Search results for "Pathogenesis"

showing 10 items of 761 documents

Innate Immune Cells' Contribution to Systemic Lupus Erythematosus

2019

Systemic lupus erythematosus (SLE) is a chronic autoimmune disease characterized by the presence of autoantibodies against nuclear antigens, immune complex deposition, and tissue damage in the kidneys, skin, heart and lung. Because of the pathogenic role of antinuclear antibodies and autoreactive T cells in SLE, extensive efforts have been made to demonstrate how B cells act as antibody-producing or as antigen-presenting cells that can prime autoreactive T cell activation. With the discovery of new innate immune cells and inflammatory mediators, innate immunity is emerging as a key player in disease pathologies. Recent work over the last decade has highlighted the importance of innate immun…

lcsh:Immunologic diseases. AllergyAnti-nuclear antibodyMini ReviewT cellImmunologyPathogenesisAntigenimmune system diseasesmedicineAnimalsHumansLupus Erythematosus SystemicImmunology and Allergydendritic cellsskin and connective tissue diseasesinnate immunitylupus (SLE)Autoimmune diseaseInnate immune systembusiness.industryInnate lymphoid cellAutoantibodymedicine.diseaseImmunity Innatemacrophage-cellmedicine.anatomical_structureImmunologyinnate lymphoid celllcsh:RC581-607businessFrontiers in Immunology
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Chlamydia trachomatis Infection and Anti-Hsp60 Immunity: The Two Sides of the Coin

2009

Chlamydia trachomatis (CT) infection is one of the most common causes of reproductive tract diseases and infertility. CT-Hsp60 is synthesized during infection and is released in the bloodstream. As a consequence, immune cells will produce anti-CT-Hsp60 antibodies. Hsp60, a ubiquitous and evolutionarily conserved chaperonin, is normally sequestered inside the cell, particularly into mitochondria. However, upon cell stress, as well as during carcinogenesis, the chaperonin becomes exposed on the cell surface (sf-Hsp60) and/or is secreted from cells into the extracellular space and circulation. Reports in the literature on circulating Hsp and anti-Hsp antibodies are in many cases short on detai…

lcsh:Immunologic diseases. Allergyanimal structuresImmunologyCardiovascular Disorders/Heart FailurePublic Health and Epidemiology/Infectious DiseasesChlamydia trachomatisPathology/Immunologychemical and pharmacologic phenomenaReviewmedicine.disease_causecomplex mixturesMicrobiologyAutoimmune DiseasesInfectious Diseases/Bacterial InfectionsPathogenesisImmune systemImmunityVirologyGeneticsmedicineAnimalsHumansImmunology/Cellular Microbiology and Pathogenesislcsh:QH301-705.5Molecular BiologyRheumatology/Autoimmunity Autoimmune and Inflammatory DiseasesAntigens BacterialbiologySettore BIO/16 - Anatomia UmanaMultiple sclerosisfungiAutoantibodyChaperonin 60Chlamydia Infectionsmedicine.diseaseHSP60 ChlamydiaMicrobiology/Immunity to Infectionslcsh:Biology (General)Immunologybiology.proteinParasitologyHSP60AntibodyDiabetes and Endocrinology/Type 1 Diabeteslcsh:RC581-607Chlamydia trachomatisPLoS Pathogens
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0127: Atrial fibrillation is associated with a marker of endothelial function and oxidative stress in patients with acute myocardial infarction

2016

Background Atrial fibrillation (AF), whether silent or symptomatic, is a frequent and severe complication of acute myocardial infarction (AMI). Asymmetric dimethylarginine (ADMA), an endogenous eNOS inhibitor, is a risk factor for endothelial dysfunction. We addressed the relationship between ADMA plasma levels and AF occurrence in AMI. Methods 273 patients hospitalized for AMI were included. Continuous electrocardiographic monitoring (CEM) ≥48 hours was recorded and ADMA was measured by High Performance Liquid Chromatography on admission blood sample. Results The incidence of silent and symptomatic AF was 39(14%) and 29 (11%), respectively. AF patients were markedly older than patients wit…

medicine.medical_specialty030204 cardiovascular system & hematologyPathogenesis03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineHeart rateMedicine030212 general & internal medicineMyocardial infarctionRisk factorEndothelial dysfunctionComputingMilieux_MISCELLANEOUSEjection fractionbusiness.industryAtrial fibrillationEndothelial function[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemmedicine.diseaseAtrial fibrillation3. Good healthMyocardial infarctionchemistryCardiologyCardiology and Cardiovascular MedicinebusinessAsymmetric dimethylarginine
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How to understand the complexity of endometriosis-related pain

