchProduct

Repeated, Intermittent Social Defeat across the Entire Juvenile Period Resulted in Behavioral, Physiological, Hormonal, Immunological, and Neurochemi…

2016

The developing brain is vulnerable to social defeat during the juvenile period. As complements of human studies, animal models of social defeat provide a straightforward approach to investigating the functional and neurobiological consequences of social defeats. Taking advantage of agonist behavior and social defeat in male golden hamster, a set of 6 experiments was conducted to investigate the consequences at multiple levels in young adulthood resulting from repeated, intermittent social defeats or “social threats” across the entire juvenile period. Male hamsters at postnatal day 28 (P28) were randomly assigned to either the social defeat, “social threat”, or arena control group, and they …

0301 basic medicinesocial threatCognitive NeuroscienceHippocampusContext (language use)cortisolpro-inflammatory cytokinesrepeated intermittent social defeatDevelopmental psychologySocial defeat03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineNeurochemicalmonoamine neurotransmittersJuvenileYoung adultOriginal Researchmale golden hamstersSocial relationadolescent bullying030104 developmental biologyjuvenileNeuropsychology and Physiological PsychologyPsychology030217 neurology & neurosurgeryGolden hamsterNeuroscienceFrontiers in Behavioral Neuroscience
researchProduct