Search results for "Oxidants"

showing 10 items of 878 documents

Effect of a two-year supplementation with low doses of antioxidant vitamins and/or minerals in elderly subjects on levels of nutrients and antioxidan…

1997

Eighty-one elderly hospitalized subjects (65 years) were recruited for a double-blind placebo-controlled study to examine low dose supplementation of antioxidant vitamins and minerals on biological and functional parameters of free radical metabolism. Subjects were randomly assigned to one of the four treatment groups, daily receiving for 2 years: placebo group; mineral group: 20 mg zinc, 100 micrograms selenium; vitamin group: 120 mg vitamin C (Vit C), 6 mg beta-carotene (beta CA), 15 mg vitamin E (Vit E); mineral and vitamin group: Zn 20 mg, Se 100 micrograms, Vit C 120 mg, beta CA 6 mg, Vit E 15 mg.Fifty-seven subjects completed the study. A large frequency of Vit C, Zn and Se deficienci…

MaleVitaminmedicine.medical_specialtyErythrocytesAntioxidantmedicine.medical_treatmentNutritional StatusMedicine (miscellaneous)chemistry.chemical_elementAscorbic AcidThiobarbituric Acid Reactive SubstancesAntioxidantsSeleniumchemistry.chemical_compoundOral administrationInternal medicinemedicineHumansVitamin ENutritional Physiological PhenomenaAgedAged 80 and overGlutathione PeroxidaseMineralsNutrition and DieteticsVitamin CSuperoxide DismutaseVitamin EVitaminsMetabolismbeta CaroteneAscorbic acidGlutathioneZincEndocrinologychemistryBiochemistryFemaleSeleniumJournal of the American College of Nutrition
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Vitamin A Deficiency Increases Protein Catabolism and Induces Urea Cycle Enzymes in Rats

2010

Chronic vitamin A deficiency induces a substantial delay in the rates of weight and height gain in both humans and experimental animals. This effect has been associated with an impaired nutrient metabolism and loss of body protein. Therefore, we analyzed the effect of vitamin A deficiency on endogenous proteolysis and nitrogen metabolism and its reversibility with all-trans retinoic acid (RA). Male weanling rats, housed in pairs, were pair-fed a vitamin A-deficient (VAD) or control diet until they were 60 d old. A group of deficient rats were further treated with daily intraperitoneal injections of all-trans RA for 10 d. Final body and tissue (i.e. liver and heart) weights were significantl…

MaleVitaminmedicine.medical_specialtyNitrogenMedicine (miscellaneous)TretinoinBiologyAntioxidantsRetinoidschemistry.chemical_compoundInternal medicinemedicineAnimalsUreaMuscle SkeletalTriglyceridesNutrition and DieteticsVitamin A DeficiencyCatabolismRetinolProtein turnoverMethylhistidinesmedicine.diseaseRatsVitamin A deficiencyProtein catabolismEndocrinologyLiverchemistryEnzyme InductionUrea cycleLipid PeroxidationEnergy sourceThe Journal of Nutrition
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Effects of β-Carotene, Retinal, Riboflavin, α-Tocopherol and Vitamins C and K1 on Sister-chromatid Exchanges Induced by 3-Amino-1-methyl-5H-pyrido[4,…

1998

The vitamins and related compounds cited in the title were investigated for their abilities to modulate sister-chromatid exchanges (SCEs) induced by Trp-P-2 or cyclophosphamide (CP) in human peripheral lymphocyte cultures in the presence of an exogenous metabolizing system from rat liver. When inducer and test substances were given simultaneously, beta-carotene, retinal and alpha-tocopherol caused a dose-dependent decrease of SCE frequencies induced by Trp-P-2 and CP. Vitamin K1, however, brought about an identical effect with Trp-P-2 only, while with CP an initial decrease of SCEs was followed by a statistically significant re-increase at higher concentrations. Vitamin C was ineffective ag…

