Search results for "PHARMACOKINETICS"

showing 10 items of 458 documents

Computer simulations for bioequivalence trials: selection of analyte in BCS drugs with first-pass metabolism and two metabolic pathways.

2010

The objective of this work is to use a computer simulation approach to define the most sensitive analyte for in vivo bioequivalence studies of all types of Biopharmaceutics Classification System (BCS) drugs undergoing first-pass hepatic metabolism with two metabolic pathways. A semi-physiological model was developed in NONMEM VI to simulate bioequivalence trials. Four BCS classes (from Class I to IV) of drugs, with three possible saturation scenarios (non-saturation, saturation and saturation of only the major route of metabolism), two (high or low) dose schemes, and six types of pharmaceutical quality for the drug products were simulated. The number of investigated scenarios was 144 (4 × 3…

DrugAnalyteChemistrymedia_common.quotation_subjectMetabolitePharmaceutical SciencePharmacologyBioequivalenceBiopharmaceutics Classification SystemModels BiologicalNONMEMBiopharmaceuticschemistry.chemical_compoundPharmacokineticsPharmaceutical PreparationsTherapeutic EquivalencyArea Under CurveComputer SimulationDrug metabolismAlgorithmsMetabolic Networks and Pathwaysmedia_commonEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
researchProduct

Computer simulations of bioequivalence trials: selection of design and analyte in BCS drugs with first-pass hepatic metabolism: linear kinetics (I).

2008

Modeling and simulation approaches are useful tools to assess the potential outcome of different scenarios in bioequivalence studies. The aim of this study is to propose a new and improved semi-physiological model for bioequivalence trial simulations and apply it for all BCS (Biopharmaceutic Classification System) drug classes with non-saturated first-pass hepatic metabolism. The semi-physiological model was developed in NONMEM VI to simulate bioequivalence trials. Parent drug and metabolite levels for both reference and test were simulated. Eight types of drugs (with high or low permeability and high or low solubility (class I to IV) and high or low intrinsic clearance) were considered in …

DrugAnalytemedia_common.quotation_subjectMetabolitePharmaceutical ScienceBioequivalencePharmacologychemistry.chemical_compoundFirst pass effectPharmacokineticsHumansComputer SimulationPharmacokineticsTissue Distributionmedia_commonDose-Response Relationship DrugChemistryNONMEMLiverNonlinear DynamicsPharmaceutical PreparationsTherapeutic EquivalencyArea Under CurveData Interpretation StatisticalDrug metabolismAlgorithmsEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
researchProduct

Biowaiver monographs for immediate-release solid oral dosage forms: Zidovudine (azidothymidine).

2012

Literature data on the properties of zidovudine relevant to waiver of in vivo bioequivalence (BE) testing requirements for the approval of immediate-release (IR) solid oral dosage forms containing zidovudine alone or in combination with other active pharmaceutical ingredients (APIs) are reviewed. Solubility, dissolution, and permeability data for zidovudine, along with its dosing schedule, therapeutic index and pharmacokinetic properties, and reports related to BE/bioavailability were all taken into consideration. Data for solubility and permeability suggest that zidovudine belongs to Class I according to the Biopharmaceutics Classification System. Also, zidovudine is not a narrow therapeut…

DrugAnti-HIV Agentsmedia_common.quotation_subjectPharmaceutical ScienceExcipientAdministration OralHIV InfectionsPharmacologyBioequivalenceDosage formPermeabilityCell LineExcipientsZidovudineDogsPharmacokineticsBIOEQUIVALÊNCIAMedicineAnimalsHumansmedia_commonActive ingredientbusiness.industryBiopharmaceutics Classification SystemSolubilityTherapeutic EquivalencyCaco-2 CellsbusinessZidovudinemedicine.drugJournal of pharmaceutical sciences
researchProduct

In vitro activity of fluconazole, voriconazole and caspofungin against clinical yeast isolates.

