Search results for "Phosphodiesterase Inhibitors"

showing 10 items of 65 documents

Inhibition of cAMP Degradation Improves Regulatory T Cell-Mediated Suppression

2009

Abstract Naturally occurring regulatory T cells (nTreg cells) are crucial for the maintenance of peripheral tolerance. We have previously shown that a key mechanism of their suppressive action is based on a contact-dependent transfer of cAMP from nTreg cells to responder T cells. Herein, we further elucidate the important role of cAMP for the suppressive properties of nTreg cells. Prevention of cAMP degradation by application of the phosphodiesterase 4 inhibitor rolipram led to strongly increased suppressive potency of nTreg cells for Th2 cells in vitro and in vivo. Detailed analyses revealed that rolipram caused, in the presence of nTreg cells, a synergistic increase of cAMP in responder T…

Lung DiseasesPhosphodiesterase InhibitorsRegulatory T cellImmunologyCellEnzyme-Linked Immunosorbent AssayMice TransgenicInflammationBiologyT-Lymphocytes RegulatoryFlow cytometryMiceTh2 CellsIn vivoCyclic AMPHypersensitivityImmune TolerancemedicineAnimalsImmunology and AllergyCells CulturedRoliprammedicine.diagnostic_testPeripheral toleranceFlow CytometryCoculture TechniquesIn vitroCyclic Nucleotide Phosphodiesterases Type 4Cell biologymedicine.anatomical_structureImmunologymedicine.symptomRoliprammedicine.drugThe Journal of Immunology
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Benzimidazolones and renzapride facilitate acetylcholine release from guinea-pig myenteric plexus via 5-HT4 receptors

1995

The effects of the 5-HT4 receptor agonists BIMU 8, BIMU 1, renzapride and of the 5-HT1p receptor agonist 5-hydroxyindalpine on basal and electrically evoked outflow of tritium were studied in guinea-pig longitudinal muscle myenteric plexus preparations preincubated with [3H]choline. Muscle contractions were recorded simultaneously. BIMU 8 caused a calcium dependent and tetrodotoxin sensitive increase in basal [3H]outflow that was assumed to represent release of [3H]acetylcholine. In addition, BIMU 8 enhanced the release of [3H]acetylcholine and twitch contractions evoked by submaximal electrical stimulation. Ondansetron (1 μmol/l) did not change the effects of BIMU 8, but DAU 6285 and tropi…

MaleAgonistIBMXPhosphodiesterase Inhibitorsmedicine.drug_classGuinea PigsMyenteric PlexusStimulationIn Vitro TechniquesPharmacologyCholineBridged Bicyclo Compoundschemistry.chemical_compoundPiperidinesmedicineAnimalsMyenteric plexusPharmacologyGeneral MedicineBridged Bicyclo Compounds HeterocyclicAcetylcholineElectric StimulationSerotonin Receptor AgonistsRenzaprideNicotinic agonistchemistryReceptors SerotoninAnesthesiaBenzamidesCholinergicBenzimidazolesFemaleSerotonin AntagonistsAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Mechanisms underlying the inhibitory effects induced by pituitary adenylate cyclase-activating peptide in mouse ileum

2005

Abstract The aim of this study was to investigate the signal transduction mechanisms underlying the inhibitory effect induced by pituitary adenylate cyclase activating peptide (PACAP-27) on the spontaneous contractile activity of longitudinal muscle of mouse ileum. Mechanical activity of ileal segments was recorded isometrically in vitro. PACAP-27 produced apamin-sensitive reduction of the amplitude of the spontaneous contractions. 9-(Tetrahydro-2-furanyl)-9H-purin-6-amine (SQ 22,536), adenylate cyclase inhibitor, or genistein and tyrphostin 25, tyrosine kinase inhibitors, had negligible effects on PACAP-27-induced inhibition. PACAP-27 effects were significantly inhibited by U-73122, phopho…

