Search results for "Phenylephrine"

showing 10 items of 47 documents

Cholinergic Responses of Ophthalmic Arteries in M3and M5Muscarinic Acetylcholine Receptor Knockout Mice

2009

PURPOSE. To determine the functional role of M 3 and M 5 muscarinic acetylcholine receptor subtypes in ophthalmic arteries using gene-targeted mice. METHODS. Muscarinic receptor gene expression was quantified in murine ophthalmic arteries using real-time PCR. To test the functional relevance of M 3 and M 5 receptors, ophthalmic arteries from mice deficient in either subtype (M3R -/- , M5R -/- , respectively) and wild-type controls were isolated, cannulated with micropipettes, and pressurized. Changes in luminal vessel diameter in response to muscarinic and nonmuscarinic receptor agonists were measured by video microscopy. RESULTS. With the use of real-time PCR, all five muscarinic receptor …

MaleNitroprussidemedicine.medical_specialtyCarbacholVasodilator AgentsCholinergic AgentsVideo RecordingGene ExpressionBiologyBradykininArticleMiceOphthalmic ArteryPhenylephrineInternal medicineMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M4medicineAnimalsRNA MessengerMice KnockoutReceptor Muscarinic M3Receptor Muscarinic M5Reverse Transcriptase Polymerase Chain ReactionMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2Muscarinic acetylcholine receptor M1AcetylcholineVasodilationEndocrinologyCholinergicCarbacholAdrenergic alpha-AgonistsAcetylcholinemedicine.drugInvestigative Opthalmology & Visual Science
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Demonstration of action-potential-producing cells in the rat pineal gland in vitro and their regulation by norepinephrine and nitric oxide

1998

There is evidence that sympathetically innervated mammalian pineal glands contain cells that exhibit action potentials. It is unknown whether ex vivo pineal glands deprived of their nervous input are still capable of firing. In the present study, multiple-unit recordings from rat pineals revealed spontaneously active cell clusters with a mean firing frequency of 1.5 +/- 0.3 Hz which could be abolished by tedrodotoxin. Regularly firing clusters showed no inherent periodicity in the minute range, whereas rhythmical clusters with periodically repeated bursts had period lengths of 12.6 min (day) and 9.5 min (night). Superfusion of norepinephrine reduced the firing frequency of both cluster type…

MaleNitroprussidemedicine.medical_specialtyPhysiologyPeriod (gene)8-Bromo Cyclic Adenosine MonophosphateAction PotentialsBiologyNitric OxideNitroargininePineal GlandNitric oxideRats Sprague-DawleyRat Pineal GlandNorepinephrine (medication)NorepinephrineBehavioral Neurosciencechemistry.chemical_compoundInternal medicinemedicineAnimalsSympathomimeticsCyclic GMPPhenylephrineInhibitory effectEcology Evolution Behavior and SystematicsNeuronsPenicillamineSulfhydryl ReagentsIsoproterenolIn vitroRatsElectrophysiologyEndocrinologychemistryAnimal Science and ZoologyEx vivomedicine.drugJournal of Comparative Physiology A: Sensory, Neural, and Behavioral Physiology
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Adrenoceptor- and cholinoceptor-mediated mechanisms in the regulation of 5-hydroxytryptamine release from isolated tracheae of newborn rabbits

1996

Abstract 1. Isolated tracheae of newborn rabbits were incubated in vitro and the outflow of 5-hydroxytryptamine (5-HT) was determined by h.p.l.c. with electrochemical detection. Evidence has previously been provided that this 5-HT outflow derives from neuroendocrine epithelial (NEE) cells of the airway mucosa. 2. Phenylephrine (1, 10 and 30 microM) enhanced the outflow of 5-HT by 80, 290 and 205%, respectively. 5-HT outflow evoked by 10 microM phenylephrine was not affected by the presence of the neurotoxin tetrodotoxin (1 microM). 3. Rauwolscine, ARC 239 (an alpha(2B)-adrenoceptor preferring antagonist), yohimbine and prazosin antagonized the effect of 10 microM phenylephrine in a concentr…

MaleSerotoninmedicine.medical_specialtyRauwolscineIn Vitro TechniquesMuscarinic Agonistschemistry.chemical_compoundIsoprenalineInternal medicineReceptors Adrenergic betaCyclic AMPmedicinePrazosinAnimalsReceptors CholinergicPhenylephrinePharmacologyForskolinMuscarineHydroxyindoleacetic AcidReceptors AdrenergicYohimbineTracheaEndocrinologyAnimals NewbornchemistryFemaleHexamethoniumRabbitsResearch Articlemedicine.drugBritish Journal of Pharmacology
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Pharmacologic pupil dilation as a predictive test for the risk for intraoperative floppy-iris syndrome.

