Search results for "Postsynaptic potential"

showing 10 items of 371 documents

Antagonists and agonists at the glycine site of the NMDA receptor for therapeutic interventions.

2003

For decades neuroreceptor research has focused on the development of NMDA glycine-site antagonists, after Johnson and Ascher found out in 1987 about the co-agonistic character of this achiral amino acid at the NMDA receptor. Contrary to the inhibitory glycine receptor (glycine(A)) the glycine binding site on the NMDA receptor (glycine(B)) is strychnine-insensitive. A great diversity of diseases showing a disturbed glutamate neurotransmission have been linked to the NMDA receptor. Glycine site antagonists have been investigated for acute diseases like stroke and head trauma as well as chronic ones like dementia and chronic pain.

PsychosisGlycinePainPharmacologyNeurotransmissionInhibitory postsynaptic potentialReceptors N-Methyl-D-AspartateGlycine bindingMemantineDrug DiscoverymedicineAnimalsHumansReceptorGlycine receptorPharmacologychemistry.chemical_classificationBinding SitesEpilepsyOrganic ChemistryGlutamate receptorGeneral Medicinemedicine.diseaseAmino acidStrokeNeuroprotective AgentsBiochemistrychemistryGlycineSchizophreniaNMDA receptorAnticonvulsantsDementiaExcitatory Amino Acid AntagonistsEuropean journal of medicinal chemistry
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Amyloid Beta-Mediated Changes in Synaptic Function and Spine Number of Neocortical Neurons Depend on NMDA Receptors

2021

Onset and progression of Alzheimer’s disease (AD) pathophysiology differs between brain regions. The neocortex, for example, is a brain region that is affected very early during AD. NMDA receptors (NMDARs) are involved in mediating amyloid beta (Aβ) toxicity. NMDAR expression, on the other hand, can be affected by Aβ. We tested whether the high vulnerability of neocortical neurons for Aβ-toxicity may result from specific NMDAR expression profiles or from a particular regulation of NMDAR expression by Aβ. Electrophysiological analyses suggested that pyramidal cells of 6-months-old wildtype mice express mostly GluN1/GluN2A NMDARs. While synaptic NMDAR-mediated currents are unaltered in 5xFAD …

QH301-705.5Amyloid betasomatosensory cortexDendritic SpinesMice TransgenicNeocortexSomatosensory systemReceptors N-Methyl-D-AspartateCatalysisArticleInorganic ChemistryAlzheimer Diseasemental disordersmedicineAnimalsBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopyNeuronsNeocortexAmyloid beta-PeptidesbiologyPyramidal Cellsmusculoskeletal neural and ocular physiologyOrganic ChemistryWild typeAmyloid betaExcitatory Postsynaptic PotentialsGeneral Medicine5xFADPathophysiologyComputer Science ApplicationsNMDARChemistryElectrophysiologyProtein Subunitsmedicine.anatomical_structurenervous systemKnockout mouseSynapsesbiology.proteinNMDA receptorbiological phenomena cell phenomena and immunityNeuroscienceAlzheimer’s diseasepsychological phenomena and processesInternational Journal of Molecular Sciences
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Quantum chemical structure-activity relationships on β-carbolines as natural monoamine oxidase inhibitors

1983

The electron density and the molecular electrostatic potential of the β-carbolines are studied using ab initioSTO-3G wave functions. The analysis was done from the point of view of a previous model built with monoamine oxidase substrates and irreversible inhibitors. The results confirm the usefulness of the model and make it possible to propose new precision to the molecular electrostatic potential patterns needed to have monoamine oxidase inhibitory activity.

Quantum chemicalChemistryMonoamine oxidaseStereochemistryBiophysicsPhysical and Theoretical ChemistryCondensed Matter PhysicsInhibitory postsynaptic potentialAtomic and Molecular Physics and OpticsInternational Journal of Quantum Chemistry
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Functional Hallmarks of GABAergic Synapse Maturation and the Diverse Roles of Neurotrophins

2011

Functional impairment of the adult brain can result from deficits in the ontogeny of GABAergic synaptic transmission. Gene defects underlying autism spectrum disorders, Rett’s syndrome or some forms of epilepsy, but also a diverse set of syndromes accompanying perinatal trauma, hormonal imbalances, intake of sleep-inducing or mood-improving drugs or, quite common, alcohol intake during pregnancy can alter GABA signaling early in life. The search for therapeutically relevant endogenous molecules or exogenous compounds able to alleviate the consequences of dysfunction of GABAergic transmission in the embryonic or postnatal brain requires a clear understanding of its site- and state-dependent …

