Search results for "Prostacyclin"

showing 10 items of 60 documents

Diabetes potentiates acetylcholine-induced relaxation in rabbit renal arteries.

2001

Abstract The response of rabbit renal arteries to acetylcholine and its endothelial modulation in diabetes were investigated. Acetylcholine induced concentration-related endothelium-dependent relaxation of renal arteries that was significantly more potent in diabetic rabbits than in control rabbits. Pretreatment with NG-nitro- l -arginine ( l -NOArg), indomethacin, or l -NOArg plus indomethacin induced partial inhibition of acetylcholine-induced relaxation. Inhibition induced by l -NOArg plus indomethacin was significantly higher in arteries from diabetic rabbits than in arteries from control rabbits. In renal arteries depolarised with KCl 30 mM and incubated with l -NOArg plus indomethacin…

MaleNitroprussidemedicine.medical_specialtyArginineEndotheliumVasodilator AgentsIndomethacinProstacyclinNitric OxideNitroarginineNitric oxideDiabetes Mellitus Experimentalchemistry.chemical_compoundRenal Arterymedicine.arteryInternal medicineMedicineAnimalsRenal arteryEnzyme InhibitorsPharmacologybusiness.industryAnti-Inflammatory Agents Non-SteroidalAcetylcholineVasodilationEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemSodium nitroprussideEndothelium VascularRabbitsbusinessAcetylcholinemedicine.drugArteryEuropean journal of pharmacology
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A polymorphism in the cyclooxygenase 2 gene as an inherited protective factor against myocardial infarction and stroke

2004

CONTEXT: Myocardial infarction (MI) and ischemic stroke are thought to be caused by matrix digestion by metalloproteinases (MMPs) leading to rupture of atherosclerotic plaques. Production of macrophage MMP-2 and MMP-9 is induced by cyclooxygenase 2 (COX-2) and prostaglandin E(2) synthesis. Although COX-2 expression may be genetically determined, the relation between COX-2 polymorphisms and the risk of MI and stroke is unclear. OBJECTIVE: To investigate the relationship between the -765G-->C polymorphism of the COX-2 gene and clinically evident plaque rupture. DESIGN, SETTING, AND PARTICIPANTS: Prospective, matched case-control study conducted between March 2002 and October 2003 among 864 pa…

MalePathologySettore MED/09 - Medicina InternaArteriosclerosisCarotid StenosiMyocardial InfarctionInfarctionProstacyclinGastroenterologyCohort StudiesCerebrovascular AccidentrteriosclerosiRisk FactorsGenotypeMedicineCarotid StenosisProspective StudiesMyocardial infarctionMembrane ProteinStrokebiologyGeneral MedicineMiddle AgedIsoenzymesStrokePhenotypeMatrix Metalloproteinase 9Matrix Metalloproteinase 2FemaleHumanmedicine.drugmedicine.medical_specialtyGenotypeArteriosclerosiInternal medicineDiabetes mellitusHumansPolymorphism Geneticbusiness.industryC-reactive proteinProstaglandin-Endoperoxide SynthaseMembrane Proteinsmedicine.diseaseEpoprostenolIsoenzymeProspective StudieAtheromaCyclooxygenase 2Prostaglandin-Endoperoxide Synthasesbiology.proteinCohort Studiebusiness
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Estrogens inhibit angiotensin II-induced leukocyte-endothelial cell interactions in vivo via rapid endothelial nitric oxide synthase and cyclooxygena…

2002

Angiotensin II (Ang II) may be a key molecule in the development of atherosclerosis. Because the incidence of coronary atherosclerosis in premenopausal women is lower than that observed in men or postmenopausal women, we have investigated the effect of estrogens on Ang II–induced leukocyte recruitment in vivo using intravital microscopy in the rat mesenteric microcirculation. Superfusion for 60 minutes with Ang II induced a significant increase in leukocyte rolling flux, adhesion, and emigration. Administration of 17-β-estradiol (17-β-E) after 30 minutes of Ang II superfusion produced a reduction of these leukocyte responses by 55.1%, 72.7%, and 70.9%, respectively, an additional 30 minutes…

MaleSelective Estrogen Receptor Modulatorsmedicine.medical_specialtyEndotheliumPhysiologyLeukocyte RollingProstacyclinCell CommunicationBiologyIn Vitro TechniquesLosartanReceptor Angiotensin Type 1Lymphatic SystemRats Sprague-DawleyAngiotensin Receptor AntagonistsCell MovementInternal medicinemedicineCell AdhesionLeukocytesAnimalsHumansSplanchnic CirculationEnzyme InhibitorsCells CulturedVenuleEstradiolAngiotensin IIEstrogen AntagonistsAntibodies MonoclonalEstrogensAngiotensin IIEpoprostenolRatsEndothelial stem cellNitric oxide synthasemedicine.anatomical_structureEndocrinologyProstaglandin-Endoperoxide Synthasesbiology.proteinEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular Medicinehormones hormone substitutes and hormone antagonistsIntravital microscopymedicine.drugCirculation research
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Staphylococcal α-toxin provokes coronary vasoconstriction and loss in myocardial contractility in perfused rat hearts: Role of thromboxane generation