2011

PURPOSE: To evaluate the effect of α1-adrenergic receptor antagonists (α1-ARAs) on pupil diameter and determine whether the diameter predicts intraoperative floppy-iris syndrome (IFIS). SETTING: Ophthalmology Section, Palermo University, Palermo, Italy. DESIGN: Prospective cohort study. METHODS: Male outpatients taking tamsulosin, α(1)-ARAs, or no α(1)-ARAs having phacoemulsification were recruited. Pupils were measured 1 month preoperatively, immediately preoperatively, and postoperatively under mesopic low (0.4 lux) and high (4.0 lux) illumination after pharmacologic dilation. The IFIS severity was graded. RESULTS: Each group comprised 50 patients. Pharmacologic dilation in both α(1)-ARA …

MaleTamsulosinmedicine.medical_specialtyMydriaticsmedicine.medical_treatmentIntraoperative floppy iris syndromeSensitivity and SpecificityPupilCohort StudiesPhenylephrineTropicamideTamsulosinPredictive Value of TestsRisk FactorsmedicinePupillary responseHumansProspective StudiesProspective cohort studyIntraoperative ComplicationsAgedSulfonamidesPhacoemulsificationbusiness.industrySettore MED/30 - Malattie Apparato VisivoDoxazosinfood and beveragesMuscle SmoothPupilPhacoemulsificationPrazosinSyndromebiochemical phenomena metabolism and nutritionmedicine.diseaseSensory SystemsSurgerycarbohydrates (lipids)OphthalmologyDrug CombinationsIfis pupil dilation tamsulosinIris DiseasesPredictive value of testsAdrenergic alpha-1 Receptor AntagonistsSurgerybusinessCohort studymedicine.drugJournal of cataract and refractive surgery
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The Janus face of chlorogenic acid on vascular reactivity: A study on rat isolated vessels

2016

Abstract Background Chlorogenic acid (CGA), the main polyphenol contained in coffee, is a major contributor to dietary polyphenol intake. Few studies reported its anti-hypertensive properties but the mechanisms are still indefinite. Purpose The present study assessed the direct effect of CGA in endothelium denuded or intact aortic rings from male Wistar rats and the mechanisms involved. Methods/Results CGA induced a direct endothelium-dependent relaxation that was significantly reduced by L-NAME (10 −4  M), indomethacin (10 −5  M) and combination of apamin (10 −7  M) and charybdotoxin (10 −7  M). Incubation of rings with CGA induced a dual effect on agonist-induced vasorelaxation. At 10 −6 …

Maleendocrine systemCharybdotoxinEndotheliumPharmaceutical ScienceVasodilation030204 cardiovascular system & hematologyPharmacologyApaminMuscle Smooth Vascular03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDrug DiscoverymedicineAnimals[CHIM]Chemical SciencesRats WistarPhenylephrineAntihypertensive AgentsComputingMilieux_MISCELLANEOUSPharmacology[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciencesRatsVasodilationmedicine.anatomical_structureComplementary and alternative medicineBiochemistrychemistry030220 oncology & carcinogenesisHypertensionMolecular MedicineSodium nitroprussideEndothelium Vascularmedicine.symptomChlorogenic AcidVasoconstrictionAcetylcholinemedicine.drug
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Fos-related antigen 2 (Fra-2) memorizes photoperiod in the rat pineal gland

2004

As the physiological role of fos-related antigen-2 (Fra-2) is largely unknown and since the pineal plays an important role in the photoperiodic control of the body, we have tested the hypothesis that Fra-2 expression is photoperiod-dependent and may be involved in imprinting photoperiod on the pineal gland and the body as a whole. To this end, we have investigated Fra-2 mRNA expression and Fra-2 protein expression under various light/dark (LD) cycles. A clear nocturnal increase occurs for both monitored parameters under all photoperiodic conditions studied. The level of Fra-2 protein expression clearly depends on photoperiod, because the amount of protein at dark onset and during the night …

Maleendocrine systemmedicine.medical_specialtyTime FactorsPhotoperiodBlotting WesternDeiodinaseFos-Related Antigen-2CREBPineal GlandRats Sprague-DawleyPhenylephrinePineal glandOrgan Culture TechniquesAcetyltransferasesInternal medicineCyclic AMPmedicineAnimalsDrug InteractionsProtein phosphorylationRNA MessengerCircadian rhythmCyclic GMPHeat-Shock ProteinsbiologyReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceIsoproterenolAdrenergic beta-AgonistsAdaptation PhysiologicalPeptide FragmentsRatsDNA-Binding ProteinsEndocrinologymedicine.anatomical_structureGene Expression RegulationIodothyronine deiodinasebiology.proteinArylalkylamineFemaleAdrenergic alpha-Agonistshormones hormone substitutes and hormone antagonistsTranscription FactorsEndocrine glandNeuroscience
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Alpha-adrenergic modulation of glutathione metabolism in isolated rat hepatocytes.

