Search results for "Oxide"

showing 10 items of 6424 documents

Prenatal Exposure to NO2 and Ultrasound Measures of Fetal Growth in the Spanish INMA Cohort

2016

This study was funded by grants from the European Union: NEWGENERIS FP6-2003- Food-3-A-016320, FP7-ENV-2011 cod 282957, HEALTH.2010.2.4.5-1; and by grants from Spain: Instituto de Salud Carlos III (Red INMA G03/176, CB06/02/0031, and FIS-FEDER PI03/1615, PI04/1509, PI04/1112, PI04/1931, PI04/2018, PI04/1436, PI05/1079, PI05/1052, PI06/1213, PI07/0314, PI08/1151, PI09/02647, PI09/02311, PI11/01007, PI11/02591, PI11/02038, PI13/1944, PI13/02429, PI14/0891, PI14/1687, and Miguel Servet CP11/00178 and MS13/00054), Conselleria de Sanitat Generalitat Valenciana, Generalitat de Catalunya (CIRIT 1999SGR 00241), Diputación Foral de Guipúzcoa (DFG/004), Departamento de Sanidad y Consumo Gobierno Vasc…

AdultMalemedicine.medical_specialtyAdolescentHealth Toxicology and MutagenesisNitrogen DioxideEmbaràsMEDLINE010501 environmental sciences01 natural sciencesUltrasonography PrenatalCohort StudiesFetal DevelopmentYoung Adult03 medical and health sciences0302 clinical medicinePregnancyFetal growthHumansMedicine030212 general & internal medicineYoung adult0105 earth and related environmental sciencesAir PollutantsPregnancyAnthropometrybusiness.industryObstetricsAire -- ContaminacióUltrasoundPublic Health Environmental and Occupational HealthAnthropometrymedicine.disease3. Good healthMaternal ExposureSpainChildren's Healthembryonic structuresCohortFemalebusinessCohort study
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Kallikrein–kinin system and fibrinolysis in hereditary angioedema due to factor XII gene mutation Thr309Lys

2009

In a subgroup of hereditary angioedema (HAE) patients with normal C1-esterase inhibitor levels, HAE is caused by a Thr309Lys mutation in the coagulation factor XII (F12) gene. The aim of this study was to examine elements of the kallikrein-kinin system ('contact system') and the downstream-linked coagulation, complement and fibrinolytic systems in the plasma of six patients with HAE caused by the Thr309Lys mutation and healthy probands. Blood samples were taken from participants during the symptom-free interval between attacks. Samples were analyzed for activity and concentrations of components of the kallikrein-kinin system and linked enzyme systems. The mean FXII clotting activity was 90%…

AdultMalemedicine.medical_specialtyAdolescentMutation MissenseKininsCoagulation Factor XIIFactor XIIaGene mutationYoung AdultInternal medicinemedicineHumansPoint MutationHereditary Angioedema Type IIIComplement Pathway ClassicalAgedAged 80 and overFactor XIIAngioedemaChemistryFibrinolysisDextran SulfateAngioedemas HereditaryPrekallikreinPrekallikreinBlood ProteinsHematologyGeneral MedicineMiddle AgedSilicon Dioxidemedicine.diseaseEnzyme ActivationEndocrinologyAmino Acid SubstitutionChromogenic CompoundsCoagulationTissue Plasminogen ActivatorHereditary angioedemaImmunologyFemaleKallikreinsmedicine.symptomcirculatory and respiratory physiologyBlood Coagulation & Fibrinolysis
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Simultaneous assessment of endothelial function, nitric oxide synthase activity, nitric oxide-mediated signaling, and oxidative stress in individuals…

2007

Abstract Background: Endothelial function is impaired in hypercholesterolemia and atherosclerosis. Based on mostly indirect evidence, this impairment is attributed to reduced synthesis or impaired biological activity of endothelium-derived nitric oxide (NO). It was the aim of this study to directly estimate and compare whole-body NO production in normo- and hypercholesterolemia by applying a nonradioactive stable isotope dilution technique in vivo. Methods: We enrolled 12 normocholesterolemic and 24 hypercholesterolemic volunteers who were all clinically healthy. To assess whole-body NO synthesis, we intravenously administered l-[guanidino-(15N2)]-arginine and determined the urinary excreti…

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIClinical BiochemistryHypercholesterolemiaVasodilationEndothelial NOSArginineDinoprostNitric OxideNitric oxideExcretionchemistry.chemical_compoundSex FactorsInternal medicinemedicineHumansCyclic GMPAgedNitratesbiologyBiochemistry (medical)Nitric Oxide Synthase Type IIIMiddle AgedNitric oxide synthaseOxidative StressEndocrinologychemistrybiology.proteinFemaleEndothelium VascularNitric Oxide SynthaseAsymmetric dimethylarginineLipoproteinClinical chemistry
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Association between polymorphisms of endothelial nitric oxide synthase gene (NOS3) and left posterior wall thickness (LPWT) of the heart in Fabry dis…

