Search results for "PHOSPHOCREATINE"

showing 10 items of 21 documents

Control of adenine nucleotide metabolism and glycolysis in vertebrate skeletal muscle during exercise.

1996

The turnover of adenosine triphosphate (ATP) in vertebrate skeletal muscle can increase more than a hundredfold during high-intensity exercise, while the content of ATP in muscle may remain virtually unchanged. This requires that the rates of ATP hydrolysis and ATP synthesis are exactly balanced despite large fluctuations in reaction rates. ATP is regenerated initially at the expense of phosphocreatine (PCr) and then mainly through glycolysis from muscle glycogen. The increased ATP turnover in contracting muscle will cause an increase in the contents of adenosine diphosphate (ADP), adenosine monophosphate (AMP) and inorganic phosphate (P(i)), metabolites that are substrates and activators o…

Adenosine monophosphatePhosphocreatinePhysical ExertionBiologyPhosphocreatineCellular and Molecular Neurosciencechemistry.chemical_compoundATP hydrolysismedicineAnimalsHomeostasisGlycolysisMuscle SkeletalMolecular BiologyPharmacologyATP synthaseAdenine NucleotidesSkeletal muscleCell BiologyAdenosine diphosphatemedicine.anatomical_structurechemistryBiochemistryVertebratesbiology.proteinMolecular Medicinemedicine.symptomEnergy MetabolismGlycolysisMuscle contractionExperientia
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Protective action of 1,3-butanediol in cerebral ischemia. A neurologic, histologic, and metabolic study.

1987

1,3-Butanediol (BD) is converted in the body to β-hydroxybutyrate, and previous studies have shown that hyperketonemia had beneficial effects in experimental models of generalized hypoxia. The aim of this study was to determine if BD would reduce brain damage following cerebral ischemia. A transient forebrain ischemia of 30-min duration was induced by the four-vessel occlusion technique in control and BD-treated rats (25 mmol/kg, i.p.; 30 min prior to ischemia). BD treatment led to significant improvement of neurologic deficit during the 72-h recovery period and reduced neuronal damage in the striatum and cortex but not in the CA1 sector of the hippocampus. Evaluation of cerebral energy me…

Blood GlucoseMalemedicine.medical_specialtyIschemiaHydroxybutyratesBlood PressureBrain damageHippocampusPhosphocreatinechemistry.chemical_compoundInternal medicinemedicine13-ButanediolAnimalsEnergy chargeButylene GlycolsCerebral CortexNeurons3-Hydroxybutyric Acidbusiness.industryBrainHypoxia (medical)medicine.diseaseCorpus StriatumRatsEndocrinologyNeurologychemistryIschemic Attack TransientLactic acidosisKetone bodiesNeurology (clinical)medicine.symptomCardiology and Cardiovascular MedicinebusinessEnergy MetabolismJournal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism
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Breakdown of high-energy phosphate compounds and lactate accumulation during short supramaximal exercise.

1987

Muscle ATP, creatine phosphate and lactate, and blood pH and lactate were measured in 7 male sprinters before and after running 40, 60, 80 and 100 m at maximal speed. The sprinters were divided into two groups, group 1 being sprinters who achieved a higher maximal speed (10.07 +/- 0.13 m X s-1) than group 2 (9.75 +/- 0.10 m X s-1), and who also maintained the speed for a longer time. The breakdown of high-energy phosphate stores was significantly greater for group 1 than for group 2 for all distances other than 100 m; the breakdown of creatine phosphate for group 1 was almost the same for 40 m as for 100 m. Muscle and blood lactate began to accumulate during the 40 m exercise. The accumulat…

High-energy phosphateAdultMalePhosphocreatinePhysiologyPhysical ExertionPhysical exerciseCreatinePhosphocreatinePhosphatesRunning03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAnimal scienceAdenosine TriphosphatePhysiology (medical)HumansOrthopedics and Sports MedicineGlycolysisMusclesPublic Health Environmental and Occupational Health030229 sport sciencesGeneral MedicineMetabolismHydrogen-Ion ConcentrationPhosphatechemistryBiochemistryLactatesMaximal exerciseEnergy MetabolismGlycolysis030217 neurology & neurosurgeryEuropean journal of applied physiology and occupational physiology
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Rats bred for low aerobic capacity become promptly fatigued and have slow metabolic recovery after stimulated, maximal muscle contractions.

