Search results for "Proband"

showing 9 items of 99 documents

Familial Hemiplegic Migraine with an ATP1A4 Mutation: Clinical Spectrum and Carbamazepine Efficacy

2020

An Italian family with familial hemiplegic migraine (FHM) with the absence of mutations in the known genes associated with this disorder, namely ATP1A2, ATP1A3, CACNA1A, and SCN1A, has recently been reported. Soon afterward, whole exome sequencing allowed the identification of the carrier status of a heterozygous ATP1A4 mutation c.1798 C >T, in four affected members of this family. Here we compare the clinical symptoms of the affected family members with those from the other FHM families linked to mutations in the known genes associated with this disorder. A further two-year follow-up, including clinical response to carbamazepine administered to the proband and the maternal grandmother due …

ProbandPediatricsmedicine.medical_specialtyATP1A4 genefamilial hemiplegic migraine; ATP1A4 gene; carbamazepine; clinical symptomsCase Reportmedicine.disease_causelcsh:RC321-57103 medical and health sciences0302 clinical medicineATP1A2ATP1A3medicine<i>ATP1A4</i> genefamilial hemiplegic migrainelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryExome sequencingFamilial hemiplegic migraine030304 developmental biologyclinical symptoms0303 health sciencesMutationbusiness.industryGeneral NeuroscienceCarbamazepinemedicine.diseaseMigrainecarbamazepinebusiness030217 neurology & neurosurgerymedicine.drugBrain Sciences
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High efficiency and clinical relevance of exome sequencing in the daily practice of neurogenetics

2021

ObjectiveTo assess the efficiency and relevance of clinical exome sequencing (cES) as a first-tier or second-tier test for the diagnosis of progressive neurological disorders in the daily practice of Neurology and Genetic Departments.MethodsSixty-seven probands with various progressive neurological disorders (cerebellar ataxias, neuromuscular disorders, spastic paraplegias, movement disorders and individuals with complex phenotypes labelled ‘other’) were recruited over a 4-year period regardless of their age, gender, familial history and clinical framework. Individuals could have had prior genetic tests as long as it was not cES. cES was performed in a proband-only (60/67) or trio (7/67) st…

ProbandPediatricsmedicine.medical_specialtyMovement disordersNeurologyNeurodegeneration with brain iron accumulation[SDV]Life Sciences [q-bio]EncephalopathyNeurogenetics03 medical and health sciences0302 clinical medicineExome SequencingGeneticsHumansMedicineExomeClinical significance030212 general & internal medicineGenetic TestingGenetics (clinical)Exome sequencingComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesbusiness.industrymedicine.diseasePhenotypeNeurology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human genetics[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurology (clinical)Nervous System Diseasesmedicine.symptombusiness030217 neurology & neurosurgery
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Personality patterns in subjects at risk for affective disorders.

1995

The main conclusions of this study on the familial links between personality patterns and affective disorders are: (1) The personality features with the greatest degree of symptomatic overlap with unipolar depression were more common among the first-degree relatives of probands with this diagnosis: thus dysthymic temperament and neuroticism are enhanced in this group of relatives compared to controls. Likewise personality features with a high degree of symptomatic overlap with bipolar affective disorder were more common among the first-degree relatives of probands with this diagnosis. Thus levels of dysthymic and cyclothymic temperament were elevated in this group of relatives compared to c…

ProbandPsychiatric Status Rating Scalesmedicine.medical_specialtyMood Disordersmedia_common.quotation_subjectDiseasemedicine.diseaseNeuroticismPersonality DisordersPsychiatry and Mental healthClinical PsychologyRisk FactorsmedicinePrevalencePersonalityHumansTemperamentBipolar disorderBig Five personality traitsmedicine.symptomPsychiatryPsychologyManiamedia_commonPsychopathology
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The impact of study design and diagnostic approach in a large multi-centre ADHD study: Part 2: Dimensional measures of psychopathology and intelligen…

