Search results for "Programmed cell death"

showing 10 items of 609 documents

Differentiation-associated apoptosis of neural stem cells is effected by Bcl-2 overexpression: impact on cell lineage determination

2001

Apoptosis is an integral part of neural development. To elucidate the importance of programmed cell death on cell lineage determination we utilized murine PCC7-Mzl cells, a model system for neural differentiation. Treatment of pluripotent PCC7-Mzl stem cells with 0.1 microM all-trans retinoic acid (RA) causes a cease of proliferation and an initiation of differentiation into neurons, glial cells and fibroblasts. Simultaneously, a fraction of the cell culture (ca. 25%) dies within 24 h by apoptosis. We transfected PCC7-Mzl cells with the human bcl-2 cDNA and generated PCC7-Mz-Bcl-2 cell lines expressing two- to tenfold higher levels of Bcl-2 than parental cells. Overexpression of Bcl-2 resul…

Programmed cell deathDNA ComplementaryHistologyCellular differentiationApoptosisTretinoinBiologyCeramidesTransfectionPathology and Forensic MedicineMiceNeurosphereTumor Cells CulturedAnimalsCell LineageElectrophoresis Agar GelNeuronsCaspase 8Stem CellsCell DifferentiationCell BiologyGeneral MedicineFibroblastsMolecular biologyCaspase 9Neural stem cellCell biologyP19 cellProto-Oncogene Proteins c-bcl-2Cell cultureCaspasesStem cellNeurogliaBiomarkersCell DivisionAdult stem cellEuropean Journal of Cell Biology
researchProduct

Toxicity of a dental adhesive compared with ionizing radiation and zoledronic acid

2015

Background: To determine the toxicity of aqueous dilutions of a universal self-priming dental adhesive (DA) and comparing these with those elicited by exposure to ionizing radiation (IR), Zoledronic acid (Z) treatment and the synergic effects of the combined treatment with IR+Z. Material and Methods: The genotoxic effect of DA was determined by the increase in the frequency of micronuclei in cytokinesis-blocked in cultured human lymphocytes before and after exposure to 2Gy of X-rays. The cytotoxic effect was studied by using the MTT cell viability test in normal prostate cell lines (PNT2) after exposure to different X-ray doses (0Gy-20Gy). The cell lines divided into different groups and tr…

Programmed cell deathDental CementsOdontologíaPharmacologymedicine.disease_causeZoledronic AcidIonizing radiationToxicologyPolymethacrylic AcidsRadiation IonizingToxicity TestsmedicineCytotoxic T cellHumansViability assayLymphocytesGeneral DentistryCells CulturedOral Medicine and PathologyDiphosphonatesChemistryResearchImidazoles:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludIn vitroOtorhinolaryngologyToxicityMicronucleus testUNESCO::CIENCIAS MÉDICASSurgeryGenotoxicity
researchProduct

A petrol ether extract of the roots of Onosma paniculatum induces cell death in a caspase dependent manner

2010

Traditional Chinese medicine (TCM) has become very popular in Western countries during the last years. Zicao, a remedy of TCM, has been traditionally used to treat cancer, and, its main constituents, naphthoquinones, have been reported to possess antitumor activity (Chen et al., 2002; Papageorgiou et al., 1999). Here, we prepared extracts of different polarities of Onosma paniculatum Bur.Franch., a plant which is amongst others used as Zicao, but, much less investigated. The extracts were analyzed concerning their growth inhibitory and apoptosis-inducing activity in various tumor cells.Cell viability was measured by XTT viability and a growth inhibition assay. Effects on the cell cycle and …

Programmed cell deathDependent mannerApoptosisOnosma paniculatumGrowth inhibitoryEtherCaspase 3Traditional Chinese medicinePharmacologyEtherPlant Rootschemistry.chemical_compoundCell Line TumorNeoplasmsDrug DiscoveryHumansCaspaseCell ProliferationPharmacologyDose-Response Relationship DrugbiologyCaspase 3Cell CycleBoraginaceaeAntineoplastic Agents Phytogenicchemistrybiology.proteinDrugs Chinese HerbalPhytotherapyJournal of Ethnopharmacology
researchProduct