2012

AIM. Muscular fatigue is a complex phenomenon affected by muscle fiber type and several metabolic and ionic changes within myocytes. Mitochondria are the main determinants of muscle oxidative capacity which is also one determinant of muscle fatigability. By measuring the concentrations of intracellular stores of high-energy phosphates it is possible to estimate the energy production efficiency and metabolic recovery of the muscle. Low intrinsic aerobic capacity is known to be associated with reduced mitochondrial function. Whether low intrinsic aerobic capacity also results in slower metabolic recovery of skeletal muscle is not known. Here we studied the influence of intrinsic aerobic capac…

Magnetic Resonance SpectroscopyAnatomy and PhysiologyPhosphocreatineEvolutionary Selectionlcsh:MedicineIsometric exerciseBreedingmetaboliset sairaudetBiochemistrychemistry.chemical_compound0302 clinical medicineTriceps surae muscleMyocyteta315lcsh:ScienceMusculoskeletal System0303 health sciencesMultidisciplinarycomplex diseaseMuscle BiochemistryAnatomyAnimal ModelsHydrogen-Ion Concentrationmedicine.anatomical_structureaerobinen kapasiteettiMuscle FatigueMuscleaerobinen suorituskykymedicine.symptomMuscle contractionMuscle ContractionResearch Articlemedicine.medical_specialtyEvolutionary ProcessessupistusominaisuudetBioenergeticsPhosphocreatinePhosphates03 medical and health sciencesModel OrganismsInternal medicinePhysical Conditioning AnimalmedicineGeneticsAnimalsskeletal muscleAdaptationBiologyAerobic capacity030304 developmental biologyEvolutionary BiologyMuscle fatiguelcsh:RSkeletal muscleElectric StimulationRatsraajalihasEndocrinologyMetabolismcontractile propertieschemistryRatlcsh:QEnergy MetabolismPhysiological ProcessesAnimal Genetics030217 neurology & neurosurgeryPLoS ONE
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Comparison of the effects of valproate, ethosuximide, phenytoin, and pentobarbital on cerebral energy metabolism in the rat.

1987

The acute effects of valproate (200 and 400 mg/kg), ethosuximide (200 and 400 mg/kg), phenytoin (25 and 50 mg/kg), and pentobarbital (30 and 60 mg/kg) on cerebral energy metabolism of rats were studied by measuring the cerebral content of energy metabolites and by evaluating the rate of metabolite utilization following decapitation. The treatments did not affect the levels of phosphocreatine (PCr), ATP, ADP, and AMP, but did enhance the glycogen or glucose stores. Pentobarbital induced a decrease in lactate, whereas valproate led to a decrease in pyruvate and an increase in lactate. Calculation of the metabolite fluxes after decapitation showed that all treatments delayed the rate of ATP ut…

MalePentobarbitalmedicine.medical_specialtyMetabolitemedicine.medical_treatmentPhosphocreatinechemistry.chemical_compoundAdenine nucleotideInternal medicinemedicineAnimalsGlycolysisPentobarbitalEpilepsyGlycogenAdenine NucleotidesValproic AcidBrainRats Inbred StrainsRatsEthosuximideAnticonvulsantEndocrinologyNeurologychemistryPhenytoinEthosuximideNeurology (clinical)Energy Metabolismmedicine.drugEpilepsia
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Growth hormone potentiates thyroid hormone effects on post-exercise phosphocreatine recovery in skeletal muscle.

