Search results for "PATHOGENESIS"

showing 10 items of 761 documents

An evidence based therapeutic approach to hereditary and acquired angioedema

2014

Purpose of review Hereditary angioedema (HAE) due to C1 esterase inhibitor (C1-INH) deficiency (HAE-C1-INH), HAE with normal C1-INH, and acquired angioedema due to C1-INH deficiency are rare but important diseases that can be associated with significant morbidity and mortality. Research into the pathogenesis of angioedema has expanded greatly and has led to new clinical trials with novel therapeutic agents and strategies. Recent findings Strategies for managing HAE-C1-INH are aimed at treating acute attacks or preventing attacks through the use of prophylactic treatment. Agents available in Europe for treating acute attacks include plasma-derived C1-INH concentrates, a bradykinin B2 recepto…

Evidence-based practiceImmunologyBradykininBioinformaticsPathogenesischemistry.chemical_compoundTherapeutic approachBradykinin B2 Receptor AntagonistsHumansImmunology and AllergyMedicineheterocyclic compoundsRandomized Controlled Trials as TopicEvidence-Based MedicineAngioedemabusiness.industryAngioedemas HereditaryAntagonistbiochemical phenomena metabolism and nutritionrespiratory systembacterial infections and mycosesmedicine.diseaserespiratory tract diseasesClinical trialchemistryHereditary angioedemaKallikreinsmedicine.symptombusinessComplement C1 Inhibitor ProteinCurrent Opinion in Allergy & Clinical Immunology
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Dupuytren contracture in diabetic hand

2011

Dupuytren's disease (DD) is a fibroproliferative disorder of unknown origin causing palmar nodules and flexion contracture of the digits. About 5% of individuals with DD are diabetic, with an increased prevalence that is proportional to the duration of the diabetes. The association with diabetes mellitus is well recorded, with a reported prevalence of between 3% and 32%. This review considers the evidence behind the association between DD and diabetes mellitus, considering the pathogenesis, clinical manifestations and therapies that can be found in the literature.

Flexion contracturemedicine.medical_specialtybusiness.industryDupuytren Contracturemedicine.medical_treatmentDiseasemedicine.diseaseDermatologySurgeryFasciotomybody regionsPathogenesisDiabetes mellitusmedicineSettore MED/33 - Malattie Apparato LocomotorebusinessDupuytren's desease diabetic hand fasciotomy dermofasciotomy
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Life History Trade-Offs and Relaxed Selection Can Decrease Bacterial Virulence in Environmental Reservoirs

2012

Pathogen virulence is usually thought to evolve in reciprocal selection with the host. While this might be true for obligate pathogens, the life histories of opportunistic pathogens typically alternate between within-host and outside-host environments during the infection-transmission cycle. As a result, opportunistic pathogens are likely to experience conflicting selection pressures across different environments, and this could affect their virulence through life-history trait correlations. We studied these correlations experimentally by exposing an opportunistic bacterial pathogen Serratia marcescens to its natural protist predator Tetrahymena thermophila for 13 weeks, after which we meas…

Food ChainEvolutionary ProcessesScienceVirulenceParallel EvolutionPathogenesisEnvironmentBiologyForms of EvolutionMicrobiologyDivergent EvolutionTetrahymena thermophilaMicrobial Ecology03 medical and health sciencesNatural Selectionexperimental evolutionSelection GeneticAdaptationBiologyMicrobial PathogensPathogenSerratia marcescensSelection (genetic algorithm)030304 developmental biologyGeneticsEvolutionary Biology0303 health sciencesMultidisciplinaryEcologyObligate030306 microbiologyHost (biology)Mechanism (biology)QRAdaptation PhysiologicalBiological EvolutionBacterial PathogensvirulenceEvolutionary EcologyMicrobial EvolutionBacterial pigmentMedicineta1181AdaptationResearch ArticlePLoS ONE
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In Candida parapsilosis the ATC1 Gene Encodes for an Acid Trehalase Involved in Trehalose Hydrolysis, Stress Resistance and Virulence

2014

An ORF named CPAR2-208980 on contig 005809 was identified by screening a Candida parapsilosis genome data base. Its 67% identity with the acid trehalase sequence from C. albicans (ATC1) led us to designate it CpATC1. Homozygous mutants that lack acid trehalase activity were constructed by gene disruption at the two CpATC1 chromosomal alleles. Phenotypic characterization showed that atc1Δ null cells were unable to grow on exogenous trehalose as carbon source, and also displayed higher resistance to environmental challenges, such as saline exposure (1.2 M NaCl), heat shock (42°C) and both mild and severe oxidative stress (5 and 50 mM H2O2). Significant amounts of intracellular trehalose were …

