Search results for "Phenotype"

showing 10 items of 1875 documents

Replacement therapy for alpha-1-protease inhibitor deficiency in PiZ subjects with chronic obstructive lung disease

1988

In a six-month multicenter feasibility and safety study, 20 patients, who all had a congenital deficiency of alpha-1-protease inhibitor (A1PI) of the PiZ phenotype accompanied by a chronic obstructive lung disease, were treated with human-plasma-derived A1PI. A weekly dose of 60 mg/kg, administered intravenously, was shown to be sufficient to maintain patient serum levels above the threshold limit of 35 percent, the serum level of healthy persons of the MZ phenotype. This is supposed to be the minimal effective level for protection against the elastolytic attack of the lung and, therefore, satisfies one of the most important criteria of feasibility of long-term replacement therapy. The glob…

AdultMalealpha 1-Antitrypsin DeficiencymedicineHumansLung Diseases ObstructiveInfusions IntravenousAgedRadial immunodiffusionClinical Trials as TopicLungPancreatic Elastasebiologybusiness.industryBlood ProteinsGeneral MedicineMiddle AgedOuchterlony double immunodiffusionTrypsinmedicine.diseaseAlpha-1 Protease Inhibitor DeficiencyObstructive lung diseasePhenotypemedicine.anatomical_structureImmunologybiology.proteinFemaleAntibodyLung Volume MeasurementsbusinessNephelometrymedicine.drugThe American Journal of Medicine
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Genotypic and phenotypic spectrum in tricho-rhino-phalangeal syndrome types I and III

2000

Tricho-rhino-phalangeal syndrome (TRPS) is characterized by craniofacial and skeletal abnormalities. Three subtypes have been described: TRPS I, caused by mutations in the TRPS1 gene on chromosome 8; TRPS II, a microdeletion syndrome affecting the TRPS1 and EXT1 genes; and TRPS III, a form with severe brachydactyly, due to short metacarpals, and severe short stature, but without exostoses. To investigate whether TRPS III is caused by TRPS1 mutations and to establish a genotype-phenotype correlation in TRPS, we performed extensive mutation analysis and evaluated the height and degree of brachydactyly in patients with TRPS I or TRPS III. We found 35 different mutations in 44 of 51 unrelated p…

AdultMaleanimal structuresAdolescentGenotypeDNA Mutational AnalysisMolecular Sequence DataLimb Deformities CongenitalBiologyOsteochondrodysplasiasPolymorphism Single NucleotideShort statureLanger–Giedion syndromeGeneticsmedicineHumansMissense mutationTricho–rhino–phalangeal syndromeGenetics(clinical)Amino Acid SequenceChildGenetics (clinical)GeneticsAnthropometryBase SequenceBrachydactylyInfantZinc FingersExonsSyndromeArticlesMiddle AgedMicrodeletion syndromemedicine.diseasePenetranceBody HeightPedigreeDNA-Binding ProteinsRadiographyPhenotypeChild PreschoolMutationTrichorhinophalangeal Syndrome Type IErythroid-Specific DNA-Binding FactorsFemalemedicine.symptomChromosomes Human Pair 8Transcription Factors
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Epimutation at human chromosome 14q32.2 in a boy with a upd(14)mat-like clinical phenotype.

2009

Recently, three reports described deletions and epimutations affecting the imprinted region at chromosome 14q32.2 in individuals with a phenotype typical for maternal uniparental disomy of chromosome 14 [upd(14)mat]. In this study, we describe another patient with upd(14)mat-like phenotype including low birth weight, neonatal feeding problems, muscular hypotonia, motor and developmental delay, small hands and feet, and truncal obesity. Conventional cytogenetic analyses, fluorescence in situ hybridization subtelomere screening, multiplex ligation-dependent probe amplification analysis of common microdeletion and microduplication syndromes, and methylation analysis of SNRPN all gave normal re…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesMolecular Sequence DataMothersBiologyMethylationPolymorphism Single NucleotideEpigenesis GeneticGenomic ImprintingIntergenic regionGeneticsmedicineHumansAbnormalities MultipleEpigeneticsChildGenetics (clinical)GeneticsChromosomes Human Pair 14Muscular hypotoniamedicine.diagnostic_testBase SequenceChromosomeUniparental DisomySubtelomerePhenotypeDifferentially methylated regionsPhenotypeMutationFemaleFluorescence in situ hybridizationClinical genetics
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One

2019

Neurofibromatosis type 1 (NF1) is an autosomal dominant disease with complete penetrance but high variable expressivity. NF1 is caused by loss-of-function mutations in the NF1 gene, a negative regulator of the RAS-MAPK pathway. The NF1 gene has one of the highest mutation rates in human disorders, which may explain the outbreak of independent de novo variants in the same family. Here, we report the co-occurrence of pathogenic variants in the NF1 and SPRED1 genes in six families with NF1 and Legius syndrome, using next-generation sequencing. In five of these families, we observed the co-occurrence of two independent NF1 variants. All NF1 variants were classified as pathogenic, according to t…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesSPRED1Neurofibromatosis 1Neurofibromin 1AdolescentCafe-au-Lait Spotsneurofibromatosis type 1eye diseasesArticlenervous system diseasesPedigreeLegius syndromePhenotypeNF1MutationHumansFemalede novo variantChildneoplasmsAdaptor Proteins Signal TransducingGenes
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Prevalence, Phenotype, and Psychosocial Well-Being in Euthyroid/Hypothyroid Thyroid-Associated Orbitopathy.