2017

Pain is the most important symptom in patients with endometriosis, and its management is truly challenging. Due to the different localization of the endometriotic lesions in the pelvis, patients suffer from visceral and somatic pain or both at the same time. There are specific and unspecific symptoms characterized by endometriosis. Specific symptoms include dysmenorrhea, cyclic and acyclic pelvic pain, dyschezia, dysuria and dyspareunia. There is also a wide range of unspecific symptoms, such as unspecific bowel and bladder complaints, the emission of pain in the legs, vegetative concomitants like vomiting, emesis, gastric disorders, headaches, dizziness, painful ovulation, irregular pelvic…

medicine.medical_specialty030219 obstetrics & reproductive medicinebusiness.industryPelvic painEndometriosisObstetrics and Gynecologymedicine.diseaseSettore MED/40 - Ginecologia E Ostetricia03 medical and health sciences0302 clinical medicinemedicine.anatomical_structurePelvic painPathogenesis of the pelvic painEndometriosis-associated painmedicineIn patient030212 general & internal medicineRadiologymedicine.symptomEndometriosibusinessClassification of endometriosiPelvis
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Ischemic type biliary lesions in histidine-tryptophan-ketoglutarate (HTK) preserved liver grafts.

2006

Ischemic type biliary lesions lead to considerable morbidity following orthotopic liver transplantation. The exact pathogenesis is unknown. One major hypothesis is that insufficient perfusion of the arterial vessels of the biliary tree, especially under perfusion with the high viscous University of Wisconsin solution, might be responsible for ischemic type biliary lesions. Due to low viscosity, HTK solution is reported to have a lower incidence of biliary complications. However, there is no data concerning ischemic type biliary lesions in HTK preserved livers. In this paper we report our results after orthotopic liver transplantation with special regard to ischemic type biliary lesions in …

medicine.medical_specialtyAdenosinemedicine.medical_treatmentAllopurinolOrgan Preservation Solutions030232 urology & nephrologyBiomedical EngineeringMedicine (miscellaneous)Bioengineering030204 cardiovascular system & hematologyLiver transplantationHTK solutionGastroenterologyPotassium ChlorideBiomaterialsPathogenesisHistidine-tryptophan-ketoglutarate03 medical and health sciences0302 clinical medicineRaffinoseIschemiaInternal medicinemedicineHumansInsulinViaspanMannitolProspective Studiesbusiness.industryImmunosuppressionGeneral MedicineOrgan PreservationMiddle AgedGlutathioneLiver TransplantationTransplantationGlucoseBile DuctsbusinessPerfusionProcaineThe International journal of artificial organs
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A2.2 Uveitis is not associated with cardiovascular disease risk factors in patients with ankylosing spondylitis

2014

Background and Objectives Ankylosing spondylitis (AS) is a chronic, autoimmune, systemic, inflammatory disease. Recently, a link has been established between autoimmune inflammatory diseases, incl. AS, and the risk of developing cardiovascular disease (CVD). Uveitis is the most frequent extraarticular location of AS - it occurs in about 25 - 40% of patients. The impact of evidence of uveitis during the course of AS still remains a challenge. Does AS with uveitis differ from AS without uveitis? The objective of this study was to detect the link between uveitis during the course of AS and the CVD risk factors in patients with AS. Materials and Methods 77 AS patients classified using the modif…

medicine.medical_specialtyAnkylosing spondylitisWaistbusiness.industryCholesterolImmunologyDiseasemedicine.diseaseGastroenterologyGeneral Biochemistry Genetics and Molecular BiologySurgeryPathogenesischemistry.chemical_compoundHigh-density lipoproteinRheumatologychemistryInternal medicineImmunology and AllergyMedicinebusinessBody mass indexUveitisAnnals of the Rheumatic Diseases
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A10.18 Lack of Association of Serum Interleukin-17 and Interleukin-23 Levels with Disease Activity in Patients with Ankylosing Spondylitis in Latvia

