Search results for "Phosphorylation"

showing 10 items of 975 documents

Impact of Oxidative Stress on the Heart and Vasculature

2017

Abstract Vascular disease and heart failure impart an enormous burden in terms of global morbidity and mortality. Although there are many different causes of cardiac and vascular disease, most causes share an important pathological mechanism: oxidative stress. In the failing heart, oxidative stress occurs in the myocardium and correlates with left ventricular dysfunction. Reactive oxygen species (ROS) negatively affect myocardial calcium handling, cause arrhythmia, and contribute to cardiac remodeling by inducing hypertrophic signaling, apoptosis, and necrosis. Similarly, oxidative balance in the vasculature is tightly regulated by a wealth of pro- and antioxidant systems that orchestrate r…

0301 basic medicinechemistry.chemical_classificationReactive oxygen speciesNecrosisVascular diseasebusiness.industryAngiogenesisOxidative phosphorylation030204 cardiovascular system & hematologymedicine.diseasemedicine.disease_causeCell biology03 medical and health sciences030104 developmental biology0302 clinical medicinechemistryApoptosisHeart failureImmunologymedicinemedicine.symptomCardiology and Cardiovascular MedicinebusinessOxidative stressJournal of the American College of Cardiology
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Evaluation of Carotenoids Protection Against Oxidative Stress in the Animal Model Caenorhabditis elegans

2019

The nematode Caenorhabditis elegans is a versatile and powerful model organism for animal experimental research and, despite being an invertebrate, displays remarkably similar molecular bases and conserved cellular pathways to those of humans. Oxidative stress is an etiological factor that influences numerous diseases, degenerative processes and aging. C. elegans has revealed as an opportune and feasible organism to investigate the antioxidant effects of different bioactives or complex food matrices, and a number of protocols have been developed by using different oxidative stressors. Carotenoids are recognized as quenchers and scavengers of reactive oxygen species, and many of their relate…

0301 basic medicinechemistry.chemical_classificationReactive oxygen specieseducation.field_of_studyAntioxidantved/biologymedicine.medical_treatmentPopulationved/biology.organism_classification_rank.speciesOxidative phosphorylationBiologymedicine.disease_causebiology.organism_classification03 medical and health sciences030104 developmental biology0302 clinical medicinechemistryBiochemistrymedicineeducationModel organismCarotenoid030217 neurology & neurosurgeryOxidative stressCaenorhabditis elegans
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An Ethanolic Extract of Ampelopsis Radix Exerts Anti-colorectal Cancer Effects and Potently Inhibits STAT3 Signaling In Vitro

2017

Colorectal cancer (CRC) is a leading cause of cancer-related morbidity and mortality worldwide. Signal transducer and activator of transcription 3 (STAT3) signaling is constantly activated in CRC, and has been proposed as a pathogenic factor and a therapeutic target of CRC. Ampelopsis Radix (AR), a traditional Chinese medicinal herb, possesses low toxicity and has long been used clinically for the treatment of cancers including CRC. Some constituents of AR have been reported to exert anticancer properties by targeting STAT3. However, the anti-CRC mode and mechanism of action of AR have not been fully elucidated. Here, we investigated the involvement of STAT3 signaling in the anti-CRC effect…

0301 basic medicinecolorectal cancerBiologyPharmacologymigrationSTAT303 medical and health sciences0302 clinical medicinemedicineCytotoxic T cellPharmacology (medical)STAT3neoplasmsOriginal ResearchPharmacologyCell growthlcsh:RM1-950apoptosisCell migrationAmpelopsis Radixinvasiondigestive system diseaseslcsh:Therapeutics. Pharmacology030104 developmental biologyMechanism of actionApoptosis030220 oncology & carcinogenesisSTAT proteinbiology.proteinPhosphorylationmedicine.symptomFrontiers in Pharmacology
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Neuroactive Steroids Reverse Tonic Inhibitory Deficits in Fragile X Syndrome Mouse Model

2018

Fragile X syndrome (FXS) is the most common form of inherited intellectual disability. A reduction in neuronal inhibition mediated by γ-aminobutyric acid type A receptors (GABAARs) has been implicated in the pathophysiology of FXS. Neuroactive steroids (NASs) are known allosteric modulators of GABAAR channel function, but recent studies from our laboratory have revealed that NASs also exert persistent metabotropic effects on the efficacy of tonic inhibition by increasing the protein kinase C (PKC)-mediated phosphorylation of the α4 and β3 subunits which increase the membrane expression and boosts tonic inhibition. We have assessed the GABAergic signaling in the hippocampus of fragile X ment…

