Search results for "Prague"

showing 10 items of 652 documents

Insight into the molecular sex dimorphism of ischaemic stroke in rat cerebral cortex: Focus on neuroglobin, sex steroids and autophagy

2020

Including sex is of paramount importance in preclinical and clinical stroke researches, and molecular studies dealing in depth with sex differences in stroke pathophysiology are needed. To gain insight into the molecular sex dimorphism of ischaemic stroke in rat cerebral cortex, male and female adult rats were subjected to transient middle cerebral artery occlusion. The expression of neuroglobin (Ngb) and other functionally related molecules involved in sex steroid signalling (oestrogen and androgen receptors), steroidogenesis (StAR, TSPO and aromatase) and autophagic activity (LC3B-II/LC3B-I ratio, UCP2 and HIF-1 alpha) was assessed in the ipsilateral ischaemic and contralateral non-ischae…

Malemedicine.medical_specialtysteroidogenesisNeuroprotectionBrain IschemiaRats Sprague-Dawley03 medical and health sciences0302 clinical medicineInternal medicineCortex (anatomy)sex steroid signallingmedicineAutophagyAnimalssex dimorphismAromataseStroke030304 developmental biologyIschemic StrokeCerebral Cortex0303 health sciencesSex Characteristicsischaemic strokebiologybusiness.industryGeneral NeuroscienceInfarction Middle Cerebral Arterymedicine.diseaseRatsAndrogen receptorStrokeDisease Models AnimalneuroglobinEndocrinologymedicine.anatomical_structureSex steroidCerebral cortexNeuroglobinbiology.proteinFemaleSteroidsbusiness030217 neurology & neurosurgery
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Neuroimmune and Mu-Opioid Receptor Alterations in the Mesocorticolimbic System in a Sex-Dependent Inflammatory Pain-Induced Alcohol Relapse-Like Rat …

2021

Evidence concerning the role of alcohol-induced neuroinflammation in alcohol intake and relapse has increased in the last few years. It is also proven that mu-opioid receptors (MORs) mediate the reinforcing properties of alcohol and, interestingly, previous research suggests that neuroinflammation and MORs could be related. Our objective is to study neuroinflammatory states and microglial activation, together with adaptations on MOR expression in the mesocorticolimbic system (MCLS) during the abstinence and relapse phases. To do so, we have used a sex-dependent rat model of complete Freund’s adjuvant (CFA)-induced alcohol deprivation effect (ADE). Firstly, our results confirm that only CFA-…

Malemedicine.medical_treatmentFreund's AdjuvantReceptors Opioid mualcohol deprivation effectNitric Oxide Synthase Type IImicroglianeuroinflammationRats Sprague-DawleyRecurrenceLimbic SystemImmunology and AllergypainPhosphorylationReceptormedia_commonMicrogliaAlcohol AbstinencealcoholMicrofilament ProteinsNF-kappa BBrief Research ReportInterleukin 10AlcoholismCytokinemedicine.anatomical_structureCytokinesFemaleμ-opioid receptorInflammation Mediatorsmedicine.medical_specialtyNeuroimmunomodulationmedia_common.quotation_subjectImmunologyPrefrontal CortexSex FactorsDownregulation and upregulationInternal medicinemedicineAnimalsNeuroinflammationbusiness.industryCalcium-Binding ProteinsAbstinenceRC581-607EndocrinologyCyclooxygenase 2mu-opioid receptorImmunologic diseases. AllergybusinessFrontiers in Immunology
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C1-ESTERASE INHIBITOR REVERSES FUNCTIONAL CONSEQUENCES OF SUPERIOR MESENTERIC ARTERY ISCHEMIA/REPERFUSION BY LIMITING REPERFUSION INJURY AND RESTORIN…

2006

Activated complement contributes significantly to reperfusion injury after ischemia. This study explores functional consequences of C1-esterase inhibitor (C1-INH) treatment after superior mesenteric artery occlusion (SMAO)/ reperfusion using intravital microscopy. Thirty anesthetized, spontaneously breathing, male Sprague-Dawley rats underwent SMAO for 60 min followed by reperfusion (4 h). C1-esterase inhibitor (100 and 200 IU/kg body weight) or saline (0.9%) was given as a single bolus before reperfusion. Sham-operated animals (n = 10) without SMAO served as controls.Systemichemodynamicsweremonitoredcontinuously,arterial bloodgasesanalyzedintermittently, andleukocyte/ endothelial interacti…

Malemedicine.medical_treatmentIschemiaPharmacologyCritical Care and Intensive Care MedicineRats Sprague-DawleyBolus (medicine)Mesenteric Artery Superiormedicine.arterymedicineAnimalsSuperior mesenteric arterySalinebusiness.industryMicrocirculationMetabolic acidosismedicine.diseaseRatsRegional Blood FlowMesenteric ischemiaReperfusion InjuryAnesthesiaEmergency MedicinebusinessComplement C1 Inhibitor ProteinReperfusion injuryIntravital microscopyShock
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Differential expression of suppressors of cytokine signaling-1, -2, and -3 in the rat hippocampus after seizure: implications for neuromodulation by …