2000

Background —Cardiac performance is severely depressed in septic shock. Endotoxin has been implicated as the causative agent in Gram-negative sepsis, but similar abnormalities are encountered in Gram-positive sepsis. We investigated the influence of the major exotoxin of Staphylococcus aureus, staphylococcal α-toxin, in isolated perfused rat hearts. Methods and Results —α-Toxin 0.25 to 1 μg/mL caused a dose-dependent increase in coronary perfusion pressure that more than doubled. In parallel, we noted a decrease in left ventricular developed pressure and the maximum rate of left ventricular pressure rise (dP/dt max ), dropping to a minimum of <60% of control. These changes were accompani…

MaleThromboxaneIndomethacinProstacyclinVentricular Function LeftHemolysin ProteinsThromboxane A2chemistry.chemical_compoundEdemaPhenylacetatesSulfonamidesHeartAzepinesPerfusionAnesthesiaLactatesVentricular pressuremedicine.symptomCardiology and Cardiovascular Medicinemedicine.drugStaphylococcus aureusmedicine.medical_specialtyBacterial ToxinsExotoxinsIn Vitro TechniquesSepsisContractilityThromboxane A2Physiology (medical)Internal medicinemedicineAnimalsMasoprocolPlatelet Activating FactorRats WistarAspirinL-Lactate Dehydrogenasebusiness.industryTriazolesmedicine.diseaseEpoprostenolMyocardial ContractionRatsEndocrinologychemistryVasoconstrictionPotassiumCoronary perfusion pressurebusinessPlatelet Aggregation InhibitorsVasoconstriction
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Modulation of adrenergic contraction of dog pulmonary arteries by nitric oxide and prostacyclin.

1999

Abstract The aim of this work was to investigate the influence of endothelium-derived nitric oxide and prostaglandins on the contractile responses of isolated dog pulmonary arteries to electrical field stimulation and noradrenaline. Electrical field stimulation (1–8 Hz, 20 v, 0.25 ms duration, for 30 s) produced frequency-dependent contractions that were abolished by tetrodotoxin, guanethidine and, prazosin (all at 10−6 M). Noradrenaline induced concentration-dependent contractions with an EC50 of 1.85 × 10−6 M. The increases in tension induced by electrical stimulation and noradrenaline were of greater magnitude in arteries denuded of endothelium. In segments with endothelium, NG-nitro- l …

Malemedicine.medical_specialtyEndotheliumArginineIndomethacinAdrenergicProstacyclinStimulationIn Vitro TechniquesPulmonary ArteryNitric OxideNitric oxidechemistry.chemical_compoundNorepinephrineDogsInternal medicinePrazosinmedicineAnimalsCyclooxygenase InhibitorsDrug InteractionsEnzyme InhibitorsGuanethidineAntihypertensive AgentsPharmacologyEpoprostenolElectric Stimulationmedicine.anatomical_structureEndocrinologyNG-Nitroarginine Methyl EsterchemistryVasoconstrictionProstaglandinsFemalemedicine.drugGeneral pharmacology
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Estradiol selectively stimulates endothelial prostacyclin production through estrogen receptor-α

2010

Estradiol (E2) acts on the endothelium to promote vasodilatation through the release of several compounds, including prostanoids, which are products of arachidonic acid metabolism. Among these, prostacyclin (PGI2) and thromboxane A2 (TXA2) exert opposite effects on vascular tone. The role of different estrogen receptors (ERs) in the PGI2/TXA2 balance, however, has not been fully elucidated. Our study sought to uncover whether E2 enhances basal production of PGI2 or TXA2 in cultured human umbilical vein endothelial cells (HUVECs), to analyze the enzymatic mechanisms involved, and to evaluate the different roles of both types of ERs (ERα and ERβ). HUVECs were exposed to E2, selective ERα (1,3…

Malemedicine.medical_specialtyEndotheliumDiarylpropionitrileEstrogen receptorProstacyclinBiologyThromboxane A2chemistry.chemical_compoundThromboxane A2EndocrinologyCytochrome P-450 Enzyme SystemInternal medicinemedicineEstrogen Receptor betaHumansMolecular BiologyCells CulturedEstradiolGroup IV Phospholipases A2Estrogen Receptor alphaEndothelial CellsProstanoidEpoprostenolIntramolecular OxidoreductasesEndocrinologymedicine.anatomical_structurechemistryCyclooxygenase 1cardiovascular systembiology.proteinFemalelipids (amino acids peptides and proteins)Endothelium VascularThromboxane-A synthaseEstrogen receptor alphamedicine.drugJournal of Molecular Endocrinology
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Mechanisms underlying the diabetes-induced hyporeactivity of the rabbit carotid artery to atrial natriuretic peptide

