Search results for "Peroxides"

showing 10 items of 162 documents

Effect of xanthine oxidase-generated extracellular superoxide on skeletal muscle force generation

2009

Skeletal muscle contractions increase superoxide anion in skeletal muscle extracellular space. We tested the hypotheses that 1) after an isometric contraction protocol, xanthine oxidase (XO) activity is a source of superoxide anion in the extracellular space of skeletal muscle and 2) the increase in XO-derived extracellular superoxide anion during contractions affects skeletal muscle contractile function. Superoxide anion was monitored in the extracellular space of mouse gastrocnemius muscles by following the reduction of cytochrome c in muscle microdialysates. A 15-min protocol of nondamaging isometric contractions increased the reduction of cytochrome c in microdialysates, indicating an …

MaleXanthine Oxidasemedicine.medical_specialtyPhysiologyOxypurinolfree radicalsSuperoxide dismutaseExtensor digitorum longus muscleMice03 medical and health sciencesGastrocnemius musclechemistry.chemical_compound0302 clinical medicineSuperoxidescontractile functionIsometric ContractionPhysiology (medical)Internal medicinemedicineAnimalsMuscle Skeletal030304 developmental biologySoleus muscle0303 health sciencesexercisebiologyMuscle fatigueSuperoxide DismutaseChemistrySuperoxideCytochromes cSkeletal muscleArticlesmusculoskeletal systemElectric StimulationMice Inbred C57BLmedicine.anatomical_structureEndocrinologyBiochemistryModels AnimalMuscle Fatiguebiology.proteinmedicine.symptomExtracellular Space030217 neurology & neurosurgeryMuscle ContractionMuscle contractionAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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Cadmium-Induced Enteropathy in Domestic Cocks: A Biochemical and Histological Study after Subchronic Exposure

2007

The biochemical and histological sequelae resulting from a diet containing 50.20 mg cadmium/kg were studied in Lohmann brown cockerels from hatching until 30 days of age. The additional cadmium chloride (CdCl(2)) to the diet induced the formation of lipid peroxides, which via a chain reaction led to accumulation of malondialdehyde in intestinal mucosa. At the end of the study (after 30 days of cadmium exposure) total protein and metallothionein levels in the intestinal mucosa and the relative ileal and duodenal weight increased. Histological data show that CdCl(2) causes an increase in number of goblet cells and granular lymphocytes in the intestinal mucosa. Down-regulation of the serotonin…

Maleinorganic chemicalsLipid Peroxidesmedicine.medical_specialtychemistry.chemical_elementCadmium chlorideToxicologychemistry.chemical_compoundCadmium ChlorideIntestinal mucosaMalondialdehydeInternal medicinemedicineAnimalsMetallothioneinEnteropathyIntestinal MucosaPoultry DiseasesPharmacologyCadmiumBody WeightHistologyGeneral Medicinemedicine.diseaseMalondialdehydeSmall intestineIntestinal DiseasesEndocrinologymedicine.anatomical_structureBiochemistrychemistryMetallothioneinChickensBasic & Clinical Pharmacology & Toxicology
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Mitochondria from females exhibit higher antioxidant gene expression and lower oxidative damage than males

2003

We have investigated the differential mitochondrial oxidative stress between males and females to understand the molecular mechanisms enabling females to live longer than males. Mitochondria are a major source of free radicals in cells. Those from female rats generate half the amount of peroxides than those of males. This does not occur in ovariectomized animals. Estrogen replacement therapy prevents the effect of ovariectomy. Mitochondria from females have higher levels of reduced glutathione than those from males. Those from ovariectomized rats have similar levels to males, and estrogen therapy prevents the fall in glutathione levels that occurs in ovariectomized animals. Oxidative damage…

Malemedicine.medical_specialtyAntioxidantOvariectomymedicine.medical_treatmentMitochondria LiverMitochondrionBiologymedicine.disease_causeDNA MitochondrialBiochemistryAntioxidantsGene Expression Regulation EnzymologicSuperoxide dismutasechemistry.chemical_compoundRNA Ribosomal 16SPhysiology (medical)Internal medicinemedicineAnimalsRats WistarDNA Primerschemistry.chemical_classificationGlutathione PeroxidaseReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseGlutathione peroxidaseEstrogensGlutathioneGlutathionePeroxidesRatsOxygenOxidative StressEndocrinologychemistryOvariectomized ratbiology.proteinRNAFemaleDismutaseReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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Ursodeoxycholic acid protects against secondary biliary cirrhosis in rats by preventing mitochondrial oxidative stress

2004

Ursodeoxycholic acid (UDCA) improves clinical and biochemical indices in primary biliary cirrhosis and prolongs survival free of liver transplantation. Recently, it was suggested that the cytoprotective mechanisms of UDCA may be mediated by protection against oxidative stress, which is involved in the development of cirrhosis induced by chronic cholestasis. The aims of the current study were 1) to identify the mechanisms involved in glutathione depletion, oxidative stress, and mitochondrial impairment during biliary cirrhosis induced by chronic cholestasis in rats; and 2) to determine the mechanisms associated with the protective effects of UDCA against secondary biliary cirrhosis. The find…