MaleIndolesPhosphodiesterase InhibitorsVasodilator AgentsMouse ileumStimulationSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundInositolEnzyme InhibitorsEstrenesRyanodineRyanodine receptorProtein-Tyrosine KinasesTyrphostinsGenisteinPyrrolidinonesCell biologyPituitary adenylate cyclase-activating peptideNG-Nitroarginine Methyl EsterPituitary Adenylate Cyclase-Activating PolypeptideThapsigarginSignal transductionCyclopiazonic acidhormones hormone substitutes and hormone antagonistsMuscle ContractionBoron Compoundsendocrine systemmedicine.medical_specialtyThapsigarginMuscular inhibitionCalcium-Transporting ATPasesIn Vitro TechniquesInositol 145-triphosphateBiologyPACAP-27 (pituitary adenylate cyclase activating peptide)IleumPhospholipase CInternal medicinemedicineAnimalsPharmacologyDose-Response Relationship DrugPhospholipase CAdenineMuscle SmoothMice Inbred C57BLEndocrinologyApaminchemistryAdenylyl Cyclase InhibitorsCalciumNitric Oxide SynthaseEuropean Journal of Pharmacology
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Pentoxifylline Prevents Loss of PP2A Phosphatase Activity and Recruitment of Histone Acetyltransferases to Proinflammatory Genes in Acute Pancreatitis

2009

Mitogen-activated protein kinases (MAPKs) are considered major signal transducers early during the development of acute pancreatitis. Pentoxifylline is a phosphodiesterase inhibitor with marked anti-inflammatory properties through blockade of extracellular signal regulated kinase (ERK) phosphorylation and tumor necrosis factor alpha production. Our aim was to elucidate the mechanism of action of pentoxifylline as an anti-inflammatory agent in acute pancreatitis. Necrotizing pancreatitis induced by taurocholate in rats and taurocholate-treated AR42J acinar cells were studied. Phosphorylation of ERK and ERK kinase (MEK1/2), as well as PP2A, PP2B, and PP2C serine/threonine phosphatase activiti…

MaleMAPK/ERK pathwayChromatin ImmunoprecipitationPhosphodiesterase InhibitorsBlotting WesternPhosphataseAnti-Inflammatory AgentsPharmacologyBiologyCell LinePentoxifyllineProinflammatory cytokineCyclic AMPPhosphoprotein PhosphatasesmedicineAnimalsPentoxifyllineRats WistarExtracellular Signal-Regulated MAP KinasesHistone AcetyltransferasesInflammationPharmacologyReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaProtein phosphatase 2medicine.diseaseCyclic Nucleotide Phosphodiesterases Type 2RatsEnzyme ActivationPancreatitisBiochemistryAcute DiseaseRNAMolecular MedicinePhosphorylationPancreatitisMitogen-Activated Protein KinasesChromatin immunoprecipitationmedicine.drugJournal of Pharmacology and Experimental Therapeutics
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Pharmacological activity of PF-904 in guinea pig in vivo, and on human bronchus and neutrophils in vitro.

1997

The effects of PF-904 (4-amino-1-ethyl-6-methylpyrazino[2,3-c][1,2,6]thiadiazine 2,2-dioxide), a pyrazinothiadiazine derivative, were examined in guinea-pig airways in vivo, in human isolated bronchus and human polymorphonuclear leukocytes. PF-904 (12.5-200 mg/kg, intraduodenal) reduced bronchoconstriction in response to histamine, arachidonic acid, platelet-activating factor (PAF) and methacholine. PF-904 (50-200 mg/kg) prevented PAF-induced airways hyperreactivity and inhibited antigen-induced bronchoconstriction, airway microvascular leakage and eosinophil lung accumulation, but antigen-induced airways hyperresponsiveness was not reduced. PF-904 (1 microM-1 mM) produced complete inhibiti…

