Search results for "OTR"

showing 10 items of 6791 documents

Adrenocorticotropic hormone and cortisol plasma levels directly correlate with childhood neglect and depression measures in addicted patients.

2008

Hypothalamic-pituitary-adrenal (HPA) axis dysfunction has been reported to be involved in vulnerability to alcohol and drug dependence in humans, possibly underlying both addictive behaviour and depression susceptibility. The aim of the present study was to investigate the possible interactions between childhood adverse experiences, depressive symptoms and HPA axis function in addicted patients, in comparison with healthy control. Eighty-two abstinent heroin or cocaine dependent patients and 44 normal controls, matched for age and sex, completed the symptoms Check List-90 (SCL-90), measuring depressive symptoms, and the Childhood Experience of Care and Abuse Questionnaire. Blood samples wer…

AdultMaleendocrine systemmedicine.medical_specialtyHypothalamo-Hypophyseal SystemHydrocortisonemedia_common.quotation_subjectStatistics as TopicMedicine (miscellaneous)Poison controlPituitary-Adrenal SystemAdrenocorticotropic hormoneHeroinNeglectLife Change EventsStress Disorders Post-TraumaticCocaine-Related DisordersAdrenocorticotropic HormoneRisk Factorschildhood neglect depression adrenocorticotropic hormoneInjury preventionmedicineHumansChild AbusePsychiatryChildDepression (differential diagnoses)media_commonPharmacologyDepressive DisorderHeroin DependenceAddictionmedicine.diseaseObject AttachmentSubstance abusePsychiatry and Mental healthFemalePsychologyArousalhormones hormone substitutes and hormone antagonistsmedicine.drugClinical psychology
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The neuroendocrinological profile of roxindole, a dopamine autoreceptor agonist, in schizophrenic patients

1995

Roxindole is a potent autoreceptor-selective dopamine agonist with additional properties as a serotonin reuptake inhibitor and 5-HT1A agonist. In order to get more insight into its mode of action in various psychiatric populations, we evaluated the effects of subchronic roxindole treatment on pituitary and adrenal hormone secretion, i.e. release of prolactin, thyroid stimulating hormone (TSH), growth hormone (GH), luteinizing hormone (LH), and cortisol. Fifteen schizophrenic patients with positive and negative symptomatology, respectively, were treated with roxindole for 28 days. Both basal and thyrotropin releasing hormone (TRH) -induced prolactin secretion diminished significantly to 26.4…

AdultMaleendocrine systemmedicine.medical_specialtyIndolesHydrocortisoneendocrine system diseasesPyridinesThyrotropinThyrotropin-releasing hormonePharmacologyDopamine agonistchemistry.chemical_compoundThyroid-stimulating hormoneAnterior pituitaryRoxindoleInternal medicinemedicineHumansPharmacologybusiness.industryLuteinizing HormoneMiddle AgedProlactinGrowth hormone secretionOxindolesProlactinEndocrinologymedicine.anatomical_structurechemistryGrowth HormoneDopamine AgonistsSchizophreniaFemaleSchizophrenic PsychologyLuteinizing hormonebusinesshormones hormone substitutes and hormone antagonistsmedicine.drugPsychopharmacology
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Subcutaneous gonadotropin therapy in male patients with hypogonadotropic hypogonadism

1991

Objective The response to subcutaneous (SC) gonadotropin replacement therapy, using human chorionic gonadotropin (hCG) and human menopausal gonadotropin (hMG) or hCG alone, was evaluated in male hypothalamic hypogonadism. Design Sixteen patients with hypothalamic hypogonadism were treated with gonadotropins for induction of puberty and normalization of spermatogenesis. The results were analyzed retrospectively. Setting The study was carried out in a clinical endocrinology department providing tertiary care and in private practices of endocrinology. Patients Eight patients with idiopathic hypogonadotropic hypogonadism and eight patients with Kallmann's syndrome in prepubertal or early pubert…

AdultMaleendocrine systemmedicine.medical_specialtyMenotropinsAdolescentmedicine.drug_classmedicine.medical_treatmentChorionic GonadotropinInjections IntramuscularHuman chorionic gonadotropinSemen qualityHypogonadotropic hypogonadismInternal medicineTestisHumansMedicineEunuchismTestosteroneSexual MaturationSpermatogenesisRetrospective StudiesChemotherapybiologyurogenital systembusiness.industryHypogonadismObstetrics and Gynecologymedicine.diseaseSpermEndocrinologyReproductive MedicineHMG-CoA reductasebiology.proteinDrug Therapy CombinationGonadotropinbusinessSpermatogenesisFertility and Sterility
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Pregnancy in an azoospermic patient with markedly elevated serum follicle-stimulating hormone levels