2012

International audience; OBJECTIVE: The aim of the study was to determine the respective impact of thyroxine and growth hormone on in vivo skeletal mitochondrial function assessed via post exercise phosphocreatine recovery. DESIGN: The hind leg muscles of 32 hypophysectomized rats were investigated using (31)P nuclear magnetic resonance spectroscopy at rest and during the recovery period following a non tetanic stimulation of the sciatic nerve. Each rat was supplemented with hydrocortisone and was randomly assigned to one of the 4 groups: the group Hx was maintained in hypopituitarism., the group HxT was treated with 1 μg/100g/day of thyroxine (T4), the group HxG with 0.2 IU/kg/day of recomb…

MalePhosphocreatineThyroid hormonesEndocrinology Diabetes and MetabolismMESH: Random AllocationThyroid GlandSkeletal muscleHypopituitarismMESH: Physical Conditioning AnimalMESH: Drug SynergismNuclear magnetic resonancechemistry.chemical_compoundRandom Allocation0302 clinical medicineEndocrinologyMESH: Human Growth HormoneMESH: AnimalsMESH : Muscle Skeletal0303 health sciencesMESH: Muscle Skeletal[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingMESH : RatsHuman Growth HormoneThyroidDrug Synergismmedicine.anatomical_structuremedicine.drugmedicine.medical_specialtyMESH : Drug SynergismMESH: RatsMESH : MaleSomatotropin030209 endocrinology & metabolismMESH: PhosphocreatinePhosphocreatineMESH : Random Allocation03 medical and health sciencesIn vivoInternal medicine[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyPhysical Conditioning AnimalMESH : Thyroxinemedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingAnimalsHumansMESH : PhosphocreatineMESH : Human Growth HormoneMitochondrionMESH : Physical Conditioning AnimalMuscle Skeletal030304 developmental biologyHydrocortisoneMESH: HumansMESH : HumansSkeletal muscleMESH : Thyroid GlandMESH: Thyroxinemedicine.diseaseMESH: MaleMESH: Thyroid GlandRatsThyroxineEndocrinologychemistryRatMESH : AnimalsTetanic stimulation[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyHormoneGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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The Etiology of Muscle Fatigue Differs between Two Electrical Stimulation Protocols

2016

International audience; Purpose: This study aimed at investigating the mechanisms involved in the force reduction induced by two electrical stimulation (ES) protocols that were designed to activate motor units differently. Methods: The triceps surae of 11 healthy subjects (8 men; age, similar to 28 yr) was activated using ES applied over the tibial nerve. Two ES protocols (conventional [CONV]: 20 Hz, 0.05 ms vs wide-pulse high-frequency [WPHF]: 80 Hz, 1 ms) were performed and involved 40 trains (6 s on-6 s off) delivered at an intensity (I-ES) evoking 20% of maximal voluntary contraction. To analyze the mechanical properties of the motor units activated at I-ES, force-frequency relation was…

MaleStimulationElectromyographytorque decreaseplateau-like behaviorh-reflexchemistry.chemical_compound0302 clinical medicinemotor unit recruitmentOrthopedics and Sports Medicinehuman motoneuronsquadriceps femorismedicine.diagnostic_testChemistryPulse (signal processing)Anatomystimulation parametersHealthy VolunteersMuscle Fatiguetriceps surae[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Femalemedicine.symptomMuscle ContractionMuscle contractionAdultRecruitment Neurophysiologicalmedicine.medical_specialtyhuman skeletal-musclePhysical Therapy Sports Therapy and RehabilitationnervePhosphocreatineYoung Adult03 medical and health sciencesInternal medicineexcitabilitymedicineHumansMuscle SkeletalMuscle fatigueElectromyography030229 sport sciencesmagnetic resonance spectroscopyElectric StimulationEndocrinologyevoked contractions[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Motor unit recruitmentTibial NerveH-reflex030217 neurology & neurosurgeryMedicine & Science in Sports & Exercise
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Influence of therapeutic and toxic doses of neuroleptics and antidepressants on energy metabolism of the isolated perfused rat brain.

