Search results for "Perl"

showing 10 items of 383 documents

Familial hypobetalipoproteinemia due to apolipoprotein B R463W mutation causes intestinal fat accumulation and low postprandial lipemia

2008

Abstract Objective Familial hypobetalipoproteinemia (FHBL) is characterized by inherited low plasma levels of apolipoprotein B (apoB)-containing lipoproteins. In this paper we investigated whether the already described APOB R463W missense mutation, a FHBL mutation able to impair the activity of microsomal triglyceride transfer protein (MTP), may cause intestinal fat accumulation and reduced postprandial lipemia. Methods Four out of five probands harboring APOB R463W mutation were compared with six healthy controls and six patients with celiac disease (CD). An oral fat load supplemented with retinyl palmitate (RP) was administered and a gastro-duodenal endoscopy with biopsy was performed. Re…

AdultMaleRetinyl Estersmedicine.medical_specialtySettore MED/09 - Medicina InternaAdolescentApolipoprotein BMutation MissenseapolipoproteinBlood lipidsHyperlipidemiasIntra-Abdominal FatBiologyMicrosomal triglyceride transfer proteinchemistry.chemical_compoundRetinyl palmitateInternal medicinemedicineHumansMissense mutationIntestinal MucosaChildVitamin ATriglyceridesApolipoproteins BTriglycerideMiddle AgedLipid MetabolismPostprandial Periodmedicine.diseasePostprandialEndocrinologychemistryHypobetalipoproteinemia Familial Apolipoprotein BB R463Wbiology.proteinFemalelipids (amino acids peptides and proteins)HypobetalipoproteinemiaDiterpenesCarrier ProteinsCardiology and Cardiovascular MedicineAtherosclerosis
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Identifying genetic risk variants for coronary heart disease in familial hypercholesterolemia: an extreme genetics approach

2015

Mutations in the low-density lipoprotein receptor (LDLR) gene cause familial hypercholesterolemia (FH), a disorder characterized by coronary heart disease (CHD) at young age. We aimed to apply an extreme sampling method to enhance the statistical power to identify novel genetic risk variants for CHD in individuals with FH. We selected cases and controls with an extreme contrast in CHD risk from 17 000 FH patients from the Netherlands, whose functional LDLR mutation was unequivocally established. The genome-wide association (GWA) study was performed on 249 very young FH cases with CHD and 217 old FH controls without CHD (above 65 years for males and 70 years of age for females) using the Ill…

AdultMaleRiskSettore MED/09 - Medicina InternaGenotypePopulationCoronary DiseaseSingle-nucleotide polymorphismGenome-wide association studyComorbidityFamilial hypercholesterolemiaQuantitative trait locusBiologymedicine.disease_causePolymorphism Single NucleotideArticleHyperlipoproteinemia Type IIYoung Adultsymbols.namesakeGene FrequencyRisk FactorsOdds RatioGeneticsmedicineHumansGenetic Predisposition to DiseaseeducationAllelesGenetics (clinical)AgedAged 80 and overGeneticsMutationeducation.field_of_studyfamilial hypercholesterolemiaPCSK9familial hypercholesterolemia; genetic risk factorgenetic risk factorGenetic VariationMiddle Agedmedicine.diseaseBonferroni correctionReceptors LDLCase-Control StudiesMutationsymbolsFemaleGenome-Wide Association StudyEuropean journal of human genetics
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Circulating mononuclear cells nuclear factor-kappa B activity, plasma xanthine oxidase, and low grade inflammatory markers in adult patients with fam…

2009

Eur J Clin Invest 2010; 40 (2): 89–94 Abstract Background  Few data are available on circulating mononuclear cells nuclear factor-kappa B (NF-kB) activity and plasma xanthine oxidase (XO) activity in heterozygous familial hypercholesterolaemia (FH). The goal of the study was to analyse circulating mononuclear cells NF-kB and plasma XO activities in FH patients. Materials and methods  Thirty FH index patients and 30 normoglycaemic normocholesterolaemic controls matched by age, gender, body mass index, abdominal circumference and homeostasis model assessment index were studied. Plasma XO and inflammatory markers were measured by standard methods. NF-kB was assayed in circulating mononuclear c…

AdultMaleRiskmedicine.medical_specialtyXanthine OxidaseApolipoprotein BLipoproteinsClinical BiochemistryBlood lipidsBiochemistryPeripheral blood mononuclear cellMonocytesHyperlipoproteinemia Type IIchemistry.chemical_compoundInternal medicineBlood plasmamedicineHumansXanthine oxidaseInflammationbiologyCholesterolInterleukin-6C-reactive proteinNF-kappa BGeneral MedicineMiddle AgedEndocrinologychemistryCardiovascular Diseasesbiology.proteinRegression Analysislipids (amino acids peptides and proteins)FemaleBiomarkersLipoproteinInterleukin-1European journal of clinical investigation
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Association between the TaqIB polymorphism in the cholesteryl ester transfer protein gene locus and plasma lipoprotein levels in familial hypercholes…

