Search results for "Predisposition"

showing 10 items of 771 documents

A genetically determined high setting of TNF-alpha influences immunologic parameters of HLA-B8,DR3 positive subjects: implications for autoimmunity.

2001

The 8.1 ancestral haplotype (AH) is a common Caucasoid haplotype carried by most people who type for HLA-B8,DR3. It seems unique in its association with a wide range of immunopathologic diseases. Healthy subjects bearing this haplotype demonstrate several alterations of immune response. This article will focus on the identification of the mechanism(s) of disease susceptibility of 8.1 AH. In 13 carriers of 8.1 AH, and 43 negative patients, enzyme immune assays serum levels of tumor necrosis factor (TNF)-alpha, soluble endothelial leukocyte adhesion molecule-1 (sELAM-1), cortisol, and interleukin(IL)-10 were determined. In addition, quantification of cytokine produced in vitro after mitogen s…

AdultMalemedicine.medical_specialtyHydrocortisonemedicine.medical_treatmentImmunologyHLA-DR3Biologymedicine.disease_causeAutoimmunityAutoimmune DiseasesHLA-B8 AntigenImmune systemHLA-DR3 AntigenInternal medicinemedicineImmunology and AllergyHumansGenetic Predisposition to DiseaseCells CulturedTumor Necrosis Factor-alphaHaplotypeInterleukinGeneral MedicineMiddle AgedInterleukin-10Interleukin 10EndocrinologyCytokineHaplotypesImmunologyTumor necrosis factor alphaFemaleE-SelectinHuman immunology
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Methionine synthase reductase (MTRR) A66G polymorphism is not related to plasma homocysteine concentration and the risk for vascular disease.

2009

Epidemiological evidence has revealed that hyperhomocysteinemia increases the risk for vascular disease. Methionine Synthase Reductase (MTRR) is one of several key enzymes in the homocysteine metabolic pathway and its mutant forms have been implicated in abnormal homocysteine accumulation. In this study, we determined total plasma homocysteine levels and MTRR A66G polymorphism in 114 patients with vascular disease: 58 patients with deep-vein thrombosis, 56 patients with arterial thrombosis, and 95 healthy subjects from the Sicilian population. Our data confirmed that, as already reported, moderately elevated t-Hcy levels are correlated with an increased risk of vascular disease. In our stud…

AdultMalemedicine.medical_specialtyHyperhomocysteinemiaMethyltransferaseHomocysteineClinical BiochemistryPopulationMethionine synthase reductase homocysteinePolymorphism Single NucleotidePathology and Forensic Medicinechemistry.chemical_compoundInternal medicineMedicineHumansGenetic Predisposition to DiseaseVascular DiseaseseducationMolecular BiologyHomocysteineAgededucation.field_of_studybiologybusiness.industryVascular disease(Methionine synthase) reductaseMiddle Agedmedicine.diseaseMTRRFerredoxin-NADP ReductaseVenous thrombosisEndocrinologyBiochemistrychemistryCase-Control Studiesbiology.proteinFemalebusinessExperimental and molecular pathology
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Genetic predisposition to thrombophilia in inflammatory bowel disease

2011

BACKGROUND Inflammatory bowel disease (IBD) is linked to a definite risk of thromboembolic events (TE), but data on the role of prothrombotic genetic mutations are conflicting. STUDY Fourteen genetic factors involved in TE pathogenesis were investigated in a homogeneous cohort of Sicilian patients with IBD with and without history of TE and in healthy controls. Forty IBD patients (21 CD, 19 UC) and 20 healthy individuals were enrolled. Genetic testing was based on the reverse hybridization principle by a commercial assay that analyzes 14 polymorphisms involved in thrombophilia and cholesterol metabolism. The rate of genetic polymorphisms and mutations was compared between IBD patients and h…