MaleNeutrophilsPhosphodiesterase InhibitorsGuinea PigsBronchiPharmacologyIn Vitro TechniquesBronchial Provocation TestsCapillary Permeabilitychemistry.chemical_compoundIn vivoSuperoxidesmedicineAnimalsHumansAnti-Asthmatic AgentsPlatelet Activating FactorRolipramPharmacologyBronchusThiadiazinesAnti-Inflammatory Agents Non-SteroidalPhosphodiesteraseBiological activityrespiratory systemBronchodilator AgentsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryPyrazinesBronchoconstrictionMethacholinemedicine.symptomBronchial HyperreactivityHistaminemedicine.drugEuropean journal of pharmacology
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In rat pinealocytes the cyclic GMP response to NO is regulated by Ca2+ and protein kinase C

1995

There is ample evidence that beta-adrenergic stimulation of cyclic GMP formation is potentiated by alpha1-adrenergic mechanisms, the latter leading to elevation of intracellular Ca2+ concentration ([Ca2+]i) and protein kinase C (PKC) activation. Recent studies have shown that nitric oxide synthase (NOS) and nitric oxide (NO) are a component of the adrenoceptor-cyclic GMP signalling pathway. The aim of the present investigation was to study the roles of alpha1-adrenergic mechanisms, Ca2+ and PKC on NO-stimulated cyclic GMP formation. To this end suspension cultures of rat pinealocytes were treated with the NO donor sodium nitroprusside (SNP) in the presence of alpha1-adrenergic agonists, [Ca…

MaleNitroprussidePhosphodiesterase InhibitorsNitric OxidePineal GlandPinealocyteNitric oxideRats Sprague-DawleyPhenylephrinechemistry.chemical_compoundCalmodulinmedicineAnimalsOuabainCyclic GMPProtein Kinase InhibitorsMolecular BiologyProtein Kinase CProtein kinase CbiologyKinaseGeneral NeuroscienceIsoproterenolPhosphodiesterasePhosphoric Monoester HydrolasesRatsNitric oxide synthaseCytosolBiochemistrychemistrybiology.proteinCalciumNeurology (clinical)Sodium nitroprussideDevelopmental Biologymedicine.drugBrain Research
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Effects of selective phosphodiesterase inhibitors on platelet-activating factor- and antigen-induced airway hyperreactivity, eosinophil accumulation,…

1996

There is currently interest in the potential use of selective inhibitors of cyclic nucleotide phosphodiesterases (PDE) in the treatment of asthma. In this study we examined the effects of three selective PDE inhibitors, milrinone (PDE III), rolipram (PDE IV) and zaprinast (PDE V), on the broncoconstriction produced by antigen and histamine, the airway hyperreactivity and microvascular leakage after aerosol exposure to platelet-activating factor (PAF) and antigen, and the antigen-induced eosinophil infiltration in guinea-pig lung. Inhaled rolipram (0.01-10 mg ml-1) inhibited dose dependently the bronchospasm produced by aerosol antigen (5 mg ml-1) an anaesthetised, ventilated guinea-pigs. Ro…

MalePhosphodiesterase InhibitorsGuinea PigsRespiratory SystemPharmacologychemistry.chemical_compoundmedicineAnimalsPlatelet Activating FactorRolipramPharmacologyPlatelet-activating factorDose-Response Relationship DrugChemistryPhosphodiesteraseGeneral Medicinerespiratory systemExtravasationAsthmaPyrrolidinonesEosinophilsImmunologyMilrinoneBronchoconstrictionmedicine.symptomZaprinastRolipramHistaminemedicine.drugHistamineNaunyn-Schmiedeberg's archives of pharmacology
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Selective action of two aporphines at alpha 1-adrenoceptors and potential-operated Ca2+ channels.

