Search results for "Phenotype"

showing 10 items of 1875 documents

Autoreactive liver-infiltrating T cells in primary biliary cirrhosis recognize inner mitochondrial epitopes and the pyruvate dehydrogenase complex.

1993

Primary biliary cirrhosis (PBC) is characterized by lymphoid infiltrates in the portal tracts of the liver and the occurrence of antimitochondrial autoantibodies in serum directed against components of the pyruvate dehydrogenase complex and the other alpha-keto acid dehydrogenase complexes. These enzymes are located on the inner mitochondrial membrane. The destruction of the biliary tract in PBC is thought to be mediated by autoreactive liver-infiltrating T cells exerting cytotoxic activity or releasing certain lymphokines. In this study the reactivity of liver infiltrating T cells was shown to a bovine pyruvate dehydrogenase complex (PDH), a purified E2 subunit (PDH-E2) and a crude prepara…

AdultMaleAdolescentBiliary cirrhosisT-LymphocytesEnzyme-Linked Immunosorbent AssayMitochondria LiverPyruvate Dehydrogenase ComplexAutoimmune hepatitisEpitopesPrimary biliary cirrhosisCell MovementmedicineCytotoxic T cellHumansCells CulturedAgedAutoantibodiesHepatologybiologyLiver Cirrhosis BiliaryAntibodies MonoclonalMiddle Agedmedicine.diseasePyruvate dehydrogenase complexPhenotypeLiverImmunologybiology.proteinFemaleAntibodyViral hepatitisCD8Journal of hepatology
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Phenotype associated with TAF2 biallelic mutations: a clinical description of four individuals and review of the literature

2021

International audience; Transcription factor IID is a multimeric protein complex that is essential for the initiation of transcription by RNA polymerase II. One of its critical components, the TATA-binding protein-associated factor 2, is encoded by the gene TAF2. Pathogenic variants of this gene have been shown to be responsible for the Mental retardation, autosomal recessive 40 syndrome. This syndrome is characterized by severe intellectual disability, postnatal microcephaly, pyramidal signs and thin corpus callosum. Until now, only three families have been reported separately. Here we report four individuals, from two unrelated families, who present with severe intellectual disability and…

AdultMaleAdolescentFoot Deformities CongenitalDevelopmental DisabilitiesAutosomal recessiveIntellectual disabilityPostnatal microcephaly[SDV.GEN] Life Sciences [q-bio]/GeneticsBiologyCorpus Callosum03 medical and health sciencesNeurodevelopmental disorderNeurodevelopmental disorderIntellectual disabilityGeneticsmedicineHumansMissense mutationGlobal developmental delayTAF2ChildGeneAllelesGenetics (clinical)Exome sequencing030304 developmental biologyGeneticsTATA-Binding Protein Associated Factors0303 health sciences[SDV.GEN]Life Sciences [q-bio]/Genetics030305 genetics & heredityGeneral Medicinemedicine.diseasePhenotypeChild PreschoolTAF2MicrocephalyFemaleTranscription Factor TFIID
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Molecular analysis of Gaucher disease: distribution of eight mutations and the complete gene deletion in 27 patients from Germany

1997

Gaucher disease is the most common lysosomal storage disease with a high prevalence in the Ashkenazi Jewish population but it is also present in other populations. The presence of eight mutations (1226G, 1448C, IVS2+1. 84GG, 1504T, 1604T, 1342C and 1297T) and the complete deletion of the beta-glucocerebrosidase gene was investigated in 25 unrelated non-Jewish patients with Gaucher's disease in Germany. In the Jewish population, three of these mutations account for more than 90% of all mutated alleles. In addition, relatives of two patients were included in our study. Restriction fragment length polymorphism analysis and sequencing of PCR products obtained from DNA of peripheral blood leukoc…

AdultMaleAdolescentGenotypePopulationBiologymedicine.disease_causeCompound heterozygosityFrameshift mutationGermanyGenotypeGeneticsmedicineHumansAlleleChildeducationGeneAllelesGenetics (clinical)GeneticsMutationeducation.field_of_studyGaucher DiseaseMiddle AgedPhenotypeChild PreschoolMutationFemaleRestriction fragment length polymorphismGene DeletionPolymorphism Restriction Fragment LengthHuman Genetics
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Association of nicotinic acetylcholine receptor subunit alpha 4 polymorphisms with nicotine dependence in 5500 Germans.

