Search results for "P-Selectin"

showing 10 items of 44 documents

Effect of boldine, secoboldine, and boldine methine on angiotensin II-induced neutrophil recruitment in vivo.

2005

AbstractAngiotensin-II (Ang-II) has inflammatory activity and is involved in different diseases associated with the cardiovascular system. This study has evaluated the effect of boldine (B), and two phenanthrene alkaloids semisynthesized by us, secoboldine (SB) and boldine methine (BM), on Ang-II-induced neutrophil recruitment. Intraperitoneal administration of 1 nM Ang-II induced significant neutrophil accumulation, which was maximal at 4–8 h. BM inhibited neutrophil infiltration into the peritoneal cavity at 4 h and 8 h by 73% and 77%, respectively, SB at 8 h by 55%, and B had no effect on this response. Although BM inhibited the release of cytokine-inducible neutrophil chemoattractant/ke…

KeratinocytesMaleChemokineAporphinesEndotheliumNeutrophilsImmunologyChemokine CXCL2InflammationPharmacologyRats Sprague-Dawleychemistry.chemical_compoundIn vivomedicineImmunology and AllergyBoldineAnimalsHumansInfusions ParenteralPlatelet Activating FactorReceptorchemistry.chemical_classificationReactive oxygen speciesbiologyMolecular StructureAngiotensin IIMonokinesInterleukin-8Endothelial CellsCell BiologyPhenanthrenesAngiotensin IIRatsP-Selectinmedicine.anatomical_structureBiochemistrychemistrybiology.proteinIntercellular Signaling Peptides and Proteinsmedicine.symptomChemokinesReactive Oxygen SpeciesChemokines CXCJournal of leukocyte biology
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Time Response of Oxidative/Nitrosative Stress and Inflammation in LPS-Induced Endotoxaemia—A Comparative Study of Mice and Rats

2017

Sepsis is a severe and multifactorial disease with a high mortality rate. It represents a strong inflammatory response to an infection and is associated with vascular inflammation and oxidative/nitrosative stress. Here, we studied the underlying time responses in the widely used lipopolysaccharide (LPS)-induced endotoxaemia model in mice and rats. LPS (10 mg/kg; from Salmonella Typhosa) was intraperitoneally injected into mice and rats. Animals of every species were divided into five groups and sacrificed at specific points in time (0, 3, 6, 9, 12 h). White blood cells (WBC) decreased significantly in both species after 3 h and partially recovered with time, whereas platelet decrease did no…

LipopolysaccharidesMale0301 basic medicinesepsis; time response; inflammation; oxidative stress; endotoxaemia; mouse; ratLipopolysaccharideNitric Oxide Synthase Type IIBacteremia030204 cardiovascular system & hematologymedicine.disease_causelcsh:ChemistrysepsisendotoxaemiaHemoglobinsLeukocyte CountMicechemistry.chemical_compound0302 clinical medicineoxidative stressratPlateletlcsh:QH301-705.5SpectroscopyGeneral MedicineComputer Science ApplicationsRespiratory burstP-SelectinSalmonella Infectionsmedicine.symptommedicine.medical_specialtyVascular Cell Adhesion Molecule-1InflammationOxidative phosphorylationArticleCatalysisInorganic ChemistrySepsis03 medical and health sciencesSpecies Specificitytime responseInternal medicineReaction TimemedicineAnimalsRats WistarPhysical and Theoretical ChemistryMolecular BiologymouseInterleukin-6Platelet CountTumor Necrosis Factor-alphabusiness.industryOrganic Chemistrymedicine.diseaseRatsMice Inbred C57BL030104 developmental biologyEndocrinologylcsh:Biology (General)lcsh:QD1-999chemistryinflammationImmunologyHemoglobinbusinessOxidative stressInternational Journal of Molecular Sciences
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Early high-dosage atorvastatin treatment improved serum immune-inflammatory markers and functional outcome in acute ischemic strokes classified as la…

