Search results for "Predisposition"

showing 10 items of 771 documents

Statistical and biological gene-lifestyle interactions of MC4R and FTO with diet and physical activity on obesity: new effects on alcohol consumption

2012

Background: Fat mass and obesity (FTO) and melanocortin-4 receptor (MC4R) and are relevant genes associated with obesity. This could be through food intake, but results are contradictory. Modulation by diet or other lifestyle factors is also not well understood. Objective: To investigate whether MC4R and FTO associations with body-weight are modulated by diet and physical activity (PA), and to study their association with alcohol and food intake. Methods: Adherence to Mediterranean diet (AdMedDiet) and physical activity (PA) were assessed by validated questionnaires in 7,052 high cardiovascular risk subjects. MC4R rs17782313 and FTO rs9939609 were determined. Independent and joint associati…

Maleconsumo de alcoholMediterranean dietEpidemiologyhumanoslcsh:MedicineMC4RCardiovascularSocial and Behavioral SciencesBody Mass IndexPsychologylcsh:ScienceBody mass indexmediana edadAged 80 and overancianoMultidisciplinaryMiddle AgedDrinking of alcoholic beveragesAlpha-Ketoglutarate-Dependent Dioxygenase FTOestilo de vidaMedicineReceptor Melanocortin Type 4Consum d'alcoholObesitatFemalePublic HealthFTOResearch Articlemedicine.medical_specialtyEstils de vidaWaistAlcohol DrinkingClinical Research DesignLifestylesAlpha-Ketoglutarate-Dependent Dioxygenase FTOExerciciBiologyInternal medicineDiabetes mellitusmedicineGenetic predispositionHumansObesitySports and Exercise MedicineLife StyleobesidadExercisePrimary CareNutritionAgedBehaviorPhysical activityproteínaslcsh:Ríndice de masa corporalProteinsnutritional and metabolic diseasesAnthropometrymedicine.diseaseObesityEndocrinologyMetabolic Disorderslcsh:QBody mass index
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Germline copy number variation in theYTHDC2gene: does it have a role in finding a novel potential molecular target involved in pancreatic adenocarcin…

2014

Abstract: Objective: The vast majority of pancreatic cancers occurs sporadically. The discovery of frequent variations in germline gene copy number can significantly influence the expression levels of genes that predispose to pancreatic adenocarcinoma. We prospectively investigated whether patients with sporadic pancreatic adenocarcinoma share specific gene copy number variations (CNVs) in their germline DNA. Patients and methods: DNA samples were analyzed from peripheral leukocytes from 72 patients with a diagnosis of sporadic pancreatic adenocarcinoma and from 60 controls using Affymetrix 500K array set. Multiplex ligation-dependent probe amplification (MLPA) assay was performed using a s…

Malecopy number variations germline alteration pancreatic cancer susceptibility YTHDC2 geneDNA Copy Number VariationsSettore MED/06 - Oncologia MedicaClinical BiochemistryAdenocarcinomaBiologyGermlinePancreatic cancerDrug DiscoverymedicineHumansGenetic Predisposition to DiseaseMultiplexProspective StudiesMultiplex ligation-dependent probe amplificationCopy-number variationAlleleGeneGerm-Line MutationAgedAdenosine TriphosphatasesAged 80 and overPharmacologyPharmacology. TherapyDNA HelicasesMiddle Agedmedicine.diseaseMolecular biologyPancreatic NeoplasmsCase-Control StudiesMolecular MedicineAdenocarcinomaFemaleMultiplex Polymerase Chain ReactionRNA HelicasesExpert Opinion on Therapeutic Targets
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Association of SUMO4 M55V polymorphism with autoimmune diabetes in Latvian patients.