Review ArticleBiologyNeurotransmissionInhibitory postsynaptic potentialgamma-Aminobutyric acidlcsh:RC321-571tonic inhibitionCellular and Molecular NeurosciencePostsynaptic potentialPresynaptic functionmedicinelcsh:Neurosciences. Biological psychiatry. Neuropsychiatryquantal analysisNGFSynaptic pharmacologyexcitatory–inhibitory balanceGABAergic synaptic transmissionBDNFbiology.proteinGABAergicsynapse developmentNeuroscienceSynapse maturationNeuroscienceNeurotrophinmedicine.drugFrontiers in Cellular Neuroscience
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Relationship between physiological excitatory and inhibitory measures of excitability in the left vs. right human motor cortex and peripheral electro…

2017

Abstract The current study was aimed at investigating the relationships of excitatory and inhibitory circuits of the left vs. right primary motor cortex with peripheral electrodermal activity (EDA). Ten healthy subjects participated in two experimental sessions. In each session, EDA was recorded for 10 min from the palmar surface of the left hand. Immediately after EDA recording, Transcranial Magnetic Stimulation (TMS) was used to probe excitatory and inhibitory circuits of the left or right primary motor cortex using two protocols of stimulation: the input-output curve for recording of motor evoked potentials, for testing excitatory circuits; the long-interval cortical inhibition (LICI) pr…

Right motor cortexAdultmedicine.medical_treatmentStimulationInhibitory postsynaptic potential050105 experimental psychologyFunctional Laterality03 medical and health sciencesElectrodermal activity0302 clinical medicineSkin Physiological PhenomenamedicineHumans0501 psychology and cognitive sciencesSkinHand musclesVegetative systemSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaGeneral Neuroscience05 social sciencesMotor CortexGalvanic Skin ResponseTranscranial Magnetic StimulationPeripheralTranscranial magnetic stimulationmedicine.anatomical_structureTMSExcitatory postsynaptic potentialPsychologyNeuroscience030217 neurology & neurosurgeryMotor cortexNeuroscience letters
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Biallelic gephyrin variants lead to impaired GABAergic inhibition in a patient with developmental and epileptic encephalopathy

2021

Abstract Synaptic inhibition is essential for shaping the dynamics of neuronal networks, and aberrant inhibition is linked to epilepsy. Gephyrin (Geph) is the principal scaffolding protein at inhibitory synapses and is essential for postsynaptic clustering of glycine (GlyRs) and GABA type A receptors. Consequently, gephyrin is crucial for maintaining the relationship between excitation and inhibition in normal brain function and mutations in the gephyrin gene (GPHN) are associated with neurodevelopmental disorders and epilepsy. We identified bi-allelic variants in the GPHN gene, namely the missense mutation c.1264G > A and splice acceptor variant c.1315-2A > G, in a patient wi…

Scaffold proteinBiologyInhibitory postsynaptic potentialEpilepsyPostsynaptic potentialGeneticsmedicineHumansMissense mutationReceptorBiologyMolecular BiologyGenetics (clinical)Brain DiseasesEpilepsyGephyrinMembrane ProteinsGeneral MedicineReceptors GABA-Amedicine.diseaseCell biologyChemistrySynapsesbiology.proteinHuman medicineReceptor clusteringCarrier ProteinsHuman Molecular Genetics
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Neural Mechanisms Underlying Human Auditory Evoked Responses Revealed By Human Neocortical Neurosolver

2022

Funding Information: This study was supported by National Institutes of Health (Grant Nos. NIBIB RO1 EB022889, NIMH RO1 MH106174). Publisher Copyright: © 2021, The Author(s). Auditory evoked fields (AEFs) are commonly studied, yet their underlying neural mechanisms remain poorly understood. Here, we used the biophysical modelling software Human Neocortical Neurosolver (HNN) whose foundation is a canonical neocortical circuit model to interpret the cell and network mechanisms contributing to macroscale AEFs elicited by a simple tone, measured with magnetoencephalography. We found that AEFs can be reproduced by activating the neocortical circuit through a layer specific sequence of feedforwar…

Sensory systemNeocortexNeuropathologyStimulus (physiology)BiologySomatosensory systemkuulohavainnotbiofysiikkamedicineAnimalsHumansRadiology Nuclear Medicine and imagingAEFBiophysical modelMEGRadiological and Ultrasound Technologymedicine.diagnostic_testhermoverkot (biologia)MagnetoencephalographyCognitionMagnetoencephalographyAuditory evoked responsesAuditory processingNeurologyAcoustic StimulationExcitatory postsynaptic potentialAuditory PerceptionEvoked Potentials Auditorykognitiivinen neurotiedeNeurology (clinical)AnatomyHNNNeuroscience
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Serotoninergic innervation of nonprincipal cells in the cerebral cortex of the lizard Podarcis hispanica.

