Search results for "Platelet Endothelial Cell Adhesion Molecule"

showing 10 items of 36 documents

PECAM-1/CD31 in infarction and longevity.

2007

: Inflammation has recently proven to be associated with the pathogenesis of atherosclerosis and inflammatory genes are good candidates for the risk of developing atherosclerosis. The early phase of atherosclerosis involves the recruitment of inflammatory cells from the circulation and their transendothelial migration. This process is mainly mediated by cellular adhesion molecules, which are expressed by the vascular endothelium and by circulating leukocytes in response to several inflammatory stimuli. Adhesion of circulating cells to the arterial surface is among the first detectable events in atherogenesis. Cellular adhesion molecules, expressed by the vascular endothelium and by circulat…

CD31MaleGenotypePopulationLongevityMyocardial InfarctionSingle-nucleotide polymorphismInflammationCoronary DiseaseBiologyPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologypolymorphismSex FactorsHistory and Philosophy of ScienceKEYWORDS: centenarianmedicineCell AdhesionSNPHumansGenetic Predisposition to DiseaseCell adhesioneducationSettore MED/04 - Patologia GeneraleAged 80 and overInflammationeducation.field_of_studyPolymorphism GeneticCell adhesion moleculeGeneral NeurosciencePlatelet Endothelial Cell Adhesion Molecule-1ItalyCase-Control StudiesImmunologycardiovascular systemCentenarianmedicine.symptomAnnals of the New York Academy of Sciences
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Implanted neonatal human dermal fibroblasts influence the recruitment of endothelial cells in mice

2012

The vascularization of new tissue within a reasonable time is a crucial prerequisite for the success of different cell- and material-based strategies. Considering that angiogenesis is a multi-step process involving humoral and cellular regulatory components, only in vivo assays provide the adequate information about vessel formation and the recruitment of endothelial cells. The present study aimed to investigate if neonatal human dermal fibroblasts could influence in vivo neovascularization. Results obtained showed that fibroblasts were able to recruit endothelial cells to vascularize the implanted matrix, which was further colonized by murine functional blood vessels after one week. The ve…

CD31MalePathologymedicine.medical_specialtyAngiogenesisCell TransplantationBiomedical EngineeringCD34Medicine (miscellaneous)Neovascularization PhysiologicInflammationAntigens CD34BiologyNitric OxideRegenerative MedicineBiomaterialsNeovascularizationHemoglobinsMiceTissue engineeringMicroscopy Electron TransmissionIn vivoReportmedicineAnimalsHumansRegenerationSkinInflammationMatrigelNeovascularization PathologicTissue EngineeringEndothelial CellsGeneral MedicineFibroblastsMice Inbred C57BLPlatelet Endothelial Cell Adhesion Molecule-1Drug CombinationsPhenotypeProteoglycansCollagenLamininmedicine.symptom
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Antibodies to PECAM-1 and glucocorticoids reduce leukocyte adhesion in adjuvant arthritis of the rat knee synovium in vivo.

2002

Objective and design: To demonstrate the effect of monoclonal antibodies to the adhesion-molecule PECAM-1 (CD31) and of prednisolone on leukocyte adhesion in rat adjuvant arthritis.¶Material: Adjuvant arthritis was induced in male CD®-rats (five groups of n=6) 18 days prior to measurements.¶Treatment: Mouse-monoclonal antibody to rat CD-31 at 200 μg/kg or prednisolone at 24 mg/kg were administered i.v. 15 minutes prior to measurements.¶Methods: Venules within the intact rat-knee synovium were focused by confocal laser scanning microscopy. Numbers of rolling and adherent leukocytes were assessed in vivo.¶Results: Induction of arthritis significantly increased rolling and adherent leukocytes …

