Search results for "Perch"
showing 10 items of 1084 documents
CCDC 244991: Experimental Crystal Structure Determination
2006
Related Article: C.Gerard, A.Mohamadou, J.Marrot, S.Brandes, A.Tabard|2005|Helv.Chim.Acta|88|2397|doi:10.1002/hlca.200590176
CCDC 722374: Experimental Crystal Structure Determination
2010
Related Article: C.Yuste, L.Canadillas-Delgado, A.Labrador, F.S.Delgado, C.Ruiz-Perez, F.Lloret, M.Julve|2009|Inorg.Chem.|48|6630|doi:10.1021/ic900599g
CCDC 193550: Experimental Crystal Structure Determination
2003
Related Article: V.V.Pavlishchuk, S.V.Kolotilov, A.W.Addison, M.J.Prushan, D.Schollmeyer, L.K.Thompson, T.Weyhermuller, E.A.Goreshnik|2003|Dalton Trans.||1587|doi:10.1039/b300539a
CCDC 190465: Experimental Crystal Structure Determination
2003
Related Article: V.V.Pavlishchuk, S.V.Kolotilov, A.W.Addison, M.J.Prushan, D.Schollmeyer, L.K.Thompson, T.Weyhermuller, E.A.Goreshnik|2003|Dalton Trans.||1587|doi:10.1039/b300539a
CCDC 879928: Experimental Crystal Structure Determination
2013
Related Article: Albert Escuer, Gina Vlahopoulou, Francesc Lloret, Franz A. Mautner|2014|Eur.J.Inorg.Chem.||83|doi:10.1002/ejic.201300910
CCDC 1045981: Experimental Crystal Structure Determination
2015
Related Article: Michele Bedin, Alavi Karim, Marcus Reitti, Anna-Carin C. Carlsson, Filip Topić, Mario Cetina, Fangfang Pan, Vaclav Havel, Fatima Al-Ameri, Vladimir Sindelar, Kari Rissanen, Jürgen Gräfenstein, Máté Erdélyi|2015|Chemical Science|6|3746|doi:10.1039/C5SC01053E
CCDC 1965760: Experimental Crystal Structure Determination
2021
Related Article: Munirathnam Manda, Himanshu Arora, Arunava Sengupta, Shashi Kant, Francesc Lloret, Rabindranath Mukherjee|2021|New J.Chem.|45|16019|doi:10.1039/D1NJ01150B
Effects of statins, fibrates, rosuvastatin, and ezetimibe beyond cholesterol: the modulation of LDL size and subclasses in high-risk patients.
2007
Increasing evidence suggests that the quality-rather than just the quantity-of low-density lipoproteins (LDLs) exerts a great influence on cardiovascular risk. LDLs comprise multiple subclasses with discrete size and density, and different physicochemical composition, metabolic behaviors, and atherogenicity. Individuals generally cluster into 2 broad subgroups. Most have a predominance of large LDLs, and some have a higher proportion of small particles. Small, dense LDLs are good predictors of cardiovascular events and progression of coronary artery disease. Their predominance has been accepted as an emerging cardiovascular risk factor by the National Cholesterol Education Program Adult Tre…
Effects of PCSK9 inhibitors on HDL cholesterol efflux and serum cholesterol loading capacity in familial hypercholesterolemia subjects: a multi-lipid…
2022
Proprotein convertase subtilisin/kexin type 9 (PCSK9), beyond regulating LDL cholesterol (LDL-c) plasma levels, exerts several pleiotropic effects by modulating lipid metabolism in extrahepatic cells such as macrophages. Macrophage cholesterol homeostasis depends on serum lipoprotein functions, including the HDL capacity to promote cell cholesterol efflux (CEC) and the serum capacity to promote cell cholesterol loading (CLC). The aim of this observational study was to investigate the effect of PCSK9 inhibitors (PCSK9-i) treatment on HDL-CEC and serum CLC in patients with familial hypercholesterolemia (FH). 31 genetically confirmed FH patients were recruited. Blood was collected and serum is…
Lipoprotein(a) Genotype Influences the Clinical Diagnosis of Familial Hypercholesterolemia
2023
: Background Evidence suggests that LPA risk genotypes are a possible contributor to the clinical diagnosis of familial hypercholesterolemia (FH). This study aimed at determining the prevalence of LPA risk variants in adult individuals with FH enrolled in the Italian LIPIGEN (Lipid Transport Disorders Italian Genetic Network) study, with (FH/M+) or without (FH/M-) a causative genetic variant. Methods and Results An lp(a) [lipoprotein(a)] genetic score was calculated by summing the number risk-increasing alleles inherited at rs3798220 and rs10455872 variants. Overall, in the 4.6% of 1695 patients with clinically diagnosed FH, the phenotype was not explained by a monogenic or polygenic cause …