Search results for "Precancerous Conditions"

showing 10 items of 81 documents

Ikivėžinių skrandžio gleivinės pokyčių paplitimas Helicobacter pylori sukelto lėtinio gastrito metu tarp Rytų Europos (Lietuvos, Latvijos) ir Azijos …

2007

The aim of the study was to compare the prevalence and severity of precancerous condition – gastric atrophy and intestinal metaplasia (IM) between Eastern European (Lithuania and Latvia) and Asian (Taiwan) countries in population older than 55 years. Methods. Patients aged 55 years and older, referred for upper endoscopy due to dyspeptic symptoms, were included in the study. Gastric biopsies were histological investigated according modified Sydney classification. Helicobacter pylori (H. pylori) was detected if any two of three methods (urease test, histology, and serology) were positive. Results. Overall 322 patients included: 52 from Taiwan (TW), 171 from Latvia (LV) and 99 from Lithuania …

Gastritis AtrophicMalemedicine.medical_specialty616.3 [udc]Helicobacter pylori ; Gastritis atrophic ; Intestinal diseases ; Metaplasia ; Precancerous conditionsBiopsyPopulationTaiwanRapid urease testintestinal metaplasiaGastroenterologyHelicobacter InfectionsAtrophyStomach NeoplasmsMetaplasiaInternal medicineGastroscopyPrevalenceHumansMedicineeducation616.33/.34 [udc]AgedMetaplasiaeducation.field_of_studyHelicobacter pyloribiologybusiness.industryStomachgastritisIntestinal metaplasia<em>Helicobacter pylori</em>; gastritis; atrophy; intestinal metaplasiaLithuaniaGeneral MedicineMiddle AgedHelicobacter pylorimedicine.diseasebiology.organism_classificationLatviaEastern europeanFemaleAtrophymedicine.symptomGastritisbusinessPrecancerous Conditions<em>Helicobacter pylori</em>
researchProduct

Management of epithelial precancerous conditions and lesions in the stomach (MAPS II): European Society of Gastrointestinal Endoscopy (ESGE), Europea…

2019

Main RecommendationsPatients with chronic atrophic gastritis or intestinal metaplasia (IM) are at risk for gastric adenocarcinoma. This underscores the importance of diagnosis and risk stratification for these patients. High definition endoscopy with chromoendoscopy (CE) is better than high definition white-light endoscopy alone for this purpose. Virtual CE can guide biopsies for staging atrophic and metaplastic changes and can target neoplastic lesions. Biopsies should be taken from at least two topographic sites (antrum and corpus) and labelled in two separate vials. For patients with mild to moderate atrophy restricted to the antrum there is no evidence to recommend surveillance. In pati…

Gastritis Atrophicmedicine.medical_specialtyAtrophic gastritisBiopsyChronic gastritisgastroenterologyGastroenterologyRisk AssessmentEndoscopy GastrointestinalChromoendoscopyHelicobacter Infections03 medical and health sciences0302 clinical medicineStomach Neoplasmsgastroenterology; precancerous conditions; stomach; gastric cancerInternal medicinemedicineHumansprecancerous conditionsMetaplasiamedicine.diagnostic_testbusiness.industryStomachgastric cancerIntestinal metaplasiamedicine.diseaseEndoscopyPatient Care ManagementEuropemedicine.anatomical_structureDysplasia030220 oncology & carcinogenesis030211 gastroenterology & hepatologyGastritismedicine.symptombusinessstomach
researchProduct

ΔNp73β is oncogenic in hepatocellular carcinoma by blocking apoptosis signaling via death receptors and mitochondria

2010

p73 belongs to the p53 family of transcription factors known to regulate cell cycle and apoptosis. The Trp73 gene has two promoters that drive the expression of two major p73 isoform subfamilies: TA and ΔN. In general, TAp73 isoforms show proapoptotic activities, whereas members of the N-terminally truncated (ΔN) p73 subfamily that lack the transactivation domain show antiapoptotic functions. We found that upregulation of ΔNp73 in hepatocellular carcinoma (HCC) correlated with reduced survival. Here, we investigated the molecular mechanisms accounting for the oncogenic role of ΔNp73 in HCC.ΔNp73β can directly interfere with the transcriptional activation function of the TA (containing the t…

Gene isoformCarcinoma HepatocellularMolecular Sequence DataApoptosisBiologyModels BiologicalTransactivationDownregulation and upregulationCell Line TumorHumansProtein IsoformsMolecular BiologyTranscription factorGenes DominantOligonucleotide Array Sequence Analysisbcl-2-Associated X ProteinRegulation of gene expressionBase SequenceSettore BIO/11Gene Expression ProfilingTumor Suppressor ProteinsLiver NeoplasmsNuclear ProteinsTumor Protein p73PromoterReceptors Death DomainCell BiologyCell cyclePrognosisMitochondriaCell biologyDNA-Binding ProteinsEnzyme ActivationGene Expression Regulation NeoplasticDrug Resistance NeoplasmCaspasesCancer researchTumor Suppressor Protein p53Signal transductionPrecancerous ConditionsSignal TransductionDevelopmental BiologyCell Cycle
researchProduct

Human papillomaviruses in oral carcinoma and oral potentially malignant disorders: a systematic review.

