Search results for "PROTEASES"

showing 10 items of 196 documents

Anti-prothrombin autoantibodies enriched after infection with SARS-CoV-2 and influenced by strength of antibody response against SARS-CoV-2 proteins

2021

Antiphospholipid antibodies (aPL), assumed to cause antiphospholipid syndrome (APS), are notorious for their heterogeneity in targeting phospholipids and phospholipid-binding proteins. The persistent presence of Lupus anticoagulant and/or aPL against cardiolipin and/or β2-glycoprotein I have been shown to be independent risk factors for vascular thrombosis and pregnancy morbidity in APS. aPL production is thought to be triggered by–among other factors–viral infections, though infection-associated aPL have mostly been considered non-pathogenic. Recently, the potential pathogenicity of infection-associated aPL has gained momentum since an increasing number of patients infected with Severe Acu…

RNA virusesPulmonologyCoronavirusesPhysiology2405 ParasitologyAntibody Response030204 cardiovascular system & hematologyBiochemistrychemistry.chemical_compoundMedical Conditions0302 clinical medicineimmune system diseasesImmune PhysiologyMedicine and Health SciencesCardiolipinMedicineBiology (General)skin and connective tissue diseasesImmune ResponsePathology and laboratory medicineCOVIDVirus Testing0303 health sciencesLupus anticoagulantImmune System Proteinsbiologymedicine.diagnostic_test2404 MicrobiologyProteasesMedical microbiologyEnzymes3. Good healthInfectious DiseasesCoagulationVirusesSARS CoV 2PathogensAntibodyResearch ArticleSARS coronavirusQH301-705.5Immunology10208 Institute of Neuropathology610 Medicine & healthSARS-CoV-2; Respiratory infections; Virus testing; serine proteases; Antibody response; Autoantibodies; Blood plasma; ImmunoassaysResearch and Analysis MethodsMicrobiologyAntibodiesRespiratory Disorders03 medical and health sciences1311 GeneticsDiagnostic MedicineAntiphospholipid syndromeVirology1312 Molecular BiologyGeneticsImmunoassaysneoplasmsMolecular BiologyAutoantibodies030304 developmental biology030203 arthritis & rheumatology2403 ImmunologyPregnancyBiology and life sciencesbusiness.industryOrganismsViral pathogensAutoantibodyProteinsRC581-607medicine.diseaseMicrobial pathogenschemistry19ImmunoassayRespiratory InfectionsImmunology2406 VirologyEnzymologyImmunologic Techniquesbiology.protein570 Life sciences; biologyParasitologyImmunologic diseases. AllergySerine ProteasesbusinessPLOS Pathogens
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Computational simulations on the binding and reactivity of a nitrile inhibitor of the SARS-CoV-2 main protease.

2021

We present a detailed computational analysis of the binding mode and reactivity of the novel oral inhibitor PF-07321332 developed against the SARS-CoV-2 3CL protease. Alchemical free energy calculations suggest that positions P3 and P4 could be susceptible to improvement in order to get a larger binding strength. QM/MM simulations unveil the reaction mechanism for covalent inhibition, showing that the nitrile warhead facilitates the recruitment of a water molecule for the proton transfer step.

Reaction mechanismNitrileLactamsProlineStereochemistrymedicine.medical_treatmentMolecular Dynamics SimulationCatalysischemistry.chemical_compoundMolecular dynamicsLeucineCatalytic DomainNitrilesMaterials ChemistrymedicineMoleculeHumansReactivity (chemistry)Protease InhibitorsBinding siteCoronavirus 3C ProteasesProteaseBinding SitesSARS-CoV-2Metals and AlloysCOVID-19General ChemistrySurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialschemistryCovalent bondCeramics and CompositesQuantum TheoryThermodynamicsChemical communications (Cambridge, England)
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Targeting SARS-CoV-2 Main Protease for Treatment of COVID-19: Covalent Inhibitors Structure-Activity Relationship Insights and Evolution Perspectives

2022

The viral main protease is one of the most attractive targets among all key enzymes involved in the SARS-CoV-2 life cycle. Covalent inhibition of the cysteine145 of SARS-CoV-2 MPRO with selective antiviral drugs will arrest the replication process of the virus without affecting human catalytic pathways. In this Perspective, we analyzed the in silico, in vitro, and in vivo data of the most representative examples of covalent SARS-CoV-2 MPRO inhibitors reported in the literature to date. In particular, the studied molecules were classified into eight different categories according to their reactive electrophilic warheads, highlighting the differences between their reversible/irreversible mech…

SARS-CoV-2COVID-19Viral Nonstructural ProteinsAntiviral AgentsSettore CHIM/08 - Chimica FarmaceuticaCOVID-19 Drug TreatmentCovalent inhibitorsMolecular Docking SimulationCysteine EndopeptidasesStructure-Activity RelationshipMain ProteaseDrug DiscoveryMolecular MedicineHumansProtease InhibitorsCysteineCoronavirus 3C Proteases
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Heteroaromatic Inhibitors of the Astacin Proteinases Meprin α, Meprin β and Ovastacin Discovered by a Scaffold-Hopping Approach.