1988

Glutathione metabolism was studied in isolated hepatocytes from 48-h starved rats. Phenylephrine (10 microM, final concentration) was incubated in the presence of a mixture of L-glutamine, glycine, L-serine, and L-methionine (at 10 times their normal plasma concentration). Alpha-adrenergic stimulation provoked a decrease in glutathione (GSH) synthesis. This effect was accompanied by an enhanced efflux of glutathione from the cells. Phenylephrine stimulated the rate of glutathione disulfide (GSSG) formation; however, this effect was clearly insufficient to explain the disappearance of GSH. Our results suggest that the decrease in cellular GSH levels observed under conditions of shock, stress…

Malemedicine.medical_specialtyAdrenergic receptorPhysiologyEndocrinology Diabetes and MetabolismStimulationIn Vitro Techniqueschemistry.chemical_compoundPhenylephrineReference ValuesPhysiology (medical)Internal medicinemedicineAnimalsCysteineAmino AcidsPhenylephrineGlutathione DisulfideRats Inbred StrainsGlutathioneMetabolismGlutathioneRatsKineticsEndocrinologymedicine.anatomical_structurechemistryLiverHepatocyteGlutathione disulfideEffluxmedicine.drugThe American journal of physiology
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Involvement of endogenous nitric oxide in the inhibition by endotoxin and interleukin-1 beta of gastric acid secretion.

1994

Administration of Escherichia coli endotoxin abolished the acid secretory response induced by a bolus injection of pentagastrin in the continuously perfused stomach of the anaesthetized rat. Likewise, acid secretion stimulated by the continuous intravenous perfusion of pentagastrin was inhibited by administration of interleukin-1 beta (IL-1 beta). In both cases pretreatment with NG-nitro-L-arginine methyl ester (L-NAME) but not dexamethasone or indomethacin substantially restored the secretory responses to pentagastrin. The actions of L-NAME were reversed by the prior administration of L-arginine but not by its enantiomer D-arginine. Even though L-NAME increased blood pressure, this does no…

Malemedicine.medical_specialtyArginineIn Vitro TechniquesArginineNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundInternal medicineEscherichia coliMedicineAnimalsSecretionRats WistarPhenylephrineHepatologybusiness.industryStomachdigestive oral and skin physiologyGastroenterologyInterleukinRatsPentagastrinEndotoxinsEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterchemistryGastric acidFemalePentagastrinbusinessmedicine.drugInterleukin-1Journal of gastroenterology and hepatology
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Nitric oxide mediates the inhibition by interleukin-1β of pentagastrin-stimulated rat gastric acid secretion

1993

Bolus injection of interleukin-1 beta (2 micrograms kg-1, i.v.) inhibited acid secretion induced by intravenous infusion of pentagastrin (8 micrograms kg-1 h-1) in the continuously perfused stomach of the anaesthetized rat. Administration of interleukin-1 beta did not modify mean systemic arterial blood pressure. Pretreatment with NG-nitro-L-arginine methyl ester (L-NAME, 2-10 mg kg-1, i.v.), but not dexamethasone (5 mg kg-1, s.c. twice over 16 h), restored the acid secretory responses to pentagastrin. The actions of L-NAME were reversed by the prior administration of L-arginine (100 mg kg-1, i.v.), but not by its enantiomer D-arginine (100 mg kg-1, i.v.). L-NAME (5 mg kg-1, i.v.) increased…

Malemedicine.medical_specialtyBlood PressureBiologyPeptide hormoneArginineNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarInfusions IntravenousPhenylephrineDexamethasonePharmacologyStomachStereoisomerismRatsPentagastrinNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologychemistryGastrointestinal hormoneGastric acidFemalePentagastrinResearch ArticleInterleukin-1medicine.drugBritish Journal of Pharmacology
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Upregulation of Phospholipase D Expression and Activation in Ventricular Pressure-Overload Hypertrophy

2005

Evidence for a role of phospholipase D (PLD) in cellular proliferation and differentiation is accumulating. We studied PLD activity and expression in normal and hypertrophic rat and human hearts. In rat heart, abdominal aortic banding (constriction to 50% of original lumen) caused hypertrophy in the left ventricle (as shown by weight index and ANP expression) by about 15% after 30 days without histological evidence of fibrosis or signs of decompensation and in the right ventricle after 100 days. The hypertrophy was accompanied by small increases of basal PLD activity and strong potentiation of stimulated PLD activity caused by 4β-phorbol-12β,13α-dibutyrate (PDB) and by phenylephrine. The mR…

Malemedicine.medical_specialtyBlotting WesternCardiomegalyBiologyGene Expression Regulation EnzymologicMuscle hypertrophyRats Sprague-DawleyDownregulation and upregulationInternal medicinePhospholipase DVentricular PressuremedicineAnimalsHumansRNA MessengerPhenylephrineProtein Kinase CProtein kinase CPharmacologyReverse Transcriptase Polymerase Chain ReactionPhospholipase DPLD2lcsh:RM1-950Body WeightRatsReceptors AdrenergicUp-RegulationEnzyme ActivationIsoenzymeslcsh:Therapeutics. Pharmacologymedicine.anatomical_structureEndocrinologyVentricleVentricular pressureMolecular Medicinelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugJournal of Pharmacological Sciences
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