Search results for "Oligodendroglioma"

showing 9 items of 19 documents

Image Analysis of Proliferating Cells in Tumors of the Human Nervous System

1994

Obtaining growth fractions from immunohistological preparations by the commonly used cell count calculation method is time consuming. For the first time, we investigated and compared the detection of proliferating cells in immunohistologically labeled tissue from tumors of the nervous system using the monoclonal antibody Ki-67 by a new computerized image analysis system and by cell count calculation. The two methods showed a high correlation (correlation index, 0.98) in 37 gliomas (2 pilocytic astrocytomas, 10 Grade II astrocytomas, 5 Grade III astrocytomas, 20 Grade IV astrocytomas and glioblastoma multiforme) and a heterogenous group of 10 additional tumors of the nervous system, includin…

PineoblastomaPathologymedicine.medical_specialtybiologybusiness.industryPrimary central nervous system lymphomaAstrocytomamedicine.diseaseKi-67GliomaImmunologymedicinebiology.proteinNeurofibromaSurgeryNeurology (clinical)OligodendrogliomabusinessAnaplastic astrocytomaNeurosurgery
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Melanoma cells release extracellular vesicle which contain H1° linker histone as well as RNA-binding proteins which bind to the H1° mRNA

2015

We previously demonstrated that G26/24 oligodendroglioma cells release EVs that contain proteins, such as FasL and TRAIL, which induce apoptosis in rat cortical neurons [1] and astrocytes [2]. We also reported that cancer cells use EVs for transferring, into the environment [3], proteins such as extracellular matrix remodelling proteases [4], and H1°, a differentiation-specific histone [5]. In particular, by releasing H1°, cells could escape differentiation cues [5]. To verify the role of EVs in releasing specific proteins and mRNAs, in this study we used as a model A375 melanoma cells. METHODS EVs were purified from cell culture media as previously reported [1, 2]. T1 RNase-protection assa…

Settore BIO/10 - BiochimicaOligodendroglioma cells extracellular vesicles (EVS) histone H1.0 RNA-binding proteins (RBPs) myelin expression factor-2 (MYEF2)Settore BIO/06 - Anatomia Comparata E Citologia
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Cancer cells can affect behaviour of neighbouring cells by transferring molecules through extracellular vesicles

2017

Most cells release into the extracellular space membrane-bound structures of different sizes, origin and composition, collectively called extracellular vesicles (EVs) [1]. Tumor cells are much more active than normal cells in producing EVs. Because of this property, they are able to transfer both nucleic acids and proteins to the surrounding normal cells, thus inducing in these latter at least some transformed behavior. We previously showed that EVs produced by G26/24 oligodendroglioma cells can horizontally transfer to their neighbours radioactive proteins [2]. In addition, EVs released by these cells contain pro-apoptotic proteins, such as TRAIL and Fas-Ligand, able to induce apoptosis in…

Settore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E CitologiaExtracellular vesicles (EVs) G26/24 oligodendroglioma cells rat cortical neurons astrocytes H1.0 histone protein H1.0 mRNA myelin expression factor-2 (MYEF2)
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Melanoma cells release extracellular vesicles which contain RNA-binding proteins able to bind the mRNA encoding histone H1°

2015

Extracellular vesicles (EVs) are produced by most prokaryotic and eukaryotic cells; tumour cells, however, release much higher amounts of EVs, which contain cancer-specific proteins and RNAs. Molecules carried by EVs are captured by surrounding cells, which then undergo profound phenotypic modifications. G26/24 oligodendroglioma cells release, for example, EVs containing FasL and TRAIL, which induce apoptosis in rat cortical neurons and astrocytes in culture. By metabolic labelling of cells, EV-mediated horizontal transfer of radioactive proteins was clearly demonstrated. Among the proteins present in EVs produced by oligodendroglioma cells, extracellular matrix remodelling proteases, and t…

Settore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E CitologiaExtracellular vesicles (EVs) G26/24 oligodendroglioma cells xtracellular matrix remodelling proteases A375 melanoma cells H1° histone RNA-protein complexes myelin expression factor-2 (MYEF2)
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RNA as a carrier of epigenetic information

2017

Both prokaryotic and eukaryotic cells release into the extracellular matrix membrane-bound structures of different sizes, origin and composition, collectively called extracellular vesicles (EVs) [1]. Tumor cells, in particular, use EVs to transfer both nucleic acids and proteins to the surrounding normal cells, thus inducing in them transformed behaviours or killing them. G26/24 oligodendroglioma cells, for example, transfer by EVs pro-apoptotic proteins, such as TRAIL and Fas-Ligand [2], extracellular matrix remodelling proteases (such as ADAMTS) [3], and even the H1.0 histone protein [4]. Another tumour cell line, with a different tissue origin (A375 melanoma cells) releases into the medi…

Settore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologiaextracellular vesicles (EVs) G26/24 oligodendroglioma cells extracellular matrix remodelling proteases H1.0 histone protein H1.0 mRNA A375 melanoma cells myelin expression factor-2 (MYEF2)
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Extracellular membrane vesicles can mediate intercellular transfer of molecules

2012

Many eukaryotic cell types, including neural cells, release into the extracellular environment vesicles of different sizes and composition. Neurons and astrocytes shed extracellular vesicles which contain FGF2 and VEGF and could be involved in interaction with endothelial cells, to form the blood-brain barrier. Also brain tumor cells, such as glioblastomas, release vesicles in the extracellular space. Microvesicles (MVs) shed from G26/24 oligodendro¬glioma cells were previously reported to contain FAS-L and to cause a reproducible, dose-dependent, inhibitory effect on neurite outgrowth, and neuronal apoptosis, when added to primary cultures of rat cortical neurons. More recently, they were …

apoptosis astrocyteRBPsSettore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologiaoligodendrogliomashedding vesicle
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Heterogeneous Pattern of Differentiation With BCAS1/NABC1 Expression in a Case of Oligodendroglioma

2020

business.industryMEDLINEGeneral MedicineComputational biologyBiologymedicine.diseasePathology and Forensic MedicineCellular and Molecular NeuroscienceText miningNeurologyExpression (architecture)medicineNeurology (clinical)OligodendrogliomabusinessJournal of Neuropathology & Experimental Neurology
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Extracellulare membrane vesicles as vehicles for cell-to-cell and cell-to-extracellular matrix functional interactions

2012

extracellular matrixSettore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologiaoligodendrogliomashedding vesicle
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Proapoptotic effects of oligodendroglioma cells on primary brain cells in culture.

2009

vesiclesoligodendroglioma cellbrain cellapoptosi
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