2003

Numerous studies have investigated the expression of various cytokine families in the CNS after brain injury. The gp130 or interleukin (IL)-6-type cytokines have received a great deal of focus, and it is clear that they exhibit an acute and robust upregulation in various brain injury models. We are interested to determine, however, whether endogenously expressed cytokines in the CNS act in a direct neuromodulatory manner. In an accompanying study, we examined the expression of five gp130 cytokines and their receptors in the lithium-pilocarpine model of status epilepticus. We follow up that study here by trying to determine if gp130 signal transduction occurs in hippocampal principal neurons…

Malemedicine.medical_treatmentPopulationSuppressor of Cytokine Signaling ProteinsHippocampal formationBiologyNeuroprotectionHippocampusRats Sprague-DawleySuppressor of Cytokine Signaling 1 ProteinSeizuresmedicineAnimalsRNA MessengerReceptors Cytokineeducationeducation.field_of_studyGeneral NeuroscienceInterleukinGlycoprotein 130RatsDNA-Binding ProteinsRepressor ProteinsCytokineGene Expression RegulationSuppressor of Cytokine Signaling 3 ProteinTrans-ActivatorsCytokinesSignal transductionCytokine receptorCarrier ProteinsNeuroscienceSignal TransductionTranscription FactorsNeuroscience
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Degradable poly(amidoamine) hydrogels as scaffolds for in vitro culturing of peripheral nervous system cells.

2012

This paper reports on the synthesis and physico-chemical, mechanical, and biological characterization of two sets of poly(amidoamine) (PAA) hydrogels with potential as scaffolds for in vivo peripheral nerve regeneration. They are obtained by polyaddition of piperazine with N,N′-methylenebis(acrylamide) or 1,4-bis(acryloyl)piperazine with 1,2-diaminoethane as cross-linking agent and exhibit a combination of relevant properties, such as mechanical strength, biocompatibility, biodegradability, ability to induce adhesion and proliferation of Schwann cells (SCs) preserving their viability. Moreover, the most promising hydrogels, that is those deriving from 1,4-bis(acryloyl)piperazine, allow the …

Materials Chemistry2506 Metals and AlloysPoly(amidoamine)Cell SurvivalBioengineeringBiocompatible MaterialsNeural cell culturingPiperazinesRats Sprague-DawleyGanglia SpinalCell AdhesionPolyaminesAnimalsCell ProliferationNeuronsAcrylamidesPolymers and PlasticTissue EngineeringTissue ScaffoldsHydrogelsPolymer applicationEthylenediaminesBiomaterialNerve RegenerationRatsHydrogelBiodegradableSchwann CellsBiotechnologyMacromolecular bioscience
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Sevoflurane Impairs Cerebral Blood Flow Autoregulation in Rats: Reversal by Nonselective Nitric Oxide Synthase Inhibition

2005

UNLABELLED In this study, we investigated the effects of 1.0 and 2.0 minimum alveolar anesthetic concentration (MAC) sevoflurane on cerebral blood flow (CBF) autoregulation before and after nonselective inhibition of nitric oxide (NO) synthase in rats. Rats were randomly assigned as follows: Group 1 (n = 8): 1.0 MAC sevoflurane; Groups 2 and 3 (n = 8 per group): 2.0 MAC sevoflurane. Assessment of autoregulation within a mean arterial blood pressure range of 140-60 mm Hg was performed by graded hemorrhage before and after administration of l-arginine methyl ester (l-NAME, 30 mg/kg IV, Groups 1 and 2) or during hypocapnia (Group 3). In 10 additional animals, brain tissue NO(2)(-) concentratio…

Methyl EthersBlood PressureVasodilationPharmacologyNitric OxideSevofluraneNitric oxideRats Sprague-DawleySevofluranechemistry.chemical_compoundHypocapniaAnimalsHomeostasisHyperventilationMedicineAutoregulationEnzyme InhibitorsCerebral HemorrhageBrain ChemistryBlood VolumeDose-Response Relationship Drugbiologybusiness.industrymedicine.diseaseRatsNitric oxide synthaseNG-Nitroarginine Methyl EsterAnesthesiology and Pain MedicineCerebral blood flowchemistryCerebrovascular CirculationAnesthesiaAnesthetics InhalationAnestheticbiology.proteinNitric Oxide Synthasebusinessmedicine.drugAnesthesia & Analgesia
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Dual modulation of striatal acetylcholine release by hyperforin, a constituent of St. John's wort.

