Search results for "PATHOGENESIS"

showing 10 items of 761 documents

Phytopathogenic Bacteria Associated with Bacterioses of Common Oak (Quercus robur L.) in Ukraine

2022

Studying the biological characteristics of the causative agents of bacteriosis of the common oak (Quercus robur L.) (to highlight the links between the key factors of pathogenesis and bacterial groups) is a new and relevant area of research. This work aims to identify the species composition of phytopathogenic bacteria associated with common oak bacteriosis and to study the morphological, cultural, and biochemical properties of pathogens. To establish the general phytosanitary state of the surveyed stands, classical forest inventory and phytopathological (visual on based on typical symptoms of the disease and macroscopic) methods were used. The properties of isolated bacteria were studied u…

Lonsdalea quercina<i>Lelliottia nimipressuralis</i>; <i>Lonsdalea quercina</i>; <i>Erwinia rhapontici</i>; <i>Pseudomonas fluorescens</i>; <i>Pseudomonas</i> sp.; <i>Quercus robur</i> L.; etiology; pathogenesisQuercus robur L.etiologypathogenesisLelliottia nimipressuralisForestryPseudomonas sp.Erwinia rhaponticiPseudomonas fluorescensForests
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Control of cytomegalovirus in bone marrow transplantation chimeras lacking the prevailing antigen-presenting molecule in recipient tissues rests prim…

1998

ABSTRACT Cytomegalovirus (CMV) infection during the transient immunodeficiency after bone marrow transplantation (BMT) develops into disease unless antiviral CD8 T cells are restored in due course. Histoincompatibility between donor and recipient is associated with increased risk. Complications may include a rejection response against the foreign major histocompatibility complex (MHC) antigens and a lack of antiviral control resulting from a misfit between donor-derived T cells and the antigenic viral peptides presented in recipient tissues. Here we have established a murine model of CMV disease after experimental BMT performed across a single MHC class I disparity. Specifically, BALB/c bon…

Lung DiseasesAdoptive cell transferImmunologyAntigen-Presenting CellsViral Pathogenesis and ImmunityCD8-Positive T-LymphocytesMajor histocompatibility complexMicrobiologyMajor Histocompatibility ComplexChimera (genetics)MiceAntigenVirologyMHC class ImedicineCytotoxic T cellAnimalsAntigen-presenting cellMice Inbred BALB CBone TransplantationbiologyChimeraVirologymedicine.anatomical_structureInsect ScienceImmunologyCytomegalovirus Infectionsbiology.proteinBone marrow
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Telomere Length in Circulating Leukocytes is Associated with Lung Function and Disease

2014

Several clinical studies suggest the involvement of premature ageing processes in chronic obstructive pulmonary disease (COPD). Using an epidemiological approach, we studied whether accelerated ageing indicated by telomere length, a marker of biological age, is associated with COPD and asthma, and whether intrinsic age-related processes contribute to the interindividual variability of lung function. Our meta-analysis of 14 studies included 934 COPD cases with 15 846 controls defined according to the Global Lungs Initiative (GLI) criteria (or 1189 COPD cases according to the Global Initiative for Chronic Obstructive Lung Disease (GOLD) criteria), 2834 asthma cases with 28 195 controls, and s…

Lung DiseasesMalePulmonary and Respiratory MedicineSpirometryNetherlands Twin Register (NTR)Vital capacityVital Capacityennenaikainen vanheneminenDiseaseCohort StudiesPathogenesisPulmonary Disease Chronic ObstructiveTelomere HomeostasisSDG 3 - Good Health and Well-beingForced Expiratory VolumeastmaLeukocytes/dk/atira/pure/keywords/cohort_studies/netherlands_twin_register_ntr_HumansCOPDMedicinekeuhkoahtaumatautiAgedAsthmatelomeeriCOPDmedicine.diagnostic_testpremature agingbusiness.industrySmokingCase-control studyta3141Middle AgedTelomerespirometriamedicine.diseaseAsthmabiologinen ikäbiological age3. Good healthrespiratory tract diseasesEuropeSpirometryCase-Control StudiesImmunologyRegression Analysis/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemalebusinessEuropean Respiratory Journal
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SPARC regulation of PMN clearance protects from pristane induced lupus and rheumatoid arthritis

2020

AbstractOne step along the pathogenesis of Systemic lupus erythematosus (SLE) is associated with polymorphonuclear leukocyte (PMN) death and their ineffective removal by M2-macrophages. The secreted protein acidic and rich in cysteine (SPARC) is a matricellular protein with unexpected immunosuppressive function in M2-macrophages and myeloid cells. To investigate the role of SPARC in autoimmunity, we adopted a pristane–induced model of lupus in mice, which recapitulates clinical manifestations of human SLE. Sparc-/- mice developed earlier and more severe renal disease, lung and liver parenchymal damage than the WT counterpart. Most prominently, Sparc-/- mice had anticipated and severe occurr…