CD31Malemedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentPrednisoloneImmunologyAnti-Inflammatory AgentsArthritisMonoclonal antibodyLeukocyte CountIn vivoInternal medicineSynovial FluidmedicineCell AdhesionLeukocytesAnimalsAntibodies BlockingGlucocorticoidsPharmacologyMicroscopy Confocalbusiness.industryAntibodies Monoclonalmedicine.diseaseArthritis ExperimentalRheumatologyRatsPlatelet Endothelial Cell Adhesion Molecule-1Rheumatoid arthritisImmunologyPrednisoloneJointsbusinessAdjuvantmedicine.drugInflammation research : official journal of the European Histamine Research Society ... [et al.]
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Paracrine Effects Influenced by Cell Culture Medium and Consequences on Microvessel-Like Structures in Cocultures of Mesenchymal Stem Cells and Outgr…

2011

Mesenchymal stem cells (MSC) from bone marrow and outgrowth endothelial cells (OEC) from peripheral blood are considered as attractive cell types for applications in regenerative medicine aiming to build up complex vascularized tissue-engineered constructs. MSC provide several advantages such as the potential to differentiate to osteoblasts and to support the neovascularization process by release of proangiogenic factors. On the other hand, the neovascularization process can be actively supported by OEC forming perfused vascular structures after co-implantation with other cell types. In this study the formation of angiogenic structures in vitro was investigated in cocultures of MSC and OEC,…

CD31medicine.medical_treatmentCellular differentiationBiomedical EngineeringFluorescent Antibody TechniqueEnzyme-Linked Immunosorbent AssayBioengineeringCD146 AntigenBiologyPolymerase Chain ReactionBiochemistryBiomaterialsParacrine signallingchemistry.chemical_compoundmedicineHumansCells CulturedGrowth factorMesenchymal stem cellEndothelial CellsCell DifferentiationMesenchymal Stem CellsFlow CytometryCoculture TechniquesCulture MediaCell biologyPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factorchemistryCell cultureImmunologyCD146Tissue Engineering Part A
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CD38/CD31, the CCL3 and CCL4 chemokines, and CD49d/vascular cell adhesion molecule-1 are interchained by sequential events sustaining chronic lymphoc…

2009

AbstractCD38 and CD49d are associated negative prognosticators in chronic lymphocytic leukemia (CLL). Despite evidence that both molecules are involved in interactions occurring between CLL and normal cells in the context of CLL-involved tissues, a functional link is still missing. Using gene expression profiles comparing CD38+CD49d+ versus CD38−CD49d− CLL cells, we showed overexpression of the CCL3 and CCL4 chemokines in cells from the former group. These chemokines were also up-regulated by CD38 signals in CLL; moreover, CCL3 was expressed by CLL cells from bone marrow biopsies (BMB) of CD38+CD49d+ but not CD38−CD49d− cases. High levels of CCR1 and, to a lesser extent, CCR5, the receptors…

Cancer ResearchChemokineChronic lymphocytic leukemiaIntegrin alpha4ApoptosisCD38immune system diseaseshemic and lymphatic diseasesReceptorsChronicMacrophages; Apoptosis; Membrane Glycoproteins; Humans; Integrin alpha4; Antigens CD38; Vascular Cell Adhesion Molecule-1; Endothelial Cells; Receptors Chemokine; Antigens CD31; Cell Survival; Bone Marrow Cells; Leukemia Lymphocytic Chronic B-Cell; Antigens CD; Up-Regulation; Chemokine CCL4; Chemokine CCL3; Cell LineChemokine CCL4Chemokine CCL3Membrane GlycoproteinsLeukemiaCell adhesion moleculehemic and immune systemsLymphocyticCDUp-RegulationPlatelet Endothelial Cell Adhesion Molecule-1Leukemiamedicine.anatomical_structureOncologyChemokineReceptors ChemokineTumor necrosis factor alphaStromal cellCell SurvivalVascular Cell Adhesion Molecule-1Bone Marrow CellsBiologyCell LineAntigens CDmedicineHumansAntigensMonocyteMacrophagesB-CellEndothelial Cellsmedicine.diseaseADP-ribosyl Cyclase 1Leukemia Lymphocytic Chronic B-CellCLL integrins chemokines CD49d CD38 prognosis.Cancer researchbiology.proteinCD31Settore MED/15 - Malattie del SangueCD38
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PECAM-1 expression in human mesothelial cells: an in vitro study.