1973

The isolated perfused rat brain was used for a comparative study of the effects of promazine, imipramine, monodesmethyl promazine and desipramine on cerebral energy metabolism. After perfusion for 30 min or 1 h the brain levels of the following substrates and metabolites were estimated: P-creatine, creatine, ATP, ADP, AMP, glycogen, glucose, glucose-6-P, fructose diphosphate, dihydroxyacetone-P, pyruvate, lactate, α-ketoglutarate, and ammonia. Drug concentrations of 5·10−6 M and 10−5 M in the perfusion medium caused a significant decrease of glucose-6-P alone. When the drug concentration was raised to a toxic range (10−4 M), reflected in the EEG by the pattern of secondary discharges, an ac…

Malemedicine.medical_specialtyImipraminePhosphocreatineBiologyPharmacologyCreatineImipramineAcetonechemistry.chemical_compoundOrganophosphorus CompoundsAmmoniaInternal medicineDesipramineTriosesmedicineAnimalsGlycolysisPyruvatesPromazinePromazinePharmacologyGlycogenDose-Response Relationship DrugDesipramineFructosephosphatesGlucosephosphatesBrainFructoseElectroencephalographyGeneral MedicineRibonucleotidesCreatineAntidepressive AgentsRatsPerfusionEndocrinologyGlucoseTranquilizing AgentschemistryLactatesKetoglutaric AcidsEnergy MetabolismPerfusionGlycolysismedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Suitability of the isolated perfused rat brain for studying effects on cerebral metabolism

1972

The concentrations of P-creatine, creatine, ATP, ADP, AMP, glycogen, glucose, glucose-6-P, fructose diphosphate, dihydroxyacetone-P, α-glycero-P, lactate and pyruvate were measured in the isolated perfused rat brain as well as in rat brain in vivo. Similar levels were observed in the isolated brain and in intact animals, and the values measured were in good accordance with those described in the literature. Only the pyruvate and lactate content were significantly higher in the isolated brain but the lactate/pyruvate ratio remained unchanged. An anesthetic or ischemia caused just the same effects on energy metabolism of the isolated rat brain as described for intact animals. Thus, 1.5 mM phe…

Malemedicine.medical_specialtyPhosphocreatineIschemiaIn Vitro TechniquesBiologyCreatinechemistry.chemical_compoundAdenosine TriphosphateIn vivoInternal medicinemedicineAnimalsGlycolysisPyruvatesBrain ChemistryPharmacologyGlycogenMonosaccharidesBrainElectroencephalographyFructoseGeneral MedicineRibonucleotidesIsolated brainCreatinemedicine.diseaseAdenosine MonophosphateRatsAdenosine DiphosphatePerfusionEndocrinologychemistrySpectrophotometryPhenobarbitalLactatesBasal MetabolismPerfusionGlycogenNaunyn-Schmiedeberg's Archives of Pharmacology
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Exercise and recovery in frog muscle: metabolism of PCr, adenine nucleotides, and related compounds

1996

The effects of exercise (swimming), fatigue, and recovery on the intracellular pH (pHi), energy-rich phosphates, and related metabolites were studied in the gastrocnemius muscle of common frogs (Rana temporaria) at 20 degrees C. Exercise caused a rapid decrease in the content of phosphocreatine (PCr) and a corresponding increase in that of Pi. The ATP level remained virtually constant for 1 min; its precipitous decrease during the following minute was associated with a rise in the contents of inosine 5'-monophosphate (IMP) and NH4+, indicating a marked activation of AMP deaminase. Five minutes of swimming caused severe fatigue, which was correlated with decreases in muscle PCr (-85%), ATP …

Malemedicine.medical_specialtyPhosphocreatinePhysiologyIntracellular pHPhysical ExertionRana temporariaPhysical exerciseBiologyPhosphocreatineGastrocnemius musclechemistry.chemical_compoundAdenosine TriphosphateInosine MonophosphateAdenine nucleotidePhysiology (medical)Internal medicinemedicineAnimalsInosineAdenine NucleotidesMusclesPhosphorusAMP deaminaseMetabolismHydrogen-Ion ConcentrationQuaternary Ammonium CompoundsEndocrinologychemistrymedicine.drugAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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