2016

Statins have beneficial effects on cerebral circulation and brain parenchyma during ischemic stroke and reperfusion. The primary hypothesis of this randomized parallel trial was that treatment with 80 mg/day of atorvastatin administered early at admission after acute atherosclerotic ischemic stroke could reduce serum levels of markers of immune-inflammatory activation of the acute phase and that this immune-inflammatory modulation could have a possible effect on prognosis of ischemic stroke evaluated by some outcome indicators. We enrolled 42 patients with acute ischemic stroke classified as large arteries atherosclerosis stroke (LAAS) randomly assigned in a randomized parallel trial to the…

Male3400P-selectinAtorvastatinInterleukin-1beta030204 cardiovascular system & hematologylaw.inventionBrain IschemiaBrain ischemiaCerebral circulation0302 clinical medicineRandomized controlled triallawAtorvastatinIntracranial ArteriosclerosiStrokeInflammation MediatorbiologyClinical Trial/Experimental StudyGeneral MedicineMiddle AgedIntracranial ArteriosclerosisIntercellular Adhesion Molecule-1Interleukin-10StrokeP-SelectinAcute DiseaseCardiologyFemaleInflammation Mediatorsmedicine.symptomE-SelectinResearch Articlemedicine.drugHumanmedicine.medical_specialtyVascular Cell Adhesion Molecule-1Inflammation03 medical and health sciencesInternal medicinemedicineHumanscardiovascular diseasesInterleukin 6AgedInflammationbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaBiomarkermedicine.diseasePhysical therapybiology.proteinbusinessBiomarkers030217 neurology & neurosurgery
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Angiotensin II-Induced Mononuclear Leukocyte Interactions with Arteriolar and Venular Endothelium Are Mediated by the Release of Different CC Chemoki…

2006

Abstract Angiotensin II (Ang-II) is associated with atherogenesis and arterial subendothelial mononuclear leukocyte infiltration. We have demonstrated that Ang-II causes the initial attachment of mononuclear cells to the arteriolar endothelium. We now report on the contribution of CC chemokines to this response. Intraperitoneal administration of 1 nM Ang-II induced MCP-1, RANTES, and MIP-1α generation, maximal at 4 h, followed by mononuclear leukocyte recruitment at 8 and 24 h. Using intravital microscopy within the rat mesenteric microcirculation 4 h after exposure to 1 nM Ang-II, arteriolar mononuclear cell adhesion was 80–90% inhibited by pretreatment with Met-RANTES, a CCR1 and CCR5 ant…

MaleCCR1EndotheliumImmunologyVascular Cell Adhesion Molecule-1Peripheral blood mononuclear cellUmbilical CordRats Sprague-DawleyLeukocyte CountCell MovementCell AdhesionLeukocytesmedicineAnimalsHumansImmunology and AllergyEndotheliumChemokine CCL5Cells CulturedChemokine CCL2Angiotensin II receptor type 1Chemokine CCL26business.industryAngiotensin IIMonocyteEpithelial Cellsmedicine.diseaseAngiotensin IIMolecular biologyRatsP-Selectinmedicine.anatomical_structureChemokines CCImmunologycardiovascular systembusinessInfiltration (medical)Intravital microscopyThe Journal of Immunology
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Aprotinin inhibits leukocyte–endothelial cell interactions after hemorrhage and reperfusion

2003

Background. The serine protease inhibitor aprotinin has been successfully used to reduce blood loss in patients undergoing cardiac operations. We studied aprotinin for its ability to modulate leukocyte– endothelial cell interactions after ischemia and reperfusion. Methods. The effects of aprotinin on leukocyte– endothelial cell interactions were observed by intravital microscopy in the rat mesenteric microcirculation and immunohistochemical analysis. The inflammatory cascade (leukocyte rolling, firm adherence, and transmigration) was studied after thrombin stimulation and after hemorrhage and reperfusion. Results. Intravenous bolus administration of aprotinin treatment (20,000 U/kg) signifi…