Cardiac Glycosides Exert Anticancer Effects by Inducing Immunogenic Cell Death

2012

Some successful chemotherapeutics, notably anthracyclines and oxaliplatin, induce a type of cell stress and death that is immunogenic, hence converting the patient's dying cancer cells into a vaccine that stimulates antitumor immune responses. By means of a fluorescence microscopy platform that allows for the automated detection of the biochemical hallmarks of such a peculiar cell death modality, we identified cardiac glycosides (CGs) as exceptionally efficient inducers of immunogenic cell death, an effect that was associated with the in- hibition of the plasma membrane Na + - and K + -dependent adenosine triphosphatase (Na + /K + -ATPase). CGs ex- acerbated the antineoplastic effects of DN…

Programmed cell deathDigoxinOrganoplatinum Compoundsmedicine.medical_treatment[SDV]Life Sciences [q-bio]Antineoplastic AgentsBiosensing TechniquesBiologyPharmacologyCardiac Glycosides03 medical and health sciencesMice0302 clinical medicineImmune systemCell Line TumorNeoplasmsmedicineAnimalsHumansAnthracyclinesComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesChemotherapyGeneral Medicinemedicine.disease3. Good healthOxaliplatinOxaliplatinCell culture030220 oncology & carcinogenesisHepatocellular carcinomaCancer cellImmunogenic cell deathmedicine.drug
researchProduct

Involvement of Microglia in Neurodegenerative Diseases: Beneficial Effects of Docosahexahenoic Acid (DHA) Supplied by Food or Combined with Nanoparti…

2021

Neurodegenerative diseases represent a major public health issue and require better therapeutic management. The treatments developed mainly target neuronal activity. However, an inflammatory component must be considered, and microglia may constitute an important therapeutic target. Given the difficulty in developing molecules that can cross the blood–brain barrier, the use of food-derived molecules may be an interesting therapeutic avenue. Docosahexaenoic acid (DHA), an omega-3 polyunsaturated fatty acid (22:6 omega-3), has an inhibitory action on cell death and oxidative stress induced in the microglia. It also acts on the inflammatory activity of microglia. These data obtained in vitro or…

Programmed cell deathDocosahexaenoic AcidsQH301-705.5microgliaApoptosisInflammationReviewPharmacologyProtective AgentsInhibitory postsynaptic potentialmedicine.disease_causeCatalysisInorganic ChemistryDrug Delivery Systemsneurodegenerative diseasemedicineAnimalsHumansBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopychemistry.chemical_classificationMicrogliabusiness.industryOrganic ChemistryNeurodegenerative DiseasesGeneral Medicinedocosahexaenoic acidnanomedicineIn vitroComputer Science ApplicationsDisease Models AnimalOxidative StressChemistryTreatment Outcomemedicine.anatomical_structurechemistryBlood-Brain BarrierinflammationDocosahexaenoic acidDietary SupplementsNanoparticlesmedicine.symptombusinessOxidative stressPolyunsaturated fatty acidInternational Journal of Molecular Sciences
researchProduct

The Sphingosine Kinase-1 Activator, K6PC-5, Attenuates the Ebola Virus Infection and the Virus Induced Cell Death

2020

Ebola virus (EBOV) is responsible for outbreaks with case-fatality rates of up to 90% and for an epidemic in West Africa with more than ten thousand deaths. EBOV glycoprotein (EBOV-GP) is the only viral surface protein and is responsible for viral entry into cells. It has been suggested to play a role in the cytopathic effects induced by the virus. Here we uncover a critical role for sphingolipids in inhibiting viral entry and virus-mediated cytotoxicity. Sphingosine kinase 1 (SphK1) catalyzes the phosphorylation of sphingosine to sphingosine-1-phosphate (S1P).  The administration of the SphK1 activator, K6PC-5, or S1P, or the overexpression of SphK1 consistently exhibited striking inhibito…

Programmed cell deathEbola virusSphingosineActivator (genetics)Biologymedicine.disease_causeVirologySphingolipidViruschemistry.chemical_compoundSphingosine kinase 1chemistryViral entrybiology.proteinmedicineSSRN Electronic Journal
researchProduct

Toxic effects induced by vanadium on sea urchin embryos

2020

Vanadium, a naturally occurring element widely distributed in soil, water and air, has received considerable interest because its compounds are often used in different applications, from industry to medicine. While the possible medical use of vanadium compounds is promising, its potential harmful effects on living organisms are still unclear. Here, for the first time, we provide a toxicological profile induced by vanadium on Paracentrotus lividus sea urchin embryos, reporting an integrated and comparative analysis of the detected effects reflecting vanadium-toxicity. At the morphological level we found a dose-dependent induction of altered phenotypes and of skeletal malformations. At the mo…

