Search results for "POF"

showing 10 items of 206 documents

Prenatal diagnosis of infantile neuronal ceroid-lipofuscinosis: a combined electron microscopic and molecular genetic approach.

1995

Based on two unrelated index patients afflicted with INCL, fetal chorion tissues were studied from subsequent pregnancies of the two respective mothers resulting in the prenatal diagnosis of INCL in two of the three pregnancies. Documentation of INCL was based on electron microscopy and DNA studies of the biopsied chorion tissue, later confirmed in the two affected fetuses after termination of their pregnancies by demonstrating INCL-specific lipopigments in post-mortem tissues, in the liver of both aborted fetuses and, additionally, in spleen and skeletal muscle of one of the affected fetuses. The autolysis of the aborted tissues, however, precluded a systematic documentation of all affecte…

MalePathologymedicine.medical_specialtyCell typeBiopsyInfantile neuronal ceroid lipofuscinosisSpleenPrenatal diagnosisBiologyConsanguinityDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesPregnancyPrenatal DiagnosisBiopsymedicineHumansreproductive and urinary physiologyFetusmedicine.diagnostic_testAborted FetusSkeletal muscleInfantAbortion InducedGeneral MedicineChorionDNAmedicine.diseasePedigreeMicroscopy Electronmedicine.anatomical_structureLiverembryonic structuresPediatrics Perinatology and Child HealthFemaleNeurology (clinical)Braindevelopment
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Probable exclusion of juvenile neuronal ceroid lipofuscinosis in a fetus at risk: an interim report.

1989

In a family with two children affected by juvenile neuronal ceroid lipofuscinosis (JNCL) an attempt was made at the prenatal diagnosis of the disorder. The following tissues from the fetus at risk were investigated by electron microscopy and were found to be free of fingerprint profiles and curvilinear bodies, typical for JNCL: uncultivated amniotic fluid cells, lymphocytes isolated from fetal blood, and fetal skin biopsy specimens. The child was born at the 34th week of gestation and was clinically normal at the age of 15 months. Postnatally, lymphocytes (isolated at the age of 6 and 15 months) and skin tissue (taken at the age of 15 months) were found to be morphologically normal. It is h…

MalePathologymedicine.medical_specialtyFetus at riskBiopsyPrenatal diagnosisBiologyNeuronal Ceroid-LipofuscinosesPregnancyRisk FactorsBiopsymedicineHumansGenetics (clinical)SkinPregnancyFetusmedicine.diagnostic_testObstetrics and GynecologyInfantmedicine.diseaseFetal DiseasesAmniocentesisAmniocentesisGestationNeuronal ceroid lipofuscinosisFemalePrenatal diagnosis
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Electron microscopic observation of tonsillar tissue as a diagnostic aid in early juvenile neuronal ceroid-lipofuscinosis.

1987

An electron microscopic observation in a tonsil of a patient with early juvenile neuronal ceroid-lipofuscinosis (NCL) demonstrated characteristic lipopigments in lymphocytes, i.e., fingerprint profiles (FPP) and granular matrixes. While numerous FPP, curvilinear profiles (CLP) and granular matrixes were found in reticulo-endothelial and plasma cells, tonsillar lymphocytes contained only FPP and granular matrixes as seen in circulating lymphocytes. These findings suggest that a tonsil biopsy, an easy and simple technique, may provide more reliable information than a skin biopsy not only for the diagnosis of but also for differentiating the clinical forms of childhood NCL.

MalePathologymedicine.medical_specialtyLymphocytePalatine TonsilInfantile neuronal ceroid lipofuscinosisBiologyLipofuscinDiagnosis Differentialstomatognathic systemDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesBiopsymedicineHumansLymphocytesChildmedicine.diagnostic_testGeneral MedicineMononuclear phagocyte systemmedicine.diseaseMicroscopy Electronmedicine.anatomical_structureTonsilPediatrics Perinatology and Child HealthSkin biopsyUltrastructureNeuronal ceroid lipofuscinosisNeurology (clinical)Braindevelopment
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The Lipofuscin Content of Nerve Cells of the Inferior Olivary Nucleus in Alzheimer's Disease

1994

Lipofuscin, the age pigment, is of interest in Alzheimer's disease because of its property to accumulate in neurons and because of the hypothesis that Alzheimer's dementia is a kind of premature ageing. The amount of intraneuronal lipofuscin in the inferior olivary nucleus of 20 brains from patients with histologically confirmed Alzheimer's disease according to the CERAD protocol and of 20 controls has been measured microfluorometrically. Patients and controls were matched for age. The amount of lipofuscin in the neurons of the inferior olivary nucleus did not differ significantly between the cases of Alzheimer's disease and the controls. The result is discussed taking the findings of previ…

MalePathologymedicine.medical_specialtygenetic structuresCognitive NeuroscienceDiseaseOlivary NucleusLipofuscinLipofuscinPathogenesisCentral nervous system diseaseDegenerative diseaseAlzheimer DiseasemedicineInferior olivary nucleusHumansDementiaAgedAged 80 and overNeuronsbusiness.industryMiddle Agedmedicine.diseasePsychiatry and Mental healthCase-Control StudiesFemalesense organsGeriatrics and GerontologyAlzheimer's diseasebusinessDementia and Geriatric Cognitive Disorders
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Ultrastructural studies of the retina in infantile neuronal ceroid-lipofuscinosis.

