Search results for "OXIDATION"

showing 10 items of 1913 documents

Fibroblast Growth Factor 21 Limits Lipotoxicity by Promoting Hepatic Fatty Acid Activation in Mice on Methionine and Choline-Deficient Diets

2014

Background & Aims Nonalcoholic fatty liver disease is a common consequence of human and rodent obesity. Disruptions in lipid metabolism lead to accumulation of triglycerides and fatty acids, which can promote inflammation and fibrosis and lead to nonalcoholic steatohepatitis. Circulating levels of fibroblast growth factor (FGF)21 increase in patients with nonalcoholic fatty liver disease or nonalcoholic steatohepatitis; therefore, we assessed the role of FGF21 in the progression of murine fatty liver disease, independent of obesity, caused by methionine and choline deficiency. Methods C57BL/6 wild-type and FGF21-knockout (FGF21-KO) mice were placed on methionine- and choline-deficient (MCD)…

Liver Cirrhosismedicine.medical_specialtyTime FactorsBiologyInfusions SubcutaneousSeverity of Illness IndexArticleHepatitischemistry.chemical_compoundAcyl-CoAMethionineNon-alcoholic Fatty Liver DiseaseInternal medicineNonalcoholic fatty liver diseasemedicineAnimalsRNA MessengerMice Knockoutchemistry.chemical_classificationHepatologyFatty acid metabolismFatty AcidsFatty liverGastroenterologyFatty acidmedicine.diseaseRecombinant ProteinsCholine DeficiencyFibroblast Growth FactorsMice Inbred C57BLDisease Models AnimalEndocrinologyLiverchemistryLipotoxicityDisease ProgressionLipid PeroxidationInflammation MediatorsSteatosisLong chain fatty acidOxidation-ReductionGastroenterology
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Allopurinol prevents cardiac and skeletal muscle damage in professional soccer players

2014

Xanthine oxidase (XO), a free radical-generating enzyme, is involved in tissue damage produced during exhaustive exercise. Our aim was to test whether allopurinol, a powerful inhibitor of XO, may be effective in preventing exercise-induced tissue damage in soccer players. Twelve soccer players were randomized into two experimental groups. One received allopurinol, before a match of the premier Spanish Football League, and the other placebo. Allopurinol prevented the exercise-induced increase in all the markers of skeletal muscle damage analyzed: creatine kinase, lactate dehydrogenase, aspartate aminotransferase, and myoglobin. Creatine kinase-MB isoenzyme and highly sensitive troponin T, sp…

Liver injurymedicine.medical_specialtybiologybusiness.industryAllopurinolPhysical Therapy Sports Therapy and Rehabilitationmedicine.diseaseCreatineGamma-glutamyltransferase activitySurgeryLipid peroxidationchemistry.chemical_compoundEndocrinologychemistryLactate dehydrogenaseInternal medicinebiology.proteinMedicineOrthopedics and Sports MedicineCreatine kinasebusinessXanthine oxidasehuman activitiesmedicine.drugScandinavian Journal of Medicine & Science in Sports
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The oxidation state of iron in silicic melt at 500 MPa water pressure

2002

Abstract The dependence of the ferric–ferrous ratio in silicate melts on oxygen fugacity was studied in the system SiO2(Qz)–NaAlSi3O8(Ab)–CaAl2Si2O8(An)–H2O using Mossbauer spectroscopy. Experiments were performed under water-saturated conditions at 500 MPa, and at temperatures of 850 and 950 °C, covering a range typical for magmatic processes. The oxygen fugacity was varied in the fO2 range from Cu–Cu2O buffer to slightly more reducing conditions than the wustite–magnetite buffer. The iron redox ratio was determined by analyzing the Mossbauer parameter distribution that was modeled based on experimental spectra collected at room temperature on the quenched samples. The obtained iron redox …

Logarithmic scaleMineralChemistryInorganic chemistryAnalytical chemistryGeologyAtmospheric temperature rangeSpectral lineSilicatechemistry.chemical_compoundGeochemistry and PetrologyMineral redox bufferOxidation stateMössbauer spectroscopyInstitut für Geowissenschaften
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Bioactivity Performance of Pure Mg after Plasma Electrolytic Oxidation in Silicate-Based Solutions

2021

The biodegradable metals, including magnesium (Mg), are a convenient alternative to permanent metals but fast uncontrolled corrosion limited wide clinical application. Formation of a barrier coating on Mg alloys could be a successful strategy for the production of a stable external layer that prevents fast corrosion. Our research was aimed to develop an Mg stable oxide coating using plasma electrolytic oxidation (PEO) in silicate-based solutions. 99.9% pure Mg alloy was anodized in electrolytes contained mixtures of sodium silicate and sodium fluoride, calcium hydroxide and sodium hydroxide. Scanning electron microscopy (SEM), energy-dispersive X-ray spectroscopy (EDX), contact angle (CA), …

