Search results for "Phosphodiesterase Inhibitors"

showing 10 items of 65 documents

Adrenergic modulation of the release of 5-hydroxytryptamine from the vascularly perfused ileum of the guinea-pig

1988

1. Isolated segments of the guinea-pig ileum were vascularly perfused and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent was determined by h.p.l.c. with electrochemical detection. Test substances were applied via the arterial perfusion medium. 2. Isoprenaline (0.1 microM) increased the outflow of 5-HT and 5-HIAA maximally by about 75% and this was antagonized by propranolol (0.1 microM). Forskolin (1-10 microM) increased the outflow of 5-HT by approximately 105% and that of 5-HIAA by approximately 55%. The phosphodiesterase inhibitor AH 21-132 (0.1-1 microM) increased the outflow of 5-HT and 5-HIAA by about 70%. Isoprenaline…

MaleSerotoninmedicine.medical_specialtyPhosphodiesterase InhibitorsAdrenergic beta-AntagonistsGuinea PigsPropranololClonidinechemistry.chemical_compoundIleumIsoprenalineInternal medicinemedicinePrazosinAnimalsNaphthyridinesPhosphodiesterase inhibitorAdrenergic alpha-AntagonistsPharmacologyForskolinColforsinIsoproterenolPhosphodiesteraseAdrenergic beta-AgonistsHydroxyindoleacetic AcidPerfusionEndocrinologychemistryTetrodotoxinEnterochromaffin cellAdrenergic alpha-AgonistsResearch Articlemedicine.drugBritish Journal of Pharmacology
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Phosphodiesterase 10A in the Rat Pineal Gland: Localization, Daily and Seasonal Regulation of Expression and Influence on Signal Transduction

2010

The cyclic nucleotide phosphodiesterase 10A (PDE10A) is highly expressed in striatal spiny projection neurons and represents a therapeutic target for the treatment of psychotic symptoms. As reported previously [J Biol Chem 2009; 284:7606–7622], in this study PDE10A was seen to be additionally expressed in the pineal gland where the levels of PDE10A transcript display daily changes. As with the transcript, the amount of PDE10A protein was found to be under daily and seasonal regulation. The observed cyclicity in the amount of PDE10A mRNA persists under constant darkness, is blocked by constant light and is modulated by the lighting regime. It therefore appears to be driven by the master cloc…

Malemedicine.medical_specialtyAANATPhosphodiesterase InhibitorsEndocrinology Diabetes and MetabolismBlotting WesternBiologyReal-Time Polymerase Chain ReactionPineal GlandRats Sprague-DawleyCellular and Molecular NeurosciencePineal glandEndocrinologyOrgan Culture TechniquesInternal medicinePapaverinemedicineCyclic AMPAnimalsImmunoprecipitationProtein kinase A signalingCyclic GMPDNA PrimersPhosphodiesterase 10A;Circadian system;Pineal gland;Norepinephrine;cAMP;cGMP;Arylalkylamine N-acetyltransferaseEndocrine and Autonomic SystemsSuprachiasmatic nucleusPhosphoric Diester HydrolasesPhosphodiesteraseImmunohistochemistryCircadian RhythmRatsddc:medicine.anatomical_structureEndocrinologySecond messenger systemRNAFemaleSuprachiasmatic NucleusPDE10ASeasonsSignal transductionAdrenergic alpha-AgonistsSignal Transduction
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Evaluation of an alternative dosing regimenwith tadalafil, three times per week, for men with erectile dysfunction: SURE study in Italy.

2007

AIM: To examine the preference for two dosing regimens of 20 mg of tadalafil, on demand or three times per week, in men affected with erectile dysfunction (ED) in Italy. METHODS: Scheduled Use versus on demand Regimen Evaluation (SURE) is a multicenter, crossover and open-label study, involving 94 urology centers in Italy. Patients aged 18 years or older affected with ED for at least 3 months were enrolled and randomized to 20 mg of tadalafil treatment on demand or three times per week for 5-6 weeks. After a 1-week washout, patients were crossed over to the alternate regimen for 5-6 weeks. A treatment preference question was used to determine the preferred treatment regimen. International I…

Malemedicine.medical_specialtyAlternate; Erectile dysfunction; On demand; SURE study; Tadalafil; Three times per week; Carbolines; Cross-Over Studies; Drug Administration Schedule; Erectile Dysfunction; Humans; Italy; Male; Middle Aged; Phosphodiesterase Inhibitors; Tadalafil; Treatment Outcome; Nephrology; EndocrinologyPhosphodiesterase Inhibitorserectile dysfunctionUrologyerectile dysfunction; SURE study; on demand; three times per week; alternate; tadalafilon demandDrug Administration Schedulelaw.inventionSettore MED/24 - UrologiaEndocrinologyRandomized controlled triallawOn demandInternal medicinealternateMedicineHumansClinical significanceDosingthree times per weekErectile dysfunction SURE Study on demand three times per week alternate tadalafilSURE studyCross-Over Studiesbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseCrossover studyTadalafilSurgeryRegimenErectile dysfunctionTreatment OutcomeItalyNephrologyAlternate; Erectile dysfunction; On demand; SURE study; Tadalafil; Three times per week; Carbolines; Cross-Over Studies; Drug Administration Schedule; Erectile Dysfunction; Humans; Italy; Male; Middle Aged; Phosphodiesterase Inhibitors; Tadalafil; Treatment Outcomebusinesstadalafilmedicine.drugCarbolines
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The effect of S-(+)-boldine on the α1-adrenoceptor of the guinea-pig aorta