1988

A 9-year-old boy who had died of infantile neuronal ceroid-lipofuscinosis had experienced retina-derived visual failure. Ophthalmologically and morphologically, his retina was severely atrophic and scarred by a dense fibrillary gliosis while photoreceptor cells had completely disappeared, cells of the bipolar layer had decreased in number and had become atrophic beyond cytologic recognition. Retinal pigment epithelial cells had undergone either atrophy or proliferation. Disease-specific granular lipopigments had accumulated in perikarya and processes of remaining cells and were infrequently associated with melanin within huge melanolipofuscin bodies and RPE cells of sessile and migrating na…

MalePathologymedicine.medical_specialtygenetic structuresInfantile neuronal ceroid lipofuscinosisCytoplasmic GranulesRetinaLipofuscinMelaninchemistry.chemical_compoundAtrophyNeuronal Ceroid-LipofuscinosesCytologymedicineHumansChildMelaninsRetinaMembranesbusiness.industryRetinalGeneral MedicinePigments Biologicalmedicine.diseaseLipidseye diseasesOphthalmologymedicine.anatomical_structurechemistryUltrastructuresense organsbusinessRetinopathyRetina (Philadelphia, Pa.)
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Further delineation of eye manifestations in homozygous 15q13.3 microdeletions including TRPM1: a differential diagnosis of ceroid lipofuscinosis.

2014

The 15q13.3 heterozygous microdeletion is a fairly common microdeletion syndrome with marked clinical variability and incomplete penetrance. The average size of the deletion, which comprises six genes including CHRNA7, is 1.5 Mb. CHRNA7 has been identified as the gene responsible for the neurological phenotype in this microdeletion syndrome. Only seven patients with a homozygous microdeletion that includes at least CHRNA7, and is inherited from both parents have been described in the literature. The aim of this study was to further describe the distinctive eye manifestations from the analysis in the three French patients diagnosed with the classical 1.5 Mb homozygous microdeletion. Patients…

MalePathologymedicine.medical_specialtygenetic structuresalpha7 Nicotinic Acetylcholine ReceptorEncephalopathyTRPM Cation ChannelsChromosome DisordersBiologyBlindnessEyePupilNeuronal Ceroid-LipofuscinosesNight BlindnessSeizuresIntellectual DisabilityRetinal DystrophiesGeneticsmedicineElectroretinographyMyopiaHumansEye AbnormalitiesChildGenetics (clinical)TRPM1Genetic Association StudiesCongenital stationary night blindnessGeneticsChromosomes Human Pair 15DystrophyEye Diseases HereditaryGenetic Diseases X-LinkedOptic NerveMicrodeletion syndromemedicine.diseasePenetranceChild PreschoolFemalesense organsDifferential diagnosisChromosome DeletionAmerican journal of medical genetics. Part A
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A case of Candida krusei peritonitis secondary to duodenal perforation due to Candida duodenitis.

2011

A case of a 62-year-old man with Candida krusei peritonitis secondary to duodenal perforation due to Candida duodenitis that was successfully treated with a 14-day course of caspofungin is reported. The potential role of Candida infection in the pathogenesis of peptic ulcers and duodenal perforation is considered. If this role is confirmed, antifungal treatment should be included in the therapeutic armamentarium of peptic disease.

MalePeptic Ulcermedicine.medical_specialtyAntifungal AgentsVeterinary (miscellaneous)PepticPeritonitisPeritonitisApplied Microbiology and BiotechnologyMicrobiologyGastroenterologyPathogenesisEchinocandinsLipopeptideschemistry.chemical_compoundMedical microbiologyDuodenitisCaspofunginInternal medicineCandida kruseimedicineHumansDuodenal DiseasesDuodenal PerforationPeptic diseaseCandidaDuodenal perforationDuodenitibiologyPeritonitibusiness.industrySmokingCandidiasisCandida Peritonitis Duodenal perforation Duodenitis Peptic disease Caspofungin SmokingMiddle Agedmedicine.diseasebiology.organism_classificationdigestive system diseasesTreatment OutcomechemistryIntestinal PerforationCaspofunginbusinessAgronomy and Crop Science
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Posaconazole against Candida glabrata Isolates with Various Susceptibilities to Fluconazole