2015

At the onset of thyroid-associated orbitopathy (TAO), most patients are hyperthyroid, while scarce data are available on euthyroid/hypothyroid TAO. The aim of this study was to assess the prevalence, phenotype, and psychosocial burden of patients with initially euthyroid/hypothyroid TAO.The medical records of 461 consecutive and unselected patients with TAO followed at a specialized joint thyroid-eye clinic were analyzed within this retrospective cross-sectional study. Main outcome measures were the prevalence of initially eu- or hypothyroid TAO as well as ophthalmic signs and symptoms, disease-specific quality of life (QoL), work impairment, and rate of psychotherapy in initially eu-/hypot…

AdultMaleendocrine systemmedicine.medical_specialtyPediatricsendocrine system diseasesAdolescentCross-sectional studyEndocrinology Diabetes and MetabolismThyroid GlandThyroiditisGraves' ophthalmopathyYoung AdultEndocrinologyQuality of lifeHypothyroidismInternal medicinemedicineOrbital DiseasesPrevalenceHumansEuthyroidAgedRetrospective StudiesAged 80 and overbusiness.industryThyroidRetrospective cohort studyMiddle Agedmedicine.diseaseThyroid DiseasesGraves OphthalmopathyPsychotherapymedicine.anatomical_structureEndocrinologyCross-Sectional StudiesPhenotypeTreatment OutcomeMultivariate AnalysisQuality of LifeRegression AnalysisFemalebusinessPsychosocialhormones hormone substitutes and hormone antagonistsThyroid : official journal of the American Thyroid Association
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Differential regulation of Fas-mediated apoptosis in both thyrocyte and lymphocyte cellular compartments correlates with opposite phenotypic manifest…

2001

Several mechanisms are probably involved in determining the evolution of autoimmune thyroid disease (AITD) towards either hypothyroidism and the clinical syndrome known as Hashimoto's thyroiditis (HT) or toward hyperthyroidism and the symptoms of Graves' disease (GD). To gain further insight into such mechanisms we performed an exhaustive comparative analysis of the expression of key molecules regulating cell death (Fas, Fas ligand [FasL], Bcl-2) and apoptosis in both thyrocytes and thyroid infiltrating lymphocytes (TILs) from patients with either GD or HT. GD thyrocytes expressed less Fas/FasL than HT thyrocytes, whereas GD TILs had higher levels of Fas/FasL than HT TILs. GD thyrocytes exp…

AdultMaleendocrine systemmedicine.medical_specialtyProgrammed cell deathFas Ligand Proteinendocrine system diseasesEndocrinology Diabetes and MetabolismLymphocyteThyroid Glandchemical and pharmacologic phenomenaApoptosisThyroiditisFas ligandAutoimmune DiseasesEndocrinologyInternal medicinemedicineHumansLymphocytesRNA Messengerfas ReceptorCellular compartmentAgedMembrane GlycoproteinsChemistryReverse Transcriptase Polymerase Chain ReactionThyroidThyroiditis Autoimmunehemic and immune systemsMiddle Agedmedicine.diseasePhenotypeThyroid DiseasesGraves DiseaseEndocrinologymedicine.anatomical_structurePhenotypeGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisFemaleThyroid : official journal of the American Thyroid Association
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Relationship of eye muscle antibodies with HLA phenotypes and thyroid-stimulating immunoglobulins in endocrine orbitopathy.

1991

The relationship between endocrine orbitopathy and Graves' hyperthyroidism is still not clairified. The purpose of this study was to elucidate the incidence of eye muscle antibodies and the relationship with HLA phenotypes and thyroid antibodies in 65 patients with endocrine orbitopathy classes 1–5. Both bovine and abdominal muscles were used as antigens in ELISA systems in which IgG and IgM antibodies were assayed. Before starting the immunosuppressant therapy, 46/65 patients (71%) showed a positive result for eye muscle antibodies. Those patients with an active disease had such antibodies more frequently. Where the ophthalmopathy was of recent onset IgM antibodies were found, whereas pati…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesExophthalmosAdolescentmedicine.medical_treatmentAutoimmune DiseasesAntigenHLA AntigensInternal medicineMedicineExophthalmosHumansAgedAutoantibodiesAutoimmune diseasebiologybusiness.industryThyroidGeneral MedicineMiddle Agedmedicine.diseaseAnti-thyroid autoantibodiesGraves Diseasemedicine.anatomical_structureEndocrinologyPhenotypeImmunoglobulin MOculomotor MusclesImmunoglobulin GImmunologybiology.proteinThyroid Stimulating ImmunoglobulinThyroglobulinFemaleAntibodymedicine.symptombusinessImmunoglobulins Thyroid-StimulatingResearch in experimental medicine. Zeitschrift fur die gesamte experimentelle Medizin einschliesslich experimenteller Chirurgie
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The Atrial Natriuretic Peptide Genetic Variant rs5068 Is Associated With a Favorable Cardiometabolic Phenotype in a Mediterranean Population