2008

Fabry disease is an X-chromosomal storage disorder due to loss-of-function mutations of the GLA gene encoding the lysosomal enzyme α-galactosidase A. Accumulating glycosphingolipid deposits disturb the function of various cells, in particular that of myocytes, arterial smooth-muscle cells, and vascular endothelium. Hypertrophic cardiomyopathy, for example measured by left posterior wall thickness (LPWT) of the heart, represents a major component of Fabry disease morbidity in adult patients. Endothelium-derived nitric oxide (eNO), produced by eNO synthase (eNOS), is a key regulator of vessel wall function and cardiovascular homeostasis. We analysed the effect of the polymorphisms c.894G > T …

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIMutation MissenseMinisatellite RepeatsYoung AdultSex FactorsGene FrequencyInternal medicineGermanyGenotypeGeneticsMedicineHumansGenetic Predisposition to DiseaseAlleleChildAllele frequencyGenetics (clinical)AgedUltrasonographyGeneticsAlpha-galactosidasePolymorphism Geneticbiologybusiness.industryMyocardiumHaplotypeHypertrophic cardiomyopathyNitric Oxide Synthase Type IIIExonsCardiomyopathy HypertrophicMiddle Agedmedicine.diseaseFabry diseaseIntronsEndocrinologyPhenotypeHaplotypesCase-Control Studiesalpha-Galactosidasebiology.proteinFabry DiseaseRegression AnalysisFemaleHypertrophy Left VentricularbusinessJournal of inherited metabolic disease
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Effects of sildenafil on human penile blood vessels.

2000

Abstract Objectives. To investigate the effects of sildenafil on human penile blood vessels and evaluate the interaction of sildenafil with neurogenic-mediated responses. Sildenafil is currently used in the treatment of erectile dysfunction. Methods. Penile dorsal arteries and deep dorsal veins were obtained from 14 multiorgan donors. Vascular rings were suspended in organ bath chambers, and the isometric tension was recorded. We then studied the effects of sildenafil on precontracted vessels and the neurogenic (noradrenergic and nitrergic) responses. Results. Sildenafil (10 −9 to 3 × 10 −6 M) caused concentration-dependent relaxation and amplified the relaxation induced by sodium nitroprus…

AdultMalemedicine.medical_specialtyAdolescentSildenafilPhosphodiesterase InhibitorsUrologyMuscle RelaxationPiperazinesSildenafil CitrateNitric oxideVeinschemistry.chemical_compoundInternal medicineMedicineHumansSulfonesGuanethidinebusiness.industrySmooth muscle contractionArteriesMiddle AgedPDE5 drug designrespiratory tract diseasesVasodilationEndocrinologymedicine.anatomical_structurechemistryPurinesVasoconstrictioncardiovascular systemSodium nitroprussidebusinessZaprinastBlood vesselmedicine.drugMuscle ContractionPenisUrology
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Effects of the association of aging and obesity on lipids, lipoproteins and oxidative stress biomarkers: a comparison of older with young men.

2011

In this study, plasma lipids, lipoproteins and markers of oxidant/antioxidant status were investigated in young (n = 45) and older (n = 40) obese men and compared to those in young (n = 65) and older (n = 55) normal weight controls. The purpose was to determine whether obesity exacerbates or not lipid, lipoprotein abnormalities and oxidative stress in older men. Our findings showed that all obese patients had increased plasma triglyceride, cholesterol, LDL-cholesterol, -triglyceride and HDL-triglyceride levels concentrations compared to controls (P < 0.01). However, the younger obese men had relatively larger and accentuated changes in plasma lipids and lipoproteins than the older patients.…

AdultMalemedicine.medical_specialtyAgingLipid PeroxidesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentLipoproteinsMedicine (miscellaneous)medicine.disease_causeAntioxidantsBody Mass IndexProtein Carbonylationchemistry.chemical_compoundInternal medicineMedicineHumansObesityAgedchemistry.chemical_classificationNutrition and DieteticsTriglyceridebusiness.industryCholesterolSuperoxide DismutaseGlutathione peroxidaseMiddle Agedmedicine.diseaseObesityLipidsOxidative StressEndocrinologychemistrylipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessBody mass indexOxidative stressBiomarkersLipoproteinNutrition, metabolism, and cardiovascular diseases : NMCD
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Age-related increase in xanthine oxidase activity in human plasma and rat tissues.

