Search results for "Perfusion"

showing 10 items of 714 documents

Pretreatment with potent P-glycoprotein ligands may increase intestinal secretion in rats.

2001

The expression of P-glycoprotein is induced in cell cultures upon exposure to various inducers. Therefore, the aim of the present study was to evaluate the in-vivo relevance of this observation, i.e. the influence of chronic pretreatments with selected drugs -- all of which are ligands to P-glycoprotein (P-gp) as demonstrated in radioligand binding studies and all of which have some or a considerable effect on P-gp expression in Caco-2 cells -- on the effective intestinal permeabilities of the model compound talinolol in rats employing in-situ single-pass intestinal perfusion of three different gut segments. Talinolol was selected, because it shows high selectivity for one of the exsorptive…

MaleColonDuodenumAdrenergic beta-AntagonistsPharmaceutical ScienceBiologyPharmacologyLigandsVinblastineJejunumPropanolamineschemistry.chemical_compoundmedicineAnimalsATP Binding Cassette Transporter Subfamily B Member 1Rats WistarP-glycoproteinIntestinal permeabilityStereoisomerismmedicine.diseaseCalcium Channel BlockersAntineoplastic Agents PhytogenicVinblastineRatsmedicine.anatomical_structureJejunumchemistryBiochemistryVerapamilDuodenumbiology.proteinVerapamilPerfusionTalinololmedicine.drugEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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Combination Therapy of Allopurinol and Dantrolene and Its Role In The Prevention of Experimental Ischemia Reperfusion Injury Of The Small Intestine.

2020

Background: The effect of different drugs on ischemia and reperfusion (I/R; induced oxygen free radical damage) was examined in small bowel tissue because the intestine is extremely sensitive to this pathology. Different drugs (allopurinol and dantrolene) can remove oxygen free radicals or inhibit the mechanisms leading to their generation, thus reducing mucosal lesions. We investigated the protective potential of combination therapy in the intestine against I/R damage. Methods: Forty-eight male Wistar rats were separated into 8 groups: one sham (control), one I/R (ischemia 60 min + reperfusion at 24 h), and 6 groups treated with allopurinol, dantrolene, or combination therapy. The grade of…

MaleCombination therapyRadicalAllopurinolTerapéuticaIschemiaAllopurinol030230 surgeryPharmacologyDantroleneDantrolene03 medical and health sciencesTratamiento médico0302 clinical medicineCirugíaIntestine SmallmedicineAnimalsRats WistarMedicamentobusiness.industrySuperoxide Dismutasemedicine.diseaseSmall intestineRatsmedicine.anatomical_structure030220 oncology & carcinogenesisReperfusion InjuryMedicamentosSurgerybusinessReperfusion injurymedicine.drugJournal of investigative surgery : the official journal of the Academy of Surgical Research
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Search for Stroke-Protecting Agents in Endothelin-1-Induced Ischemic Stroke Model in Rats

2012

Background and Objective. Ischemic stroke may initiate a reperfusion injury leading to brain damage cascades where inflammatory mechanisms play a major role. Therefore, the necessity for the novel stroke-protecting agents whose the mechanism of action is focused on their anti-inflammatory potency is still on the agenda for drug designers. Our previous studies demonstrated that cerebrocrast (a 1,4-dihydropyridine derivative) and mildronate (a representative of the aza-butyrobetaine class) possessed considerable anti-inflammatory and neuroprotective properties in different in vitro and in vivo model systems. The present study investigated their stroke-protecting ability in an endothelin-1 (ET…

MaleDihydropyridinesDrug Evaluation PreclinicalInfarctionBrain damagePharmacologyNeuroprotectionIn vivomedicineAnimalsRats WistarStrokeEndothelin-1business.industryGeneral Medicinemedicine.diseaseRatsStrokeDisease Models AnimalNeuroprotective AgentsMechanism of actionendothelin-1; ischemic stroke; neurodegeneration; protection; cerebrocrast; mildronateDrug Therapy Combinationmedicine.symptombusinessReperfusion injuryEx vivoMethylhydrazinesMedicina; Volume 48; Issue 10; Pages: 77
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Temporal trends in prehospital management of ST-segment elevation myocardial infarction from 2002 to 2010 in Cote d’Or: Data from the RICO registry (…

2012

Summary Background Myocardial infarction with ST-segment elevation (STEMI) is a medical emergency requiring specific management, with the main aim of achieving reperfusion as quickly as possible. Guidelines from medical societies have defined optimal management, with proven efficacy on morbi-mortality. Aims Our study aimed to evaluate trends in practices between 2002 and 2010 in the emergency management of STEMI in a single French department, namely Cote d’Or. Methods All patients admitted with a first STEMI to one of the six participating coronary care units (private or public) in Cote d’Or since January 2001 were included in a prospective registry (obseRvatoire des Infarctus de Cote d’Or …

MaleEmergency Medical ServicesFirst medical contactTime delaysTime FactorsMyocardial InfarctionTemporal trendsÉvolutionSTEMIHumansST segmentMedicineProspective StudiesRegistriesMyocardial infarctionManagement practicesEmergency managementbusiness.industryGeneral MedicineMiddle AgedDélaismedicine.diseaseOptimal managementTime delaysReperfusionFemaleFranceMedical emergencyCardiology and Cardiovascular MedicinebusinessArchives of Cardiovascular Diseases
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Cordycepin protects against cerebral ischemia/reperfusion injury in vivo and in vitro.

