Search results for "Prefrontal Cortex"

showing 10 items of 323 documents

Activation of a ΔFOSB dependent gene expression pattern in the dorsolateral prefrontal cortex of patients with major depressive disorder

2010

Abstract Background A ΔFOSB mediated transcriptional response in the nucleus accumbens (NAc) is induced by chronic social stress in rodent and a 50% down-regulation of ΔFOSB has been also reported in the NAc of eight depressed subjects. To evaluate the role of ΔFOSB in the prefrontal cortex which is critically involved in negative cognitive bias associated with major depressive disorder (MDD) we have quantified the mRNA levels of ΔFOSB and of five of its major target genes in the Brodmann area 46 from 24 patients with MDD (11 with psychotic symptoms) and 12 controls. Method Expression of the six genes has been quantified by a real-time quantitative PCR method: ΔFOSB , GRIA2 (encoding the Gl…

Malemedicine.medical_specialtyDown-RegulationGene ExpressionPrefrontal CortexNerve Tissue ProteinsAMPA receptorNucleus accumbensNucleus AccumbensInternal medicineBrodmann area 46medicineHumansReceptors AMPAGRIA2Prefrontal cortexAgedSecretogranin IIIDepressive DisorderDepressive Disorder MajorbiologyDepressionMiddle AgedAntidepressive AgentsDorsolateral prefrontal cortexPsychiatry and Mental healthClinical PsychologyEndocrinologymedicine.anatomical_structurePCP4Case-Control Studiesbiology.proteinFemalePsychologyProto-Oncogene Proteins c-fosNeuroscienceJournal of Affective Disorders
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A feasibility study of application and potential effects of a single session transcranial Direct Current Stimulation (tDCS) on competitive anxiety, m…

2020

Objective : To examine feasibility and potential effects of a single session tDCS over the dorsolateral prefrontal cortex (DLPFC) on competitive anxiety, mood state, and autonomic and endocrine stress responses in elite archer athletes under a real world competition. Methods : Twelve male elite archers volunteered to participate in this pilot trial. Participants were randomized in order to take left anodal DLPFC, left cathodal DLPFC, or sham stimulation (the F3 or F4 areas according to the 10/20 EEG International System) in a within-subject study design. This study included three official competitions. About 45 min before the competition, the tDCS stimulation process was started and the par…

Malemedicine.medical_specialtyHydrocortisonemedia_common.quotation_subjectmedicine.medical_treatmentCompetitive anxietyPrefrontal CortexExperimental and Cognitive PsychologyStimulationSalivary cortisolAudiologyElectroencephalographyAnxietyTranscranial Direct Current StimulationCompetition (biology)Salivary alpha-amylase03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineMedicineHumans0501 psychology and cognitive sciences050102 behavioral science & comparative psychologymedia_commonTranscranial direct-current stimulationbiologymedicine.diagnostic_testbusiness.industryAthletes05 social sciencesbiology.organism_classificationTranscranial direct current stimulation (tDCS)Dorsolateral prefrontal cortexmedicine.anatomical_structureAthletesBrain stimulationAnxietyFeasibility Studiesmedicine.symptomalpha-AmylasesbusinessElite athlete030217 neurology & neurosurgery
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Lasting downregulation of the lipid peroxidation enzymes in the prefrontal cortex of mice susceptible to stress-induced anhedonia

2015

International audience; Antioxidant enzymes and lipid peroxidation in the brain are involved in neuropsychiatric pathologies, including depression. 14- or 28-day chronic stress model induced a depressive syndrome defined by lowered reward sensitivity in C57BL/6J mice and changed gene expression of peroxidation enzymes as shown in microarray assays. We studied how susceptibility or resilience to anhedonia is related to lipid peroxidation in the prefrontal cortex (PFC). With 14-day stress, a comparison of the activities of catalase (CAT), superoxide dismutase (SOD), glutathione peroxidase (GPX) and accumulation of malondialdehyde (MDA) revealed a decrease of the first two measures in suscepti…

Malemedicine.medical_specialtyImipramineAnhedoniaLipid peroxidationDown-RegulationMotor ActivityMicroarrayHippocampusImipraminePrefrontal cortexGene Expression Regulation EnzymologicSuperoxide dismutaseLipid peroxidationFood PreferencesMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalMalondialdehydeInternal medicinemedicineAnimalsChronic stresschemistry.chemical_classificationGlutathione PeroxidasebiologySuperoxide Dismutasebusiness.industryGene Expression ProfilingGlutathione peroxidaseAnhedoniaResilience PsychologicalCatalaseMalondialdehydeAggressionEndocrinologychemistrybiology.protein[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptombusinessChronic stress depression modelStress Psychologicalmedicine.drug
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Dopamine acting through D2 receptors modulates the expression of PSA-NCAM, a molecule related to neuronal structural plasticity, in the medial prefro…

2008

A "neuroplastic" hypothesis proposes that changes in neuronal structural plasticity may underlie the aetiology of depression and the action of antidepressants. The medial prefrontal cortex (mPFC) is affected by this disorder and shows an intense expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a plasticity-associated molecule, which is expressed mainly in interneurons. The monoamines serotonin, dopamine and noradrenaline are the principal targets of antidepressant action. Pharmacological manipulation of serotonin levels regulates synaptophysin and PSA-NCAM expression in the adult mPFC. However, the involvement of structural plasticity on the antidepress…

