Search results for "PLATELET ACTIVATION"

showing 10 items of 121 documents

Stem cell-secreted factor therapy regenerates the ovarian niche and rescues follicles.

2021

Background Ovarian senescence is a normal age-associated phenomenon, but increasingly younger women are affected by diminished ovarian reserves or premature ovarian insufficiency. There is an urgent need for developing therapies to improve ovarian function in these patients. In this context, previous studies suggest that stem cell–secreted factors could have regenerative properties in the ovaries. Objective This study aimed to test the ability of various human plasma sources, enriched in stem cell–secreted factors, and the mechanisms behind their regenerative properties, to repair ovarian damage and to promote follicular development. Study Design In the first phase, the effects of human pla…

Ovarian CortexOvaryBone Marrow CellsPrimary Ovarian InsufficiencyPremature ovarian insufficiencyHematopoietic Cell Growth Factors03 medical and health sciencesMicePlasma0302 clinical medicineOvarian FollicleMice Inbred NODGranulocyte Colony-Stimulating FactorMedicineAnimalsHumans030212 general & internal medicinePlatelet activationOvarian ReserveStem Cell Factor030219 obstetrics & reproductive medicinebusiness.industryCell growthStem CellsOvaryInfant NewbornObstetrics and GynecologyBone Marrow Stem CellCell cycleFetal BloodDisease Models Animalmedicine.anatomical_structureCancer researchHeterograftsFemaleStem cellbusinessAmerican journal of obstetrics and gynecology
researchProduct

Hemostatic Alterations in Peripheral Arteriopathies

1985

The most common cause of obliterative disease in limb arteries is slowly progressive arteriosclerosis which is eventually superimposed by thrombosis.

Pathologymedicine.medical_specialtybusiness.industryMedicinePlatelet activationArteriosclerosisDiseasebusinessmedicine.diseaseThrombosisPeripheral
researchProduct

Serum brain-derived neurotrophic factor and platelet activation evaluated by soluble P-selectin and soluble CD-40-ligand in patients with acute myoca…

2009

Little is known about the role of neurotrophins (NT) under adult vascular homeostasis in normal and pathological conditions. The NT family, including nerve growth factor and brain-derived neurotrophic factor (BDNF) are expressed in atherosclerotic vessels. Previous studies demonstrated that plasma BDNF levels were increased in the coronary circulation in patients with unstable angina. However, the role of BDNF during the onset and evolution of unstable angina remains to be elucidated. The objective of this study was to evaluate the relationship between BDNF, functional parameters and biological markers associated with inflammatory processes and platelet activation. BDNF serum levels were as…

PharmacologyBrain-derived neurotrophic factor0303 health sciencesmedicine.medical_specialtyP-selectinUnstable anginabusiness.industry030204 cardiovascular system & hematologymedicine.disease03 medical and health sciencesCoronary circulation0302 clinical medicineNerve growth factorEndocrinologymedicine.anatomical_structureNeurotrophic factorsInternal medicineImmunologymedicinePharmacology (medical)Platelet activationMyocardial infarctionbusiness030304 developmental biologyFundamental & Clinical Pharmacology
researchProduct

The effects of the nitric oxide donors molsidomine and SIN-1 on human polymorphonuclear leucocyte functionin vitro andex vivo

1992

The nitrovasodilator and nitric oxide donor molsidomine and its metabolite SIN-I dilate vascular smooth muscle and inhibit platelet activation by increasing intracellular concentrations of cyclic GMP We have therefore studied the effects of molsidomine and SIN-I on isolated human polymorphonuclear leucocytes (PMN)in vitro andex vivo. In vitro molsidomine dose-dependently reducedβ-glucuronidase release and the generation of superoxide anions from non-activated and from FMLP- or PAF-stimulated human PMNs. SIN-1 was equally effective in reducing (β-glucuronidase release and totally inhibited oxygen radical generation at a concentration of 580 μmol · l−1. In a double-blind, placebo-controlled, …