2011

Cordycepin, (3'-deoxyadenosine), a bioactive compound of Cordyceps militaris, has been shown to exhibit many pharmacological actions, such as anti-inflammatory, antioxidative and anticancer activities. Little is known about the neuroprotective action of cordycepin as well as its molecular mechanisms. In this study, cordycepin was investigated for its neuroprotective potential in mice with ischemia following 15 min of the bilateral common carotid artery occlusion and 4h of reperfusion. The effect of cordycepin was also studied in mice brain slices treated with oxygen-glucose deprivation (OGD) injury. Our results showed that cordycepin was able to prevent postischemic neuronal degeneration an…

MaleExcitatory Amino AcidsIschemiaCell CountPharmacologyNeuroprotectionHippocampusBrain IschemiaSuperoxide dismutaseBrain ischemiachemistry.chemical_compoundMiceIn vivoMalondialdehydemedicineAnimalsPharmacologyNeuronsbiologyCordycepinDeoxyadenosinesbusiness.industrySuperoxide DismutaseGlutamate receptormedicine.diseaseOxygenGlucoseBiochemistrychemistryReperfusion Injurybiology.proteinMatrix Metalloproteinase 3businessReperfusion injuryPropidiumEuropean journal of pharmacology
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Acute in vivo administration of a fish oil-containing emulsion improves post-ischemic cardiac function in n-3-depleted rats

2006

International audience; A novel i.v. lipid preparation (MCT:FO) containing 80% medium chain-triacylglycerols and 20% fish oil was recently developed to rapidly replenish cell membrane phospholipids with omega 3 (n-3) polyunsaturated fatty acids (PUFA). In regard of this property, we investigated the effect of a single i.v. administration of MCT:FO on the recovery of cardiac function after ischemia in control and n-3-depleted rats. Results were compared with those obtained either with a control preparation, where FO was replaced by triolein (MCT:OO), or with saline. Saline (1 ml) or lipid preparation (also 1 ml) was injected as a bolus via the left saphenous vein. After 60 min the heart was …

MaleFat EmulsionsTime Factorsmedicine.medical_treatmentMyocardial IschemiaWistar030204 cardiovascular system & hematologyPharmacologyLIPID PREPARATIONchemistry.chemical_compound0302 clinical medicineBolus (medicine)MESH: Fatty Acids Omega-3Heart Rate[SDV.IDA]Life Sciences [q-bio]/Food engineeringMedicineMESH: AnimalsMESH: Oxygen ConsumptionTrioleinMESH: Heart RateSalineOMEGA3-DEFICIENCYOmega-3chemistry.chemical_classification0303 health sciencesFatty AcidsHeartGeneral Medicine[SDV.IDA] Life Sciences [q-bio]/Food engineeringFish oilMESH: Myocardial Reperfusion Injury3. Good healthLactatesMESH: Myocardial IschemiaIntravenousPolyunsaturated fatty acidCardiac function curveFat Emulsions Intravenousmedicine.medical_specialtyMESH: Rats[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringACIDE GRAS POLYINSATURE OMEGA3IschemiaMESH: Fish OilsMyocardial Reperfusion InjuryMESH: Coronary CirculationMESH: Lactates03 medical and health sciencesISCHEMIA-REPERFUSIONFish OilsOxygen ConsumptionIn vivoCoronary CirculationMESH: Analysis of VarianceFatty Acids Omega-3[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyGeneticsAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRats WistarLANGENDORFF030304 developmental biologyAnalysis of Variancebusiness.industryBody WeightMESH: Time FactorsMESH: Rats Wistarmedicine.diseaseMESH: MaleRatsMESH: Body WeightMESH: Fat Emulsions IntravenousSurgeryMESH: Heartchemistrybusiness
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Enzymic control of irreversible binding of metabolically activated benzo(a)pyrene in perfused rat liver by monooxygenase activity.

1977

Addition of [3H]-benzo(a)pyrene to the perfusion medium of isolated rat livers results in irreversible binding of radioactivity to DNA, RNA and protein. Binding to DNA accounted for about 0.1% of the total radioactivity which was bound in livers from animals treated with oil or saline and was increased by a factor of 3–5 after pretreatment of the animals with β-naphthoflavone or with phenobarbital. When the inhibitiors of monooygenase activity, α-naphthoflavone or metyrapone, were present in the perfusion medium, irreversible binding was reduced in livers from both β-naphthoflavone- and phenobarbital-pretreated animals, irrespective of the inhibitor used.

