Search results for "PATHOGENESIS"

showing 10 items of 761 documents

Local skin necroses after intramuscular injection -Experimental animal studies-.

1977

The pathogenesis of local skin necroses after intramuscular injection of various drugs such as phenylbutazone (Embolia cutis medicamentosa, Nicolau's syndrome) is not clear. In an attempt to simulate this clinical feature experiments were performed on the rabbit ear lobe. A 20% phenylbutazone solution was injected paraarterial, intraarterial and paraarterial after perforation of the vessel. The drug produced a violent inflammation with all kinds of application. The local inflammation induced by paraarterial injection resulted in a fine scarring. Both other kinds of application produced necroses or even perforations. The histological examinations in these cases revealed massive destructions …

MalePathologymedicine.medical_specialtyNecrosisPerforation (oil well)InflammationDermatologyDermatitis ContactInjections IntramuscularSkin DiseasesPathogenesisLesionNecrosismedicinePhenylbutazoneAnimalsSkinbusiness.industryGeneral MedicineSurgerymedicine.anatomical_structurePhenylbutazoneBlood VesselsFemaleRabbitsmedicine.symptombusinessIntramuscular injectionmedicine.drugArteryArchives for dermatological research = Archiv fur dermatologische Forschung
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Neocortical Variation of Abeta Load in Fully Expressed, Pure Alzheimer's Disease

2010

The relationship between amyloid-beta (A beta) deposition and tau-related neurofibrillary changes is a key issue in the pathogenesis of Alzheimer's disease (AD). The aim of this study was to investigate the extent and cortical distribution of A beta and tau pathology, their mutual links and their correlation with the duration of the disease in thirty-nine patients with fully expressed AD. By tau immunohistochemistry, we identified different patterns of distribution of neurofibrillary changes that were ascribed to Braak stage V and VI. The disease duration was longer in patients at Braak stage VI than in those at V. Morphometric analysis carried out in several neocortical areas demonstrated …

MalePathologymedicine.medical_specialtyTau proteinNeocortextau ProteinsPathogenesisSuperior temporal gyrusAlzheimer Diseasemental disordersmedicineHumansSenile plaquesAgedAged 80 and overNeocortexAmyloid beta-PeptidesbiologyGeneral NeuroscienceGeneral MedicinePsychiatry and Mental healthClinical Psychologymedicine.anatomical_structureGene Expression RegulationCerebral cortexbiology.proteinDisease ProgressionFemaleGeriatrics and GerontologyPrimary motor cortexPsychologyNeuroscienceBraak staging
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Experimental autoimmune hepatitis: Disease induction, time course and t-cell reactivity

1990

This study describes a murine model of autoimmune hepatitis: experimental autoimmune hepatitis. Experimental autoimmune hepatitis could be induced most effectively in male C57BL/6 mice by intraperitoneal immunization with the 100,000 g supernatant of syngeneic liver homogenate (S-100) in complete Freund's adjuvant. BALB/C and C3H mice were less susceptible than C57BL/6 mice. Experimental autoimmune hepatitis could not be induced in Lewis rats. Intraperitoneal immunization was more effective than intramuscular or subcutaneous injections, and the amount of protein administered above a threshold was of little influence. A single intraperitoneal injection of S-100 in complete Freund's adjuvant …

MalePathologymedicine.medical_specialtyTime FactorsNecrosisT-Lymphocytesmedicine.medical_treatmentFreund's AdjuvantIntraperitoneal injectionAutoimmune hepatitisLymphocyte ActivationAutoimmune DiseasesHepatitisPathogenesisMiceNecrosisSpecies SpecificitymedicineAnimalsAutoimmune diseaseHepatitisMice Inbred BALB CMice Inbred C3HHepatologybusiness.industryLiver cellS100 Proteinsmedicine.diseaseMice Inbred C57BLLiverFreund's adjuvantImmunologyFemalemedicine.symptombusinessHepatology
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Decreased Paraoxonase-2 Expression in Human Carotids During the Progression of Atherosclerosis

2008

Objective— Many gene products involved in oxidation and inflammation are implicated in the pathogenesis of atherosclerosis. We investigated paraoxonase 2 (PON2), 5-lipoxygenase (5-LO), and 5-LO activating protein (FLAP) expression and malondialdehyde (MDA) levels in carotid lesions to assess their involvement in plaque formation. Methods and Results— We measured gene expression and MDA levels in atherosclerotic plaques from 59 patients undergoing carotid endarterectomy, and in plaque-adjacent tissue from 41/59 patients. Twenty-three fetal carotids and 6 mammary arteries were also investigated. Real-time polymerase chain reaction and immunohistochemistry revealed decreased PON2 expression i…