1995

Objective To assess the possibility of achieving a pregnancy in an azoospermic patient with markedly elevated serum FSH level. Design A case report. Setting In vitro fertilization program at the Instituto Valenciano de Infertilidad. Patient An azoospermic patient with small testes and serum FSH level (38.7 mlU/mL) higher than three times normal. Testicular biopsy revealed Sertoli cell-only syndrome with focal spermatogenesis. Interventions Intracytoplasmic microinjection of testicular tissue-extracted spermatozoa. Main Outcome Measurements: Fertilization rate, cleavage rate, clinical pregnancy. Results Eight of 11 (73%) intact oocytes showed two pronuclei. All of them cleaved normally. Four…

AdultMaleendocrine systemmedicine.medical_specialtyMicroinjectionsmedicine.drug_classmedicine.medical_treatmentFertilization in VitroBiologyIntracytoplasmic sperm injectionAndrologyFollicle-stimulating hormonePregnancyInternal medicineTestismedicineHumansMicroinjectionAzoospermiaPregnancySertoli CellsIn vitro fertilisationurogenital systemObstetrics and GynecologyOligospermiamedicine.diseaseSpermatozoaEndocrinologyReproductive MedicineOocytesFemaleFollicle Stimulating HormoneGonadotropinSpermatogenesisFertility and Sterility
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Time-related effects of thyrotropin-releasing hormone (TRH) on the pituitary-thyroid axis and extrathyroidal targets.

2009

Thyrotropin-releasing hormone (TRH) is a tripeptide and acts as a stimulator of the pituitary-thyroid axis as well as having a great number of well defined extrathyroidal functions. Studies in experimental animals have shown, that TRH also has a role as a neuromodulator within the autonomous nervous system. In this study we analyzed the effects following peripheral administration of TRH (200 micrograms, 400 micrograms) in patients with endocrinological disorders and in healthy females and males. By means of a questionnaire, patients were asked about possible (side-) effects; ventilatory and cardiovascular monitoring was performed during steady state. The pulsatile TSH-secretion pattern was …

AdultMaleendocrine systemmedicine.medical_specialtyTime Factorsendocrine system diseasesEndocrinology Diabetes and MetabolismThyrotropin-releasing hormoneThyrotropinStimulationEndocrine System DiseasesNorepinephrine (medication)Pituitary thyroid axisEndocrinologyHeart RateInternal medicineSurveys and QuestionnairesHeart rateInternal MedicinemedicineHumansThyrotropin-Releasing Hormonebusiness.industryRespirationThyroidGeneral Medicinemedicine.anatomical_structureEpinephrineEndocrinologyFemalebusinesshormones hormone substitutes and hormone antagonistsmedicine.drugHormoneExperimental and clinical endocrinology
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Gonadotropin and Testosterone Secretion in Normal Human Males After Stimulation With Gonadotropin-Releasing Hormone (Gnrh) or Potent Gnrh Analogs Usi…

1978

Gonadotropin-releasing hormone (GnRH) and some potent long-acting GnRH analogs, applied by different routes of administration, were tested in six healthy human males. The effects on gonadotropin secretion were compared with the one after intravenous (i.v.) bolus injection of 25 microgram of GnRH. The net increase of luteinizing hormone (deltaLH) in serum produced by 25 microgram of GnRH i.v. was matched by subcutaneous (s.c.) injection of 100 microgram of GnRH, dissolved in 20% gelatin or without gelatin; 5 microgram of D-Ser (TBU)6-des-Gly10-GnRH-ethylamide i.v.; 5 microgram of D-Leu6-des-Gly10-GnRH-ethylamide i.v.; and 50 microgram of D-Trp6-des Gly10-GnRH-ethylamide given pernasally (p.n…

AdultMaleendocrine systemmedicine.medical_specialtyTime Factorsmedicine.drug_classMicrogramStimulationGonadotropin-releasing hormoneGonadotropin-Releasing HormoneInternal medicinemedicineHumansInfusions ParenteralTestosteroneAdministration IntranasalChemistryObstetrics and GynecologyLuteinizing HormoneHormonesGonadotropin secretionEndocrinologyReproductive MedicineInjections IntravenousNasal administrationFollicle Stimulating HormoneGonadotropinLuteinizing hormonePituitary Hormone-Releasing Hormoneshormones hormone substitutes and hormone antagonistsHormoneFertility and Sterility
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Subclinical hyperthyroidism due to a thyrotropin receptor (TSHR) gene mutation (S505R).