AdultMalemedicine.medical_specialtyIBDAngiotensinogenPeptidyl-Dipeptidase AThrombophiliaInflammatory bowel diseaseGastroenterologyPolymorphism (computer science)Risk FactorsInternal medicineDiabetes mellitusGenotypeGenetic predispositionmedicineHumansThrombophiliaGenetic Predisposition to DiseaseAllele frequencySicilyGenetic testingAgedPolymorphism Geneticmedicine.diagnostic_testbusiness.industryGastroenterologyMiddle Agedmedicine.diseaseInflammatory Bowel Diseasesdigestive system diseasesMutationFemalebusiness
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Lack of genetic association of neutral endopeptidase (NEP) with complex regional pain syndrome (CRPS)

2010

Complex regional pain syndrome (CRPS) is a condition that is characterized by severe pain and exaggerated neurogenic inflammation, which may develop after injury or surgery. Neurogenic inflammation is mediated by neuropeptides, such as calcitonin gene-related peptide (CGRP) and substance P (SP) that are released from nociceptors. Genetic factors may play a role in CRPS as was suggested by the occurrence of familial cases and several genetic association studies investigating mainly the human leukocyte antigen (HLA) system. Here we investigated the role of neutral endopeptidase (NEP), a key enzyme in neuropeptide catabolism. NEP dysfunction resulting in reduced inactivation of neuropeptides m…

AdultMalemedicine.medical_specialtyLinkage disequilibrium5' Flanking RegionSubstance PHuman leukocyte antigenBiologyCalcitonin gene-related peptideLinkage Disequilibriumchemistry.chemical_compoundInternal medicinemedicineHumansGenetic Predisposition to DiseaseDinucleotide RepeatsPromoter Regions GeneticNeprilysinGenetic Association StudiesGenetic associationNeurogenic inflammationPolymorphism GeneticGeneral NeurosciencefungiMiddle Agedmedicine.diseaseCRPS Pain NEP Association reflex sympathetic dystrophy syndrome type-i facilitated neurogenic inflammation nociceptive abnormalities alzheimers-disease neprilysin gene rat model enkephalinase prevalence dystoniaEndocrinologyComplex regional pain syndromechemistryCase-Control StudiesFemaleNeprilysinComplex Regional Pain Syndromes
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A Novel Loss of Function Mutation of PCSK9 Gene in White Subjects With Low-Plasma Low-Density Lipoprotein Cholesterol

2007

Objectives— The PCSK9 gene, encoding a pro-protein convertase involved in posttranslational degradation of low-density lipoprotein receptor, has emerged as a key regulator of plasma low-density lipoprotein cholesterol. In African-Americans two nonsense mutations resulting in loss of function of PCSK9 are associated with a 30% to 40% reduction of plasma low-density lipoprotein cholesterol. The aim of this study was to assess whether loss of function mutations of PCSK9 were a cause of familial hypobetalipoproteinemia and a determinant of low-plasma low-density lipoprotein cholesterol in whites. Methods and Results— We sequenced PCSK9 gene in 18 familial hypobetalipoproteinemia subjects and i…

AdultMalemedicine.medical_specialtyNonsense mutationBiologymedicine.disease_causePolymorphism Single NucleotideRisk AssessmentSensitivity and SpecificityStatistics NonparametricWhite Peopleloss of function mutationHypobetalipoproteinemiaschemistry.chemical_compoundPCSK9 GeneGene FrequencyInternal medicinemedicineHumansGenetic Predisposition to DiseaseMutationhypocholesterolemiaCholesterolIncidencePCSK9Serine EndopeptidasesCholesterol LDLmedicine.diseaseHypocholesterolemiaEndocrinologyfamilial hypobetalipoproteinemiachemistryCodon NonsensePCSK9 geneCase-Control Studiesfamilial hypobetalipoproteinemia hypocholesterolemia loss of function mutation PCSK9 genefamilial hypobetalipoproteinemia; hypocholesterolemia; loss of function mutation; PCSK9 gene.FemaleProprotein ConvertasesHypobetalipoproteinemiaProprotein Convertase 9Cardiology and Cardiovascular MedicineLipoprotein
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Incidence of Crohn's disease and CARD15 mutation in a small township in Sicily.