MalePathologymedicine.medical_specialtycarotid plaquemedicine.medical_treatment5-Lipoxygenase-Activating ProteinsInflammationCarotid endarterectomySettore MED/22 - Chirurgia VascolareRisk AssessmentSensitivity and SpecificitySeverity of Illness Indexparaoxonase 2 (PON2PathogenesisCohort StudiesTissue Culture TechniquesatherosclerosiPredictive Value of TestsGene expressionmedicineHumansCarotid StenosisMammary ArteriesAgedoxidative streEndarterectomy CarotidArachidonate 5-Lipoxygenasebusiness.industryVascular diseaseAryldialkylphosphataseMembrane ProteinsMiddle Agedmedicine.diseaseAtherosclerosismedicine.anatomical_structureCarotid ArteriesGene Expression RegulationCirculatory systemDisease ProgressionImmunohistochemistryFemalemedicine.symptomCardiology and Cardiovascular MedicinebusinessCarrier Proteinsfetal carotid and mammary arteryBiomarkersBlood vessel
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The Lipofuscin Content of Nerve Cells of the Inferior Olivary Nucleus in Alzheimer's Disease

1994

Lipofuscin, the age pigment, is of interest in Alzheimer's disease because of its property to accumulate in neurons and because of the hypothesis that Alzheimer's dementia is a kind of premature ageing. The amount of intraneuronal lipofuscin in the inferior olivary nucleus of 20 brains from patients with histologically confirmed Alzheimer's disease according to the CERAD protocol and of 20 controls has been measured microfluorometrically. Patients and controls were matched for age. The amount of lipofuscin in the neurons of the inferior olivary nucleus did not differ significantly between the cases of Alzheimer's disease and the controls. The result is discussed taking the findings of previ…

MalePathologymedicine.medical_specialtygenetic structuresCognitive NeuroscienceDiseaseOlivary NucleusLipofuscinLipofuscinPathogenesisCentral nervous system diseaseDegenerative diseaseAlzheimer DiseasemedicineInferior olivary nucleusHumansDementiaAgedAged 80 and overNeuronsbusiness.industryMiddle Agedmedicine.diseasePsychiatry and Mental healthCase-Control StudiesFemalesense organsGeriatrics and GerontologyAlzheimer's diseasebusinessDementia and Geriatric Cognitive Disorders
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New insights into autoantibody profiles from immune privileged sites in the eye: a glaucoma study.

2011

Glaucoma is a chronic neurodegenerative disease and one of the leading causes of blindness. Autoantibody based immune processes are assumed to be involved in its pathogenesis. However, it is still unclear to what extent autoantibody patterns found in the eye (aqueous humor) are congruent to systemic autoantibodies (blood). Consistency would underline the specificity of known serum antibody markers for glaucoma. In this study we used antigen microarrays to analyze autoantibody reactivities in sera and corresponding aqueous humor samples of primary open-angle glaucoma patients (N=37) and non-glaucomatous controls (N=31). Compared to control subjects several divergent immunoreactivities were i…

MalePathologymedicine.medical_specialtygenetic structuresImmunologyGlaucomaDiseaseCataract ExtractionEyePathogenesisAqueous HumorBehavioral NeuroscienceImmune systemAntigenArtificial IntelligencemedicineHumansAntigensAgedAutoantibodiesbiologyEndocrine and Autonomic Systemsbusiness.industryMicroarray analysis techniquesAutoantibodymedicine.diseaseMicroarray Analysiseye diseasesArea Under CurveImmunoglobulin GImmunologybiology.proteinFemaleOcular Hypertensionsense organsNeural Networks ComputerAntibodybusinessAlgorithmsGlaucoma Open-AngleBrain, behavior, and immunity
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Cytokine gene expression in the tunica albuginea of patients with Peyronie's disease. Pilot study with a control group.

2012

Introduction and objective Cytokines may be involved in the pathogenesis of Peyronie's disease (PD). In this case, biological drugs, acting on specific cytokines, could be adopted in the pharmacological treatment of the disease. Materials and Methods Twenty PD patients and 8 patients affected by congenital recurvatum penis (control group) who underwent corporoplasty were enrolled in the study Histological examination and measurement by Real Time PCR of the expression of the encoding genes for IL-2, IL-4, IL-6, IL-10, IL-13, TGF-β, TNF-α and IFN-γ were performed on the removed tunica albuginea (TA). For the normalization of data GAPDH (glucerldehyde-3fosfatehydrogenase) and GUSB (β-glucuroni…

MalePathologymedicine.medical_specialtymedicine.medical_treatmentPenile IndurationGene ExpressionInflammationPilot ProjectsSettore MED/24 - UrologiaPathogenesisTunica albuginea (ovaries)Young AdultCytokines Induratio Penis Plastica PEYRONIE's disease gene expression Tunica albuginea Biological drugsMedicineHumansbusiness.industryGeneral Medicinemedicine.diseaseHousekeeping genemedicine.anatomical_structureCytokineReal-time polymerase chain reactionCavernous tissueCytokinesPeyronie's diseasemedicine.symptombusinessPenis
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The effect of the pro-inflammatory cytokine tumor necrosis factor-alpha on human joint capsule myofibroblasts.