MalePulmonary and Respiratory MedicineSerine Proteinase InhibitorsEndotheliumHemorrhageInflammationLeukocyte RollingCell CommunicationPharmacologyMicrocirculationRats Sprague-DawleyAprotininThrombinLeukocytesmedicineAnimalsMesenteryAprotininbusiness.industryMicrocirculationThrombinImmunohistochemistryRatsEndothelial stem cellP-Selectinmedicine.anatomical_structureReperfusionImmunologySurgeryEndothelium Vascularmedicine.symptomCardiology and Cardiovascular Medicinebusinesshormones hormone substitutes and hormone antagonistsIntravital microscopymedicine.drugThe Annals of Thoracic Surgery
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Simvastatin Inhibits Inflammatory Properties ofStaphylococcus aureusα-Toxin

2002

Background—Simvastatin, a 3-hydroxy-methylglutaryl coenzyme A reductase inhibitor, has been shown to lower serum cholesterol levels in clinical use. Moreover, statins exert beneficial effects in vascular diseases by inhibition of leukocyte rolling, adherence, and transmigration. The aim of this study was to determine if pretreatment with simvastatin attenuatesStaphylococcus aureusα-toxin–induced increase in leukocyte-endothelial interactions during exotoxemia.Methods and Results—The effects of simvastatin on leukocyte-endothelial cell interactions were observed by intravital microscopy in the rat mesenteric microcirculation. Simvastatin (50 or 100 μg/kg) was administered 18 hours before the…

MaleSimvastatinNitric Oxide Synthase Type IIIP-selectinEndotheliumBacterial ToxinsToxemiaInflammationLeukocyte RollingPharmacologyMicrocirculationRats Sprague-DawleyHemolysin ProteinsMesenteric VeinsVenulesCell MovementCulture TechniquesPhysiology (medical)Cell AdhesionLeukocytesmedicineAnimalsMicroscopy Videobusiness.industryAnti-Inflammatory Agents Non-SteroidalHemodynamicsStaphylococcal InfectionsImmunohistochemistryRatsEndothelial stem cellP-Selectinmedicine.anatomical_structureSimvastatinImmunologyEndothelium VascularHydroxymethylglutaryl-CoA Reductase InhibitorsNitric Oxide Synthasemedicine.symptomCardiology and Cardiovascular MedicinebusinessIntravital microscopymedicine.drugCirculation
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Modification of human platelet adhesion on biomaterial surfaces by protein preadsorption under static and flow conditions.

2004

Biomaterial-induced thrombosis remains one of the main complications of vascular implant devices. Preadsorbed proteins on the biomaterial/blood interface will modify the adhesion and activation of platelets (PTLs) during the initial contact-phase. Our results clearly show that PTL-adherence on biomaterials is influenced not only by protein preadsorption, but also by flow conditions. The covalent coating of TCPS and glass by phosphorylcholine (PC) induces a significant decrease of PTL adhesion but leads to a slight, but nevertheless significant activation of PTL, which was detected by the induction of P-selectin expression using FACS analysis. Methodologically, the visualization of PTL adhes…

Materials scienceBiomedical EngineeringBiophysicschemistry.chemical_elementBioengineeringBiocompatible MaterialsCalciumIn Vitro TechniquesBiomaterialsBlood cellPlatelet AdhesivenessIn vivoMaterials TestingmedicineHumansPlateletPhosphorylcholineBiomaterialThrombosisAdhesionBlood ProteinsFlow CytometryPlatelet ActivationBiomechanical PhenomenaBlood Vessel ProsthesisP-Selectinmedicine.anatomical_structurechemistryembryonic structuresImmunologyHemorheologyBiophysicsAdsorptionProtein adsorptionJournal of materials science. Materials in medicine
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Effects of transdermal hormone replacement therapy on levels of soluble P- and E-selectin in postmenopausal healthy women

2002

Abstract Objective: To study the adhesion molecule pattern in postmenopausal women who were not receiving hormone replacement therapy (HRT), HRT users, and fertile women. Design: Case-control study. Setting: Second University of Naples, Naples, Italy. Patient(s): Fifty healthy naturally postmenopausal women and 20 fertile women. Intervention(s): Twenty-six women received no HRT and 24 received continuous transdermal 17β−estradiol, 0.05 mg/d, plus oral acetate nomegestrol, 5 mg/d. Main Outcome Measure(s): Levels of the soluble forms of intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), E-selectin, and P-selectin. Result(s): Women who did not received HRT …