2020

Abstract Astacin metalloproteinases, in particular meprins α and β, as well as ovastacin, are emerging drug targets. Drug‐discovery efforts have led to the development of the first potent and selective inhibitors in the last few years. However, the most recent compounds are based on a highly flexible tertiary amine scaffold that could cause metabolic liabilities or decreased potency due to the entropic penalty upon binding to the target. Thus, the aim of this study was to discover novel conformationally constrained scaffolds as starting points for further inhibitor optimization. Shifting from flexible tertiary amines to rigid heteroaromatic cores resulted in a boost in inhibitory activity. …

ScaffoldTertiary amineStereochemistryCell SurvivalAntineoplastic Agentsscaffold hoppingMatrix metalloproteinaseScaffold hoppinghydroxamate01 natural sciencesBiochemistryHydrocarbons AromaticmetalloproteinasesStructure-Activity RelationshipmeprinVery Important PaperDrug DiscoveryTumor Cells CulturedHumansProtease InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsAminesPharmacologyDose-Response Relationship DrugMolecular StructureFull Paper010405 organic chemistryChemistryOrganic ChemistryMetalloendopeptidasesFull PapersovastacinRecombinant Proteinsheteroaromatics0104 chemical sciences010404 medicinal & biomolecular chemistryMetalloproteasesMolecular MedicineAstacinDrug Screening Assays AntitumorSelectivityChemMedChem
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Type-II Transmembrane Prolyl Dipeptidases and Matrix Metalloproteinases in Membrane Vesicles of Active Endothelial Cells

2006

Endothelia cells in sparse culture are migratory and increase the production of gelatinases of serine- and metallo-classes in membrane vesicles. Collectively, proteases associated with membrane vesicles degrade extracellular matrix components including type-I and type-IV collagens, laminin and fibronectin. Inhibitor studies suggest the existence of small gelatinases that were derived from these serine- and metallo-proteases. Thus, further studies are warranted to demonstrate the cooperative action of metallo- and serine proteases on cell surfaces and in extracellular vesicles during endothelial cell migration in 3D collagenous matrices, and potential proteolytic activation mechanism for the…

SerineExtracellular matrixFibronectinGelatinasesProteasesbiologyChemistryLamininbiology.proteinMatrix metalloproteinaseTransmembrane proteinCell biology
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Melanoma cells release extracellular vesicles which contain RNA-binding proteins able to bind the mRNA encoding histone H1°

2015

Extracellular vesicles (EVs) are produced by most prokaryotic and eukaryotic cells; tumour cells, however, release much higher amounts of EVs, which contain cancer-specific proteins and RNAs. Molecules carried by EVs are captured by surrounding cells, which then undergo profound phenotypic modifications. G26/24 oligodendroglioma cells release, for example, EVs containing FasL and TRAIL, which induce apoptosis in rat cortical neurons and astrocytes in culture. By metabolic labelling of cells, EV-mediated horizontal transfer of radioactive proteins was clearly demonstrated. Among the proteins present in EVs produced by oligodendroglioma cells, extracellular matrix remodelling proteases, and t…

Settore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E CitologiaExtracellular vesicles (EVs) G26/24 oligodendroglioma cells xtracellular matrix remodelling proteases A375 melanoma cells H1° histone RNA-protein complexes myelin expression factor-2 (MYEF2)
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RNA as a carrier of epigenetic information

2017

Both prokaryotic and eukaryotic cells release into the extracellular matrix membrane-bound structures of different sizes, origin and composition, collectively called extracellular vesicles (EVs) [1]. Tumor cells, in particular, use EVs to transfer both nucleic acids and proteins to the surrounding normal cells, thus inducing in them transformed behaviours or killing them. G26/24 oligodendroglioma cells, for example, transfer by EVs pro-apoptotic proteins, such as TRAIL and Fas-Ligand [2], extracellular matrix remodelling proteases (such as ADAMTS) [3], and even the H1.0 histone protein [4]. Another tumour cell line, with a different tissue origin (A375 melanoma cells) releases into the medi…