0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyNeuroactive steroidGABAA receptor (GABAAR)fragile XInhibitory postsynaptic potentialTonic (physiology)lcsh:RC321-571tonic inhibition03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineInternal medicinemedicineMolecular Biologylcsh:Neurosciences. Biological psychiatry. NeuropsychiatryProtein kinase COriginal ResearchChemistryphosphorylationDentate gyrusFMR1030104 developmental biologyEndocrinologyMetabotropic receptorGABAergicneurosteroidbenzodiazepine030217 neurology & neurosurgeryNeuroscienceFrontiers in Molecular Neuroscience
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Cytoprotective Effects of Dinitrosyl Iron Complexes on Viability of Human Fibroblasts and Cardiomyocytes

2019

Nitric oxide (NO) is an important signaling molecule that plays a key role in maintaining vascular homeostasis. Dinitrosyl iron complexes (DNICs) generating NO are widely used to treat cardiovascular diseases. However, the involvement of DNICs in the metabolic processes of the cell, their protective properties in doxorubicin-induced toxicity remain to be clarified. Here, we found that novel class of mononuclear DNICs with functional sulfur-containing ligands enhanced the cell viability of human lung fibroblasts and rat cardiomyocytes. Moreover, DNICs demonstrated remarkable protection against doxorubicin-induced toxicity in fibroblasts and in rat cardiomyocytes (H9c2 cells). Data revealed t…

0301 basic medicinedonors nitric oxideCellOxidative phosphorylationdinitrosyl iron complexesheart diseaseMitochondrionNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicinePharmacology (medical)Viability assayInner mitochondrial membranecell viabilityOriginal Researchchemistry.chemical_classificationPharmacologyReactive oxygen specieslcsh:RM1-950GlutathioneCell biology030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistry030220 oncology & carcinogenesismembrane potentialFrontiers in Pharmacology
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Evidence That Graves' Ophthalmopathy Immunoglobulins Do Not Directly Activate IGF-1 Receptors

2018

Background: Graves' ophthalmopathy (GO) pathogenesis involves thyrotropin (TSH) receptor (TSHR)-stimulating autoantibodies. Whether there are autoantibodies that directly stimulate insulin-like growth factor 1 receptors (IGF-1Rs), stimulating insulin-like growth factor receptor antibodies (IGFRAbs), remains controversial. This study attempted to determine whether there are stimulating IGFRAbs in patients with GO. Methods: Immunoglobulins (Igs) were purified from normal volunteers (NV-Igs) and patients with GO (GO-Igs). The effects of TSH, IGF-1, NV-Igs, and GO-Igs on pAKT and pERK1/2, members of pathways used by IGF-1R and TSHR, were compared in orbital fibroblasts from GO patients (GOFs) a…

0301 basic medicineendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismImmunoglobulins030209 endocrinology & metabolismCell LineReceptor IGF Type 1PathogenesisGraves' ophthalmopathy03 medical and health sciences0302 clinical medicineEndocrinologymedicineHumansImmunology Autoimmunity and Graves' OphthalmopathyPhosphorylationReceptorbiologybusiness.industryAutoantibodyReceptors Thyrotropinmedicine.diseaseeye diseasesGraves Ophthalmopathy030104 developmental biologyImmunologybiology.proteinlipids (amino acids peptides and proteins)AntibodybusinessProto-Oncogene Proteins c-akthormones hormone substitutes and hormone antagonists
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Phosphorylated immunoreceptor tyrosine-based activation motifs and integrin cytoplasmic domains activate spleen tyrosine kinase via distinct mechanis…

2018

Spleen tyrosine kinase (Syk) is involved in cellular adhesion and also in the activation and development of hematopoietic cells. Syk activation induced by genomic rearrangement has been linked to certain T-cell lymphomas, and Syk inhibitors have been shown to prolong survival of patients with B-cell lineage malignancies. Syk is activated either by its interaction with a double-phosphorylated immunoreceptor tyrosine-based activation motif (pITAM), which induces rearrangements in the Syk structure, or by the phosphorylation of specific tyrosine residues. In addition to its immunoreceptor function, Syk is activated downstream of integrin pathways, and integrins bind to the same region in Syk a…