2006

Background: The incidence of Crohn's disease (CD) has been shown to be lower in Southern than in Northern Europe. Data on the frequency of the NOD2/CARD15 mutations for Mediterranean area are very scant. Aim: To determine the incidence of CD from 1979 to 2002 in a township in Sicily together with the allele frequency of NOD2/CARD15 mutations in patients, family members and controls, and to determine the allele frequency of these mutations in sporadic CD from other areas of Sicily in comparison with a control population. Methods: Casteltermini is a small town close to Agrigento (Sicily) with a population of 9,130 inhabitants. All the diagnoses of inflammatory bowel disease (IBD) made from 19…

AdultMalemedicine.medical_specialtyPathologyAdolescentEpidemiologyPopulationNod2 Signaling Adaptor ProteinInflammatory bowel diseaseGastroenterologyCrohn DiseaseGene FrequencyInternal medicineEpidemiologyPrevalencemedicineHumansGenetic Predisposition to DiseaseRisk factoreducationAllele frequencySicilyNOD2/CARD15Crohn's diseaseeducation.field_of_studybusiness.industryIncidence (epidemiology)IncidenceMiddle Agedmedicine.diseaseUlcerative colitisdigestive system diseasesCrohn's diseaseMutationColitis UlcerativeFemalebusinessEuropean journal of epidemiology
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Superficial thrombophlebitis in varicose vein disease: the particular role of methylenetetrahydrofolate reductase.

2010

Background The purpose of this study was to compare the genetic background of superficial (SVT) and deep vein thrombosis (DVT). Methods Factor V (FV)-Leiden (G16891A)-, factor II(G20210A)-mutations, protein C- and S, as well as methylenetetrahydrofolate reductase (MTHFR) polymorphisms at C677T and A1298C, and serum homocysteine levels (hcy) were determined in 29 patients with SVT and 26 with DVT. Findings FV- and –II-mutations were less frequent in patients with SVT (2/3) compared with DVT (9/5), respectively ( P < 0.002 in case of FV). However, the frequency of the MTHFR C677T polymorphism was significantly higher in patients with SVT compared with DVT (CT 12 versus 10, and TT 7 versus …

AdultMalemedicine.medical_specialtyPathologyDeep veinMutation MissenseDiseaseGastroenterologyVaricose VeinsInternal medicineVaricose veinsGenotypemedicineHumansSuperficial thrombophlebitisGenetic Predisposition to DiseaseHomocysteineAgedAged 80 and overMethylenetetrahydrofolate Dehydrogenase (NADP)Venous Thrombosisbiologybusiness.industryFactor VGeneral MedicineMiddle AgedThrombophlebitismedicine.diseaseThrombosisVenous thrombosismedicine.anatomical_structureAmino Acid SubstitutionMethylenetetrahydrofolate reductasebiology.proteinFemaleProthrombinmedicine.symptomCardiology and Cardiovascular MedicinebusinessPhlebology
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Frequency of NOD2/CARD15 variants in both sporadic and familial cases of Crohn's disease across Italy. An Italian Group for Inflammatory Bowel Diseas…

2004

Abstract Background. Three variants of the NOD 2 /CARD 15 gene are strongly associated with susceptibility to Crohn’s disease; however, striking racial and geographic differences of their frequency have been described. Aims. We have compared the allele frequencies of familial cases of Crohn’s disease recruited in a multicentre study across Italy, in order to disclose possible geographic heterogeneity. Moreover, we also compared the allele frequencies in sporadic cases of Crohn’s disease and healthy controls from Southern Italy with those reported in other two populations from Central and Northern Italy. Subjects and Methods. A total of 731 subjects were genotyped for the polymorphism of thr…