LuminescencePlasma GasesPharmaceutical ScienceSodium silicate02 engineering and technologymagnesium01 natural sciencesdegradation rateAnalytical Chemistrychemistry.chemical_compoundCoated Materials BiocompatibleCoatingDrug DiscoveryMagnesiumPhosphorusPlasma electrolytic oxidation021001 nanoscience & nanotechnologyAnti-Bacterial AgentsBody FluidsSolutionsChemistry (miscellaneous)Sodium hydroxideMolecular Medicine0210 nano-technologyOxidation-ReductionStaphylococcus aureusMaterials scienceplasma electrolytic oxidationCell SurvivalOxidechemistry.chemical_elementMicrobial Sensitivity Testsengineering.material010402 general chemistryElectrolysisArticleCorrosionlcsh:QD241-441biocompatibilitylcsh:Organic chemistryCell Line TumorHumansPhysical and Theoretical Chemistryantibacterial propertiesElectrodesAnodizingSilicatesOrganic ChemistrySpectrometry X-Ray Emission0104 chemical sciencessilicate bathchemistryengineeringCalciumNuclear chemistryMolecules
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Transcription factor NRF2 regulates miR-1 and miR-206 to drive tumorigenesis

2013

The mechanisms by which deregulated nuclear factor erythroid-2–related factor 2 (NRF2) and kelch-like ECH-associated protein 1 (KEAP1) signaling promote cellular proliferation and tumorigenesis are poorly understood. Using an integrated genomics and 13C-based targeted tracer fate association (TTFA) study, we found that NRF2 regulates miR-1 and miR-206 to direct carbon flux toward the pentose phosphate pathway (PPP) and the tricarboxylic acid (TCA) cycle, reprogramming glucose metabolism. Sustained activation of NRF2 signaling in cancer cells attenuated miR-1 and miR-206 expression, leading to enhanced expression of PPP genes. Conversely, overexpression of miR-1 and miR-206 decreased the exp…

Lung NeoplasmsCell SurvivalNF-E2-Related Factor 2Citric Acid CycleMice NudeBiologymedicine.disease_causeMiceRNA interferenceCarcinoma Non-Small-Cell LungCell Line TumormicroRNAGene expressionmedicineAnimalsHumansTranscription factor3' Untranslated RegionsCell ProliferationOligonucleotide Array Sequence AnalysisRegulation of gene expressionBinding SitesBase SequenceGeneral MedicineMolecular biologyHDAC4Cell biologyTumor BurdenGene Expression Regulation NeoplasticMicroRNAsCell Transformation NeoplasticGlucoseRNA InterferenceHistone deacetylaseCarcinogenesisTranscriptomeOxidation-ReductionNeoplasm TransplantationResearch Article
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Oestradiol or genistein rescues neurons from amyloid beta-induced cell death by inhibiting activation of p38.

2007

Oestrogenic compounds have been postulated as neuroprotective agents. This prompted us to investigate their mechanism action in neurons in primary culture. Cells were pretreated with physiological concentrations of 17-beta estradiol (0.2 nm) or with nutritionally relevant concentrations of genistein (0.5 microm), and 48 h later treated with 5 microm of amyloid beta (Abeta) for 24 h. We found that Abeta increased oxidative stress, measured as peroxide levels or oxidized glutathione/reduced glutathione ratio, which in turn, caused phosphorylation of p38 MAP kinase. Amyloid beta subsequently induced neuronal death. Inhibiting the MAP kinase pathway prevented cell death, confirming the role of …

MAPK/ERK pathwayAgingProgrammed cell deathmedicine.medical_specialtyAmyloid betaCell Survivalp38 mitogen-activated protein kinasesGenisteinPhytoestrogensIn Vitro Techniquesmedicine.disease_causeNeuroprotectionp38 Mitogen-Activated Protein Kinaseschemistry.chemical_compoundInternal medicinemedicineAnimalsCells CulturedCerebral CortexNeuronsAmyloid beta-PeptidesbiologyCell DeathEstradiolEstrogensCell BiologyGlutathioneGenisteinMitochondriaRatsOxidative StressEndocrinologychemistrybiology.proteinOxidation-ReductionOxidative stressAging cell
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F-2-isoprostanes: review of analytical methods

2006

International audience; F2-isoprostanes (F2-isoPs) represent a new family of biomarkers for oxidative stress generated by free radical attack of membrane-bounded arachidonic acid. Esterified F2-isoPs can be found in tissue or plasma lipids whereas the free form F2-isoPs, hydrolyzed by phospholipase, is mainly present in body fluids. The extent of systematic damage due to oxidative stress within the body can be assessed by the determination of plasma or urine F2-isoPs. The determination of F2-isoPs in clinical practice is not often used due to the complexity to extract the compounds from their biologic matrixes before the analysis step. In most of published protocols, extraction procedure is…