Malemedicine.medical_specialtyCirrhosisCardiolipinsGlutamate-Cysteine LigaseBiliary cirrhosisPopulationBiologymedicine.disease_causeMembrane Potentialschemistry.chemical_compoundPrimary biliary cirrhosisInternal medicinemedicineCardiolipinAnimalsRats Wistareducationeducation.field_of_studyCholestasisHepatologyLiver Cirrhosis BiliaryUrsodeoxycholic AcidCystathionine gamma-LyaseGlutathionemedicine.diseaseGlutathioneUrsodeoxycholic acidMitochondriaPeroxidesRatsOxidative StressEndocrinologyLiverchemistryChronic DiseaseHepatocytesOxidation-ReductionOxidative stressmedicine.drugHepatology
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REDUCED OXIDATIVE STRESS DURING ACELLULAR REPERFUSION OF THE RAT LIVER AFTER HYPOTHERMIC OSCILLATING PERFUSION

1999

Background ATP resynthesis during reperfusion after liver preservation has been shown to be well correlated with the function of transplanted grafts. Nevertheless, the advantages of a cellular energy charge loading during the preservation period are yet not fully understood. This study evaluates the effects of different nucleotide levels at the end of preservation on metabolic changes and oxidative stress during reperfusion. Methods Two experimental groups were chosen reflecting different energy charge states after preservation: static cold storage for 10 hr and hypothermic oxygenated oscillating perfusion for 10 hr. In both experimental groups, normothermic ex vivo acellular reperfusion ov…

Malemedicine.medical_specialtyCold storageHypothermiaLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundSuperoxidesMalondialdehydeRats Inbred BNInternal medicinemedicineAnimalsEnergy chargeLiver preservationCryopreservationTransplantationbiologyTumor Necrosis Factor-alphaChemistrySuperoxideLiver cellOrgan PreservationMalondialdehydeLiver GlycogenRatsPerfusionOxidative StressEndocrinologyLiverBiochemistryReperfusionbiology.proteinLipid PeroxidationBiomarkersTransplantation
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Oxidant/antioxidant status in obese children compared to pediatric patients with type 1 diabetes mellitus.

2009

Codoner-Franch P, Pons-Morales S, Boix-Garcia L, Valls-Belles V. Oxidant/antioxidant status in obese children compared to pediatric patients with type 1 diabetes mellitus. Background: Type 1 diabetes (T1D) mellitus and obesity are recognized risk factors for cardiovascular disease (CVD). A common mechanism underlying an increased risk for endothelial dysfunction in these two metabolic diseases is oxidative stress. Objective: To evaluate and compare the oxidant/antioxidant defense systems in children affected with T1D or obesity in order to determine the importance of oxidative stress before the emergence of complications. Subjects: Children with T1D (n = 20) or obesity (n = 22), without com…

Malemedicine.medical_specialtyLipid PeroxidesAntioxidantErythrocytesAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentalpha-TocopherolProtein oxidationmedicine.disease_causeLipid peroxidationProtein Carbonylationchemistry.chemical_compoundInternal medicineMalondialdehydeInternal MedicinemedicineHumansObesityProspective StudiesChildRetrospective Studieschemistry.chemical_classificationType 1 diabetesGlutathione Peroxidasebusiness.industryGlutathione peroxidaseMalondialdehydemedicine.diseasebeta CaroteneObesityGlutathioneOxidative StressEndocrinologyDiabetes Mellitus Type 1chemistryPediatrics Perinatology and Child HealthFemaleLipid PeroxidationbusinessOxidative stressPediatric diabetes
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Absence of lipid peroxidation as determined by ethane exhalation in rats treated with 2, 3, 7, 8-tetrachlorodibenzo-p-dioxin (TCDD).

1985

The exhalation of ethane is widely used as an indicator of in vivo lipid peroxidation. To test the hypothesis that lipid peroxidative events are involved in the toxicity of 2, 3, 7, 8-tetrachlorodibenzo-p-dioxin (TCDD), we administered a lethal dose of TCDD (60 μg/kg), IP to male Sprague Dawley rats (160–180 g) and measured by gas chromatography the exhalation of ethane into the atmosphere of a closed all-glass exposure chamber. TCDD-treated rats exhaled only slightly more ethane than control rats at a single time point 7 days following TCDD administration. Since the exhalation of ethane is the net result of the endogenous production of the gas and its metabolic degradation, the latter was …

Malemedicine.medical_specialtyLipid PeroxidesPolychlorinated DibenzodioxinsHealth Toxicology and MutagenesisEndogenyToxicologyDioxinsLipid peroxidationchemistry.chemical_compoundCytochrome P-450 Enzyme SystemIn vivoInternal medicinemedicineAnimalsheterocyclic compoundsEthaneChemistryLethal doseExhalationRats Inbred StrainsGeneral MedicineMetabolismTetrachlorodibenzo-p-dioxinRatsstomatognathic diseasesEndocrinologyBiochemistryToxicityArchives of toxicology
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Pregnenolone sulfate, a naturally occurring excitotoxin involved in delayed retinal cell death.