Settore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologiaextracellular vesicles (EVs) G26/24 oligodendroglioma cells extracellular matrix remodelling proteases H1.0 histone protein H1.0 mRNA A375 melanoma cells myelin expression factor-2 (MYEF2)
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Novel pathogenic mechanism of microbial metalloproteinases: liberation of membrane-anchored molecules in biologically active form exemplified by stud…

1996

Certain membrane-anchored proteins, including several cytokines and cytokine receptors, can be released into cell supernatants through the action of endogenous membrane-bound metalloproteinases. The shed molecules are then able to fulfill various biological functions; for example, soluble interleukin-6 receptor (sIL-6R) can bind to bystander cells, rendering these cells sensitive to the action of IL-6. Using IL-6R as a model substrate, we report that the metalloproteinase from Serratia marcescens mimics the action of the endogenous shedding proteinase. Treatment of human monocytes with the bacterial protease led to a rapid release of sIL-6R into the supernatant. This effect was inhibitable …

Staphylococcus aureusProteasesmedicine.medical_treatmentImmunologyBiologyMatrix metalloproteinaseMicrobiologyMonocytesSubstrate SpecificityAntigens CDChlorocebus aethiopsmedicineAnimalsHumansReceptorSerratia marcescensMetalloproteinaseProteaseMembrane ProteinsMetalloendopeptidasesBiological activityBacterial InfectionsReceptors InterleukinListeria monocytogenesReceptors Interleukin-6Recombinant ProteinsBlotInfectious DiseasesSolubilityBiochemistryPseudomonas aeruginosaParasitologySignal transductionResearch ArticleSignal TransductionInfection and Immunity
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Inactivation of the ftsH gene of Lactobacillus plantarum WCFS1: Effects on growth, stress tolerance, cell surface properties and biofilm formation

2012

FtsH proteins are ubiquitous membrane-bound, ATP-dependent metalloproteases of the AAA family. In eubacteria, FtsH is involved in protein quality control under stress conditions. Lactobacillus plantarum is a widespread lactic acid bacterium that is encountered in several fermented food, including dairy products, vegetables and meat. In the present work the expression of the ftsH gene of L. plantarum was studied by quantitative real time RT-PCR in bacterial cultures subjected to various abiotic stresses. Both oxidative stress and addition of a membrane-fluidizing agent induced ftsH transcription, while a depletion of carbon-source repressed its mRNA level. Mutants deprived of the FtsH protea…

Surface Propertiesmedicine.medical_treatmentMutantReal-Time Polymerase Chain Reactionmedicine.disease_causeMicrobiologyMicrobiologyATP-Dependent ProteasesBacterial ProteinsStress PhysiologicalTranscription (biology)medicineGeneProteasebiologyReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingTemperatureBiofilmbiology.organism_classificationBiochemistryBiofilmsSaltsProtein qualityGene DeletionLactobacillus plantarumOxidative stressLactobacillus plantarumMicrobiological Research
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The Swedish dilemma - the almost exclusive use of APPswe-based mouse models impedes adequate evaluation of alternative β-secretases.

2022

Abstract Alzheimer's disease (AD) is the most common form of dementia, however incurable so far. It is widely accepted that aggregated amyloid β (Aβ) peptides play a crucial role for the pathogenesis of AD, as they cause neurotoxicity and deposit as so-called Aβ plaques in AD patient brains. Aβ peptides derive from the amyloid precursor protein (APP) upon consecutive cleavage at the β- and γ-secretase site. Hence, mutations in the APP gene are often associated with autosomal dominant inherited AD. Almost thirty years ago, two mutations at the β-secretase site were observed in two Swedish families (termed Swedish APP (APPswe) mutations), which led to early-onset AD. Consequently, APPswe was …

SwedenProteasesbiologyBACE1-ASNeurotoxicityMice TransgenicCell Biologymedicine.diseaseCathepsin BPathogenesisAmyloid beta-Protein PrecursorDisease Models AnimalADAMTS4Alzheimer Diseasemental disordersbiology.proteinAmyloid precursor proteinmedicineAnimalsHumansAmyloid Precursor Protein SecretasesMolecular BiologyAmyloid precursor protein secretaseNeuroscienceBiochimica et biophysica acta. Molecular cell research
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