2011

Human papillomavirus (HPV) in oral carcinoma (OSCC) and potentially malignant disorders (OPMD) is controversial. The primary aim was to calculate pooled risk estimates for the association of HPV with OSCC and OPMD when compared with healthy oral mucosa as controls. We also examined the effects of sampling techniques on HPV detection rates. Methods: Systematic review was performed using PubMed (January 1966-September 2010) and EMBASE (January 1990-September 2010). Eligible studies included randomized controlled, cohort and cross-sectional studies. Pooled data were analysed by calculating odds ratios, using a random effects model. Risk of bias was based on characteristics of study group, appr…

HPVprecancerAlphapapillomavirusmeta-analysiCohort StudiespremalignancyBiasSettore MED/28 - Malattie OdontostomatologicheRisk Factorscase–controlHumansbiopsyhumanProspective StudiesRandomized Controlled Trials as Topiclichen planuerythroplakiaadultPapillomavirus Infectionsoral cancerCell Transformation ViralControl GroupsCross-Sectional StudiesleukoplakiacytologyMouth NeoplasmsPrecancerous ConditionsOral diseases
researchProduct

Benign, preinvasive and invasive ductal breast lesions. A comparative study with quantitative techniques: morphometry, image- and flow cytometry.

1999

The histological distinction between ductal hyperplasia of the breast, atypical ductal hyperplasia and ductal carcinoma in situ is difficult and subjective. To gain a better understanding of these lesions, we performed a comparative study comprising 20 cases of ductal hyperplasia without atypia, 20 cases of ductal hyperplasia with atypia, and 30 cases of ductal carcinoma in situ (well-, moderately- and poorly-differentiated), using quantitative techniques: image cytometry analysis, morphometry and DNA analysis, and DNA flow cytometry. Our results confirm that the mean nuclear area and volume progressively decreased from ductal carcinoma in situ to ductal hyperplasia without atypia. The diff…

In situPathologymedicine.medical_specialtyBreast NeoplasmsCell SeparationPathology and Forensic MedicineFlow cytometryAtypiaImage Processing Computer-AssistedMedicineHumansDuctal Hyperplasiaskin and connective tissue diseasesneoplasmsDNA Image CytometryImage CytometryCell NucleusPloidiesintegumentary systemmedicine.diagnostic_testbusiness.industryCarcinoma Ductal BreastCell BiologyDNA NeoplasmDuctal carcinomaHyperplasiamedicine.diseaseFlow Cytometrybody regionsImage CytometryFemalebusinessPrecancerous ConditionsCarcinoma in SituPathology, research and practice
researchProduct

Management of laryngeal precancerous lesions.

2012

Abstract Objective The identification of precancerous lesions is the basis of an early diagnosis, and of a treatment that allows, in the great part of cases, the preservation of organ functions. The aims of this study were: the evaluation of the less invasive treatment for precancerous lesion of the larynx to minimize the recurrences, the estimation of number of further operation required. Methods A prospective study was done on patients with clinical diagnosis of laryngeal precancerosis. The patients were treated by a transoral endoscopic approach with direct microlaryngoscopy (DML) doing an excision-biopsy with cold blade, consisting in excision of the whole visible lesion with vocal liga…

LarynxAdultMalemedicine.medical_specialtyBiopsyLaryngoscopyPrecancerouLesionLarynx; Cancer; Precancerous; Precancerosis; LaryngoscopyBiopsyMedicineHumansProspective StudiesProspective cohort studyLaryngeal NeoplasmsCancerAgedAged 80 and overmedicine.diagnostic_testLaryngoscopybusiness.industryCancerGeneral MedicineMiddle Agedmedicine.diseaseSettore MED/32 - AudiologiaSurgerySettore MED/31 - Otorinolaringoiatriamedicine.anatomical_structureTreatment OutcomeOtorhinolaryngologyLigamentCordectomyPrecancerosiSurgeryFemaleLarynxmedicine.symptomNeoplasm Recurrence LocalbusinessPrecancerous ConditionsAuris, nasus, larynx
researchProduct

Hypoxia-inducible factor 1 alpha expression increases during colorectal carcinogenesis and tumor progression

2008

Abstract Background Hypoxia-inducible factor 1 alpha (HIF-1α) is involved in processes promoting carcinogenesis of many tumors. However, its role in the development of colorectal cancer is unknown. To investigate the significance of HIF-1α during colorectal carcinogenesis and progression we examined its expression in precursor lesions constituting the conventional and serrated pathways, as well as in non-metastatic and metastatic adenocarcinomas. Methods Immunohistochemistry and Western blot is used to analyse HIF-1α expression in normal colonic mucosa, hyperplastic polyps (HPP), sessile serrated adenomas (SSA), low-grade (TA-LGD) and high-grade (TA-HGD) traditional adenomas as well as in n…