0301 basic medicinekinaasitCell signalingentsyymitIntegrinsintegrinIntegrinAmino Acid MotifsMutation MissenseSykPeptidechemical and pharmacologic phenomenaBiochemistryspleen tyrosine kinase (Syk)environment and public healthBiokemia solu- ja molekyylibiologia - Biochemistry cell and molecular biology03 medical and health sciencesProtein DomainsLääketieteen bioteknologia - Medical biotechnologyenzyme kineticshemic and lymphatic diseasescell signalingHumansSyk KinaseTyrosinePhosphorylationCell adhesionMolecular Biologychemistry.chemical_classificationsoluviestintäintegriinit030102 biochemistry & molecular biologybiologyChemistryta1182hemic and immune systemsCell Biology3. Good healthCell biologyEnzyme Activationenzymes and coenzymes (carbohydrates)030104 developmental biologyAmino Acid SubstitutionCytoplasmbiology.proteinPhosphorylationPeptidessurface plasmon resonance (SPR)Signal Transduction
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New Laboratory Protocol to Determine the Oxidative Stress Profile of Human Nasal Epithelial Cells Using Flow Cytometry

2021

Several studies have shown the importance of oxidative stress (OS) in respiratory disease pathogenesis. It has been reported that the nasal epithelium may act as a surrogate for the bronchial epithelium in several respiratory diseases involving OS. However, the sample yields obtained from nasal biopsies are modest, limiting the number of parameters that can be determined. Flow cytometry has been widely used to evaluate cellular OS profiles. It has the advantage that analyses can be performed using a small amount of sample. Therefore, we aimed to set up a new method based on flow cytometry to assess the oxidative profile of human nasal epithelial cells which could be used in research on resp…

0301 basic medicinelcsh:MedicineOxidative phosphorylationrare respiratory diseasesmedicine.disease_causeArticleFlow cytometryNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineoxidative stressreactive oxygen specieschemistry.chemical_classificationReactive oxygen speciesmedicine.diagnostic_testSuperoxidebusiness.industryflow cytometrylcsh:RGeneral MedicineGlutathioneMolecular biologynasal epithelium030104 developmental biologychemistry030220 oncology & carcinogenesisbusinessPeroxynitriteOxidative stressJournal of Clinical Medicine
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Oxidative Stress in Non-Dialysis-Dependent Chronic Kidney Disease Patients

2021

Background: Cardiovascular complications are the leading cause of morbidity and mortality at any stage of chronic kidney disease (CKD). Moreover, the high rate of cardiovascular mortality observed in these patients is associated with an accelerated atherosclerosis process that likely starts at the early stages of CKD. Thus, traditional and non-traditional or uremic-related factors represent a link between CKD and cardiovascular risk. Among non-conventional risk factors, particular focus has been placed on anaemia, mineral and bone disorders, inflammation, malnutrition and oxidative stress and, in this regard, connections have been reported between oxidative stress and cardiovascular disease…

0301 basic medicinemedicine.medical_specialtyAntioxidantHealth Toxicology and Mutagenesismedicine.medical_treatment030232 urology & nephrologyRenal functionInflammationOxidative phosphorylationmedicine.disease_causeArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicinecardiovascular diseaseMalondialdehydeInternal medicineHumansMedicineoxidative stressRenal Insufficiency Chronicbusiness.industryPublic Health Environmental and Occupational HealthRGlutathionemedicine.diseaseMalondialdehyde030104 developmental biologyEndocrinologychemistry8-Hydroxy-2'-DeoxyguanosineChronic DiseaseMedicinemedicine.symptombusinessOxidation-ReductionOxidative stresschronic kidney diseaseKidney diseaseInternational Journal of Environmental Research and Public Health
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Oxidative stress biomarkers in Fabry disease: is there a room for them?

2020

Abstract Background Fabry disease (FD) is an X-linked lysosomal storage disorder, caused by deficient activity of the alpha-galactosidase A enzyme leading to progressive and multisystemic accumulation of globotriaosylceramide. Recent data point toward oxidative stress signalling which could play an important role in both pathophysiology and disease progression. Methods We have examined oxidative stress biomarkers [Advanced Oxidation Protein Products (AOPP), Ferric Reducing Antioxidant Power (FRAP), thiolic groups] in blood samples from 60 patients and 77 healthy controls. Results AOPP levels were higher in patients than in controls (p < 0.00001) and patients presented decreased levels of…

0301 basic medicinemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentGlobotriaosylceramideOxidative phosphorylationDiseasemedicine.disease_cause03 medical and health scienceschemistry.chemical_compound0302 clinical medicinelysoGb3Internal medicinemedicineHumansFabry diseaseOriginal Communicationbusiness.industryBiomarkermedicine.diseaseFabry diseasePathophysiologyOxidative Stress030104 developmental biologyEndocrinologyNeurologychemistryAdvanced oxidation protein productsalpha-GalactosidaseMutationNeurology (clinical)businessBiomarkers030217 neurology & neurosurgeryOxidative stressJournal of Neurology
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