2002

Extracts of the medicinal plant St. John's wort (Hypericum perforatum) are widely used for the treatment of mild to moderate depression. Hyperforin, a constituent of St. John's wort, is known to inhibit the sodium-dependent uptake of catecholamines and amino acids into synaptic nerve endings, probably by interference with mechanisms controlling the synaptic sodium concentration. Because de novo synthesis of acetylcholine (ACh) is dependent on sodium-dependent high-affinity choline uptake, we studied the effect of hyperforin on choline (Ch) uptake in vitro and on striatal ACh release in vivo using microdialysis. In rat brain synaptosomes, hyperforin inhibited high-affinity choline uptake wit…

MicrodialysisPharmacologyMotor ActivityPhloroglucinolCholineRats Sprague-Dawleychemistry.chemical_compoundBridged Bicyclo CompoundsIn vivomedicineCholineAnimalsReceptors CholinergicIC50PharmacologyChemistryTerpenesHypericum perforatumBiological TransportAcetylcholineCorpus StriatumAnti-Bacterial AgentsRatsHyperforinSystemic administrationMolecular MedicineAcetylcholineHypericummedicine.drugThe Journal of pharmacology and experimental therapeutics
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Stimulation of hippocampal acetylcholine release by hyperforin, a constituent of St. John’s Wort

2004

Abstract Extracts of the medicinal plant St. John’s Wort ( Hypericum perforatum ) are widely used in the therapy of affective disorders and have been reported to exert antidepressant, anxiolytic, and cognitive effects in experimental and clinical studies. We here report that hyperforin, the major active constituent of the extract, increases the release of acetylcholine from rat hippocampus in vivo as determined by microdialysis. Hippocampal acetylcholine levels were increased by 50–100% following the systemic administration of pure hyperforin at doses of 1 and 10 mg/kg. The effect was almost completely suppressed by local perfusion with calcium-free buffer or with tetrodotoxin (1 μM). We co…

Microdialysismedicine.drug_classMicrodialysisTetrodotoxinPhloroglucinolPharmacologyHippocampusAnxiolyticRats Sprague-DawleyBridged Bicyclo Compoundschemistry.chemical_compoundmedicineAnimalsAnesthetics LocalNeurotransmitterPlant ExtractsTerpenesGeneral NeuroscienceHypericum perforatumAcetylcholineAnti-Bacterial AgentsRatsHyperforinchemistryAntidepressantCholinergicHypericumAcetylcholinemedicine.drugNeuroscience Letters
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Evidence for a relationship between mitochondrial Complex I activity and mitochondrial aldehyde dehydrogenase during nitroglycerin tolerance: effects…

2012

The medical use of nitroglycerin (GTN) is limited by patient tolerance. The present study evaluated the role of mitochondrial Complex I in GIN biotransformation and the therapeutic effect of mitochondrial antioxidants. The development of GIN tolerance (in rat and human vessels) produced a decrease in mitochondrial 02 consumption. Co-incubation with the mitochondria-targeted antioxidant mitoquinone (MQ 10(-6) mol/L) or with glutathione ester (GEE, 10(-4) mol/L) blocked GTN tolerance and the effects of GTN on mitochondrial respiration and aldehyde dehydrogenase 2 (ALDH-2) activity. Biotransformation of GTN depended on the mitochondria being functionally active, particularly mitochondrial Comp…

Mitochondrial ROSMaleAntioxidantmedicine.medical_treatmentAldehyde dehydrogenaseMitochondrionmedicine.disease_causeBiochemistryAntioxidantsRats Sprague-Dawleychemistry.chemical_compoundMiceNitroglycerinCyclic GMPAortaBiotransformationbiologyDrug ToleranceGlutathioneMitochondriaVasodilationBiochemistrycardiovascular systemAntioxidantcirculatory and respiratory physiologyBiophysicsIn Vitro TechniquesALDH-2Nitric oxideCell LineOxygen ConsumptionRotenoneRespirationmedicineHuman Umbilical Vein Endothelial CellsAnimalsHumansElectron Transport Complex IDose-Response Relationship DrugNitric oxideGlutathioneCell BiologyAldehyde DehydrogenaseRatschemistryOxidative stressMutationbiology.proteinReactive Oxygen SpeciesOxidative stressBiochimica et biophysica acta
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Preparation of two sets of 5,6,7-trioxygenated dihydroflavonol derivatives as free radical scavengers and neuronal cell protectors to oxidative damag…

2009

An unusual class of 5,6,7-trioxygenated dihydroflavonols (3a-e and 4a-j) were designed and prepared. Their antioxidative properties were assessed by examining their capacities in several in vitro models, including superoxide anion and 1,1-diphenyl-2-picrylhydrazyl (DPPH) radical scavenging, rat liver homogenate lipid peroxidation inhibition, PC12 cells protection from oxidative damage, and xanthine oxidase inhibition. These dihydroflavonols displayed positive quenching abilities towards O(2)(-) and DPPH free radicals, in which the majority exhibited superior antioxidant properties to Vitamin C. cis-Configurated compound (+/-)-3e demonstrated remarkable inhibition to LPO with an IC(50) value…

Models MolecularXanthine OxidaseAntioxidantFlavonolsmedicine.drug_classDPPHmedicine.medical_treatmentClinical BiochemistryMolecular ConformationPharmaceutical Sciencemedicine.disease_causeBiochemistryPC12 CellsAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipDrug DiscoverymedicineAnimalsXanthine oxidaseMolecular BiologyXanthine oxidase inhibitorNeuronsSuperoxideOrganic ChemistryFree Radical ScavengersFree radical scavengerRatschemistryBiochemistryMolecular MedicineLipid PeroxidationReactive Oxygen SpeciesOxidative stressBioorganicmedicinal chemistry
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