2007

This study assessed the role of xanthine oxidase in vascular ageing. A positive correlation between xanthine oxidase activity and age was found in human plasma. Similar results were found in rat plasma. Xanthine oxidase expression and activity in homogenates from the aortic wall were significantly higher in samples from old rats than in their young counterparts (p<0.01). In rat skeletal muscle homogenates both xanthine oxidase expression and activity showed a similar age-related profile. Superoxide production by xanthine oxidase in aortic rings was higher in aged rats. Uric acid, the final product of xanthine oxidase has been proposed as a risk factor for coronary heart disease and an indep…

AdultMalemedicine.medical_specialtyAgingXanthine Oxidasemedicine.disease_causeBiochemistrychemistry.chemical_compoundSuperoxidesInternal medicinemedicineAnimalsHumansProspective StudiesRats WistarXanthine oxidaseMuscle SkeletalAortaAgedSuperoxideSkeletal muscleGeneral MedicineGlutathioneMiddle Agedmedicine.diseaseRatsEndocrinologymedicine.anatomical_structurechemistryAgeingHeart failureUric acidFemaleOxidative stressFree radical research
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Antenatal Steroids and Antioxidant Enzyme Activity in Preterm Infants: Influence of Gender and Timing

2009

Antenatal steroids have improved the survival of preterm infants; however, the mechanism of action is not fully understood. We aimed to establish an association between antenatal steroids and antioxidant activity and postnatal oxidative stress. In a prospective cohort study, extremely preterm neonates receiving antenatal steroids (CORT) or not (NOCORT) were enrolled. An association between antenatal steroids and activities of antioxidant enzymes and glutathione cycle enzymes in cord blood was found. In addition, reduced oxidative stress (GSH/GSSG ratio, CORT vs. NOCORT, 35.68 + or - 12.20 vs. 28.38 + or - 9.92; p < 0.01) and, decreased oxidation of proteins (ortho-tyrosine/phenylalanine rat…

AdultMalemedicine.medical_specialtyAntioxidantAdolescentPhysiologymedicine.medical_treatmentClinical BiochemistryBiologymedicine.disease_causeBiochemistryAntioxidantsYoung Adultchemistry.chemical_compoundSex FactorsAdrenal Cortex HormonesPregnancyInternal medicinemedicineHumansProspective StudiesProspective cohort studyMolecular BiologyGeneral Environmental SciencePregnancySuperoxide DismutaseInfant NewbornRetinopathy of prematurityCell BiologyGlutathioneCatalasemedicine.diseaseOxidative StressEndocrinologyBronchopulmonary dysplasiachemistryCord bloodGeneral Earth and Planetary SciencesFemaleInfant PrematureOxidative stressAntioxidants &amp; Redox Signaling
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Nitric oxide metabolites and oxidative stress in mild essential hypertension

2010

Many papers have showed non univocal data about oxidative stress status and nitric oxide metabolites in essential hypertension. Considering this preamble we examined the total antioxidant status (TAS), the lipid peroxidation (LP), expressed as thiobarbituric acid-reactive substances (TBARS), the stable end products of nitric oxide (NOx) and LP/NOx ratio in 25 subjects with untreated mild essential hypertension. The obtained data show a significant increase in TBARS (p < 0.001) and NOx (p < 0.001) in hypertensives and no variation in TAS and in TBARS/NOx ratio. None of these parameters was statistically related to the metabolic parameters or to the blood pressure values. The high level of li…

AdultMalemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentBlood PressureEssential hypertensionmedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsNitric oxideEssential hypertension.Lipid peroxidationchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineTBARSHumansNOxOxidate streNitric oxideHematologyMiddle Agedmedicine.diseaseOxidative StressBlood pressureEndocrinologychemistryBiochemistryHypertensionFemaleLipid PeroxidationCardiology and Cardiovascular MedicineOxidative stressClinical Hemorheology and Microcirculation
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Lipid peroxidation and nitric oxide metabolites in sedentary subjects and sportsmen before and after a cardiopulmonary test.

2012

Our aim was to investigate the effects of an exercise test on some indices of oxidative status and endothelial function, in trained and untrained subjects. We examined lipid peroxidation, nitric oxide metabolites (NOx) and their ratio before and after a cardiopulmonary test, using a cycloergometer. We enrolled 60 male subjects who practiced sport unprofessionally, subdivided in two groups (A and B) according to the values of VO2max. Group A included sportsmen with poor or fair aerobic fitness (VO2max 39 ml/Kg/min). The control group included 19 male sedentary subjects. Lipid peroxidation was evaluated by detection of the thiobarbituric acid-reactive substances (TBARS); the NOx were evaluate…

AdultMalemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentOxidative phosphorylationmedicine.disease_causeNitric OxideThiobarbituric Acid Reactive SubstancesNitric oxideLipid peroxidationchemistry.chemical_compoundGriess testPhysiology (medical)Internal medicineTBARSMedicineAerobic exerciseHumansNO exercise cardiopulmonary testbusiness.industryHematologyOxygenOxidative StressEndocrinologychemistryPhysical therapyExercise TestLipid PeroxidationSedentary BehaviorCardiology and Cardiovascular MedicinebusinessOxidative stressSportsClinical hemorheology and microcirculation
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