2013

OBJECTIVE We hypothesized that the minor allele of the atrial natriuretic peptide (ANP) genetic variant rs5068 is associated with a favorable cardiometabolic phenotype in a general Mediterranean population. RESEARCH DESIGN AND METHODS We genotyped a random sample of the residents of Ventimiglia di Sicilia, Sicily, for rs5068. RESULTS Genotype frequencies of rs5068 are AA, 93.5%; AG, 6.4%; and GG, 0.1%. All subsequent analyses are AA versus AG+GG. After adjusting for age and sex, the minor G allele is associated with lower BMI (estimate [SE]: −1.7 kg/m2 [0.8], P = 0.04). In the AG+GG group, males with HDL cholesterol levels <40 mg/dL are less frequent (P = 0.05) and obesity tends to …

AdultMalegenetic variantCardiovascular and Metabolic Riskmedicine.medical_specialtySettore MED/09 - Medicina InternaGenotypeEndocrinology Diabetes and MetabolismPopulationBlood PressureGene Frequencycardiometabolic phenotypeAtrial natriuretic peptideInternal medicineNatriuretic Peptide Brainatrial natriuretic peptideInternal MedicinemedicineHumansAlleleeducationAllele frequencyAgedOriginal ResearchAdvanced and Specialized Nursingeducation.field_of_studybusiness.industryOdds ratioMiddle Agedmedicine.diseaseGenotype frequencyMinor allele frequencyPhenotypeEndocrinologyFemaleMetabolic syndromebusinessAtrial Natriuretic FactorDiabetes Care
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Epigenetic differences arise during the lifetime of monozygotic twins.

2005

Monozygous twins share a common genotype. However, most monozygotic twin pairs are not identical; several types of phenotypic discordance may be observed, such as differences in susceptibilities to disease and a wide range of anthropomorphic features. There are several possible explanations for these observations, but one is the existence of epigenetic differences. To address this issue, we examined the global and locus-specific differences in DNA methylation and histone acetylation of a large cohort of monozygotic twins. We found that, although twins are epigenetically indistinguishable during the early years of life, older monozygous twins exhibited remarkable differences in their overall…

AdultMalemedicine.medical_specialtyADNRestriction MappingTwinsMonozygotic twinX-inactivationEpigenesis GeneticHistonesX Chromosome InactivationSurveys and QuestionnairesGenotypemedicineHumansEpigeneticsOligonucleotide Array Sequence AnalysisGeneticsAnalysis of VarianceMultidisciplinarybiologyReverse Transcriptase Polymerase Chain ReactionElectrophoresis CapillaryGene Expression Regulation DevelopmentalAcetylationNucleic acid amplification techniqueDNASequence Analysis DNATwins MonozygoticDNA MethylationExpressió gènicaFenotipHistonePhenotypeSpainDNA methylationbiology.protein5-MethylcytosineCommentaryMedical geneticsBessonsFemaleGene expressionNucleic Acid Amplification TechniquesProceedings of the National Academy of Sciences of the United States of America
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Gender specific associations of the Trp64Arg mutation in the beta3-adrenergic receptor gene with obesity-related phenotypes in a Mediterranean popula…

2001

Abstract. Corella D, Guillen M, Portoles O, Sorli JV, Alonso V, Folch J, Saiz C (School of Medicine, University of Valencia, Valencia, Spain). Gender specific associations of the Trp64Arg mutation in the β3-adrenergic receptor gene with obesity-related phenotypes in a Mediterranean population: interaction with a common lipoprotein lipase gene variation. J Intern Med 2001; 250: 348–360. Objective. To investigate the association between the Trp64Arg β3-adrenergic receptor (ADRB3) mutation and obesity-related phenotypes in a Mediterranean Spanish population considering the effect of other genetic and environmental factors. Design and subject. Cross-sectional study in 1063 (476 men and 587 wome…

AdultMalemedicine.medical_specialtyAdolescentDNA Mutational AnalysisPopulationBiologyArginineGenetic determinismBody Mass IndexSex FactorsPolymorphism (computer science)Internal medicineGenotypeInternal MedicinemedicineHumansGenetic Predisposition to DiseaseObesityAlleleeducationAllelesAgedLipoprotein lipaseeducation.field_of_studyGenetic Carrier ScreeningBody WeightTryptophanGenetic VariationMiddle Agedmedicine.diseaseObesityLipoprotein LipasePhenotypeEndocrinologySpainReceptors Adrenergic beta-3FemaleBody mass indexJournal of Internal Medicine
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