PharmacologyMolsidomineChemistrySuperoxideMetabolitehemic and immune systemsGeneral MedicinePharmacologyNitric oxidechemistry.chemical_compoundBiochemistryIn vivomedicinePharmacology (medical)Platelet activationNitrovasodilatorEx vivomedicine.drugEuropean Journal of Clinical Pharmacology
researchProduct

The sGC stimulator riociguat inhibits platelet function in washed platelets but not in whole blood

2015

Background and Purpose Stimulation of soluble guanylyl cyclase (sGC) is a valuable therapeutic strategy for the treatment of several cardiovascular diseases. The sGC stimulator riociguat has been approved for the treatment of two forms of pulmonary hypertension. Platelets contain large amounts of sGC and play a key role in the regulation of haemostasis. Therefore, we investigated the effects of riociguat on platelet function. Experimental Approach The effect of riociguat treatment on human platelet activation and aggregation was investigated. The sGC-specific effects of riociguat were determined by comparing wild-type and platelet-specific sGC-knockout mice. Key Results Riociguat induced cG…

Pharmacologymedicine.medical_specialtyChemistrymedicine.diseaseRiociguatPulmonary hypertensionEndocrinologyInternal medicinecardiovascular systemmedicinePlateletPlatelet activationSoluble guanylyl cyclaseReceptorIloprostmedicine.drugWhole bloodBritish Journal of Pharmacology
researchProduct

Platelet function testing in pigs using the Multiplate® Analyzer.

2019

PLOS ONE 14(8), e0222010 (2019). doi:10.1371/journal.pone.0222010

Platelet AggregationSwinePhysiology030204 cardiovascular system & hematologyPlatelet inhibitionPharmacologyAntiplatelet Therapy0302 clinical medicineOral administrationAnimal CellsPig ModelsMedicine and Health SciencesMedicinePlateletOral AdministrationRoutes of AdministrationMammalsMultidisciplinaryPharmaceuticsQREukaryotaHematologyAnimal ModelsClopidogrelBody FluidsBloodExperimental Organism SystemsVertebratesMedicineAnatomyCellular Typesmedicine.drugResearch ArticleBlood PlateletsPlateletsPlatelet Function TestsScience610Research and Analysis Methods03 medical and health sciencesAnimal modelDrug TherapyAnimalsddc:610Platelet activationBlood CoagulationApplication methodsPharmacologyBlood CellsAspirinbusiness.industryOrganismsBiology and Life Sciences030208 emergency & critical care medicineCell BiologyPlatelet ActivationReference valuesAmniotesPurinergic P2Y Receptor AntagonistsAnimal StudiesbusinessPlatelet Aggregation InhibitorsPloS one
researchProduct

Insights in ChAdOx1 nCoV-19 vaccine-induced immune thrombotic thrombocytopenia

2021

Abstract SARS-CoV-2 vaccine ChAdOx1 nCoV-19 (AstraZeneca) causes a thromboembolic complication termed vaccine-induced immune thrombotic thrombocytopenia (VITT). Using biophysical techniques, mouse models, and analysis of VITT patient samples, we identified determinants of this vaccine-induced adverse reaction. Super-resolution microscopy visualized vaccine components forming antigenic complexes with platelet factor 4 (PF4) on platelet surfaces to which anti-PF4 antibodies obtained from VITT patients bound. PF4/vaccine complex formation was charge-driven and increased by addition of DNA. Proteomics identified substantial amounts of virus production-derived T-REx HEK293 proteins in the ethyle…

ProteomicsAntigen-Antibody ComplexPlatelet Factor 4Extracellular TrapsBiochemistryEpitopesMiceSinus Thrombosis IntracranialMedicinePlateletCell Line TransformedMicroscopybiologyHematologymedicine.anatomical_structureSpike Glycoprotein CoronavirusAntibodyDrug ContaminationVirus CultivationGenetic VectorsImmunologyAdenoviridaeProinflammatory cytokineImaging Three-DimensionalImmune systemAntigenChAdOx1 nCoV-19AnimalsHumansPlatelet activationB cellAutoantibodiesInflammationPurpura Thrombocytopenic IdiopathicSARS-CoV-2business.industryCOVID-19Cell BiologyPlatelet ActivationPlatelets and ThrombopoiesisDynamic Light ScatteringHEK293 CellsImmunoglobulin GImmunologybiology.proteinCapsid ProteinsbusinessCapillary Leak SyndromePlatelet factor 4Extravasation of Diagnostic and Therapeutic MaterialsBlood
researchProduct

Selective estrogen receptor modulators and risk for coronary heart disease.