2006

Small ubiquitin-related modifier (SUMO4), located in IDDM5, has been identified as a potential susceptibility gene for type 1 diabetes mellitus (T1DM). The novel polymorphism M55V, causing an amino acid change in the evolutionarily conserved met55 residue has been shown to activate the nuclear factor kappaB (NF-kappaB), hence the suspected role of SUMO4 in the pathogenicity of T1DM. The M55V polymorphism has been shown to be associated with susceptibility to T1DM in Asians, but not in Caucasians. Latent autoimmune diabetes in adults (LADA) is a slowly progressive form of T1DM and SUMO4 M55V has not been studied in LADA to date. The current study aims to test whether Latvians are similar to …

Maleendocrine system diseasesAdolescentHuman leukocyte antigenBiologyPolymerase Chain ReactionPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular Biologylaw.inventionAutoimmune DiseasesMethionineHistory and Philosophy of ScienceGene Frequencyimmune system diseaseslawmedicineHumansGenetic Predisposition to DiseaseAlleleChildPolymerase chain reactionAllelesGeneticsType 1 diabetesGeneral NeuroscienceInfantmedicine.diseasePathogenicityLatviaDiabetes Mellitus Type 1Autoimmune diabetesCase-Control StudiesChild PreschoolSmall Ubiquitin-Related Modifier ProteinsAmino acid changeFemaleRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthAnnals of the New York Academy of Sciences
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Microsatellite allele 5 of MHC class I chain-related gene a increases the risk for insulin-dependent diabetes mellitus in latvians.

2006

: Insulin-dependent diabetes mellitus (IDDM) is one of the most common chronic diseases. It is an autoimmune, polygenic disease, associated with several genes on different chromosomes. The most important gene is human leukocyte antigen (HLA), also known as major histocompatibility complex (MHC), which is located on chromosome 6p21.3. HLA-DQ8/DR4 and DQ2/DR3 are positively associated with IDDM and DQ6 is negatively associated with IDDM in most Caucasian populations. The MICA gene is located in the MHC class I region and is expressed by monocytes, keratinocytes, and endothelial cells. Sequence determination of the MICA gene identifies 5 alleles with 4, 5, 6, and 9 repetitions of GCT or 5 repe…

Maleendocrine system diseasesAdolescentHuman leukocyte antigenMajor histocompatibility complexGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceGene FrequencyDiabetes mellitusHLA-DQ AntigensMHC class ImedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneAllelesbiologyGeneral NeuroscienceHistocompatibility Antigens Class IInfant Newbornnutritional and metabolic diseasesChromosomeInfantmedicine.diseaseLatviaDiabetes Mellitus Type 1Child PreschoolImmunologybiology.proteinMicrosatelliteFemaleMicrosatellite RepeatsAnnals of the New York Academy of Sciences
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Stronger Association of Common Variants in TCF7L2 Gene with Nonobese Type 2 Diabetes in the Latvian Population

2012

Polymorphisms in the gene coding for transcription factor 7 like 2 (TCF7L2) are recognized as the strongest common genetic risk factors for type 2 diabetes (T2D) across multiple ethnicities. This study was conducted to evaluate an association between TCF7L2 variants and diabetes susceptibility in the population of Latvia. We genotyped 4 single nucleotide polymorphisms (SNP) rs7901695, rs7903146, rs11196205 and rs12255372 in 1 093 controls and 1 043 diabetic subjects. Association with T2D was found for 3 SNPs rs7901695, rs7903146 and rs12255372 in the whole sample (under an additive genetic model, the adjusted odds ratios (OR) were 1.26, 95% CI [1.08-1.48], P=0.003; OR=1.32, 95% CI [1.12-1.5…

Maleendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismPopulationSingle-nucleotide polymorphismType 2 diabetesBiologyPolymorphism Single NucleotideBody Mass IndexEndocrinologyPolymorphism (computer science)Databases GeneticGenetic modelInternal MedicinemedicineHumansSNPGenetic Predisposition to DiseaseObesityeducationGenetic Association StudiesGeneticseducation.field_of_studynutritional and metabolic diseasesGeneral MedicineOdds ratioMiddle Agedmedicine.diseaseLatviaDiabetes Mellitus Type 2Case-Control StudiesFemaleTranscription Factor 7-Like 2 ProteinTCF7L2Experimental and Clinical Endocrinology & Diabetes
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Tumor Necrosis Factor-α Allele 2 Shows an Association with Insulin-Dependent Diabetes Mellitus in Latvians