MaleVitaminmedicine.medical_specialtyRiboflavinT-Lymphocytesmedicine.medical_treatmentRiboflavinAscorbic AcidToxicologyAntioxidantsRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsHumansVitamin ETocopherolCyclophosphamideCells Cultured030304 developmental biology0303 health sciencesVitamin CChemistryVitamin ERetinolVitamin K 1VitaminsGeneral Medicinebeta CaroteneAscorbic acidRats3. Good healthEndocrinologyBiochemistry030220 oncology & carcinogenesisRetinaldehydealpha-TocopherolSister Chromatid ExchangeCarbolinesMutagensFood ScienceFood and Chemical Toxicology
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Inhibition of Xanthine Oxidase by Phenolic Conjugates of Methylated Quinic Acid

2003

The caffeoyl conjugates of prenylhydroquinone glucoside and of quinic acid, either in the carboxyl-free or carboxymethyl forms, isolated from Phagnalon rupestre (Asteraceae), showed inhibitory activity on lipid peroxidation induced by Fe 2+/ascorbate and by CCl4/NADPH in rat liver microsomes, with IC50 values ranging from 3 to 11 microM. After having demonstrated their effect on the xanthine oxidase-regulated superoxide production, the active compounds were tested for the direct inhibition of this enzyme. Methylated dicaffeoylquinic conjugates competitively inhibited the enzyme and the highest potency was obtained for the 4,5-diester, with an IC50 value of 3.6 microM, nearly ten times lower…

MaleXanthine OxidaseAntioxidantStereochemistrymedicine.medical_treatmentQuinic AcidPharmaceutical ScienceAsteraceaeAntioxidantsAnalytical ChemistryLipid peroxidationInhibitory Concentration 50chemistry.chemical_compoundPhenolsGlucosideDrug DiscoverymedicineAnimalsEnzyme InhibitorsRats WistarXanthine oxidasePharmacologybiologyPlant ExtractsSuperoxideOrganic ChemistryQuinic acidXanthineHydroquinonesRatsComplementary and alternative medicinechemistryBiochemistryEnzyme inhibitorMicrosomes Liverbiology.proteinMolecular MedicineLipid PeroxidationPhytotherapyPlanta Medica
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Antioxidant activity of anti-inflammatory plant extracts

2002

The antioxidant properties of twenty medical herbs used in the traditional Mediterranean and Chinese medicine were studied. Extracts from Forsythia suspensa, Helichrysum italicum, Scrophularia auriculata, Inula viscosa, Coptis chinensis, Poria cocos and Scutellaria baicalensis had previously shown anti-inflammatory activity in different experimental models. Using free radical-generating systems H. italicum. I. viscosa and F. suspensa protected against enzymatic and non-enzymatic lipid peroxidation in model membranes and also showed scavenging property on the superoxide radical. All extracts were assayed at a concentration of 100 microg/ml. Most of the extracts were weak scavengers of the hy…

MaleXanthine OxidaseErythrocytesAntioxidantmedicine.drug_classmedicine.medical_treatmentHelichrysum italicumAntioxidantsGeneral Biochemistry Genetics and Molecular BiologyAnti-inflammatoryRats Sprague-DawleyLipid peroxidationchemistry.chemical_compoundmedicineAnimalsGeneral Pharmacology Toxicology and PharmaceuticsXanthine oxidaseForsythia suspensaPlants MedicinalbiologyTraditional medicineDeoxyribosePlant ExtractsAnti-Inflammatory Agents Non-SteroidalFree Radical ScavengersGeneral MedicineCoptis chinensisbiology.organism_classificationRatsBiochemistrychemistryMicrosomes LiverScutellaria baicalensisLipid PeroxidationMedicine TraditionalAminopyrine N-DemethylaseLife Sciences
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Complex I dysfunction and tolerance to nitroglycerin: an approach based on mitochondrial-targeted antioxidants.

2006

Nitroglycerin (GTN) tolerance was induced in vivo (rats) and in vitro (rat and human vessels). Electrochemical detection revealed that the incubation dose of GTN (5×10 −6 mol/L) did not release NO or modify O 2 consumption when administered acutely. However, development of tolerance produced a decrease in both mitochondrial O 2 consumption and the K m for O 2 in animal and human vessels and endothelial cells in a noncompetitive action. GTN tolerance has been associated with impairment of GTN biotransformation through inhibition of aldehyde dehydrogenase (ALDH)-2, and with uncoupling of mitochondrial respiration. Feeding rats with mitochondrial-targeted antioxidants (mitoquinone [MQ]) and i…