AdultMalemedicine.medical_specialtyPathologyGenotypeNod2 Signaling Adaptor ProteinDiseaseInflammatory bowel diseaseGastroenterologyInflammatory bowel diseaseNOD2Frameshift mutationGeneticCrohn DiseaseGene FrequencyPolymorphism (computer science)NOD2Internal medicineMedicineHumansGenetic Predisposition to DiseaseCARD15Frameshift MutationAllele frequencyAgedCrohn's diseasePolymorphism GeneticHepatologybusiness.industrySignificant differenceGastroenterologyIntracellular Signaling Peptides and ProteinsMiddle Agedmedicine.diseaseCrohn's diseaseItalyFemalebusinessCarrier Proteins
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Polymorphisms in the SCD1 gene are associated with indices of stearoyl CoA desaturase activity and obesity: a prospective study.

2013

Scope The serum fatty acid (FA) composition is influenced by dietary fat and the endogenous production of FAs. Stearoyl CoA desaturase 1 (SCD1) is the rate-limiting enzyme catalyzing the synthesis of MUFAs from saturated FAs. Variations in SCD1 activity have been associated with obesity, diabetes, or inflammation. We evaluated the associations between genetic variation of the SCD1 gene, SCD1 activity, intake of oil, and obesity in a population-based prospective study in southern Spain. Methods and results We collected phenotypic, metabolic, nutritional, and genetic information. The type of dietary fat was assessed from samples of cooking oil taken from the participants’ kitchens and analyze…

AdultMalemedicine.medical_specialtyPopulationSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideInternal medicineGenetic variationmedicineHumansPlant OilsGenetic Predisposition to DiseaseCookingObesityProspective StudieseducationOlive OilPhospholipidschemistry.chemical_classificationeducation.field_of_studyFatty acidMiddle Agedmedicine.diseaseObesityDietary FatsEnzyme assayStearoyl-CoA DesaturaseEndocrinologyBiochemistrychemistryHaplotypesSpainbiology.proteinlipids (amino acids peptides and proteins)Stearoyl-CoA desaturase-1Stearoyl-CoA DesaturaseFood ScienceBiotechnologyMolecular nutritionfood research
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Confirmation that the AKT1 (rs2494732) genotype influences the risk of psychosis in cannabis users.

2012

Background Cannabis use is associated with an increased risk of psychosis. One study has suggested that genetic variation in the AKT1 gene might influence this effect. Methods In a case-control study of 489 first-episode psychosis patients and 278 control subjects, we investigated the interaction between variation at the AKT1 rs2494732 single nucleotide polymorphism and cannabis use in increasing the risk of psychosis. Results The rs2494732 locus was not associated with an increased risk of a psychotic disorder, with lifetime cannabis use, or with frequency of use. We did, however, find that the effect of lifetime cannabis use on risk of psychosis was significantly influenced by the rs24947…

AdultMalemedicine.medical_specialtyPsychosisMarijuana AbuseEpisode of CarePolymorphism Single NucleotideRisk AssessmentOddsInternational Classification of DiseasesRisk FactorsLondonSettore MED/48 -Scienze Infermierist. e Tecn. Neuro-Psichiatriche e Riabilitat.medicineConfidence IntervalsOdds RatioHumansGenetic Predisposition to DiseaseGenetic TestingGene–environment interactionPsychiatrySettore MED/25 - PsichiatriaBiological PsychiatryDemographybiologyCase-control studyOdds ratiomedicine.diseasebiology.organism_classificationConfidence intervalPsychotic DisordersSocioeconomic FactorsCase-Control StudiesFemaleGene-Environment InteractionAKT1 gene cannabis use gene environment interaction psychosis schizophrenia signaling pathwaysCannabisRisk assessmentPsychologyProto-Oncogene Proteins c-aktDemographyBiological psychiatry
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