Fungal PhysiologyMutantGlycobiologyTrehalase activitylcsh:MedicineMicrobiologiaPathogenesisPathology and Laboratory MedicineCandida parapsilosisBiochemistrychemistry.chemical_compoundNucleic AcidsMicrobial PhysiologyMedicine and Health SciencesTrehalaseTrehalaselcsh:ScienceFungal BiochemistryCandida albicansCandidaMultidisciplinaryVirulencebiologyOrganic CompoundsSalt ToleranceCatalaseEnzymesChemistryPhysical SciencesResearch ArticleGenes FungalMolecular Sequence DataCarbohydratesMycologyMicrobiologyMicrobiologyFungal ProteinsAmino Acid SequenceHeat shockGlycoproteinslcsh:ROrganismsFungiChemical CompoundsWild typeTrehaloseBiology and Life Sciencesbiology.organism_classificationTrehaloseYeastOxidative StressMetabolismchemistryProteolysisEnzymologylcsh:QHeat-Shock ResponsePLoS ONE
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The Cell Cycle-Specific Growth-Inhibitory Factor Produced by Actinobacillus actinomycetemcomitans Is a Cytolethal Distending Toxin

1998

ABSTRACT Actinobacillus actinomycetemcomitans has been shown to produce a soluble cytotoxic factor(s) distinct from leukotoxin. We have identified in A. actinomycetemcomitans Y4 a cluster of genes encoding a cytolethal distending toxin (CDT). This new member of the CDT family is similar to the CDT produced by Haemophilus ducreyi . The CDT from A. actinomycetemcomitans was produced in Escherichia coli and was able to induce cell distension, growth arrest in G 2 /M phase, nucleus swelling, and chromatin fragmentation in HeLa cells. The three proteins, CDTA, -B and -C, encoded by the cdt locus were all required for toxin activity. Antiserum raised against recombinant CDTC completely inhibited …

G2 PhaseCytolethal distending toxin[SDV]Life Sciences [q-bio]Bacterial ToxinsMolecular Sequence DataRestriction MappingImmunologyMitosismedicine.disease_causeAggregatibacter actinomycetemcomitansMicrobiologyVirulence factorMicrobiologyEscherichia colimedicineHumansAmino Acid SequenceCloning MolecularEscherichia coliBase SequencebiologyToxinACTIVITEAggregatibacter actinomycetemcomitansGENETIQUECell cyclebiology.organism_classificationGrowth InhibitorsRecombinant Proteins[SDV] Life Sciences [q-bio]Infectious DiseasesGenes BacterialMultigene FamilyActinobacillusMolecular and Cellular PathogenesisParasitologyHaemophilus ducreyiHeLa CellsInfection and Immunity
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Immunological insights into the pathogenesis of active CMV infection in non-immunosuppressed critically ill patients.

2011

Dissociation of cytomegalovirus (CMV) DNA loads between the lower respiratory tract and blood, with high levels in the former compartment and low or undetectable levels in the latter, commonly occurs during active CMV infection in critically ill patients despite the presence of high frequencies of CMV-specific IFN-γ-producing CD8+ and CD4+ T cells in blood. Data presented in this case report suggest that inter-compartmental differences in interleukin-10 (IL-10) levels may, in part, explain the pathobiology of this phenomenon. In the absence of ganciclovir treatment, a significant correlation was observed between IL-10 levels and CMV DNA loads in lower respiratory tract specimens (P = 0.016)…

GanciclovirMalemedicine.medical_treatmentCritical IllnessRespiratory SystemCytomegalovirusPathogenesisVirologymedicineHumansInterleukin 6AgedAged 80 and overLungbiologyvirus diseasesImmunosuppressionMiddle AgedViral LoadVirologyInterleukin-10Interleukin 10Infectious Diseasesmedicine.anatomical_structureBloodImmunologyCytomegalovirus Infectionsbiology.proteinFemaleBronchoalveolar Lavage FluidCD8Respiratory tractmedicine.drugJournal of medical virology
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Lipid chaperones and associated diseases: a group of chaperonopathies defining a new nosological entity with implications for medical research and pr…

2020

AbstractFatty acid–binding proteins (FABPs) are lipid chaperones assisting in the trafficking of long-chain fatty acids with functions in various cell compartments, including oxidation, signaling, gene-transcription regulation, and storage. The various known FABP isoforms display distinctive tissue distribution, but some are active in more than one tissue. Quantitative and/or qualitative changes of FABPs are associated with pathological conditions. Increased circulating levels of FABPs are biomarkers of disorders such as obesity, insulin resistance, cardiovascular disease, and cancer. Deregulated expression and malfunction of FABPs can result from genetic alterations or posttranslational mo…