2006

Aim: To identify the molecular defect by which non-autoimmune subclinical hyperthyroidism was caused in a 6-mo-old infant who presented with weight loss. Methods: Congenital non-autoimmune hyperthyroidism is caused by activating germline mutations in the thyrotropin receptor (TSHR) gene. Therefore, the TSHR gene was sequenced directly from the patient's genomic DNA. Results: Molecular analysis revealed a heterozygous point mutation (S505R) in the TSHR gene as the underlying defect. Conclusion: A constitutively activating mutation in the TSHR gene has to be considered not only in patients with severe congenital non-autoimmune hyperthyroidism, but also in children with subclinical non-autoimm…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesAsymptomaticHyperthyroidismThyrotropin receptorTSHR Gene MutationGermline mutationInternal medicineMedicineMissense mutationHumansPoint MutationGeneSubclinical infectionbusiness.industryPoint mutationInfantReceptors ThyrotropinGeneral Medicineeye diseasesPedigreeEndocrinologyPediatrics Perinatology and Child HealthCancer researchFemalemedicine.symptombusinesshormones hormone substitutes and hormone antagonistsActa paediatrica (Oslo, Norway : 1992)
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Prospective study on thyroid autoimmunity and dysfunction related to chronic hepatitis C and interferon therapy.

1997

This study was designed to assess patients with chronic hepatitis C (CHC) for the presence of thyroid autoimmunity and dysfunction, to evaluate the risk of thyroid disorders associated with interferon (IFN) therapy, and to survey the outcome of possible treatment-related thyroid injury. Out of 104 consecutive untreated patients (30 women and 74 men; mean age, 52.7 years), 8 (7.7%) were found seropositive for thyroid autoantibodies (ThyAb), whereas seropositivity in healthy controls was 1/98 (1.3%). The relative increase in risk of developing thyroid autoimmunity associated with CHC was 760% (95% Cl, 220–1300%). No patients had abnormalities of thyroid function tests, but on IFN treatment, 3…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismThyroid GlandThyrotropinInterferon alpha-2Thyroid function testsGastroenterologyIodide PeroxidaseThyroiditisAutoimmune DiseasesAutoimmune thyroiditisEndocrinologyInternal medicinemedicineHumansProspective StudiesAutoantibodiesAutoimmune diseasemedicine.diagnostic_testbusiness.industryThyroidInterferon-alphaMiddle Agedmedicine.diseaseHepatitis CThyroid DiseasesAnti-thyroid autoantibodiesRecombinant ProteinsDiscontinuationThyroxinemedicine.anatomical_structureImmunologyTriiodothyronineFemaleThyroid functionbusiness
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Graves' Autoantibodies Exhibit Different Stimulating Activities in Cultures of Thyrocytes and Orbital Fibroblasts Not Reflected by Clinical Assays

2021

Background: The pathogenesis of Graves' hyperthyroidism (GH) and associated Graves' orbitopathy (GO) appears to involve stimulatory autoantibodies (thyrotropin receptor [TSHR]-stimulating antibodies [TSAbs]) that bind to and activate TSHRs on thyrocytes and orbital fibroblasts. In general, measurement of circulating TSHR antibodies by clinical assays correlates with the status of GH and GO. However, most clinical measurements of TSHR antibodies use competitive binding assays that do not distinguish between TSAbs and antibodies that bind to but do not activate TSHRs. Moreover, clinical assays for TSAbs measure stimulation of only one signaling pathway, the cyclic adenosine monophosphate (cAM…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGraves' diseaseThyrotropinStimulationEndocrinologyimmune system diseasesInternal medicinemedicineHumansSecretionImmunology Autoimmunity and Graves' OphthalmopathyAutoantibodiesbiologyKinaseChemistryFibroblastsMiddle Agedmedicine.diseaseGraves Diseaseeye diseasesIn vitroGraves OphthalmopathyEndocrinologyThyroid Epithelial Cellsbiology.proteinFemaleThyroglobulinSignal transductionAntibodyThyroid
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Analytical performance and clinical utility of a bioassay for thyroid-stimulating immunoglobulins.

2013

Abstract The analytical performance and the clinical utility of a thyrotropin receptor (TSHR)–stimulating immunoglobulin (TSI) bioassay were compared with those of a TSHR-binding inhibitory immunoglobulin (TBII) assay. Limits of detection (LoD) and quantitation (LoQ), assay cutoff, and the half-maximal effective concentration (EC50) were measured. Dilution analysis was performed in sera of hyperthyroid patients with Graves disease (GD) during antithyroid treatment (ATD). Titer was defined as the first dilution step at which measurement of TSI or TBII fell below the assay cutoff. The LoD, LoQ, cutoff, and EC50 of the bioassay were 251-, 298-, 814-, and 827-fold lower than for the TBII assay.…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesSerial dilutionGraves' diseaseCHO CellsThyrotropin receptorYoung AdultCricetulusAntithyroid AgentsLimit of DetectionInternal medicineCricetinaemedicineBioassayAnimalsHumansProspective StudiesAgedDetection limitbiologyDose-Response Relationship DrugChemistryReproducibility of ResultsGeneral MedicineMiddle Agedmedicine.diseaseeye diseasesGraves DiseaseTiterEndocrinologybiology.proteinThyroid Stimulating ImmunoglobulinBiological AssayFemaleAntibodyBiomarkersImmunoglobulins Thyroid-StimulatingAmerican journal of clinical pathology
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