LungSystemic lupus erythematosusbusiness.industryMatricellular proteinArthritisDendritic cellmedicine.diseasemedicine.disease_causeAutoimmunityPathogenesismedicine.anatomical_structureRheumatoid arthritisImmunologymedicineCancer researchMacrophagebusiness
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Morphological and biochemical features of Borrelia burgdorferi pleomorphic forms

2015

The spirochaete bacterium Borrelia burgdorferi sensu lato is the causative agent of Lyme disease, the most common tick-borne infection in the northern hemisphere. There is a long-standing debate regarding the role of pleomorphic forms in Lyme disease pathogenesis, while very little is known about the characteristics of these morphological variants. Here, we present a comprehensive analysis of B. burgdorferi pleomorphic formation in different culturing conditions at physiological temperature. Interestingly, human serum induced the bacterium to change its morphology to round bodies (RBs). In addition, biofilm-like colonies in suspension were found to be part of B. burgdorferi’s normal in vitr…

Lyme Diseaseta1183ta1182Biologymedicine.diseasebiology.organism_classificationbacterial infections and mycosesMicrobiologyVirologyStandardMicrobiologyPathogenesisCell wallLyme diseaseCell WallBorrelia burgdorferiCell and Molecular Biology of Microbespleomorphic bacteriamedicineSpirochaeteLyme disease microbiologyHumansCell envelopeBorrelia burgdorferiBacteriaMicrobiology
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Heat shock proteins in fibrosis and wound healing: Good or evil?

2014

Heat shock proteins (HSPs) are key regulators of cell homeostasis, and their cytoprotective role has been largely investigated in the last few decades. However, an increasing amount of evidence highlights their deleterious effects on several human pathologies, including cancer, in which they promote tumor cell survival, proliferation and drug resistance. Therefore, HSPs have recently been suggested as therapeutic targets for improving human disease outcomes. Fibrotic diseases and cancer share several properties; both pathologies are characterized by genetic alterations, uncontrolled cell proliferation, altered cell interactions and communication and tissue invasion. The discovery of new HSP…

MAPK/ERK pathwayPulmonary FibrosisCellApoptosisBiologyCell Physiological PhenomenaTransforming Growth Factor beta1PathogenesisFibrosisNeoplasmsHeat shock proteinmedicineHumansHSP70 Heat-Shock ProteinsPharmacology (medical)HSP90 Heat-Shock ProteinsHSP110 Heat-Shock ProteinsHSP47 Heat-Shock ProteinsHeat-Shock ProteinsPharmacologyWound HealingCell growthCancerEndomyocardial Fibrosismedicine.diseaseFibrosisHeat-Shock Proteins Smallmedicine.anatomical_structureImmunologyCancer researchCollagenWound healingPharmacology &amp; Therapeutics
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Candida albicans Yeast and Hyphae are Discriminated by MAPK Signaling in Vaginal Epithelial Cells

2011

We previously reported that a bi-phasic innate immune MAPK response, constituting activation of the mitogen-activated protein kinase (MAPK) phosphatase MKP1 and c-Fos transcription factor, discriminates between the yeast and hyphal forms of Candida albicans in oral epithelial cells (ECs). Since the vast majority of mucosal Candida infections are vaginal, we sought to determine whether a similar bi-phasic MAPK-based immune response was activated by C. albicans in vaginal ECs. Here, we demonstrate that vaginal ECs orchestrate an innate response to C. albicans via NF-κB and MAPK signaling pathways. However, unlike in oral ECs, the first MAPK response, defined by c-Jun transcription factor acti…

MAPK/ERK pathwaylcsh:MedicineYeast and Fungal ModelsPathogenesisSignal transductionMolecular cell biologyCandida albicansGranulocyte Colony-Stimulating FactorCandida albicanslcsh:ScienceImmune Response0303 health sciencesMultidisciplinarybiologyCandidiasisNF-kappa BSignaling cascadesObstetrics and GynecologyCorpus albicansInnate ImmunityHost-Pathogen InteractionInfectious DiseasesVaginaCytokinesMedicineFemaleSignal transductionCandidalysinResearch ArticleMAPK signaling cascadesMAP Kinase Signaling SystemUrologyImmunologySexually Transmitted DiseasesHyphaeMycologyMicrobiologyMicrobiologyImmune Activation03 medical and health sciencesModel OrganismsHumansTranscription factorBiology030304 developmental biologyInnate immune systemChemokine CCL20030306 microbiologyGenitourinary InfectionsInterleukin-6lcsh:RImmunityFungiMouth MucosaImmune DefenseEpithelial Cellsbiology.organism_classificationImmunity InnateCCL20Immune Systemlcsh:QClinical ImmunologyPLoS ONE
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Virulent synergistic effect between Enterococcus faecalis and Escherichia coli assayed by using the Caenorhabditis elegans model.