2009

Polymorphisms in the CHRNA4 gene coding the nicotinic acetylcholine receptor subunit alpha 4 have recently been suggested to play a role in the determination of smoking-related phenotypes. To examine this hypothesis, we conducted a genetic association study in three large samples from the German general population (N(1)=1412; N(2)=1855; N(3)=2294). Five single-nucleotide polymorphisms in CHRNA4 were genotyped in 5561 participants, including 2707 heavily smoking cases (regularly smoking at least 20 cigarettes per day) and 2399 never-smoking controls (or=100 cigarettes over lifetime). We examined associations of the polymorphisms with smoking case-control status and with the extent of nicotin…

AdultMaleAdolescentGenotypeProtein subunitBiologyPharmacologyReceptors NicotinicPolymorphism Single NucleotideWhite PeopleGermanyGeneticsmedicineHumansGenetic Predisposition to DiseaseNicotine dependenceAgedPharmacologyAged 80 and overTobacco Use DisorderMiddle Agedmedicine.diseaseNicotinic acetylcholine receptorPhenotypeMolecular MedicineFemaleSmoking CessationThe pharmacogenomics journal
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Efficacy of Rapamycin as Inducer of Hb F in Primary Erythroid Cultures from Sickle Cell Disease and β-Thalassemia Patients.

2015

Phenotypic improvement of hemoglobinopathies such as sickle cell disease and β-thalassemia (β-thal) has been shown in patients with high levels of Hb F. Among the drugs proposed to increase Hb F production, hydroxyurea (HU) is currently the only one proven to improve the clinical course of these diseases. However, Hb F increase and patient's response are highly variable, indicating that new pharmacological agents could be useful for patients not responding to HU or showing a reduction of response during long-term therapy. In this study we evaluated the efficacy of rapamycin, a lypophilic macrolide used for the prevention of acute rejection in renal transplant recipients, as an inducer of Hb…

AdultMaleAdolescentGenotypeThalassemiaClinical BiochemistryCellDiseaseAnemia Sickle Cellbeta-GlobinsPharmacologyBiologyYoung Adultalpha-GlobinsIn vivomedicineHumansHydroxyureaInducergamma-GlobinsGenetics (clinical)Cells CulturedFetal HemoglobinAgedErythroid Precursor CellsSirolimusBiochemistry (medical)beta-ThalassemiaClinical courseHematologyMiddle Agedmedicine.diseasePhenotypeMolecular biologyIn vitromedicine.anatomical_structureGene Expression RegulationMutationFemaleHemoglobin
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Gluten Stimulation Induces an in vitro Expansion of Peripheral Blood Tγδ Cells from HLA-DQ2-Positive Subjects of Families of Patients with Celiac Dis…

1998

The intestinal gluten sensitivity formally known as celiac disease (CD) is characterized by an evident involvement of local immune response and it is associated with the expression of HLA-DQ2 allele. The major role in the disease seems to be played by the T lymphocyte population bearing gamma delta T cell receptor (T gamma delta cells) which are increased both in peripheral blood and intestinal mucosae of celiac patients. In this paper data on the effects of in vitro gluten stimulation on lymphocytes expressing the T gamma delta phenotype are reported. Gluten seems to be able to induce the expansion of the T gamma delta cell population both in CD patients and their HLA-DQ2-positive asymptom…

AdultMaleAdolescentGlutensT-LymphocytesImmunologyEnzyme-Linked Immunosorbent AssayStimulationDiseasePolymerase Chain ReactionImmune systemHLA-DQ AntigensGeneticsHumansMedicineReceptors ImmunologicAlleleChildCells CulturedGenetics (clinical)chemistry.chemical_classificationbusiness.industryHLA-DQ2nutritional and metabolic diseasesMiddle AgedGlutendigestive system diseasesIn vitroPeripheral bloodCeliac DiseasePhenotypechemistryChild PreschoolImmunologyLeukocytes MononuclearFemaleInterleukin-4businessExperimental and Clinical Immunogenetics
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Frequency and phenotype of SPG11 and SPG15 in complicated hereditary spastic paraplegia

2009

Background: Hereditary spastic paraplegias (HSP) are clinically and genetically highly heterogeneous. Recently, two novel genes, SPG11 ( spatacsin ) and SPG15 ( spastizin ), associated with autosomal recessive HSP, were identified. Clinically, both are characterised by complicated HSP and a rather similar phenotype consisting of early onset spastic paraplegia, cognitive deficits, thin corpus callosum (TCC), peripheral neuropathy and mild cerebellar ataxia. Objective: To compare the frequency of SPG11 and SPG15 in patients with early onset complicated HSP and to further characterise the phenotype of SPG11 and SPG15. Results: A sample of 36 index patients with early onset complicated HSP and …