MaleantioxidantAntioxidantPhysiologyUbiquinonemedicine.medical_treatmentMuscle RelaxationVasodilator AgentsAldehyde dehydrogenasePharmacologyMitochondrionmedicine.disease_causeAntioxidantsMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundNitroglycerinDrug toleranceoxidative stressCyclic GMPchemistry.chemical_classificationbiologyAldehyde Dehydrogenase MitochondrialDrug ToleranceGlutathioneMitochondriamitochondriaBiochemistrycardiovascular systemCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyMuscle ContractionendotheliumIn Vitro TechniquesMitochondrial ProteinsOrganophosphorus CompoundsOxygen ConsumptionRespirationmedicineAnimalsHumansReactive oxygen speciesElectron Transport Complex IDose-Response Relationship DrugEndothelial CellsGlutathioneAldehyde DehydrogenasenitroglycerinRatsOxidative Stresschemistrybiology.proteinReactive Oxygen SpeciesOxidative stressCirculation research
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Is there a role for antioxidant carotenoids in limiting self-harming immune response in invertebrates?

2007

Innate immunity relies on effectors, which produce cytotoxic molecules that have not only the advantage of killing pathogens but also the disadvantage of harming host tissues and organs. Although the role of dietary antioxidants in invertebrate immunity is still unknown, it has been shown in vertebrates that carotenoids scavenge cytotoxic radicals generated during the immune response. Carotenoids may consequently decrease the self-harming cost of immunity. A positive relationship between the levels of innate immune defence and circulating carotenoid might therefore be expected. Consistent with this hypothesis, we show that the maintenance and use of the prophenoloxidase system strongly cor…

MaleantioxidantMESH : Immunity Natural[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyAntioxidantsMESH: Linear ModelsMESH: AmphipodaHemolymphMESH : Linear ModelsHemolymphMESH: AnimalsMESH : FemaleCarotenoidchemistry.chemical_classificationbiologyEffectorMonophenol Monooxygenasefood and beveragesProphenoloxidaseMESH : AmphipodaAgricultural and Biological Sciences (miscellaneous)MESH : Monophenol Monooxygenase[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyMESH : AntioxidantsFemaleGeneral Agricultural and Biological SciencesResearch ArticleMESH: Monophenol MonooxygenaseMESH : Maleimmune costsecological immunologyMESH : Hemolymph[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyImmune systemImmunityAnimalsAmphipodaMESH: Immunity NaturalMESH : CarotenoidsInnate immune systemMESH: HemolymphMESH: Antioxidants[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologybiochemical phenomena metabolism and nutritionbiology.organism_classificationCarotenoidsImmunity InnateMESH: MaleGammarus pulexchemistryImmunologyMESH: CarotenoidsLinear ModelsbacteriaMESH : AnimalsMESH: Female
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Prenatal exposure to residential air pollution and infant mental development: modulation by antioxidants and detoxification factors.

2011

Background: Air pollution effects on children’s neurodevelopment have recently been suggested to occur most likely through the oxidative stress pathway. Objective: We aimed to assess whether prenatal exposure to residential air pollution is associated with impaired infant mental development, and whether antioxidant/detoxification factors modulate this association. Methods: In the Spanish INfancia y Medio Ambiente (INMA; Environment and Childhood) Project, 2,644 pregnant women were recruited during their first trimester. Nitrogen dioxide (NO2) and benzene were measured with passive samplers covering the study areas. Land use regression models were developed for each pollutant to predict aver…

Malecognitionvegetablesnitrogen dioxideHealth Toxicology and Mutagenesisaromatic hydrocarbonsEnvironmental pollutionvitamin DPrenatal Nutritional Physiological PhenomenaInfants -- CreixementBayley Scales of Infant DevelopmentAntioxidantsPregnancyEnvironmental healthVitamin D and neurologyMedicineHumansPrenatal Nutritional Physiological Phenomenachild developmentPregnancyAir Pollutantsbusiness.industryResearchPublic Health Environmental and Occupational HealthInfantfruitintelligencemedicine.diseaseChild developmentInfants -- AlimentacióConfidence intervalMaternal ExposureEnvironmental chemistrybreast-feedingVitamines en nutrició humanaNodrissons -- AlimentacióFemaleenvironmental pollutionbusinessBreast feedingEnvironmental health perspectives
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The Effect of a Mediterranean Diet on the Incidence of Cataract Surgery