2009

Introduction Previous studies have shown that the number of myoblastically differentiated fibroblasts known as myofibroblasts (MFs) is significantly increased in stiff joint capsules, indicating their crucial role in the pathogenesis of post-traumatic joint stiffness. Although the mode of MFs' function has been well defined for different diseases associated with tissue fibrosis, the underlying mechanisms of their regulation in the pathogenesis of post-traumatic joint capsule contracture are largely unknown. Methods In this study, we examined the impact of the pro-inflammatory cytokine tumor necrosis factor-alpha (TNF-α) on cellular functions of human joint capsule MFs. MFs were challenged w…

MalePathologymedicine.medical_treatmentFluorescent Antibody TechniqueGene ExpressionDinoprostExtracellular matrixPathogenesisElbow JointImmunology and AllergyCells CulturedReverse Transcriptase Polymerase Chain ReactionAntibodies MonoclonalMiddle AgedImmunohistochemistryExtracellular MatrixCytokinemedicine.anatomical_structureAntirheumatic AgentsCytokinesTumor necrosis factor alphaFemaleHip Jointmedicine.symptomInflammation MediatorsMyofibroblastmusculoskeletal diseasesmedicine.medical_specialtyanimal structuresContractureDiclofenacImmunologyBlotting Westernmacromolecular substancesBiologyCollagen Type IDinoprostoneRheumatologyJoint capsuleResearch articlemedicineHumansAgedCell ProliferationCyclooxygenase 2 InhibitorsTumor Necrosis Factor-alphaProstaglandins FFibroblastsActinsInfliximabCyclooxygenase 2Joint stiffnessContractureJoint CapsuleArthritis researchtherapy
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A case of Candida krusei peritonitis secondary to duodenal perforation due to Candida duodenitis.

2011

A case of a 62-year-old man with Candida krusei peritonitis secondary to duodenal perforation due to Candida duodenitis that was successfully treated with a 14-day course of caspofungin is reported. The potential role of Candida infection in the pathogenesis of peptic ulcers and duodenal perforation is considered. If this role is confirmed, antifungal treatment should be included in the therapeutic armamentarium of peptic disease.

MalePeptic Ulcermedicine.medical_specialtyAntifungal AgentsVeterinary (miscellaneous)PepticPeritonitisPeritonitisApplied Microbiology and BiotechnologyMicrobiologyGastroenterologyPathogenesisEchinocandinsLipopeptideschemistry.chemical_compoundMedical microbiologyDuodenitisCaspofunginInternal medicineCandida kruseimedicineHumansDuodenal DiseasesDuodenal PerforationPeptic diseaseCandidaDuodenal perforationDuodenitibiologyPeritonitibusiness.industrySmokingCandidiasisCandida Peritonitis Duodenal perforation Duodenitis Peptic disease Caspofungin SmokingMiddle Agedmedicine.diseasebiology.organism_classificationdigestive system diseasesTreatment OutcomechemistryIntestinal PerforationCaspofunginbusinessAgronomy and Crop Science
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T Helper Cell Subsets in the Pleural Fluid of Tuberculous Patients Differentiate Patients With Non-Tuberculous Pleural Effusions

2021

BackgroundTuberculous pleural effusion (TPE) is one of the most common forms of extrapulmonary tuberculosis (Tb). Patients with TPE or malignant pleural effusions (MPE) frequently have a similar lymphocytic pleural fluid profile. Since the etiology of PE in various diseases is different, identifying the cellular components may provide diagnostic clues for understanding the pathogenesis.ObjectiveWe determined the frequency of T helper (Th) subtypes in the PEs for differentiation of Tb and non-Tb patients.MethodsThirty patients with TPE, 30 patients with MPE, 14 patients with empyema (EMP), and 14 patients with parapneumonic effusion (PPE) were enrolled between December 2018 and December 2019…

MalePleural effusionLYMPHOCYTESGastroenterologyParapneumonic effusionPathogenesistuberculouspleural effusionT-Lymphocyte Subsets1108 Medical MicrobiologyIMMUNE-RESPONSEImmunology and AllergyMedicineIL-2 receptorOriginal Researchmedicine.diagnostic_testMESOTHELIAL CELLSFOXP3Exudates and TransudatesT-Lymphocytes Helper-InducerTuberculosis PleuralT helper celldifferentiationMiddle Agedmedicine.anatomical_structure1107 ImmunologyfrequencyFemaleTH17 CELLSADENOSINE-DEAMINASELife Sciences & Biomedicinemedicine.medical_specialtyImmunologyDIAGNOSISFlow cytometryDiagnosis DifferentialPredictive Value of TestsInternal medicineT helperHumansLymphocyte CountScience & Technologybusiness.industryRC581-607medicine.diseaseEmpyemaROC CurveFeasibility StudiesImmunologic diseases. AllergybusinessFrontiers in Immunology
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