2007

Predicting the clinical outcome of a systemic mycosis is often a difficult task, especially when microbiological resistance is one of the factors contributing to therapeutic failure. Some of these factors are host-related--e.g. immune state, site and severity of infection, poor compliance to therapy--while others are associated with the drug's characteristics--e.g. dosage, type of compound (fungistatic/fungicidal), pharmacokinetic properties and drug-drug interactions. In the last few years, clinicians have been confronted with the problem of selecting the most appropriate antifungal therapy for systemic infections and have highlighted the need for a reliable method to assay the in vitro su…

DrugAntifungal AgentsSystemic mycosismedia_common.quotation_subjectMicrobial Sensitivity TestsBiologyPharmacologyPeptides Cyclicchemistry.chemical_compoundEchinocandinsLipopeptidesPharmacokineticsCaspofunginDrug Resistance FungalmedicineHumansPharmacology (medical)Fluconazolemedia_commonCandidaPharmacologyVoriconazoleTriazolesYeastIn vitroInfectious DiseasesPyrimidinesOncologychemistryVoriconazoleCaspofunginFluconazolemedicine.drugJournal of chemotherapy (Florence, Italy)
researchProduct

Long-Circulating Hyaluronan-Based Nanohydrogels as Carriers of Hydrophobic Drugs

2018

[EN] Nanohydrogels based on natural polymers, such as polysaccharides, are gaining interest as vehicles for therapeutic agents, as they can modify the pharmacokinetics and pharmacodynamics of the carried drugs. In this work, hyaluronan-riboflavin nanohydrogels were tested in vivo in healthy rats highlighting their lack of toxicity, even at high doses, and their different biodistribution with respect to that of native hyaluronan. They were also exploited as carriers of a hydrophobic model drug, the anti-inflammatory piroxicam, that was physically embedded within the nanohydrogels by an autoclave treatment. The nanoformulation was tested by intravenous administration showing an improvement of…

DrugBiodistributionmedia_common.quotation_subjectRiboflavinPharmaceutical Sciencelcsh:RS1-441Pharmacokinetic02 engineering and technologyPharmacologyPiroxicam030226 pharmacology & pharmacyArticleNanohydrogelsLong circulatinglcsh:Pharmacy and materia medica03 medical and health sciencesPiroxicam0302 clinical medicineBiodistributionPharmacokineticsIn vivomedicineHyaluronanbiodistribution; hyaluronan; hydrophobic drugs; nanohydrogels; pharmacokinetic; piroxicam; riboflavinmedia_commonChemistry021001 nanoscience & nanotechnologyHydrophobic drugsToxicityCirculation time0210 nano-technologymedicine.drug
researchProduct

Biowaiver Monographs for Immediate Release Solid Oral Dosage Forms: Piroxicam

2014

ABSTRACT Literature and experimental data relevant to the decision to allow a waiver of in vivo bioequivalence (BE) testing for the approval of immediate release (IR) solid oral dosage forms containing piroxicam in the free acid form are reviewed. Piroxicam solubility and permeability, its therapeutic use and therapeutic index, pharmacokinetic properties, data related to the possibility of excipient interactions and reported BE/bioavailability (BA), and corresponding dissolution data are taken into consideration. The available data suggest that according to the current biopharmaceutics classification system (BCS) and all current guidances, piroxicam would be assigned to BCS Class II. The ex…

DrugChemistry Pharmaceuticalmedia_common.quotation_subjectBiological AvailabilityPharmaceutical ScienceExcipientBioequivalencePharmacologyPiroxicamDosage formBiopharmaceuticsArthritis RheumatoidExcipientsFood-Drug InteractionsPiroxicamPharmacokineticsmedicineAnimalsHumansTissue Distributionmedia_commonChemistryAnti-Inflammatory Agents Non-SteroidalStereoisomerismBiopharmaceutics Classification SystemRatsBioavailabilityIntestinal AbsorptionSolubilityTherapeutic EquivalencyCaco-2 CellsHalf-Lifemedicine.drugJournal of Pharmaceutical Sciences
researchProduct

In-situ forming gel-like depot of a polyaspartamide-polylactide copolymer for once a week administration of Sulpiride

2015

Abstract Objectives An in-situ forming gel-like depot, prepared by using an appropriate polyaspartamide-polylactide graft copolymer, has been employed to release in a sustained way sulpiride. Methods α,β-poly(N-2-hydroxyethyl)-D,L-aspartamide-g-polylactic acid (PHEA-g-PLA) has been used as a polymer component. Its physicochemical properties make possible to dissolve it in N-methyl-2-pyrrolidone, with the obtainment of a solution able to form a gel-like depot once injected into a physiological medium. Cell compatibility of PHEA-g-PLA depot has been investigated, using murine dermal fibroblasts as cell model. 3-(4,5-Dimethylthiazol-2-yl)-5-(3-carboxymethoxyphenyl)-2-(4-sulfophenyl)-2H-tetrazo…

DrugDepotPolymersmedia_common.quotation_subjectChemistry PharmaceuticalPolyesterssulpiridePharmaceutical SciencePharmacologyCell Linechemistry.chemical_compoundDrug Delivery SystemsPharmacokineticsPolylactic acidmedicineFluorescence microscopeCopolymerAnimalsViability assayRats Wistarpolylactic acidgraft copolymermedia_commonPharmacologyin-situ forming depotRatsDrug LiberationchemistryRabbitsSulpiridePeptidesαβ-poly(N-2-hydroxyethyl)-DL-aspartamidemedicine.drug
researchProduct

Hepatocytes--the choice to investigate drug metabolism and toxicity in man: in vitro variability as a reflection of in vivo.