2011

Contains fulltext : 97437.pdf (Publisher’s version ) (Open Access) BACKGROUND: The International Multi-centre ADHD Genetics (IMAGE) project with 11 participating centres from 7 European countries and Israel has collected a large behavioural and genetic database for present and future research. Behavioural data were collected from 1068 probands with ADHD and 1446 unselected siblings. The aim was to describe and analyse questionnaire data and IQ measures from all probands and siblings. In particular, to investigate the influence of age, gender, family status (proband vs. sibling), informant, and centres on sample homogeneity in psychopathological measures. METHODS: Conners' Questionnaires, St…

ProbandResearch designMale110 012 Social cognition of verbal communicationIntelligencePerception and Actions Mental Health [DCN 1]MedizinSocial Sciencescentre effectsDevelopmental psychology2738 Psychiatry and Mental Health0302 clinical medicinelcsh:PsychiatryMulticenter Studies as Topicsibling designChildATTENTION-DEFICIT/HYPERACTIVITY DISORDERMental DisordersWechsler ScalesWechsler Adult Intelligence Scale10058 Department of Child and Adolescent PsychiatryDiagnostic and Statistical Manual of Mental DisordersPsychiatry and Mental healthPhenotypeConduct disorderResearch DesignRELIABILITYFemaleFamily RelationsPsychologyClinical psychologyPsychopathologyResearch ArticleAdultlcsh:RC435-571DEFICIT HYPERACTIVITY DISORDERTWIN610610 Medicine & health150 000 MR Techniques in Brain Function03 medical and health sciencesmulti-centre studyADHD multi-centre studymedicineCriterion validityAttention deficit hyperactivity disorderHumansADHDGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersSiblingMETAANALYSISFamily HealthPsychiatric Status Rating ScalesCHILD PSYCHIATRIC-PATIENTSSiblingsPARENTmedicine.disease030227 psychiatryAttention Deficit Disorder with HyperactivityCONDUCT DISORDERCRITERION VALIDITY030217 neurology & neurosurgery
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Genotype–phenotype correlation in a new Fabry-disease-causing mutation

2019

Background: Fabry disease (FD) is a rare X-linked inherited lysosomal storage disorder caused by &#945;-galactosidase A deficiency leading to intracellular glycosphingolipid accumulation. FD manifestation is multisystem, and can differ depending on disease-related genetic variants. Currently, more than 700 different FD-causing mutations have been identified in the human GLA gene. We identified a novel mutation in a Lithuanian family with classical manifestations of Fabry disease, revealing severe effects to the cardiovascular systems of heterozygous women. Case presentation: A 49-year-old woman underwent echocardiography due to progressive dyspnea that lasted seven years, reduced physical a…

Probandmedicine.medical_specialtyAbdominal painMedicine (General)α-galactosidase ACase ReportLeft ventricular hypertrophyGastroenterologyclassical manifestationR5-920Internal medicinemedicineGLA geneFabry diseasemedicine.diagnostic_testbusiness.industryCardiac arrhythmiaGeneral MedicineFabry disease ; α-galactosidase A ; GLA gene ; novel mutation ; classical manifestationmedicine.diseaseFabry diseaseHyperintensityMutation (genetic algorithm)<i>GLA</i> geneRenal biopsymedicine.symptomnovel mutationbusiness
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PRENATAL IDENTIFICATION OF A HETEROZYGOUS STATUS IN TWO FETUSES AT RISK FOR GLUCOSE–GALACTOSE MALABSORPTION

1996

Glucose-galactose malabsorption (GGM) is an autosomal recessive disorder which presents with severe osmotic diarrhoea shortly after birth. Two proband siblings with GGM were previously demonstrated to contain a missense mutation (D28N) in the Na + -dependent glucose/galactose cotransporter (SGLTI) that accounts for the defect in sugar absorption. Prenatal screening for GGM was performed in two subsequent pregnancies in this large consanguineous family. The first exon of the SGLTI gene was PCR-amplified from genomic DNA and screened for the presence of the D28N mutation by EcoRV restriction digestion. The proband's sibling was heterozygous and a cousin was not a carrier of the D28N mutation.…