1993

Abstract Contractions evoked by noradrenaline (1 μM) or a depolarizing solution of 60 mM KCI were concentration dependently depressed by the aporphine alkaloids (S)-boldine and (R)-apomorphine in rataorta. Both drugs had a greater inhibitory potency on the contraction elicited by noradrenaline. Dose-response curves for noradrenaline were shifted to the right in presence of (S)-boldine. (R)-Apomorphine acted by a complex mechanism at α 1 -adrenoceptors and its inhibitory effects was irreversible. The conformational features of these alkaloids may explain their different behaviour at α 1 -adrenoceptors. In Ca 2+ -free solution, the alkaloids inhibited the contraction evoked by noradrenaline b…

MaleReceptor complexAporphinesApomorphineStereochemistryPhosphodiesterase InhibitorsMolecular ConformationIn Vitro TechniquesMuscle Smooth VascularPotassium Chloridechemistry.chemical_compoundNorepinephrineRadioligand AssaymedicinePrazosinBoldineAnimalsAporphineRats WistarEvoked PotentialsPharmacologyMembranesAlkaloidDihydropyridinePhosphodiesteraseReceptors Adrenergic alphaRatsAntitussive AgentsMechanism of actionchemistryCattleCalcium Channelsmedicine.symptommedicine.drugMuscle ContractionEuropean journal of pharmacology
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Multiple actions of glaucine on cyclic nucleotide phosphodiesterases, α1-adrenoceptor and benzothiazepine binding site at the calcium channel

1992

1. In the present study, the properties of glaucine (an aporphine structurally related to papaverine) were compared with those of papaverine, diltiazem, nifedipine and prazosin. The work includes functional studies on rat isolated aorta contracted with noradrenaline, caffeine or KCl, and a determination of the affinity of glaucine at calcium channel binding sites of alpha-adrenoceptors, by use of [3H]-(+)-cis-diltiazem, [3H]-nitrendipine and [3H]-prazosin binding to cerebral cortical membranes. The effects of glaucine on the different molecular forms of cyclic nucleotide phosphodiesterases (PDE) isolated from bovine aorta were also determined. 2. Contraction evoked by noradrenaline (1 micro…

MaleReceptor complexAporphinesPhosphodiesterase InhibitorsStereochemistryAorta ThoracicIn Vitro TechniquesPharmacologyBinding CompetitiveMuscle Smooth VascularNorepinephrineRadioligand Assaychemistry.chemical_compoundPrazosinmedicineAnimalsRats WistarPharmacologyPapaverineVoltage-dependent calcium channelChemistryCalcium channelDihydropyridinePhosphodiesterasePrazosinReceptors Adrenergic alphaGlaucineRats3'5'-Cyclic-AMP Phosphodiesterasescardiovascular systemCattleCalcium ChannelsResearch ArticleMuscle Contractionmedicine.drugBritish Journal of Pharmacology
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Investigations of the dual contractile/relaxant properties showed by antioquine in rat aorta.

1993

1. In the present study we assessed the activity of antioquine, a bisbenzyltetrahydroisoquinoline alkaloid isolated from Pseudoxandra sclerocarpa, by examining its effects on the contractile activity of rat isolated aorta, specific binding of [3H]-(+)-cis-diltiazem, [3H]-nitrendipine and [3H]-prazosin to cerebral cortical membranes and the different molecular forms of cyclic nucleotide phosphodiesterases (PDE) isolated from bovine aorta. 2. Contractions in rat aorta induced by high concentrations of KCl (80 mM) and noradrenaline (1 microM) were inhibited by antioquine in a concentration-dependent manner (0.1 microM- 300 microM). The alkaloid appeared more potent against KCl-induced contract…

MaleReceptor complexmedicine.medical_specialtyPhosphodiesterase InhibitorsMuscle RelaxationReceptors Drugchemistry.chemical_elementAorta ThoracicCalciumIn Vitro TechniquesBenzylisoquinolinesCalcium in biologyMuscle Smooth VascularNorepinephrineRadioligand AssayAlkaloidsCytosolInternal medicineCaffeinemedicinePrazosinExtracellularAnimalsRats WistarPharmacologyCyclic nucleotide phosphodiesteraseChemistryPhosphoric Diester HydrolasesCalcium channelDihydropyridineCalcium Channel BlockersPyrrolidinonesRatsKineticsEndocrinologyBiophysicsCalciumCattleRoliprammedicine.drugMuscle ContractionResearch ArticleBritish journal of pharmacology
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