1996

Mesothelial cells are actively involved in inflammatory processes by expressing a set of cell adhesion molecules (CAMs). Transmigration of leukocytes into inflamed tissues requires a chemotactic stimulus and engagement of platelet-endothelial cell adhesion molecule-1 (PECAM-1). To investigate the kinetics involved in peritonitis, pure cultures of mesothelial cells are necessary. In previous studies, we have found that human mesothelial cells (HOMES) show a weak constitutive expression of PECAM-1, which cannot be further stimulated by cytokines. It is known that all serous cavities and body fluids contain numerous macrophages which strongly express this adhesion molecule. To identify the cel…

Cell SeparationIn Vitro TechniquesEpitheliumPathology and Forensic MedicineInterferon-gammaE-selectinmedicineHumansCell adhesionMolecular BiologyCells CulturedbiologyChemistryCell adhesion moleculeTumor Necrosis Factor-alphaMonocyteEpithelial CellsCell BiologyGeneral MedicineCell sortingMolecular biologyImmunohistochemistryRecombinant ProteinsCell biologyPlatelet Endothelial Cell Adhesion Molecule-1Microscopy Electronmedicine.anatomical_structureCell culturebiology.proteinNeural cell adhesion moleculeOmentumMesothelial CellInterleukin-1Pathobiology : journal of immunopathology, molecular and cellular biology
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Effects of Cytokines on the Expression of Cell Adhesion Molecules by Cultured Human Omental Mesothelial Cells

1995

Cultured mesothelial cells (HOMES) are very responsive to the proinflammatory cytokines, interleukin (IL)-1 and tumor necrosis factor-alpha (TNF-alpha). E-selectin, ICAM-1 and VCAM-1 are known to play an important role, because they are presented by diverse cell types, for example endothelial cells (ECs), and interact with co-responding ligands on white blood cell membranes. In this study, the expression of ICAM-1, VCAM-1, E-selectin as well as PECAM-1 on cultured HOMES was studied over 5, 24, 48 and 72 h exposure to IL-1 beta, interferon-gamma and TNF-alpha. In previous studies we have shown that IL-1 beta and TNF-alpha increase the expression of ICAM-1, E-selectin and VCAM-1 on the cytopl…

Cell typeTime Factorsmedicine.medical_treatmentUmbilical veinImmunophenotypingPathology and Forensic MedicineProinflammatory cytokineImmunoenzyme TechniquesmedicineHumansFluorescent Antibody Technique IndirectMolecular BiologyCells CulturedChemistryCell adhesion moleculeInterleukinEpithelial CellsCell BiologyGeneral MedicineIntercellular Adhesion Molecule-1Cell biologyPlatelet Endothelial Cell Adhesion Molecule-1CytokineMicroscopy FluorescenceCell cultureCytokinesTumor necrosis factor alphaE-SelectinCell Adhesion MoleculesOmentumPathobiology
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Lymphatic endothelial progenitors bud from the cardinal vein and intersomitic vessels in mammalian embryos.

2012

Abstract The lymphatic vasculature preserves tissue fluid balance by absorbing fluid and macromolecules and transporting them to the blood vessels for circulation. The stepwise process leading to the formation of the mammalian lymphatic vasculature starts by the expression of the gene Prox1 in a subpopulation of blood endothelial cells (BECs) on the cardinal vein (CV) at approximately E9.5. These Prox1-expressing lymphatic endothelial cells (LECs) will exit the CV to form lymph sacs, primitive structures from which the entire lymphatic network is derived. Until now, no conclusive information was available regarding the cellular processes by which these LEC progenitors exit the CV without co…