2017

Background: Cataract is a leading cause of vision impairment worldwide, and surgery is the only available treatment. The process that initiates lens opacification is dependent on the oxidative stress experienced by the lens components. A healthy overall dietary pattern, with the potential to reduce oxidative stress, has been suggested as a means to decrease the risk of developing cataract. We aimed to investigate the hypothesis that an intervention with a Mediterranean diet (MedDiet) rather than a low-fat diet could decrease the incidence of cataract surgery in elderly subjects. Methods: We included 5802 men and women (age range: 55-80 years) from the Prevencion con Dieta Mediterronea study…

Malecumplimiento del pacienteMediterranean dietmedicine.medical_treatmenthumanosExtra-virgin olive oilestudios de seguimientoDiet MediterraneanDieta mediterràniaCataract surgeryAntioxidantslaw.invention0302 clinical medicineMediterranean diet; PREDIMED; cataract; cataract surgery; nuts; extra-virgin olive oil; low-fat diet; antioxidantsRandomized controlled trialCirurgia de cataracteslawRisk FactorsPrevalenceNutsAge-related cataract030212 general & internal medicineDiet Fat-Restrictedmediana edadAged 80 and overCiències de la salutancianoLow-fat dietNutrition and Dieteticsdietaresultado del tratamientoIncidence (epidemiology)IncidenceHazard ratioVitaminsMetaanalysisMiddle AgedCiencias de la saludcatarataTreatment OutcomeFemaleCiencias de la Salud [Materias Investigacion]lcsh:Nutrition. Foods and food supplyRiskPREDIMEDmedicine.medical_specialtyZeaxanthinlcsh:TX341-641incidenciaArticleCataractAssociation03 medical and health sciencesMediterranean cookingInternal medicineMediterranean dietCuina mediterràniamedicinefactores de riesgoHumansOlive OilAgedCataractes -- CirugiaNuclear cataractProportional hazards modelbusiness.industryPreventionHealth sciencesCataract surgeryConfidence intervalDietSurgerynuecesClinical trialFood Science; Nutrition & Dietetics; Nutrition and Dietetics030221 ophthalmology & optometryCataractesPatient CompliancebusinessFood ScienceFollow-Up Studies
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Metformin increases APP expression and processing via oxidative stress, mitochondrial dysfunction and NF-κB activation: Use of insulin to attenuate m…

2015

AbstractClinical and experimental biomedical studies have shown Type 2 diabetes mellitus (T2DM) to be a risk factor for the development of Alzheimer's disease (AD). This study demonstrates the effect of metformin, a therapeutic biguanide administered for T2DM therapy, on β-amyloid precursor protein (APP) metabolism in in vitro, ex vivo and in vivo models. Furthermore, the protective role of insulin against metformin is also demonstrated. In LAN5 neuroblastoma cells, metformin increases APP and presenilin levels, proteins involved in AD. Overexpression of APP and presenilin 1 (Pres 1) increases APP cleavage and intracellular accumulation of β-amyloid peptide (Aβ), which, in turn, promotes ag…

Maleendocrine system diseasesmedicine.medical_treatmentmedicine.disease_causeAntioxidantsNF-κBAmyloid beta-Protein PrecursorAspartic Acid EndopeptidasesInsulinBiguanideNF-kappa BBrainAlzheimer's diseaseMetforminMetforminMitochondriaProtein TransportAntioxidantmedicine.drugmetformin T2DM Alzheimer's diseaseAdultmedicine.medical_specialtyProgrammed cell deathmedicine.drug_classOxidative phosphorylationBiologyAntidiabetic drugModels BiologicalPresenilinInternal medicineCell Line Tumormental disordersmedicinePresenilin-1AnimalsHumansMolecular BiologyCell NucleusSettore MED/04 - Patologia GeneraleAmyloid beta-PeptidesInsulinAdenylate KinaseOxidative Stress Pathwaynutritional and metabolic diseasesCell BiologyHydrogen PeroxideMice Inbred C57BLEndocrinologyGene Expression RegulationCytoprotectionOxidative stressLeukocytes MononuclearAmyloid Precursor Protein SecretasesOxidative stressBiochimica et Biophysica Acta (BBA) - Molecular Cell Research
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