MaleHealth Toxicology and MutagenesisIn Vitro TechniquesToxicologychemistry.chemical_compoundBenzopyrene HydroxylasemedicineAnimalsBenzopyrene HydroxylaseFlavonoidsMetyraponeRNAGeneral MedicineDNARatsBiochemistrychemistryBenzo(a)pyreneLiverPhenobarbitalPyreneRNAPhenobarbitalAryl Hydrocarbon HydroxylasesPerfusionDNAmedicine.drugProtein BindingArchives of toxicology
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Dynamics of tumor oxygenation and red blood cell flux in response to inspiratory hyperoxia combined with different levels of inspiratory hypercapnia.

2002

Abstract Background and purpose : Increasing arterial oxygen partial pressure (pO 2 ) by breathing hyperoxic gases is an effective means of improving tumor oxygenation, although the efficacy of adding CO 2 to the inspiratory gas has been discussed controversially. This study aimed at analyzing the impact of different inspiratory CO 2 fractions on the time course of oxygenation and perfusion changes in experimental tumors during and after inspiratory hyperoxia. Material and methods : Perfusion and oxygenation of rat DS-sarcomas were studied during spontaneous breathing of pure oxygen or hyperoxic gas mixtures containing different CO 2 fractions (1, 2.5 or 5%). Red blood cell (RBC) flux was a…

MaleHyperoxiaHypercapniaRats Sprague-DawleyOxygen ConsumptionmedicineLaser-Doppler FlowmetryAnimalsRadiology Nuclear Medicine and imagingHyperoxiaChemistryOxygen Inhalation TherapyHematologyOxygenationTumor OxygenationCarbon DioxideRatsOxygenPerfusionBlood pressureOncologyRegional Blood FlowAnesthesiaBreathingCarbogen BreathingSarcoma Experimentalmedicine.symptomHypercapniaPerfusionRadiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology
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Changes in microregional perfusion, oxygenation, ATP and lactate distribution in subcutaneous rat tumours upon water-filtered IR-A hyperthermia

1995

The effect of hyperthermia on microcirculatory and metabolic parameters in s.c. DS-sarcomas of different sizes on the hind foot dorsum of SD-rats was investigated. Hyperthermia was carried out using a novel water-filtered, infrared-A radiation technique. Heating was performed at a rate of 0.5 degrees C/min until 44 degrees C was achieved in the tumour centre, which was maintained for 60 min. Using a multichannel laser Doppler flowmeter, red blood cell flux could be assessed continuously and at several sites within the tumour tissue simultaneously. Substantial inter-site variations in laser Doppler flux (LDF) were observed during hyperthermia which were independent of tumour size, site of me…

MaleHyperthermiaCancer ResearchPathologymedicine.medical_specialtyInfrared RaysPhysiologyRats Sprague-DawleyTumour tissueLaser doppler fluxAdenosine TriphosphateSingle sitePhysiology (medical)Laser-Doppler FlowmetrymedicineAnimalsDistribution (pharmacology)Lactic Acidbusiness.industryChemistryMicrocirculationTemperatureHyperthermia InducedOxygenationmedicine.diseaseRatsOxygenPerfusionRed blood cellGlucosemedicine.anatomical_structureLactatesFemaleSarcoma ExperimentalNuclear medicinebusinessPerfusionBlood Flow VelocityInternational Journal of Hyperthermia
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Spontaneous release of endogenous 5-hydroxytryptamine and 5-hydroxyindoleacetic acid from the isolated vascularly perfused ileum of the guinea-pig

1987

The spontaneous release of 5-hydroxytryptamine and its metabolite 5-hydroxyindoleacetic acid from the enterochromaffin cells of the small intestine into the portal circulation was investigated in vitro using the vascularly perfused ileum of the guinea-pig. The release of 5-hydroxytryptamine decreased by 70% in a calcium-free medium and by 35% in the presence of tetrodotoxin. Inhibition of monoamine oxidase activity by pargyline (100 microM) had no effect on the spontaneous release of 5-hydroxytryptamine although it caused a 75% reduction in the outflow of 5-hydroxyindoleacetic acid. Imipramine (1 microM), an inhibitor of neuronal uptake of 5-hydroxytryptamine, reduced the 5-hydroxyindoleace…

MaleImipramineSerotoninmedicine.medical_specialtyMonoamine oxidaseMetaboliteGuinea PigsMyenteric PlexusIleumTetrodotoxinIn Vitro Techniqueschemistry.chemical_compoundIleumInternal medicinemedicineAnimalsPortal VeinCatabolism5-Hydroxyindoleacetic acidGeneral NeuroscienceTryptophanHydroxyindoleacetic AcidPargylinePerfusionmedicine.anatomical_structureEndocrinologyPargylinechemistryEnterochromaffin cellCalciumMethyldopaSerotoninmedicine.drugNeuroscience
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