2013

Background Ankylosing spondylitis (AS) is a clinically well-known chronic inflammatory disease of the axial skeleton and peripheral joints. The pathogenesis of this disease still remains a challenge. Determination of cytokine profile and its role involved in AS pathogenesis give an opportunity to extend the targeted therapeutic approach. Interleukin-17 (IL-17) and interleukin-23 (IL-23) are cytokines of interest in the investigation of the pathogenesis of spondyloarthritides although their importance in AS is not clearly defined. Objectives to investigate levels of IL-17 and IL-23 in a group of AS and in a demographically matched group of healthy subjects and its association with the diseas…

medicine.medical_specialtyAnkylosing spondylitismedicine.diagnostic_testbusiness.industryImmunologyInflammationDiseasemedicine.diseaseGastroenterologyGeneral Biochemistry Genetics and Molecular BiologyPathogenesisRheumatologyInternal medicineErythrocyte sedimentation rateImmunologyInterleukin 23Immunology and AllergyMedicineInterleukin 17medicine.symptombusinessBASDAIAnnals of the Rheumatic Diseases
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Magnesium and Alzheimer’s Disease

2015

Environmental factors, including nutrition and metal elements, are implicated in the pathophysiology of Alzheimer’s disease (AD). Several in vitro and in vivo data indicate a role for magnesium (Mg) in many biological and clinical aspects of AD. Mg deficiency, aside from having a negative impact on the energy production pathways required by the mitochondria to generate adenosine triphosphate, also affects many biochemical mechanisms vital for neuronal properties and synaptic plasticity, including the response of N-methyl- d -aspartate receptors to excitatory amino acids, stability, and viscosity of the cell membrane. Mg also has an action as a mild calcium antagonist, and as an antioxidant …

medicine.medical_specialtyAntioxidantChemistrymedicine.medical_treatmentchemistry.chemical_elementInflammationMitochondrionCalciummedicine.disease_causePathogenesisEndocrinologyInternal medicineSynaptic plasticitymedicinemedicine.symptomCognitive declineOxidative stress
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Primary paraesophageal Ewing’s sarcoma: an uncommon case report and literature review

2015

Ewing’s sarcoma is a rare and highly aggressive cancer most frequently arising in people under 20 years of age. We report an uncommon case of primary paraesophageal Ewing’s sarcoma in a 25-year-old male harboring the infrequent EWSR1/ERG fusion transcript with multiple splice variants coexisting in the same tumor. The patient was totally refractory to chemotherapy and died 17 months after diagnosis. We underscore the need for better understanding of the molecular pathogenesis of the disease and improved systemic therapy options.

medicine.medical_specialtyChemotherapyPathologyrecurrenceParaesophagealbusiness.industrymedicine.medical_treatmentMolecular pathogenesisEwing's sarcomaCase ReportDiseasemedicine.diseaseDermatologySystemic therapyOncologyFusion transcriptimmunohistochemistryMedicinePharmacology (medical)SarcomaEwing’s sarcomabusinessfusion genesOncoTargets and Therapy
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Complement activation by oxidatively modified low-density lipoproteins

1999

Background Oxidatively modified low-density lipoproteins (LDLs) have been implicated in the pathogenesis of atherosclerosis and are found in human vascular lesions. There is increasing evidence that complement activation may also play a role in atherogenesis. Activated complement proteins have been demonstrated to be present in early atherosclerotic lesions, and lipids isolated from lesions have been shown to activate complement, hence their designation as lesion complement activator (LCA). The question now arose whether oxidized LDLs would also activate complement. Material and methods The complement-activating capacity of a lesion complement activator preparation and of minimally as well …

medicine.medical_specialtyClinical BiochemistryInflammationImmunoelectrophoresis030204 cardiovascular system & hematologyBiochemistryLipid peroxidationPathogenesisLesion03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicine030304 developmental biology0303 health sciencesmedicine.diagnostic_testChemistryVascular diseaseGeneral Medicinemedicine.diseaseComplement systemComplement (complexity)EndocrinologyBiochemistrylipids (amino acids peptides and proteins)medicine.symptomEuropean Journal of Clinical Investigation
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