2001

Abstract Cholesteryl ester transfer protein (CETP) facilitates the exchange of triglycerides (TG) and cholesteryl ester between lipoprotein particles. Subjects with familial hypercholesterolemia (FH) have been reported to have higher CETP activities, which could contribute to the lower high-density lipoprotein-cholesterol (HDL-C) levels and increased cardiovascular risk observed in some of these patients. Several polymorphisms have been reported in the CETP locus; the common TaqlB polymorphism is associated, in normolipidemic subjects, with decreased CETP activity and levels and with increased HDL-C levels. No data is available on the influence of this polymorphism in FH subjects. We have e…

AdultMaleSite-Specific DNA-Methyltransferase (Adenine-Specific)medicine.medical_specialtyGenotypeApolipoprotein BLipoproteinsEndocrinology Diabetes and MetabolismPopulationFamilial hypercholesterolemiaHyperlipoproteinemia Type IIchemistry.chemical_compoundEndocrinologyInternal medicineCholesterylester transfer proteinmedicineHumanseducationNational Cholesterol Education ProgramAllelesGlycoproteinseducation.field_of_studyPolymorphism Geneticbiologymedicine.diagnostic_testmedicine.diseaseCholesterol Ester Transfer ProteinsCholesterolEndocrinologychemistryCardiovascular DiseasesSpainbiology.proteinCholesteryl esterFemalelipids (amino acids peptides and proteins)Carrier ProteinsLipid profileLipoproteinMetabolism
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Significance of hyperlactatemia in acute hypnotic drug poisoning

1981

Lactate concentration, fibrinogen and fibrin(ogen) -- degradation-products in central venous blood were analysed in 35 unconscious patient with acute hypnotic drug poisoning (HDP) and compared with the results in 13 healthy control persons undergoing the same degree of forced diuresis via central venous catheters. Blood samples were taken on admission and at 12 h intervals up to 36 h after admission. Patients with HDP were attributed to the categories of moderate intoxications (n = 17) and severe intoxications (n = 18) according to their clinical condition. On admission, blood lactate was significantly higher in severe intoxication (3.90 +/- 2.94 mmol/l) as compared to the control group (1.…

AdultMaleUnconsciousnessFibrinogenFibrinFibrin Fibrinogen Degradation ProductsDrug DiscoverymedicineBlood lactateHumansHypnotics and SedativesIn patientGenetics (clinical)biologybusiness.industryUnconsciousnessHemodynamicsFibrinogenGeneral MedicineVenous bloodHypnotic drugAnesthesiaLactatesbiology.proteinMolecular MedicineFemaleHyperlactatemiamedicine.symptombusinessmedicine.drugKlinische Wochenschrift
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Increased oxidative stress levels and normal antioxidant enzyme activity in circulating mononuclear cells from patients of familial hypercholesterole…

2010

Familial hypercholesterolemia (FH) is a clinical condition with high risk for developing atherosclerosis. Increased oxidative stress (OS) and FH have been related to atherosclerosis, but no data are available on levels of OS and antioxidant enzyme activity in circulating mononuclear cells (CMCs) from FH patients. Circulating mononuclear cells are important mediators in atherosclerosis development, and chronically increased blood OS present in FH can induce modification in CMC activity. The objective of the study was to analyze the OS levels in CMCs from FH patients and controls. We have selected 30 nonrelated FH index patients and 30 normoglycemic and normocholesterolemic controls matched b…

AdultMaleXanthine Oxidasemedicine.medical_specialtyAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentFamilial hypercholesterolemiamedicine.disease_causeAntioxidantsHyperlipoproteinemia Type IISuperoxide dismutasechemistry.chemical_compoundEndocrinologyMalondialdehydeInternal medicinemedicineHumansXanthine oxidasechemistry.chemical_classificationGlutathione PeroxidaseGlutathione DisulfidebiologySuperoxide DismutaseGlutathione peroxidaseGlutathioneMiddle AgedAtherosclerosisCatalasemedicine.diseaseGlutathioneOxidative StressEndocrinologychemistryCatalaseLeukocytes Mononuclearbiology.proteinFemaleOxidation-ReductionOxidative stressMetabolism
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Increased plasma xanthine oxidase activity is related to nuclear factor kappa beta activation and inflammatory markers in familial combined hyperlipi…

2010

Abstract Background and aims Xanthine oxidase (XO) has been described as one of the major enzymes producing free radicals in blood. Oxidative stress and inflammatory processes have been implicated in the pathogenesis of endothelial dysfunction and the progression of atherosclerosis but until now, there is little data about the influence of vascular prooxidant systems and inflammation in familial combined hyperlipidemia (FCH). Our goal was to evaluate whether XO activity was altered in FCH and if it was related to the inflammatory process represented by NFkB, IL-6 and hsCRP, and assessing the correlation between XO activity and insulin resistance (IR). Method and results 40 Non-related subje…