2008

ABSTRACT We investigated the in vitro activities of posaconazole (POS), fluconazole (FLC), amphotericin B (AMB), and caspofungin (CAS) against four clinical isolates of Candida glabrata with various susceptibilities to FLC (FLC MICs ranging from 1.0 to >64 μg/ml). POS MICs ranged from ≤0.03 to 0.5 μg/ml; AMB MICs ranged from 0.25 to 2.0 μg/ml, while CAS MICs ranged from 0.03 to 0.25 μg/ml. When FLC MICs increased, so did POS MICs, although we did not observe any isolate with a POS MIC greater than 0.5 μg/ml. Time-kill experiments showed that POS, FLC, and CAS were fungistatic against all isolates, while AMB at eight times the MIC was fungicidal against three out of four isolates of C. gl…

MalePosaconazoleAntifungal AgentsCandida glabrataMicrobial Sensitivity TestsDrug resistanceBiologyKidneyMicrobiologyMicechemistry.chemical_compoundDrug Resistance Fungalhemic and lymphatic diseasesAmphotericin BmedicineAnimalsHumansExperimental TherapeuticsPharmacology (medical)FluconazolePharmacologyCandida glabrataExperimental modelCandidiasisFungi imperfectiTriazolesbiology.organism_classificationTreatment OutcomeInfectious DiseaseschemistryCaspofunginFluconazolemedicine.drugAntimicrobial Agents and Chemotherapy
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Retinal oxidation, apoptosis and age- and sex-differences in the mnd mutant mouse, a model of neuronal ceroid lipofuscinosis

2004

Retinal degeneration is an early and progressive event in many forms of neuronal ceroid lipofuscinoses (NCLs), a heterogeneous group of neurodegenerative disorders with unknown pathogenesis. We here used the mutant motor neuron degeneration (mnd) mouse, a late-infantile NCL variant, to investigate the retinal oxidative state and apoptotic cell death as a function of age and sex. Total superoxide dismutase (SOD) activities and thiobarbituric acid-reactive substance (TBARS) levels revealed progressive increases in retinal oxyradicals and lipid peroxides of mnd mice of both sexes. Female mnd retinas showed a higher oxidation rate and consistently exhibited the 4-hydroxy-2-nonenal (4-HNE)-adduc…

MaleRetinal degenerationPathologymedicine.medical_specialtyApoptosisBiologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesRetinaMiceMice Neurologic Mutantschemistry.chemical_compoundSex FactorsNeuronal Ceroid-LipofuscinosesIn Situ Nick-End LabelingmedicineAnimalsOuter nuclear layerMolecular BiologyAldehydesRetinaTUNEL assayLipid peroxideCaspase 3Superoxide DismutaseGeneral NeuroscienceRetinal DegenerationRetinalmedicine.diseaseImmunohistochemistryEnzyme ActivationMice Inbred C57BLDisease Models AnimalOxidative Stressmedicine.anatomical_structureBiochemistrychemistryCaspasesFemaleNeuronal ceroid lipofuscinosisNeurology (clinical)Oxidation-ReductionOxidative stressDevelopmental BiologyBrain Research
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Posaconazole Activity against Candida glabrata after Exposure to Caspofungin or Amphotericin B

2008

ABSTRACT We evaluated the effects of sequential therapy with caspofungin (CAS) or amphotericin B (AMB) followed by posaconazole (POS) against Candida glabrata . The susceptibilities to POS of yeast cells pre-exposed to CAS or AMB were identical to those of untreated cells as shown by standard Clinical and Laboratory Standards Institute broth dilution, cell viability, and disk diffusion methods. We then investigated the activity of sequential regimens in an experimental model of disseminated candidiasis. CAS given at 1 mg/kg/day for 2 days followed by POS at either 15 or 30 mg/kg/day significantly reduced the counts compared to the controls, but this treatment was not superior to the use of …

MaleSettore MED/07 - Microbiologia E Microbiologia ClinicaPosaconazoleAntifungal Agentsmedicine.drug_classAntibioticsColony Count MicrobialCandida glabrataMicrobial Sensitivity TestsBiologyPharmacologyKidneyDrug Administration ScheduleMicrobiologyEchinocandinsLipopeptidesMicechemistry.chemical_compoundCaspofunginAmphotericin BAmphotericin BmedicineAnimalsHumansExperimental TherapeuticsPharmacology (medical)Viability assayPharmacologyCandida glabrataPosaconazole Candida glabrataCandidiasisTriazolesbacterial infections and mycosesbiology.organism_classificationDisseminated CandidiasisRegimenTreatment OutcomeInfectious DiseaseschemistryCaspofunginmedicine.drugAntimicrobial Agents and Chemotherapy
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