2007

Coronary heart disease (CHD) is the leading cause of death in women in most countries. Atherosclerosis is the main biological process determining CHD. Clinical data support the notion that CHD is sensitive to estrogens, but debate exists concerning the effects of the hormone on atherosclerosis and its complications. Selective estrogen receptor modulators (SERMs) are compounds capable of binding the estrogen receptor to induce a functional profile distinct from estrogens. The possibility that SERMs may shift the estrogenic balance on cardiovascular risk towards a more beneficial profile has generated interest in recent years. There is considerable information on the effects of SERMs on disti…

Selective Estrogen Receptor Modulatorsmedicine.medical_specialtyEndotheliumEstrogen receptorCoronary DiseaseDiseaseCoronary Artery DiseaseRisk FactorsInternal medicinemedicineAnimalsHumansRaloxifenePlatelet activationCause of deathHemostasisbusiness.industryObstetrics and GynecologyEstrogensGeneral MedicineLipidsEndocrinologymedicine.anatomical_structureSelective estrogen receptor modulatorEndothelium Vascularbusinesshormones hormone substitutes and hormone antagonistsHormonemedicine.drugClimacteric : the journal of the International Menopause Society
researchProduct

Alteration of serotonin system by polychlorinated biphenyls exposure.

2011

Although commercial production of polychlorinated biphenyls (PCBs) was banned in 1979, PCBs continue to be an environmental and health concern due to their high bioaccumulation and slow degradation rates. In fact, PCBs are still present in our food supply (fish, meat, and dairy products). In laboratory animals, exposure to single PCB congener or to mixtures of different congeners induces a variety of physiological alterations. PCBs cross the placenta and even exposure at low level is harmful for the foetus by leading to neurodevelopment alterations. Serotonin system which regulates many physiological functions from platelet activation to high cerebral processes and neurodevelopment is one o…

SerotoninChemistryNeurotoxicityfood and beveragesPhysiologyBrainCell BiologyEnvironmental exposureEnvironmental Exposuremedicine.diseasePolychlorinated BiphenylsCellular and Molecular NeuroscienceCongenerBioaccumulationEnvironmental chemistryToxicitymedicineAnimalsHumansSerotoninPlatelet activation5-HT receptorNeurochemistry international
researchProduct

Cardiovascular effects of air pollution

2017

Air pollution is composed of particulate matter (PM) and gaseous pollutants, such as nitrogen dioxide and ozone. PM is classified according to size into coarse particles (PM 10), fine particles (PM 2.5) and ultrafine particles. We aim to provide an original review of the scientific evidence from epidemiological and experimental studies examining the cardiovascular effects of outdoor air pollution. Pooled epidemiological studies reported that a 10 μg/m 3 increase in long-term exposure to PM 2.5 was associated with an 11% increase in cardiovascular mortality. Increased cardiovascular mortality was also related to long-term and short-term exposure to nitrogen dioxide. Exposure to air pollution…

Time FactorsCardiovascular mortalityOzoneair pollutionAir pollution030204 cardiovascular system & hematology010501 environmental sciencesmedicine.disease_causeCardiovascular SystemRisk Assessment01 natural sciencesArticleToxicology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsEnvironmental healthUltrafine particleAnimalsHumansoxidative stressMedicineNitrogen dioxidePlatelet activationBlood CoagulationAir quality index0105 earth and related environmental sciencesparticulate matterPollutantAir Pollutantsbusiness.industryEnvironmental ExposureGeneral MedicineParticulatesPrognosisPlaque Atheroscleroticmyocardial infarctionchemistryCardiovascular DiseasesReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessArchives of Cardiovascular Diseases
researchProduct