Programmed cell deathEmbryo NonmammalianEnvironmental EngineeringHealth Toxicology and Mutagenesis0208 environmental biotechnologyVanadium-stressVanadiumchemistry.chemical_elementApoptosis02 engineering and technology010501 environmental sciences01 natural sciencesParacentrotus lividusDevelopmental abnormalityCellular stress responseHeat shock proteinAutophagyAnimalsHumansEnvironmental ChemistrySettore BIO/06 - Anatomia Comparata E Citologia0105 earth and related environmental sciencesHeat shock proteinsbiologyChemistryAutophagyPublic Health Environmental and Occupational HealthVanadiumGeneral MedicineGeneral Chemistrybiology.organism_classificationPollution020801 environmental engineeringCell biologyApoptosisParacentrotus lividus embryosToxicityParacentrotusChemosphere
researchProduct

Sea urchin embryos exposed to cadmium as an experimental model for studying the relationship between autophagy and apoptosis

2014

The sea urchin embryo is a suitable model that offers an excellent opportunity to investigate different defence strategies activated in stress conditions. We previously showed that cadmium accumulates in a dose- and time-dependent manner into embryonic cells, activating different stress and defence mechanisms, including the synthesis of HSPs and the onset of apoptosis and/or autophagy. In this paper we investigated the functional relationship between autophagy and apoptosis, evaluating apoptosis signals in cadmium-exposed Paracentrotus lividus embryos with inhibited autophagy. We found that the inhibition of autophagy produced the concurrent reduction of apoptosis, suggesting that the two p…

Programmed cell deathEmbryo NonmammalianImmunocytochemistryApoptosisAquatic ScienceBiologyOceanographyApoptosis Autophagy Stress Cadmium Sea urchin EmbryoParacentrotus lividusAutophagyAnimalsSettore BIO/06 - Anatomia Comparata E CitologiaTUNEL assayCaspase 3AutophagyEmbryoGeneral Medicinebiology.organism_classificationPollutionEmbryonic stem cellCell biologyApoptosisModels AnimalParacentrotusWater Pollutants ChemicalCadmiumMarine Environmental Research
researchProduct

Pro-inflammatory T helper 17 directly harms oligodendrocytes in neuroinflammation.

2021

Significance Multiple sclerosis (MS) is a neuroinflammatory, demyelinating disease that represents one of the most frequent causes of irreversible disability in young adults. Treatment options to halt disability are limited. We discovered that T helper (Th)17 cells in contact with oligodendrocytes produce higher levels of glutamate and induce significantly greater oligodendrocyte damage than their Th2 counterpart. Blockade of CD29, which is linked to glutamate release pathways and expressed in high levels on Th17 cells, preserved human oligodendrocyte processes from Th17-mediated injury. Our data thus provide evidence for the direct and deleterious attack of Th17 cells on the myelin compart…

Programmed cell deathEncephalomyelitis Autoimmune ExperimentalCentral nervous systemFreund's AdjuvantoligodendrocytesMice Transgenicglutamate03 medical and health sciencesMyelinMice0302 clinical medicineImmunology and Inflammationintravital microscopymedicineAnimalsNeuroinflammation030304 developmental biologyInflammationMice Knockout0303 health sciencesMultidisciplinaryChemistryMultiple sclerosisGlutamate receptorMembrane ProteinsCD29Biological SciencesCD29 blockademedicine.disease420Oligodendrocyte3. Good healthCell biologyDNA-Binding ProteinsMice Inbred C57BLOligodendrogliamedicine.anatomical_structurePertussis ToxinTh17 CellsMyelin-Oligodendrocyte Glycoprotein030217 neurology & neurosurgeryProceedings of the National Academy of Sciences of the United States of America
researchProduct

The role of FAS/FASLig and apoptosis in β-cell death in animal models: relevance for human IDDM

1998

Programmed cell deathEndocrinologyText miningbusiness.industryApoptosisEndocrinology Diabetes and MetabolismDiabetes mellitusMedicineRelevance (information retrieval)businessBioinformaticsmedicine.diseaseBiochemistryDiabetes / Metabolism Reviews
researchProduct