Gene isoformChaperonotherapyBiomedical ResearchDiseaseBioinformaticsFatty Acid-Binding ProteinsBiochemistryModels BiologicalFatty acid–binding proteinsFatty acid-binding proteinPathogenesisInsulin resistanceSettore BIO/10 - BiochimicaMedicineAnimalsHumansDiseasePathologicalLipid chaperonesbusiness.industrySettore BIO/16 - Anatomia UmanaCancerCell BiologyChaperonopathiesmedicine.diseaseLipidslipids (amino acids peptides and proteins)Metabolic syndromePerspective and Reflection ArticlebusinessLipid chaperone-associate pathologiesMolecular ChaperonesCell stresschaperones
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Goodpasture antigen-binding protein, the kinase that phosphorylates the goodpasture antigen, is an alternatively spliced variant implicated in autoim…

2000

The non-collagenous C-terminal domain of the alpha(3) chain of collagen IV is the autoantigen in Goodpasture disease, an autoimmune disorder described only in humans. Specific N-terminal phosphorylation is a biological feature unique to the human domain when compared with other homologous domains lacking immunopathogenic potential. We have recently cloned from a HeLa-derived cDNA library a novel serine/threonine kinase (Goodpasture antigen-binding protein (GPBP)) that phosphorylates the N-terminal region of the human domain (Raya, A. Revert, F, Navarro, S. and Saus J. (1999) J. Biol. Chem. 274, 12642-12649). We show here that the pre-mRNA of GPBP is alternatively spliced in human tissues an…

Gene isoformCollagen Type IVMolecular Sequence DataBiologyProtein Serine-Threonine KinasesBiochemistryAutoantigensSerinePathogenesisTwo-Hybrid System TechniquesHumansAmino Acid SequencePhosphorylationMolecular BiologyCeramide Transfer ProteinBase SequenceKinasecDNA libraryCell BiologyDNACeramide transportMolecular biologyRecombinant ProteinsAlternative SplicingBiochemistryPhosphorylationCollagenThe Journal of biological chemistry
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Gamma-secretase modulation with Abeta42-lowering nonsteroidal anti-inflammatory drugs and derived compounds.

2006

The amyloid-beta (Abeta) peptides and specifically the highly amyloidogenic isoform Abeta42 appear to be key agents in the pathogenesis of familial and sporadic forms of Alzheimer's disease (AD). The final step in the generation of Abeta from the amyloid precursor protein is catalyzed by the multiprotein complex gamma-secretase, which constitutes a prime drug target for prevention and therapy of the disease. However, highly potent gamma-secretase inhibitors that block formation of all Abeta peptides have provoked troubling side effects in preclinical animal models of AD. This toxicity can be readily explained by the promiscuous substrate specificity of gamma-secretase and its essential role…

Gene isoformendocrine systemClinical Trials as TopicNonsteroidalAmyloid beta-Peptidesmedicine.drug_classAnti-Inflammatory Agents Non-SteroidalPharmacologyIbuprofenAmyloid β peptideAnti-inflammatoryPathogenesischemistry.chemical_compoundNeurologychemistryAlzheimer DiseasemedicineAnimalsHumansNeurology (clinical)γ secretaseAmyloid Precursor Protein SecretasesGamma secretasemedicine.drugNeuro-degenerative diseases
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Preemptive CD8 T-Cell Immunotherapy of Acute Cytomegalovirus Infection Prevents Lethal Disease, Limits the Burden of Latent Viral Genomes, and Reduce…

1998

ABSTRACT In the immunocompetent host, primary cytomegalovirus (CMV) infection is resolved by the immune response without causing overt disease. The viral genome, however, is not cleared but is maintained in a latent state that entails a risk of virus recurrence and consequent organ disease. By using murine CMV as a model, we have shown previously that multiple organs harbor latent CMV and that reactivation occurs with an incidence that is determined by the viral DNA load in the respective organ (M. J. Reddehase, M. Balthesen, M. Rapp, S. Jonjic, I. Pavic, and U. H. Koszinowski. J. Exp. Med. 179:185–193, 1994). This predicts that a therapeutic intervention capable of limiting the load of lat…

Genes Viralmedicine.medical_treatmentImmunologyViral Pathogenesis and ImmunityGenome ViralCD8-Positive T-LymphocytesBiologymedicine.disease_causeMicrobiologyVirusMiceImmune systemRecurrenceRisk FactorsVirologyVirus latencymedicineAnimalsHumansCytotoxic T cellLungCells CulturedBone Marrow TransplantationMice Inbred BALB CCytomegalovirusImmunotherapyViral Loadmedicine.diseaseVirologyVirus LatencyDisease Models AnimalInsect ScienceAcute DiseaseCytomegalovirus InfectionsDNA ViralImmunologyFemaleImmunotherapyViral loadCD8Journal of Virology
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