2002

The present study was designed to investigate the neurosteroid pregnenolone sulfate (PS), known for its ability to modulate NMDA receptors and interfere with acute excitotoxicity, in delayed retinal cell death. Three hours after exposure of the isolated and intact retina to a 30-min PS pulse, DNA fragmentation as assessed by genomic DNA gel electrophoresis and a modified in situ terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end-labeling (TUNEL) method appeared concurrently with an increase in superoxide dismutase (SOD) activity and thiobarbituric acid-reactive substances (TBARS) levels. At 7 h, the increased amount of DNA laddering was accompanied by a higher number of TUN…

Malemedicine.medical_specialtyNeurotoxinsExcitotoxicityApoptosisDNA FragmentationDNA ladderingBiologymedicine.disease_causeBiochemistryReceptors N-Methyl-D-AspartateThiobarbituric Acid Reactive SubstancesRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundAdjuvants ImmunologicSuperoxidesInternal medicinemedicineTBARSIn Situ Nick-End LabelingAnimalsCycloheximideRats WistarProgesteroneProtein Synthesis InhibitorsTUNEL assayEstradiolL-Lactate DehydrogenaseDehydroepiandrosterone SulfateSuperoxide DismutaseRatsEndocrinologychemistryApoptosisPregnenolonePregnenoloneDNA fragmentationLipid PeroxidationPregnenolone sulfateReactive Oxygen Speciesmedicine.drugJournal of neurochemistry
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Chronic Therapy With Isosorbide-5-Mononitrate Causes Endothelial Dysfunction, Oxidative Stress and a Marked Increase in Vascular Endothelin-1 Express…

2011

Aims Isosorbide-5-mononitrate (ISMN) is one of the most frequently used compounds in the treatment of coronary artery disease predominantly in the USA. However, ISMN was reported to induce endothelial dysfunction, which was corrected by vitamin C pointing to a crucial role of reactive oxygen species (ROS) in causing this phenomenon. We sought to elucidate the mechanism how ISMN causes endothelial dysfunction and oxidative stress in vascular tissue. Methods and results Male Wistar rats ( n = 69 in total) were treated with ISMN (75 mg/kg/day) or placebo for 7 days. Endothelin (ET) expression was determined by immunohistochemistry in aortic sections. Isosorbide-5-mononitrate infusion caused si…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIIsosorbide DinitratePharmacologymedicine.disease_causeBiochemistryMicechemistry.chemical_compoundSuperoxidesEnosInternal medicinePhysiology (medical)medicineAnimalsNitric Oxide DonorsEnzyme InhibitorsRats WistarEndothelial dysfunctionCyclic GMPAortaMice KnockoutNADPH oxidaseEndothelin-1biologybusiness.industryNADPH Oxidasesmedicine.diseasebiology.organism_classificationEndothelin 1BosentanRatsNitric oxide synthaseEndothelial stem cellOxidative StressNG-Nitroarginine Methyl EsterEndocrinologychemistryApocyninbiology.proteinEndothelium VascularCardiology and Cardiovascular MedicineEndothelin receptorbusinessOxidative stressSignal Transductionmedicine.drugFree Radical Biology and Medicine
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Resveratrol Reverses Endothelial Nitric-Oxide Synthase Uncoupling in Apolipoprotein E Knockout Mice

2010

A crucial cause of the decreased bioactivity of nitric oxide (NO) in cardiovascular diseases is the uncoupling of the endothelial NO synthase (eNOS) caused by the oxidative stress-mediated deficiency of the NOS cofactor tetrahydrobiopterin (BH(4)). The reversal of eNOS uncoupling might represent a novel therapeutic approach. The treatment of apolipoprotein E knockout (ApoE-KO) mice with resveratrol resulted in the up-regulation of superoxide dismutase (SOD) isoforms (SOD1-SOD3), glutathione peroxidase 1 (GPx1), and catalase and the down-regulation of NADPH oxidases NOX2 and NOX4 in the hearts of ApoE-KO mice. This was associated with reductions in superoxide, 3-nitrotyrosine, and malondiald…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIISOD3SOD2ResveratrolAntioxidantsSuperoxide dismutaseMicechemistry.chemical_compoundApolipoproteins ESuperoxidesEnosMalondialdehydeInternal medicineStilbenesmedicineAnimalsGTP CyclohydrolaseMice KnockoutPharmacologychemistry.chemical_classificationReactive oxygen speciesbiologyReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseChemistrySuperoxideMyocardiumTetrahydrobiopterinbiology.organism_classificationBiopterinIsoenzymesOxidative StressEndocrinologyBiochemistryResveratrolbiology.proteinRNATyrosineMolecular Medicinemedicine.drugJournal of Pharmacology and Experimental Therapeutics
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