Nomegestrolmedicine.medical_specialtyAdministration Cutaneougenetic structuresP-selectinmedicine.drug_classHormone Replacement Therapymedicine.medical_treatmentHRTVascular Cell Adhesion Molecule-1Administration Cutaneouschemistry.chemical_compoundInternal medicineE-selectinmedicineHumansICAM-1biologyEstradiolbusiness.industryCase-control studyObstetrics and GynecologyHormone replacement therapy (menopause)Megestrolmedicine.diseaseIntercellular Adhesion Molecule-1AtherosclerosisMenopausePostmenopauseP-SelectinEndocrinologyReproductive MedicinechemistryEstrogenCase-Control Studiesbiology.proteinAdhesion molecules; Atherosclerosis; HRTFemalebusinessCase-Control StudieE-SelectinAdhesion moleculesHuman
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Clinical correlates of change in inflammatory biomarkers: The Framingham Heart Study

2013

Objectives: Traditional clinical risk factors are associated with inflammation cross-sectionally, but associations of longitudinal variation in inflammatory biomarkers with corresponding changes in clinical risk factors are incompletely described. We sought to analyze clinical factors associated with change in inflammation in the community.Methods: We studied 3013 Framingham Offspring (n = 2735) and Omni Cohort (n = 278) participants (mean age 59 years, 55% women, 9% ethnic/racial minority) who attended two consecutive examination cycles (mean 6.7 years apart). We selected ten inflammatory biomarkers representing distinctive biological functions: C-reactive protein (CRP), intercellular adhe…

OncologyMaleBLOOD-PRESSUREIsoprostanesFramingham Heart StudyRisk FactorsMyocardial infarctionOXIDATIVE STRESSskin and connective tissue diseasesChemokine CCL2Biological markersbiologyLongitudinal studiesMiddle AgedIntercellular Adhesion Molecule-1Inflammatory biomarkersC-REACTIVE PROTEINP-SelectinADIPOSE-TISSUEMassachusettsCardiovascular DiseasesCARDIOVASCULAR-DISEASEFemalemedicine.symptomCardiology and Cardiovascular MedicineVasculitisVasculitismedicine.medical_specialtyInflammationArticleInternal medicinemedicineHumansReceptors Tumor Necrosis Factor Type IIInterleukin 6AgedInflammationbusiness.industryInterleukin-6PERIPHERAL ARTERIAL-DISEASEC-reactive proteinOsteoprotegerinADHESION MOLECULE-1medicine.diseasePHOSPHOLIPASE A(2)Blood pressurePLASMA-CONCENTRATIONMYOCARDIAL-INFARCTIONImmunology1-Alkyl-2-acetylglycerophosphocholine Esterasebiology.proteinsense organsbusinessBiomarkers
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P-Selectin Glycoprotein Ligand-1 as a Potential Target for Humoral Immunotherapy of Multiple Myeloma (Supplementry Material)

2009

Monoclonal antibodies (mAbs), successfully adopted in the treatment of several haematological malignancies, have proved almost ineffective in multiple myeloma (MM), because of the lack of an appropriate antigen for targeting and killing MM cells. Here, we demonstrate that PSGL1, the major ligand of P-Selectin, a marker of plasmacytic differentiation expressed at high levels on normal and neoplastic plasma cells, may represent a novel target for mAb-mediated MM immunotherapy. The primary effectors of mAb-induced cell-death, complement-mediated lysis (CDC) and antibody-dependent cellmediated cytotoxicity (ADCC), were investigated using U266B1 and LP1 cell-lines as models. Along with immunolog…

PharmacologyAntibody-dependent cell-mediated cytotoxicityCancer Researchbiologymedicine.drug_classmedicine.medical_treatmentImmunotherapyMonoclonal antibodyMolecular biologymedicine.anatomical_structureOncologyAntigenDrug Discoverymedicinebiology.proteinP-selectin glycoprotein ligand-1Bone marrowAntibodyCytotoxicityCurrent Cancer Drug Targets
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