2004

The mechanism of serotoninergic transmission in the neo- and archicortex of mammals kis complex, including both synaptic and nonsynaptic components, direct actions on principal cells, and indirect effects mediated by GABAergic interneurons. Here we studied the termination pattern and synaptic organization of the serotoninergic afferents in the cerebral cortex of the lizard, Podarcis hispanica, which is considered to correspond in part to the mammalian hippocampal formation, with the aim of unraveling basic, phylogenetically preserved rules in the connectivity of this pathway. We demonstrate that serotoninergic afferents, visualized by immunostaining for serotonin itself, establish multiple …

SerotoninHippocampal formationInhibitory postsynaptic potentialSerotonergicPodarcis hispanicaNerve FibersmedicineAnimalsNeuropeptide YTissue DistributionOpioid peptidegamma-Aminobutyric AcidCerebral CortexbiologyStaining and LabelingGeneral NeuroscienceLizardsbiology.organism_classificationMicroscopy Electronmedicine.anatomical_structureParvalbuminsCerebral cortexSynapsesbiology.proteinImmunologic TechniquesGABAergicEndorphinsNeuroscienceParvalbuminThe Journal of comparative neurology
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Unconventional ligands and modulators of nicotinic receptors

2002

Evidence gathered from epidemiologic and behavioral studies have indicated that neuronal nicotinic receptors (nAChRs) are intimately involved in the pathogenesis of a number of neurologic disorders, including Alzheimer's disease, Parkinson's disease, and schizophrenia. In the mammalian brain, neuronal nAChRs, in addition to mediating fast synaptic transmission, modulate fast synaptic transmission mediated by the major excitatory and inhibitory neurotransmitters glutamate and GABA, respectively. Of major interest, however, is the fact that the activity of the different subtypes of neuronal nAChR is also subject to modulation by substances of endogenous origin such as choline, the tryptophan …

SerotoninNeuroactive steroidPsychotomimetic drugReceptors NicotinicNeurotransmissionPharmacologyBiologyKynurenic AcidLigandsInhibitory postsynaptic potentialCholineCellular and Molecular Neurosciencechemistry.chemical_compoundKynurenic acidmental disordersmedicineAnimalsHumansPhencyclidineAnestheticsAmyloid beta-PeptidesGalantamineGeneral NeuroscienceGlutamate receptorNicotinic agonistnervous systemchemistryHallucinogensSteroidsNeurosciencemedicine.drugJournal of Neurobiology
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Kinetic Properties of Cl−Uptake Mediated by Na+-Dependent K+-2Cl−Cotransport in Immature Rat Neocortical Neurons

2007

GABA, the main inhibitory neurotransmitter in the adult nervous system, evokes depolarizing membrane responses in immature neurons, which are crucial for the generation of early network activity. Although it is well accepted that depolarizing GABA actions are caused by an elevated intracellular Cl−concentration ([Cl−]i), the mechanisms of Cl−accumulation in immature neurons are still a matter of debate. Using patch-clamp, microfluorimetric, immunohistochemical, and molecular biological approaches, we studied the mechanism of Cl−uptake in Cajal-Retzius (CR) cells of immature [postnatal day 0 (P0) to P3] rat neocortex. Gramicidin-perforated patch-clamp and 6-methoxy-N-ethylquinolinium-microfl…

Sodium-Potassium-Chloride SymportersNeocortexStimulationBiologyChloridesmedicineAnimalsSolute Carrier Family 12 Member 2Rats Wistargamma-Aminobutyric AcidNeuronsNeocortexGeneral NeuroscienceExcitatory Postsynaptic PotentialsCell DifferentiationDepolarizationArticlesRatsKineticsmedicine.anatomical_structureAnimals NewbornBiochemistryExcitatory postsynaptic potentialBiophysicsGABAergicCotransporterIntracellularBumetanidemedicine.drugThe Journal of Neuroscience
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