2006

Insulin-dependent diabetes mellitus (IDDM) is one of the most common chronic diseases. It is an autoimmune disease. Genes contributing the most for development of IDDM are located on chromosome 6p21.3 in the region called the major histocompatibility complex (MHC). HLA-DQ8/DR4 and DQ2/DR3 have shown positive association with IDDM, while DQ6 has negative association with IDDM in most Caucasian populations. The location of the tumor necrosis factor alpha (TNF-alpha) gene in the MHC suggests the role of TNF in the etiology of IDDM as an autoimmune disease. The TNF region contains several polymorphisms that are associated with different levels of TNF-alpha production and susceptibility to autoi…

Maleendocrine systemmedicine.medical_specialtyAdolescentendocrine system diseasesDiseaseMajor histocompatibility complexPolymerase Chain ReactionGeneral Biochemistry Genetics and Molecular BiologyGene FrequencyHistory and Philosophy of Scienceimmune system diseasesDiabetes mellitusInternal medicinemedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneAllelesAutoimmune diseasebiologyTumor Necrosis Factor-alphabusiness.industryGeneral NeuroscienceHistocompatibility Antigens Class IInfant NewbornInfantnutritional and metabolic diseasesmedicine.diseaseLatviaDiabetes Mellitus Type 1EndocrinologyChild Preschoolbiology.proteinEtiologyFemaleTumor necrosis factor alphabusinessMicrosatellite RepeatsAnnals of the New York Academy of Sciences
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Longitudinal interactions between brain and cognitive measures on reading development from 6 months to 14 years

2017

Dyslexia is a neurobiological disorder impairing learning to read. Brain responses of infants at genetic risk for dyslexia are abnormal already at birth, and associations from infant speech perception to preschool cognitive skills and reading in early school years have been documented, but there are no studies showing predicting power until adolescence. Here we show that in at-risk infants, brain activation to pseudowords at left hemisphere predicts 44% of reading speed at 14 years, and even improves the prediction after taking into account neurocognitive preschool measures of letter naming, phonology, and verbal short-term memory. The association between infant brain responses and reading …

Maleevent-related potentialsspeech perceptionlukeminenDevelopmental psychologyDyslexiaBehavioral NeuroscienceCognition0302 clinical medicineReading (process)Longitudinal StudiesaivotutkimusChildEvoked PotentialsRapid automatized namingta515media_commoninfants05 social sciencesBrainElectroencephalographyCognitionChild PreschoolSpeech PerceptionFemalePsychologySpeech perceptionAdolescentCognitive Neurosciencemedia_common.quotation_subjectExperimental and Cognitive PsychologypitkittäistutkimusLanguage Development050105 experimental psychology03 medical and health scienceschildrenEvent-related potentialmedicineLearning to readHumansdysleksiaGenetic Predisposition to Disease0501 psychology and cognitive sciencesDyslexiaInfantmedicine.diseaseReadingpsykologiset testitlukutaitolukihäiriötNeurocognitive030217 neurology & neurosurgeryNeuropsychologia
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Association between a SLC23A2 gene variation, plasma vitamin C levels, and risk of glaucoma in a Mediterranean population

2011

Purpose Several dietary factors have been associated with glaucoma. Among them, dietary antioxidant intake (i.e., vitamin C and vitamin A) in association with glaucoma has been analyzed, but with mixed results. Genetic factors may play a role in modulating the effect of dietary antioxidant intake on glaucoma; however, nutrigenetic studies in this field are scarce. Our aim was to study the association between selected polymorphisms in key proteins related to vitamin C and vitamin A concentrations and primary open-angle glaucoma (POAG). Methods We performed a case-control study matched for age, sex, and bodyweight. We recruited 300 subjects (150 POAG cases and 150 controls) from a Mediterrane…