AdultMaleAdolescentHereditary spastic paraplegiaGenes RecessiveCompound heterozygosityCorpus callosumCorpus CallosumYoung AdultGene FrequencyIntellectual DisabilitySpasticHumansMedicineMutation frequencyAllele frequencyGenetic Association StudiesPolymorphism GeneticCerebellar ataxiaSpastic Paraplegia Hereditarybusiness.industryProteinsmedicine.diseasePhenotypePsychiatry and Mental healthPhenotypeMutationImmunologyFemaleSurgeryNeurology (clinical)medicine.symptomCarrier ProteinsbusinessNeuroscienceJournal of Neurology, Neurosurgery & Psychiatry
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Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies

2012

Identification of mutations in the HOGA1 gene as the cause of autosomal recessive primary hyperoxaluria (PH) type III has revitalized research in the field of PH and related stone disease. In contrast to the well-characterized entities of PH type I and type II, the pathophysiology and prevalence of type III is largely unknown. In this study, we analyzed a large cohort of subjects previously tested negative for type I/II by complete HOGA1 sequencing. Seven distinct mutations, among them four novel, were found in 15 patients. In patients of non-consanguineous European descent the previously reported c.700+5G>T splice-site mutation was predominant and represents a potential founder mutation, w…

AdultMaleAdolescentIn silicoCell Culture TechniquesMedizinGene ExpressionContext (language use)Biologymedicine.disease_causeArticlePrimary hyperoxaluriaKidney CalculiGeneticsmedicineHumansGenetic TestingGeneGenetics (clinical)Genetic testingGeneticsMutationmedicine.diagnostic_testGenetic heterogeneityOxo-Acid-LyasesMiddle Agedmedicine.diseasePhenotypePedigreeHyperoxaluria PrimaryMutationFemale
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Genetic and environmental influences on longitudinal changes in leisure-time physical activity from adolescence to young adulthood.

2013

The aim of this study was to estimate genetic and environmental influences on the longitudinal evolution of leisure-time physical activity habits from adolescence to young adulthood. Data were gathered at four time points, at mean ages 16.2, 17.1, 18.6, and 24.5 years. At baseline, the sample comprised 5,216 monozygotic and dizygotic twins, born 1975–1979, and, at the last follow-up point, of 4,531 monozygotic and dizygotic twins. Physical activity volume was assessed as frequency of leisure-time physical activity and participants were categorized into three groups: inactive, moderately active, and active. Genetic and environmental influences were estimated using a multivariate, longitudina…

AdultMaleAdolescentLeisure timePhysical activityEnvironmentMotor ActivitySocial EnvironmentDizygotic twinsDevelopmental psychology03 medical and health sciencesYoung Adult0302 clinical medicineLeisure ActivitiesTwins DizygoticHumansLongitudinal StudiesProspective StudiesRegistriesYoung adult10. No inequalityta315Genetics (clinical)030304 developmental biology0303 health sciencesModels GeneticObstetrics and Gynecologyta3141030229 sport sciencesTwins MonozygoticHeritabilityPhenotypePediatrics Perinatology and Child HealthFemaleGene-Environment InteractionPsychologyTwin Research and Human Genetics
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HLA antigen, gene, and haplotype frequencies in Thailand.

1978

Antigen, gene, and haplotype frequencies as well as phenotype distribution of the HLA system were studied in a series of 213 individuals in northern Thailand. The series consisted of 160 northern Thais, 23 Thai individuals from various other regions of Thailand, and 25 persons of Chinese origin. Most frequently found were the alleles HLA-A11 and HLA-Bw40 and the haplotype HLA-A2, B-. Phenotype distribution followed a Hardy-Weinberg expectation. Significant differences were found especially between our results for the alleles of locus B and the results of a series from Bangkok reported by Chiewsilp and Chanarat (1976).

AdultMaleAdolescentLocus (genetics)Human leukocyte antigenBiologyThaisAntigenGene FrequencyHLA Antigensparasitic diseasesGeneticsHumansAlleleGeneGenetics (clinical)AllelesAgedGeneticsHaplotypeMiddle Agedbiology.organism_classificationThailandPhenotypePhenotypeFemalegeographic locationsHuman genetics
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