LipopolysaccharidesCancer ResearchPathologymedicine.medical_specialtyColorectal cancerColonic PolypsMouse model of colorectal and intestinal cancermedicine.disease_causelcsh:RC254-282chemistry.chemical_compoundWestern blotCell Line TumorGeneticsMedicineHumansNeoplasm Metastasismedicine.diagnostic_testbusiness.industrymedicine.diseaseHypoxia-Inducible Factor 1 alpha Subunitlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensImmunohistochemistryVascular endothelial growth factorCell Transformation NeoplasticOncologychemistryHyperplastic PolypTumor progressionDisease ProgressionImmunohistochemistrybusinessCarcinogenesisColorectal NeoplasmsPrecancerous ConditionsResearch ArticleBMC Cancer
researchProduct

Expression of L- and M2-pyruvate kinases in proliferating oval cells and cholangiocellular lesions developing in the livers of rats fed a methyl-defi…

1994

Male outbred Sprague-Dawley rats were fed a choline-deficient diet containing 0.1% w/w DL-ethionine (CDE) for up to 22 weeks. The expression of the pyruvate kinase isoenzymes L (L-PK) and M2 (M2-PK) was immunohistochemically analyzed in liver slices from rats killed 4, 10, 14 and 22 weeks after starting the treatment. M2-PK was detected in bile duct epithelial cells of untreated rats and in proliferating oval cells, cholangiofibroses and cholangiofibromas of CDE-fed animals. Thus, M2-PK can be viewed as a positive marker of the bile duct epithelial/oval cell compartment. L-PK, a parenchymal cell-specific protein in untreated rat liver, was not present in proliferating oval cells, but was co…

MaleCancer ResearchPathologymedicine.medical_specialtyNecrosisLiver cytologyPyruvate KinaseCellBiologyCholangiocarcinomaRats Sprague-DawleyNecrosisLiver Neoplasms ExperimentalParenchymamedicineAnimalsEthionineBile ductAntibodies MonoclonalGeneral MedicineImmunohistochemistryEpitheliumCholine DeficiencyRatsBile Ducts Intrahepaticmedicine.anatomical_structureBile Duct NeoplasmsLiverHepatocytemedicine.symptomPrecancerous ConditionsCell DivisionPyruvate kinaseCarcinogenesis
researchProduct

HPV DNA and survivin expression in epithelial oral carcinogenesis: a relationship?

2004

Abstract HPV has been thought to be involved in the development of several oral diseases, such as premalignant mucosal lesions and oral carcinoma. Survivin is a recently characterized IAP protein, which is abundantly expressed in most solid and haematological malignancies, but undetectable in normal adult tissues. Aim of this study was to investigate survivin expression and HPV presence in oral premalignant lesions and oral carcinoma. 47 samples of oral tissue including 11 squamous cell carcinomas (OSCC), 16 oral leukoplakias (OL) and 20 normal oral mucosa specimens, after investigation of HPV presence by nested PCR (consensus MY/GP primers) and viral genotype identification by direct seque…

MaleCancer ResearchPathologymedicine.medical_specialtySurvivinCellBiologymedicine.disease_causeInhibitor of Apoptosis ProteinsSurvivinmedicineCarcinomaHumansPapillomaviridaeHPV infectionvirus diseasesCell Transformation Viralmedicine.diseasefemale genital diseases and pregnancy complicationsEpitheliumNeoplasm Proteinsstomatognathic diseasesCell Transformation Neoplasticmedicine.anatomical_structureOncologyEpidermoid carcinomaDNA ViralCarcinoma Squamous CellCancer researchImmunohistochemistryFemaleMouth NeoplasmsLeukoplakia OralOral SurgeryCarcinogenesisMicrotubule-Associated ProteinsPrecancerous ConditionsOral Oncology
researchProduct

The rat liver foci bioassay: II. Investigations on the dose-dependent induction of ATPase-deficient foci by vinyl chloride at very low doses

1985

In order to study the dose-dependence of the genotoxic effect of vinyl chloride (VC) hepatocellular ATPase-deficient foci were evaluated after subchronic exposure of newborn rats. Wistar rats were exposed from day 1 after birth over 10 weeks to 10, 40, 70, 150, 500 and 2000 p.p.m. VC (8 h/day; 5 days/week). One week after cessation of exposure hepatic ATPase-deficient foci were quantitated. For a subsequent investigation lower dose range groups of female and male Wistar and Sprague-Dawley rats were exposed (8 h/day; 5 days/week) to 2.5, 5, 10, 20, 40 and 80 p.p.m. VC. Exposure started at day 3 of life and lasted for 3 weeks. After cessation of exposure the animals were maintained for 10 wee…

MaleCancer ResearchPathologymedicine.medical_specialtyVinyl CompoundsATPaseVinyl ChlorideDose dependenceVinyl chlorideAndrologychemistry.chemical_compoundLiver Neoplasms ExperimentalSex FactorsSpecies SpecificitymedicineAnimalsBioassayCarcinogenAdenosine TriphosphatasesDose-Response Relationship DrugbiologyLow doseRats Inbred StrainsGeneral MedicineRatsLinear relationshipLiverchemistryRat liverbiology.proteinBiological AssayFemalePrecancerous ConditionsCarcinogenesis
researchProduct