2008

5 pages; International audience; BACKGROUND: The role of enterococci in the pathogenesis of polymicrobial infections is still debated. The purpose of this study was to evaluate the effect of virulent enterococci in the presence or absence of Escherichia coli strains in the in vivo Caenorhabditis elegans model. PRINCIPAL FINDINGS: This study demonstrated that there was a synergistic effect on virulence when an association of enterococci and E. coli (LT50 = 1.6 days+/-0.1 according to the tested strains and death of nematodes in 4 days+/-0.5) was tested in comparison with enterococci alone (LT50 = 4.6 days+/-0.1 and death in 10.4 days+/-0.6) or E. coli alone (LT50 = 2.1+/-0.9 and deaths 6.6+/…

MESH : Virulence FactorsInfectious Diseases/Gastrointestinal InfectionsMESH : Escherichia colilcsh:MedicineMESH : Genotypemedicine.disease_causeMESH: Regression AnalysisPathogenesisMESH: GenotypeInfectious Diseases/Bacterial InfectionsMESH : Regression AnalysisGenotype[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisEnterococcus faecalis[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: AnimalsMESH : Anti-Bacterial AgentsMESH : Enterococcus faecalislcsh:ScienceCaenorhabditis elegans0303 health sciencesMultidisciplinarybiologyMESH: Escherichia coliBacterial Infections3. Good healthAnti-Bacterial AgentsMicrobiology/Immunity to InfectionsMESH : Bacterial InfectionsGastroenterology and Hepatology/Gastrointestinal Infections[SDV.IMM]Life Sciences [q-bio]/ImmunologyRegression AnalysisMicrobiology/Cellular Microbiology and PathogenesisResearch ArticleMESH: Enterococcus faecalis[SDV.IMM] Life Sciences [q-bio]/ImmunologyGenotypeMESH: Bacterial InfectionsVirulence FactorsVirulenceEnterococcus faecalisMicrobiologyMESH : Caenorhabditis elegans03 medical and health sciencesIn vivoMESH: Anti-Bacterial AgentsMESH: Caenorhabditis elegansmedicineEscherichia coliAnimalsCaenorhabditis elegansEscherichia coli030304 developmental biologyMESH: Virulence Factors030306 microbiologylcsh:RMicrobiology/Medical Microbiology[SDV.EE.IEO] Life Sciences [q-bio]/Ecology environment/Symbiosisbiology.organism_classificationMESH : Disease Models AnimalDisease Models AnimalEnterococcuslcsh:QMESH : AnimalsMESH: Disease Models Animal[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisPloS one
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Diminished Vitamin D Receptor Protein Levels in Crohn´s Disease Fibroblasts: Effects of Vitamin D

2020

Vitamin D (VD) deficiency has been associated to Crohn&acute

Male0301 basic medicineCrohn’s diseaselcsh:TX341-641vitamin DCalcitriol receptorArticlePathogenesis03 medical and health sciences0302 clinical medicineCrohn DiseaseCell MovementFibrosisfibroblastsVitamin D and neurologyAnimalsHumansMedicineFibroblastWound HealingCrohn's diseaseNutrition and Dieteticsbusiness.industryfibrosisEpithelial CellsVitamin D Deficiencymedicine.diseasedigestive system diseasesIntestinesMice Inbred C57BLDisease Models Animal030104 developmental biologymedicine.anatomical_structureGene Expression Regulationvitamin D receptor (VDR)Cancer researchReceptors CalcitriolFemale030211 gastroenterology & hepatologyWound healing assaybusinesslcsh:Nutrition. Foods and food supplyFood ScienceHormoneCrohn´s diseaseNutrients
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Magnesium in Infectious Diseases in Older People

2021

Reduced magnesium (Mg) intake is a frequent cause of deficiency with age together with reduced absorption, renal wasting, and polypharmacotherapy. Chronic Mg deficiency may result in increased oxidative stress and low-grade inflammation, which may be linked to several age-related diseases, including higher predisposition to infectious diseases. Mg might play a role in the immune response being a cofactor for immunoglobulin synthesis and other processes strictly associated with the function of T and B cells. Mg is necessary for the biosynthesis, transport, and activation of vitamin D, another key factor in the pathogenesis of infectious diseases. The regulation of cytosolic free Mg in immune…

Male0301 basic medicineEpstein-Barr Virus InfectionsAgingSettore MED/09 - Medicina InternaX-Linked Combined Immunodeficiency DiseaseReviewX-Linked Combined Immunodeficiency Diseasesinfectious diseasesCommunicable DiseasePathogenesis0302 clinical medicineEpstein-Barr Virus Infectionoxidative stressMedicineMagnesium030212 general & internal medicineVitamin DCation Transport ProteinsImmunodeficiencyInfectious diseaseNutrition and DieteticsbiologyFemalemedicine.symptomAntibodylcsh:Nutrition. Foods and food supplyHumanlcsh:TX341-641InflammationCommunicable DiseasesVirus03 medical and health sciencesImmune systemImmunityHumansAgedInflammationSARS-CoV-2business.industryCOVID-19medicine.disease030104 developmental biologyCation Transport ProteinImmunologyPrimary immunodeficiencybiology.proteinOxidative strebusinessMagnesium DeficiencyFood ScienceNutrients
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