Probandmedicine.medical_specialtyFetusMalabsorptionmedicine.diagnostic_testObstetrics and GynecologyBiologymedicine.diseaseExonEndocrinologyGlucose-galactose malabsorptionInternal medicinemedicineAmniocentesisMissense mutationAlleleGenetics (clinical)Prenatal Diagnosis
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A novel mutation (Thr116IIe) in the presenilin 1 gene in a patient with early-onset Alzheimer's disease

2004

We report a novel presenilin 1 (PSN1) mutation (Thr116Ile) in a woman with early onset Alzheimer's disease (AD). This mutation was not found in 100 healthy controls, indicating that this is not a common polymorphism. The patient presented with forgetfulness at age 45, followed over the next 3 years by a worsening of the memory loss and frequent episodes of confusion and spatial disorientation. Neuroimaging studies were consistent with AD. The analysis of the family's pedigree showed that the proband was apparently the only member affected. Because the early death of several close relatives (i.e. the mother and the grandmother) and the demonstration that the father is not a mutation carrier,…

ThreonineProbandDNA Mutational AnalysisDiseaseBioinformaticsGenetic analysisPresenilinMutation CarrierAlzheimer DiseasePolymorphism (computer science)Presenilin-1medicineHumansEarly-onset Alzheimer's diseaseIsoleucineGeneticsbusiness.industryMembrane ProteinsMiddle Agedmedicine.diseaseNeurologyMutationMutation (genetic algorithm)FemaleSettore MED/26 - NeurologiaNeurology (clinical)businessAlzheimer's Disease Novel mutation Presenilin 1
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LA JUSTICIA EN LA EDAD MEDIA: LA PERVIVENCIA DE LOS PRINCIPIOS INQUISITIVOS Y DE LIBRE APORTACIÓN DE PRUEBA

2017

L'oggetto di studio è la sopravvivenza dei principi che dirigono il onus probandi, ed il modo in cui lo fanno, in diritto romano classico e postclásico nel diritto spagnolo; in particolare, il principio inquisitorio nel Diritto visigoto (Editto di Eurico), Breviario di Alarico e la Lex Visigothorum, e il principio di libera produzione di prova, da un punto di vista estrutturale, nella fonte valenzana medievale: els Furs. El objeto de estudio es el estudio pervivencia de los principios que rigen la carga de la prueba, y el modo en que lo hacen, en derecho romano clásico y postclásico en el derecho español; en concreto, el inquisitivo en el Derecho visigodo (Edicto de Eurico, Breviario de Ala…

diritto visigóticoderecho visigodolcsh:Lawdirittolibera apportazione di provadiritto romanoromano-onusprobandi-principioprincipio inquisitivoderecho romanofurs.onus probandilibre aportación de pruebaprincipio inquisitoriolcsh:KVergentis. Revista de Investigación de la Cátedra Internacional Conjunta Inocencio III
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The revised ghent nosology; reclassifying isolated ectopia lentis

2014

Inherited ectopia lentis (EL) is most commonly caused by Marfan syndrome (MFS), a multisystemic disorder caused by mutations in FBN1. Historically the diagnosis for patients with EL who have no systemic features of MFS is isolated EL (IEL). However, the Ghent nosology for MFS was updated in 2010 and made some important alterations. In particular, patients with EL and a FBN1 mutation are now categorically diagnosed with MFS, if their mutation has previously been described with aortic dilation/dissection. This carries significant systemic implications, as many patients previously diagnosed with IEL are now reclassified. We provide a review of all published cases of IEL caused by FBN1 mutation…

musculoskeletal diseasesProbandMarfan syndromeNosologycongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtybusiness.industrymedicine.disease3. Good healthDissectionGeneticsMedicineIn patientChinese familyAortic dilationbusinessEctopia lentisGenetics (clinical)Clinical Genetics
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