1996

1. The cardiovascular activity of S-(+)-boldine, an aporphine alkaloid structurally related to papaverine, was determined. The work includes functional studies on guinea-pig isolated aorta contracted with noradrenaline, caffeine, KCl or Ca2+, and on guinea-pig trachea contracted with acetylcholine or histamine. 2. S-(+)-boldine inhibited in a concentration-dependent manner the contractile response evoked by noradrenaline (10 microM) in guinea-pig aorta (IC50 = 1.4 +/- 0.2 microM) while the KCl depolarizing solution (60 mM)- or the Ca2+ (1 mM)-induced contractions were only partially affected by boldine up to 300 microM. In contrast, papaverine relaxed noradrenaline (NA), KCl or Ca2+ induced…

Malemedicine.medical_specialtyAporphinesContraction (grammar)Phosphodiesterase InhibitorsMuscle RelaxationGuinea PigsAorta ThoracicIn Vitro TechniquesPhosphatidylinositolsMuscle Smooth Vascularchemistry.chemical_compoundCaffeinePapaverineReceptors Adrenergic alpha-1Internal medicinemedicine.arterymedicinePrazosinAnimalsBoldineThoracic aortaAdrenergic alpha-AntagonistsPharmacologyPapaverineParasympatholyticsCalcium Channel BlockersTracheaEndocrinologychemistryCompetitive antagonistAdrenergic alpha-1 Receptor AntagonistsHistamineAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Role of K+ and Ca2+ fluxes in the cerebroarterial vasoactive effects of sildenafil

2007

The aim of this study was to assess the role of K(+) and Ca(2+) fluxes in the cerebroarterial vasoactive effects of the phosphodiesterase-5 inhibitor sildenafil. We used isolated rabbit basilar arteries to assess the effects of extracellular K(+) raising on sildenafil-induced vasodilatation, and studied the pharmacological interaction of sildenafil with selective modulators of membrane K(+) and Ca(2+) channels. Expression of Kv1 subunits of K(+) channels was assessed at messenger and protein levels. Parallel experiments were carried out with zaprinast for comparison. Sildenafil (10 nM-0.1 mM) induced concentration-dependent relaxation of endothelin-1 (10 nM)-precontracted arteries, which wa…

Malemedicine.medical_specialtyCalcium Channels L-Typemedicine.drug_mechanism_of_actionPhosphodiesterase InhibitorsVasodilationIn Vitro TechniquesPharmacologyPiperazinesSildenafil Citratechemistry.chemical_compoundInternal medicinemedicineAnimalsChannel blockerRNA MessengerSulfonesPharmacologyTetraethylammoniumDose-Response Relationship DrugChemistryDepolarization3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterIberiotoxinEndocrinologyPurinesBasilar ArterycGMP-specific phosphodiesterase type 5PotassiumShaker Superfamily of Potassium ChannelsCalciumRabbitsZaprinastPhosphodiesterase 5 inhibitorEuropean Journal of Pharmacology
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Differential effects of isoliquiritigenin and YC-1 in rat aortic smooth muscle.

1997

We investigated the effects of isoliquiritigenin and YC-1 (3-(5'-hydroxymethyl-2'-furyl)-1-benzyl indazole) on tension in endothelial-free rat aortic rings precontracted with phenylephrine (3 microM). Both compounds induced a concentration-dependent relaxation (EC50 of YC-1 1.9 microM and of isoliquiritigenin 9.4 microM). The effects developed faster with YC-1 than with isoliquiritigenin, and the effects of YC-1 were potentiated by isoliquiritigenin (10 microM). 1H-[1,2,4]Oxadiazolo[4,3-a]quinoxalin-1-one (30 microM) inhibited the effect of YC-1, but not of isoliquiritigenin. These results suggest that the effects of YC-1 are due to stimulation of soluble guanylyl cyclase activity, whereas …

Malemedicine.medical_specialtyIndazolesPhosphodiesterase InhibitorsMuscle RelaxationStimulationMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundChalconeChalconesAldehyde ReductaseInternal medicinemedicineAnimalsEnzyme InhibitorsPhenylephrinePharmacologybiologyDose-Response Relationship DrugChemistryBiological activityRatsDose–response relationshipEndocrinologyCarotid ArteriesMechanism of actionEnzyme inhibitorGuanylate Cyclasebiology.proteinFemalemedicine.symptomSoluble guanylyl cyclaseIsoliquiritigeninPlatelet Aggregation Inhibitorsmedicine.drugMuscle ContractionEuropean journal of pharmacology
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Characterization of two different Ca2+ entry pathways dependent on depletion of internal Ca2+ pools in rat aorta