EndotheliumMesenchymegovernment.form_of_governmentRecombinant Fusion ProteinsImmunologyEmbryonic DevelopmentMice TransgenicBiologyBiochemistryMiceMicroscopy Electron TransmissionCell MovementVascular BiologymedicineAnimalsLymph sacsProgenitor cellEmbryonic Stem CellsHomeodomain ProteinsMice KnockoutBuddingMembrane GlycoproteinsCommon cardinal veinsTumor Suppressor ProteinsfungiCell BiologyHematologyAnatomyAdherens JunctionsCadherinsEmbryo MammalianCoronary VesselsCell biologyPlatelet Endothelial Cell Adhesion Molecule-1Lymphatic Endotheliummedicine.anatomical_structureLymphatic systemgovernmentsense organsEndothelium LymphaticBlood
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Timing effect of intramyocardial hydrogel injection for positively impacting left ventricular remodeling after myocardial infarction

2015

Intramyocardial injection of various injectable hydrogel materials has shown benefit in positively impacting the course of left ventricular (LV) remodeling after myocardial infarction (MI). However, since LV remodeling is a complex, time dependent process, the most efficacious time of hydrogel injection is not clear. In this study, we injected a relatively stiff, thermoresponsive and bioabsorbable hydrogel in rat hearts at 3 different time points - immediately after MI (IM), 3 d post-MI (3D), and 2 w post-MI (2W), corresponding to the beginnings of the necrotic, fibrotic and chronic remodeling phases. The employed left anterior descending coronary artery ligation model showed expected infar…

InjectionTime FactorsMacrophageMyocardial InfarctionInfarction02 engineering and technology030204 cardiovascular system & hematologyCardiac tissue engineeringAntigens CD31Hydrogel Polyethylene Glycol DimethacrylateHeart Ventricle0302 clinical medicineFibrosisMyocardial infarctionInflammation MediatorVentricular RemodelingIntervention timing021001 nanoscience & nanotechnologyPlatelet Endothelial Cell Adhesion Molecule-1Neutrophil InfiltrationMechanics of MaterialsSelf-healing hydrogelsCardiologyCytokinesFemalemedicine.symptomInflammation Mediators0210 nano-technologymedicine.medical_specialtyMaterials scienceTime FactorHeart VentriclesBiophysicsInflammationBioengineeringCeramics and CompositeAnterior Descending Coronary ArteryArticleInjectionsBiomaterials03 medical and health sciencesInternal medicinemedicineAnimalsMechanics of MaterialVentricular remodelingCytokineActinAnimalMacrophagesMyocardiummedicine.diseaseBiomaterialInjectable materialActinsHydrogelRats Inbred LewCeramics and CompositesLigation
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In Vitro Expression of the Endothelial Phenotype: Comparative Study of Primary Isolated Cells and Cell Lines, Including the Novel Cell Line HPMEC-ST1…

2002

Endothelial cell lines are commonly used in in vitro studies to avoid problems associated with the use of primary endothelial cells such as the presence of contaminating cells, the difficulty in obtaining larger numbers of cells, as well as the progressive loss of cell viability and expression of endothelial markers in the course of in vitro propagation. We have analyzed the characteristics defining distinctive endothelial phenotypes in the cell lines EA.hy926, ECV304, EVLC2, HAEND, HMEC-1, ISO-HAS-1 and a cell line recently generated in our laboratory, HPMEC-ST1.6R, and have compared these phenotypes with those found in primary human endothelial cells isolated from umbilical vein (HUVEC), …

LipopolysaccharidesCD31Cell SurvivalAngiogenesisCD34Vascular Cell Adhesion Molecule-1Antigens CD34Enzyme-Linked Immunosorbent AssayBiologyPolymerase Chain ReactionBiochemistryCell Linevon Willebrand FactorCell AdhesionHumansMicroscopy Phase-ContrastViability assayLungCells CulturedChemokine CCL2SkinMatrigelNeovascularization PathologicInterleukin-6Reverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaCell adhesion moleculeInterleukin-8TemperatureGranulocyte-Macrophage Colony-Stimulating FactorCell BiologyIntercellular Adhesion Molecule-1ImmunohistochemistryCell biologyLipoproteins LDLPlatelet Endothelial Cell Adhesion Molecule-1Endothelial stem cellDrug CombinationsPhenotypeCell cultureImmunologyProteoglycansCollagenEndothelium VascularLamininE-SelectinCardiology and Cardiovascular MedicineInterleukin-1Microvascular Research
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