Malegenetic structuresMediterranean RegionAscorbic AcidPolymorphism Single Nucleotideeye diseasesGenetics PopulationGene FrequencyRisk FactorsCase-Control StudiesHumansFemaleGenetic Predisposition to DiseaseVitamin ASodium-Coupled Vitamin C TransportersGlaucoma Open-AngleResearch ArticleAged
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The association of variants in PNPLA3 and GRP78 and the risk of developing hepatocellular carcinoma in an Italian population

2016

// Daniele Balasus 1, * , Michael Way 2, * , Caterina Fusilli 3 , Tommaso Mazza 3 , Marsha Y. Morgan 2 , Melchiorre Cervello 4 , Lydia Giannitrapani 1 , Maurizio Soresi 1 , Rosalia Agliastro 5 , Manlio Vinciguerra 2, 6 , Giuseppe Montalto 1, 4 1 Biomedical Department of Internal Medicine and Medical Specialties, University of Palermo, Palermo, Italy 2 Institute for Liver & Digestive Health, Division of Medicine, Royal Free Campus, University College London, London, UK 3 IRCCS Casa Sollievo della Sofferenza, Bioinformatics Unit, San Giovanni Rotondo (FG), Italy 4 Institute of Biomedicine and Molecular Immunology, National Research Council (C.N.R.), Palermo, Italy 5 Immunohematology and Trans…

Malehepatitis C virusSettore MED/09 - Medicina InternaGenome-wide association studyCohort StudiesLiver diseasesingle nucleotide polymorphisms0302 clinical medicineGene FrequencyRisk FactorsEpidemiologyhepatitis C viruEndoplasmic Reticulum Chaperone BiPSicilyHeat-Shock ProteinsLiver NeoplasmsTransfusion medicineHepatitis Chepatocellular carcinomaMiddle Aged3. Good healthOncologyrisk factor030220 oncology & carcinogenesisHepatocellular carcinomaCohort030211 gastroenterology & hepatologyFemaleResearch Papergenetic variantmedicine.medical_specialtyCarcinoma HepatocellularGenotypeSingle-nucleotide polymorphismPolymorphism Single Nucleotide03 medical and health sciencesInternal medicinemedicineHumansGenetic Predisposition to DiseaseAllelesAgedbusiness.industrygenetic variantsMembrane ProteinsLipasemedicine.diseasedigestive system diseasesSurgerybusiness
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Genetic Instability and Intratumoral Heterogeneity in Neuroblastoma with MYCN Amplification Plus 11q Deletion

2013

Background/Aim Genetic analysis in neuroblastoma has identified the profound influence of MYCN amplification and 11q deletion in patients’ prognosis. These two features of high-risk neuroblastoma usually occur as mutually exclusive genetic markers, although in rare cases both are present in the same tumor. The purpose of this study was to characterize the genetic profile of these uncommon neuroblastomas harboring both these high-risk features. Methods We selected 18 neuroblastomas with MNA plus 11q loss detected by FISH. Chromosomal aberrations were analyzed using Multiplex Ligation-dependent Probe Amplification and Single Nucleotide Polymorphism array techniques. Results and Conclusion Thi…

Malelcsh:MedicineMutually exclusive eventsGenetic analysisPediatricsGenetic profileChromosome BreakpointsNeuroblastomaGene duplicationPathologylcsh:ScienceChildGeneticsOncogene ProteinsN-Myc Proto-Oncogene ProteinMultidisciplinaryNuclear ProteinsOncologyChild PreschoolCytogenetic AnalysisMedicineFemaleChromosome DeletionResearch ArticleGenetic MarkersBiologyPolymorphism Single NucleotideCytogeneticsDiagnostic MedicineNeuroblastomamedicineGeneticsCancer GeneticsHumansIn patientGenetic Predisposition to DiseaseneoplasmsBiologyClinical GeneticsChromosomes Human Pair 11lcsh:RGene AmplificationInfantmedicine.diseaseGenetic markerPediatric OncologyMycn amplificationCancer researchlcsh:QBiomarkersGeneral PathologyPLoS ONE
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