MASS SPECTROMETRYIsoprostaneBiophysicsPharmaceutical ScienceMass spectrometry01 natural sciencesBiochemistryIMMUNOASSAYchemistry.chemical_compound[ CHIM.ORGA ] Chemical Sciences/Organic chemistryPlasma lipidsQUANTITATIVE ANALYSISNEUROPROSTANESample preparationComputingMilieux_MISCELLANEOUSChromatography010405 organic chemistryChemistry[CHIM.ORGA]Chemical Sciences/Organic chemistryL IPID PEROXIDATION010401 analytical chemistryExtraction (chemistry)[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences[CHIM.ORGA] Chemical Sciences/Organic chemistry3. Good health0104 chemical sciencesClinical Practice[SDV.SP] Life Sciences [q-bio]/Pharmaceutical sciencesF2-IsoprostanesMolecular MedicineFree formISOPROSTANE
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Anticancer Activity, Reduction Mechanism and G-Quadruplex DNA Binding of a Redox-Activated Platinum(IV)–Salphen Complex

2022

Aiming at reducing the unselective cytotoxicity of Pt(II) chemotherapeutics, a great deal of effort has been concentrated into the design of metal-containing drugs with different anticancer mechanisms of action. Inert Pt(IV) prodrugs have been proposed to be a valid alternative as they are activated by reduction directly into the cell releasing active Pt(II) species. On the other hand, a promising strategy for designing metallodrugs is to explore new potential biological targets rather than canonical B-DNA. G-quadruplex nucleic acid, obtained by self-assembly of guanine-rich nucleic acid sequences, has recently been considered an attractive target for anticancer drug design. Therefore, comp…

MDPt(IV) complexesOrganic ChemistryAntineoplastic AgentsDNAGeneral MedicineDFTPt(IV) complexes; G-quadruplex; DFT; MDCatalysisComputer Science ApplicationsG‐quadruplexG-QuadruplexesInorganic ChemistrySettore CHIM/03 - Chimica Generale E InorganicaProdrugsPhysical and Theoretical ChemistryOxidation-ReductionMolecular BiologySpectroscopyPlatinumInternational Journal of Molecular Sciences
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Mitochondrial and redox dysfunction in post-menopause as risk factor of neurodegenerative disease: a pilot study testing the role of a validated Japa…

2020

During the menopause women may experience increased oxidative stress and decreased antioxidant capacity and, together with the decline of neurosteroids, this represents a risk factor for Alzheimer's disease. The aim of the present study was to test a functional food (FPP-ORI, Osato Research Institute, Gifu, Japan) on redox and mitochondrial efficiency in post-menopausal women. The study population consisting of 69 untreated post-menopausal women were given supplements as follows: Group A was given a multivitamin (MV) 1c 2 times a day, and group B was given FPP 4.5 g 2 times a day. Group C consisted of 23 fertile premenopausal women as the control group. The tests carried out on entry, and a…

MDAmenopausePilot ProjectsAntioxidantsElectron Transport Complex IVFPP-ORIJapanFunctional FoodRisk FactorsMalondialdehydeBDNF; COX activity; FPP-ORI; GPx; MDA; SOD1; menopause; mitochondria; redox dysfunctionHumansGPxBrain-Derived Neurotrophic FactorNeurodegenerative DiseasesSOD1PostmenopauseCOX activitymitochondriaOxidative StressBDNFLeukocytes Mononuclearredox dysfunctioncFemaleCOX activity.Oxidation-Reductionredox dysfunction
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Di- and Tetrairon(III) μ-Oxido Complexes of an N3S-Donor Ligand: Catalyst Precursors for Alkene Oxidations

2019

The new di- and tetranuclear Fe(III) μ-oxido complexes [Fe 4 (μ-O) 4 (PTEBIA) 4 ](CF 3 SO 3 ) 4 (CH 3 CN) 2 ] (1a), [Fe 2 (μ-O)Cl 2 (PTEBIA) 2 ](CF 3 SO 3 ) 2 (1b), and [Fe 2 (μ-O)(HCOO) 2 (PTEBIA) 2 ](ClO 4 ) 2 (MeOH) (2) were prepared from the sulfur-containing ligand (2-((2,4-dimethylphenyl)thio)-N,N-bis ((1-methyl-benzimidazol-2-yl)methyl)ethanamine (PTEBIA). The tetrairon complex 1a features four μ-oxido bridges, while in dinuclear 1b, the sulfur moiety of the ligand occupies one of the six coordination sites of each Fe(III) ion with a long Fe-S distance of 2.814(6) A. In 2, two Fe(III) centers are bridged by one oxido and two formate units, the latter likely formed by methanol oxidati…

MECHANISMFe-S interactionoxidation116 Chemical sciencesThio-rautaSULFURHomogeneous catalysis02 engineering and technology010402 general chemistry01 natural sciencesMedicinal chemistrythioetherCatalysislcsh:Chemistrychemistry.chemical_compoundThioetheriron-oxo complexAcetonitrileta116Fe-S interaction; homogeneous catalysis; iron-oxo complex; oxidation; thioetherOriginal Researchchemistry.chemical_classificationeetteritFUNCTIONAL-MODELCOORDINATIONPEROXIDEAlkeneLigandACTIVE-SITEhapettuminenGeneral Chemistrykompleksiyhdisteet021001 nanoscience & nanotechnology540COPPER-COMPLEXEShomogeneous catalysis0104 chemical sciencesChemistrychemistrylcsh:QD1-999katalyysiACIDOXO0210 nano-technologySelectivityNONHEME IRON CATALYSTSFrontiers in Chemistry
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