1998

Ryanodine (10 microM), thapsigargin (1 microM) and cyclopiazonic acid (10 microM) produced a slow, sustained contractile response in rat aorta that only can be observed in Ca2+-containing solution. In Ca2+-free medium, no response to the drugs was obtained, which suggests that the contraction elicited in presence of Ca2+ is mainly due to the contribution of extracellular influx. This Ca2+ entry does not depend on the opening of dihydropyridine-dependent Ca2+-channels for nimodipine does not affect this. Noradrenaline (1 microM) induced a biphasic response in Ca2+-free medium that was mediated by two different Ca2+ compartments, one of which is common to caffeine (10 mM), and is also deplete…

Malemedicine.medical_specialtyIndolesThapsigarginContraction (grammar)Phosphodiesterase InhibitorsVasodilator AgentsAorta ThoracicIn Vitro TechniquesMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundCaffeineInternal medicinemedicine.arterymedicineExtracellularAnimalsVasoconstrictor AgentsRats WistarCa2 entryNimodipinePharmacologyAortaRyanodineRyanodine receptorGeneral MedicineRatsEndocrinologychemistryBiophysicsThapsigarginCalciumCalcium ChannelsCyclopiazonic acidMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Stroke after tadalafil use

2013

No abstract available

Malemedicine.medical_specialtyNeurologyStroke; tadalafil Phosphodiesterase inhibitorsDermatologyTadalafilInternal medicinemedicineHumansStrokeNeuroradiologymedicine.diagnostic_testCerebral infarctionbusiness.industrySettore MED/44 - Medicina Del LavoroBrainMagnetic resonance imagingGeneral MedicineMiddle AgedPhosphodiesterase 5 Inhibitorsmedicine.diseaseMagnetic Resonance ImagingTadalafilStrokePsychiatry and Mental healthSexual Dysfunction PhysiologicalSexual dysfunctionCardiologyNeurology (clinical)Neurosurgerymedicine.symptombusinesstadalafil Phosphodiesterase inhibitorsmedicine.drugCarbolines
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Relaxant effect of sildenafil in the rabbit basilar artery

2005

We hypothesized that sildenafil, inhibitor of phosphodiesterase-5 (PDE-5), interacts with the nitric oxide (NO)-cGMP pathway in the cerebral arteries and shows vasoactive effects. To prove it in the isolated rabbit basilar artery, we compared the effects of sildenafil with other PDE-5 inhibitors, assessed the endothelial dependence of the vasoactive responses, and used modulators of the cGMP and cAMP signaling processes. Sildenafil (10 nM-0.1 mM) induced concentration-dependent relaxations of endothelin-1 (10 nM)-precontracted basilar artery, which were partially inhibited both in endothelium-denuded arteries and in arteries precontracted by depolarization with KCl (50 mM). Endothelin-1 (1 …

Malemedicine.medical_specialtyPhosphodiesterase InhibitorsPhysiologySildenafilVasodilator AgentsCerebral arteriesVasodilationIn Vitro TechniquesPiperazinesSildenafil Citratechemistry.chemical_compound3'5'-Cyclic-GMP PhosphodiesterasesQuinoxalinesmedicine.arteryInternal medicinemedicineBasilar arteryAnimalsSulfonesCyclic Nucleotide Phosphodiesterases Type 5PharmacologyOxadiazolesDose-Response Relationship DrugPhosphoric Diester HydrolasesPDE5 drug designVasodilationNG-Nitroarginine Methyl EsterEndocrinologychemistryGuanylate CyclasePurinesBasilar Arterycardiovascular systemMolecular MedicineRabbitsSodium nitroprussideNitric Oxide SynthaseSoluble guanylyl cyclaseZaprinastSignal Transductionmedicine.drugVascular Pharmacology
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EAU guidelines on the treatment and follow-up of non-neurogenic male lower urinary tract symptoms including benign prostatic obstruction.

2013

To present a summary of the 2013 version of the European Association of Urology guidelines on the treatment and follow-up of male lower urinary tract symptoms (LUTS). We conducted a literature search in computer databases for relevant articles published between 1966 and 31 October 2012. The Oxford classification system (2001) was used to determine the level of evidence for each article and to assign the grade of recommendation for each treatment modality. Men with mild symptoms are suitable for watchful waiting. All men with bothersome LUTS should be offered lifestyle advice prior to or concurrent with any treatment. Men with bothersome moderate-to-severe LUTS quickly benefit from α1-blocke…

Malemedicine.medical_specialtyPhosphodiesterase Inhibitorsmedicine.medical_treatmentUrologyUrologyProstatic HyperplasiaMuscarinic AntagonistsSeverity of Illness Indexchemistry.chemical_compound5-alpha Reductase InhibitorsLower Urinary Tract SymptomsLower urinary tract symptomsmedicineNocturiaHumansWatchful WaitingTransurethral resection of the prostateUrinary retentionProstatectomybusiness.industryTransurethral Resection of Prostatemedicine.diseaseDutasterideTreatment OutcomechemistryAdrenergic alpha-1 Receptor AntagonistsUrological AgentsProstate surgeryStentsmedicine.symptombusinessUrinary CatheterizationRisk Reduction BehaviorWatchful waitingEuropean urology
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