AdultMaleXanthine Oxidasemedicine.medical_specialtyFree RadicalsEndocrinology Diabetes and Metabolismmedicine.medical_treatmentHyperlipidemia Familial CombinedMedicine (miscellaneous)Inflammationmedicine.disease_causechemistry.chemical_compoundInsulin resistanceMalondialdehydeInternal medicineHyperlipidemiamedicineHumansXanthine oxidaseInflammationNutrition and Dieteticsmedicine.diagnostic_testInterleukin-6business.industryInsulinNF-kappa BMiddle AgedAtherosclerosismedicine.diseaseLipidsOxidative StressC-Reactive ProteinLogistic ModelsEndocrinologychemistryMultivariate AnalysisUric acidFemaleEndothelium VascularLipid PeroxidationInsulin Resistancemedicine.symptomCardiology and Cardiovascular MedicineLipid profilebusinessBiomarkersOxidative stressNutrition, Metabolism and Cardiovascular Diseases
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Effectiveness of cascade filtration plasmapheresis in two patients affected by familial hypercholesterolemia

1995

Hypercholesterolemia has been recognised as a primary risk factor for coronary heart disease. Reduction of plasma levels of total and LDL cholesterol has been shown to decrease coronary atherosclerosis. Plasmapheresis represents an useful non-pharmacological tool to treat severe hypercholesterolemias. We have evaluated the effectiveness of a system of plasmapheresis using a cascade filtration method in two young male subjects (aged 16 and 26 years) with homozygous familial hypercholesterolemia. Both showed severe coronary atherosclerosis as determined by angiography. Procedures were performed at intervals of 7 days in each case. We observed a mean reduction of plasma levels of total cholest…

AdultMalemedicine.medical_specialtyAdolescentApolipoprotein Bmedicine.medical_treatmentFamilial hypercholesterolemiaFibrinogenGastroenterologyHyperlipoproteinemia Type IIchemistry.chemical_compoundInternal medicinemedicineHumansHyperlipoproteinemia Type IIRisk factorCoronary atherosclerosisbiologybusiness.industryCholesterolCholesterol LDLPlasmapheresisHematologyGeneral Medicinemedicine.diseaseEndocrinologychemistrybiology.proteinlipids (amino acids peptides and proteins)PlasmapheresisbusinessFiltrationmedicine.drugJournal of Clinical Apheresis
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Insulin resistance and oxidative stress in familial combined hyperlipidemia.

2007

Oxidative stress is associated with atherosclerosis. Familial combined hyperlipidemia (FCH) is considered as a human model of primary dyslipidemia and atherosclerosis frequently associated with insulin resistance (IR), but there are few data on its possible relation to oxidative stress. The objective of this study was to evaluate oxidative stress status using different markers in subjects with FCH assessing its possible correlation with anthropometric parameters and IR. This was a cross-sectional study. A cohort of 40 FCH patients (20 with IR (HOMAor=3.2) and 20 without IR (HOMA3.2)), and 20 healthy volunteers were included, all of them non-diabetic, normotensive and non-smokers. We measure…

AdultMalemedicine.medical_specialtyAdolescentHyperlipidemia Familial Combinedmedicine.disease_causeAnthropometric parametersInsulin resistanceInternal medicinemedicineHumansAgedGlutathione Disulfidebusiness.industryVascular diseasenutritional and metabolic diseasesDeoxyguanosineMiddle Agedmedicine.diseaseAtherosclerosisGlutathioneLipidsFamilial combined hyperlipidemiaOxidative StressEndocrinology8-Hydroxy-2'-DeoxyguanosineCohortFemaleInsulin ResistanceCardiology and Cardiovascular MedicinebusinessDyslipidemiaOxidative stressAtherosclerosis
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Small and dense LDL in Familial Combined Hyperlipidemia and N291S polymorphism of the lipoprotein lipase gene

2009

18 pagesm 2 figures. -- PMID: 19335919 [PubMed]

AdultMalemedicine.medical_specialtyApolipoprotein BAdolescentEndocrinology Diabetes and MetabolismPopulationClinical BiochemistryHyperlipidemia Familial CombinedMutation MissenseBlood lipidschemistry.chemical_compoundYoung AdultHigh-density lipoproteinEndocrinologyInternal medicinePrevalenceMedicineShort PaperHumanseducationlcsh:RC620-627AgedBiochemistry medicalLipoprotein lipaseeducation.field_of_studyPolymorphism Geneticmedicine.diagnostic_testbiologyCholesterolbusiness.industryBiochemistry (medical)Cholesterol LDLMiddle AgedAtherosclerosisLipidslcsh:Nutritional diseases. Deficiency diseasesLipoprotein LipaseEndocrinologychemistrySpainLow-density lipoproteinCase-Control Studiesbiology.proteinFemalelipids (amino acids peptides and proteins)businessLipid profile
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