Malemedicine.medical_specialtyInterneuronDopamineSynaptophysinPrefrontal CortexNeural Cell Adhesion Molecule L1Synaptic TransmissionDopamine agonistRats Sprague-DawleyDevelopmental NeuroscienceDopamineDopamine receptor D2Internal medicinePhenethylaminesmedicineAnimalsNeuronsAnalysis of VarianceMicroscopy ConfocalNeuronal PlasticityGlutamate DecarboxylaseReceptors Dopamine D2ChemistryDopaminergicDopamine antagonistImmunohistochemistryRatsmedicine.anatomical_structureEndocrinologynervous systemNeurologyDopamine receptorDopamine AgonistsSialic AcidsDopamine AntagonistsHaloperidolNeural cell adhesion moleculeNeurosciencemedicine.drugExperimental Neurology
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Chronic Fluoxetine Treatment Increases the Expression of PSA-NCAM in the Medial Prefrontal Cortex

2006

Recent hypotheses suggest that changes in neuronal structure and connectivity may underlie the etiology of depression. The medial prefrontal cortex (mPFC) is affected by depression and shows neuronal remodeling during adulthood. This plasticity may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), which is intensely expressed in the adult mPFC. As the expression of PSA-NCAM is increased by serotonin in other cerebral regions, antidepressants acting on serotonin reuptake may influence PSA-NCAM expression and thus counteract the effects of depression by modulating neuronal structural plasticity. Using immunohistochemistry, we have studied the relationship…

Malemedicine.medical_specialtyInterneuronFluorescent Antibody TechniquePrefrontal CortexCell CountNeural Cell Adhesion Molecule L1urologic and male genital diseasesSerotonergicRats Sprague-Dawleychemistry.chemical_compoundFluoxetineInternal medicinemedicineNeuropilAnimalsPrefrontal cortexNeurotransmitter5-HT receptorNeuronsPharmacologyAnalysis of VarianceRatsPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologyGene Expression Regulationnervous systemchemistryReceptors SerotoninSialic AcidsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeSerotoninPsychologyNeuroscienceNeuropsychopharmacology
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Streptozotocin diabetic mice display depressive-like behavior and alterations in the structure, neurotransmission and plasticity of medial prefrontal…

2015

Diabetes mellitus patients are at increased risk of developing depression, although the neurobiological bases of this comorbidity are not yet fully understood. These patients show CNS alterations, similar to those found in major depression, including changes in the structure and neurotransmission of excitatory neurons. However, although depressive patients and animal models also display alterations in inhibitory networks, little is known about the effects of diabetes on interneurons. Our main objective was to study the impact of diabetes on interneurons of the medial prefrontal cortex (mPFC), one of the regions most affected by major depression. For this purpose we have induced diabetes wit…

Malemedicine.medical_specialtyInterneuronGlutamate decarboxylaseGreen Fluorescent ProteinsSynaptophysinPrefrontal CortexMice TransgenicNeural Cell Adhesion Molecule L1NeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionDiabetes Mellitus ExperimentalInterneuronsInternal medicinemedicineAnimalsPrefrontal cortexDepressive DisorderNeuronal PlasticitybiologyGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyGeneral NeuroscienceDendritesTail suspension testEndocrinologymedicine.anatomical_structurenervous systemExcitatory postsynaptic potentialSynaptophysinbiology.proteinSialic AcidsPsychologyNeuroscienceBrain research bulletin
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Changes in brain oxidative metabolism induced by inhibitory avoidance learning and acute administration of amitriptyline

2007

The effects of antidepressant drugs on memory have been somewhat ignored, having been considered a mere side effect of these compounds. However, the memory impairment caused by several antidepressants could be considered to form part of their therapeutic effects. Amitriptyline is currently one of the most prescribed tricyclic antidepressants, and exerts marked anticholinergic and antihistaminergic effects. In this study, we evaluated the effects of inhibitory avoidance (IA) learning and acute administration of amitriptyline on brain oxidative metabolism. Brain oxidative metabolism was measured in several limbic regions using cytochrome oxidase (CO) quantitative histochemistry. Amitriptyline…

Malemedicine.medical_specialtyMammillary bodyAmitriptylineClinical BiochemistryInfralimbic cortexCentral nervous systemAntidepressive Agents TricyclicToxicologyBiochemistryElectron Transport Complex IVMiceBehavioral NeuroscienceLimbic systemInternal medicineAvoidance LearningAnimalsMedicineAmitriptylinePrefrontal cortexBiological PsychiatryPharmacologybusiness.industryDentate gyrusBrainDiagonal band of Brocamedicine.anatomical_structureEndocrinologybusinessOxidation-Reductionmedicine.drugPharmacology Biochemistry and Behavior
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Changes in histone acetylation in the prefrontal cortex of ethanol-exposed adolescent rats are associated with ethanol-induced place conditioning