2007

The pharmaceutical industry is committed to marketing safer drugs with fewer side effects, predictable pharmacokinetic properties and quantifiable drug-drug interactions. Drug metabolism is a major determinant of drug clearance and interindividual pharmacokinetic differences, and an indirect determinant of the clinical efficacy and toxicity of drugs. Progressive advances in the knowledge of metabolic routes and enzymes responsible for drug biotransformation have contributed to understanding the great metabolic variations existing in human beings. Phenotypic as well genotypic differences in the expression of the enzymes involved in drug metabolism are the main causes of this variability. How…

DrugDiclofenacDrug-Related Side Effects and Adverse Reactionsmedia_common.quotation_subjectBiologyPharmacologyIn Vitro TechniquesToxicologyModels BiologicalPharmacokineticsCytochrome P-450 Enzyme SystemIn vivoGenetic variationHumansDrug InteractionsPharmacokineticsBiotransformationCells Culturedmedia_commonMolecular StructureAnti-Inflammatory Agents Non-SteroidalCytochrome P450Genetic VariationGeneral MedicineIn vitroPharmaceutical PreparationsToxicityInactivation Metabolicbiology.proteinHepatocytesDrug metabolismMetabolic Networks and PathwaysChemico-biological interactions
researchProduct

Drug biotransformation by human hepatocytes. In vitro/in vivo metabolism by cells from the same donor.

2001

Abstract Background/Aims : Cultured human hepatocytes are considered a close model to human liver. However, the fact that hepatocytes are placed in a microenvironment that differs from that of the cell in the liver raises the question: to what extent does drug metabolism in vitro reflect that of the liver in vivo? This issue was examined by investigating the in vitro and in vivo metabolism of aceclofenac, an analgesic/anti-inflammatory drug. Methods : Hepatocytes isolated from programmed liver biopsies were incubated with aceclofenac, and the metabolites formed were investigated by HPLC. During the course of clinical recovery, patients were given the drug, and the metabolites, largely prese…

DrugDiclofenacHepatologymedia_common.quotation_subjectHydrolysisAnti-Inflammatory Agents Non-SteroidalMetabolismPharmacologyBiologyIn vitromedicine.anatomical_structureBiochemistryPharmacokineticsIn vivoHepatocytemedicineHepatocytesAceclofenacHumansDrug metabolismBiotransformationCells Culturedmedia_commonmedicine.drugJournal of hepatology
researchProduct

Assessing drug-drug interactions through therapeutic drug monitoring when administering oral second-generation antipsychotics.

2016

Second-generation antipsychotics (SGAs) are frequently co-prescribed with drug metabolic inducers and inhibitors. SGA pharmacokinetic drug-drug interactions (DDIs) with inducers and inhibitors have not received enough attention in the literature but can be studied in by using therapeutic drug monitoring (TDM).The limited information available on oral SGA pharmacokinetic DDIs is reviewed. A systematic literature search on the available oral SGA TDM studies is completed. By integrating TDM studies with the information on in vitro metabolism studies, case report/series and prospective studies, a table is provided to manage average SGA patients taking inducers or inhibitors by using TDM and/or …

DrugDrug-Related Side Effects and Adverse Reactionsmedia_common.quotation_subjecttherapeutic drug monitoringAdministration OralPharmacologyToxicology030226 pharmacology & pharmacyDrug interactions03 medical and health sciences0302 clinical medicinePharmacokineticsinhibitorsMedicineHumansProspective cohort studyClozapinemedia_commonLurasidoneinducersPharmacologyRisperidonemedicine.diagnostic_testDose-Response Relationship Drugbusiness.industrysecond-generation antipsychoticsGeneral MedicineDrug interactions; inducers; inhibitors; pharmacokinetics; second-generation antipsychotics; therapeutic drug monitoring030227 psychiatryTherapeutic drug monitoringQuetiapineAntidepressive Agents Second-GenerationDrug Monitoringbusinesspharmacokineticsmedicine.drugAntipsychotic Agents
researchProduct