2012

Alcohol drinking during adolescence can induce long-lasting effects on the motivation to consume alcohol. Abnormal plasticity in reward-related processes might contribute to the vulnerability of adolescents to drug addiction. We have shown that binge-like ethanol treatment in adolescent rats induces alterations in the dopaminergic system and causes histone modifications in brain reward regions. Considering that histone acetylation regulates transcriptional activity and contributes to drug-induced alterations in gene expression and behavior, we addressed the hypothesis that ethanol is capable of inducing transcriptional changes by histone modifications in specific gene promoters in adolescen…

Malemedicine.medical_specialtyPrefrontal CortexHDAC inhibitionChromatin remodelingHistonesCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicineConditioning PsychologicalmedicineAnimalsEpigeneticsRats WistarConditioned place aversionPharmacologyEthanolbiologyHistone modificationsAge FactorsAcetylationSodium butyrateRatsAdolescenceHistone Deacetylase InhibitorsHistoneEndocrinologychemistryBiochemistryAcetylationbiology.proteinBrain stimulation rewardBinge-like ethanol treatmentHistone deacetylaseFOSBNeuropharmacology
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Temporal abnormalities in children with developmental dyscalculia.

2012

Recent imaging studies have associated Developmental dyscalculia (DD) to structural and functional alterations corresponding Parietal and the Prefrontal cortex (PFC). Since these areas were shown also to be involved in timing abilities, we hypothesized that time processing is abnormal in DD. We compared time processing abilities between 10 children with pure DD (8 years old) and 11 age-matched healthy children. Results show that the DD group underestimated duration of a sub-second scale when asked to perform a time comparison task. The timing abnormality observed in our DD participants is consistent with evidence of a shared fronto-parietal neural network for representing time and quantity.…

Malemedicine.medical_specialtyPsychometricsPsychometricsDevelopmental DisabilitiesStatistics as TopicDyscalculiaAudiologyNeuropsychological TestsDevelopmental psychologyPerceptual DisordersSettore M-PSI/04 - Psicologia Dello Sviluppo E Psicologia Dell'EducazioneDevelopmental DyscalculiaDevelopmental and Educational PsychologymedicineReaction TimeHumansDyscalculia/complications Neuropsychological TestsPrefrontal cortexChildTime processingAnalysis of VarianceNeuropsychology and Physiological PsychologyTemporal IntegrationCase-Control StudiesAcalculiaDevelopmental DyscalculiaTime PerceptionFemaleAnalysis of variancemedicine.symptomAbnormalityPsychologyAnalysis of Variance; Case-Control Studies; Child; Developmental Disabilities; Dyscalculia; Female; Humans; Male; Neuropsychological Tests; Perceptual Disorders; Psychometrics; Reaction Time; Statistics as Topic; Time Perception; Neuropsychology and Physiological Psychology; Developmental and Educational PsychologyDevelopmental neuropsychology
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Time processing in children with Tourette's syndrome.

2010

Background: Tourette syndrome (TS) is characterized by dysfunctional connectivity between prefrontal cortex and sub-cortical structures, and altered meso-cortical and/or meso-striatal dopamine release. Since time processing is also regulated by fronto-striatal circuits and modulated by dopaminergic transmission, we hypothesized that time processing is abnormal in TS. Methods: We compared time processing abilities between nine children with TS-only (i.e. without major psychiatric comorbidities) and 10 age-matched healthy children, employing a time reproduction task in which subjects actively reproduce different temporal intervals, and a time comparison task in which subjects judge whether a …

Malemedicine.medical_specialtyTic severityPhysiologicalCognitive NeuroscienceMatched-Pair AnalysisShort-term memoryPoison controlExperimental and Cognitive PsychologyTourette's syndrome temporal processing time comparison time reproduction tic severityTic severity; Time comparison; Time reproduction; Tourette's syndromel; Case-Control Studies; Child; Humans; Imitative Behavior; Male; Matched-Pair Analysis; Pattern Recognition Physiological; Psychomotor Performance; Reference Values; Time Perception; Tourette Syndrome; Cognitive Neuroscience; Experimental and Cognitive Psychology; Neuropsychology and Physiological PsychologyPattern RecognitionAudiologyTourette's syndromelTourette syndromeArts and Humanities (miscellaneous)Reference ValuesDevelopmental and Educational PsychologyMemory spanmedicineHumansPrefrontal cortexChildTic severity; Time comparison; Time reproduction; Tourette's syndromel; Case-Control Studies; Child; Humans; Imitative Behavior; Male; Matched-Pair Analysis; Pattern Recognition Physiological; Psychomotor Performance; Reference Values; Time Perception; Tourette Syndrome; Neuropsychology and Physiological Psychology; Experimental and Cognitive Psychology; Developmental and Educational Psychology; Arts and Humanities (miscellaneous); Cognitive NeuroscienceIntelligence quotientWorking memoryCognitionmedicine.diseaseImitative BehaviorNeuropsychology and Physiological PsychologyCase-Control StudiesPattern Recognition PhysiologicalTime reproductionTime PerceptionTime comparisonPsychologyPsychomotor PerformanceCognitive psychologyTourette SyndromeBrain and cognition
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