2010

Abstract Atrial natriuretic peptide (ANP) plays an important role in the pathophysiology of the vascular complications in diabetes. The working hypothesis was that diabetes might modify the vascular actions of ANP in isolated rabbit carotid arteries and the mechanisms involved in these actions. ANP (10 −12 –10 −7  M) induced a relaxation of precontracted carotid arteries, which was lower in diabetic than in control rabbits. In arteries from both groups of animals, endothelium removal increased the ANP-induced relaxation. Isatin inhibited the relaxation to ANP both in arteries with and without endothelium. Carotid arteries from diabetic rabbits showed a decreased natriuretic peptide receptor…

Malemedicine.medical_specialtyEndotheliummedicine.drug_classThromboxaneDown-RegulationProstacyclinVasodilationDiabetes Mellitus ExperimentalRandom AllocationAtrial natriuretic peptideDiabetes mellitusInternal medicinemedicineNatriuretic peptideAnimalsReceptorPharmacologybusiness.industrymedicine.diseaseVasodilationCarotid Arteriesmedicine.anatomical_structureEndocrinologycardiovascular systemRabbitsbusinessAtrial Natriuretic Factorhormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologymedicine.drugPharmacological Research
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Effects of zinc acexamate on blood flow and prostanoid levels in the gastric mucosa of the rat

1989

The effects of the new antiulcer compound zinc acexamate on blood flow and prostanoid levels in the gastric mucosa have been studied. Zinc acexamate (30 and 300 mg/kg) dose-dependently prevents the reduction induced by the perfusion of noradrenaline (3.5 micrograms/kg.min, 30 min) in gastric mucosal blood flow, as measured by 3H-aniline clearance. Zinc acexamate pretreatment also increases the levels of prostaglandin E2 in the gastric mucosa of the rat, both under control conditions and after infusion with noradrenaline. The levels of thromboxane A2 and prostacyclin were not modified by zinc acexamate. These results confirm the importance of microcirculation in pathogenesis and the idea tha…

Malemedicine.medical_specialtyMetabolic Clearance RateClinical BiochemistryProstacyclinBiologyMicrocirculationNorepinephrinechemistry.chemical_compoundThromboxane A2Internal medicinemedicineGastric mucosaAnimalsProstaglandin E2Chromatography High Pressure LiquidAminocaproatesStomachProstanoidRats Inbred StrainsCell BiologyAnti-Ulcer AgentsRatsEndocrinologymedicine.anatomical_structurechemistryGastric MucosaRegional Blood FlowAminocaproic AcidProstaglandinsPerfusionmedicine.drugProstaglandins, Leukotrienes and Essential Fatty Acids
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Synergistic platelet antiaggregatory effects of the adenylate cyclase activator iloprost and the guanylate cyclase activating agent SIN-1 in vivo

1993

The aim of our study was to evaluate the platelet antiaggregatory and hemodynamic effects of the stable prostacyclin analog iloprost and the NO-donor SIN-1, an active metabolite of molsidomine. The number of circulating platelets was determined in anesthetized male Wistar rats as a measure of in vivo platelet aggregation. Platelet count decreased from 648 +/- 25 to 476 +/- 15 x 10(3) platelets/microliter and from 578 +/- 36 to 411 +/- 40 (mean +/- SEM) in response to two repetitive injections of collagen (70 micrograms/kg body weight). Treatment with SIN-1 bolus injections (0.3 or 1 mg/kg bw) and/or continuous i.v. infusion of iloprost (0.2 or 0.4 micrograms/kg bw/min) was initiated 15 min …

Malemedicine.medical_specialtyMolsidominePlatelet AggregationPlatelet aggregationBlood PressureProstacyclinNitric Oxidechemistry.chemical_compoundIn vivoInternal medicinemedicineAnimalsPlateletIloprostRats WistarAntihypertensive AgentsActive metaboliteChemistryDrug SynergismHematologyRatsEnzyme ActivationEndocrinologyGuanylate CyclaseMolsidomineAdenylyl Cyclase InhibitorsPlatelet Aggregation InhibitorsSignal TransductionIloprostmedicine.drugGuanylate cyclaseThrombosis Research
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Diabetes impairs the atrial natriuretic peptide relaxant action mediated by potassium channels and prostacyclin in the rabbit renal artery.

2012

Diabetes is associated with increased prevalence of hypertension, cardiovascular and renal disease. Atrial natriuretic peptide (ANP) plays an important role in cardiovascular pathophysiology and is claimed to have cardioprotective and renoprotective effect in diabetic patients. The working hypothesis was that alloxan-induced diabetes might modify the vascular effects of ANP in isolated rabbit renal arteries and the mechanisms involved in such actions. Plasma ANP levels were higher in diabetic rabbits than in control rabbits. ANP (10(-12)-10(-7)M) induced a relaxation of precontracted renal arteries, which was lower in diabetic than in control rabbits. In arteries from both groups of animals…

Malemedicine.medical_specialtyPotassium ChannelsEndotheliumProstacyclinIn Vitro TechniquesDiabetes Mellitus ExperimentalGlibenclamideThromboxane A2chemistry.chemical_compoundThromboxane A2Renal ArteryAtrial natriuretic peptidemedicine.arteryDiabetes mellitusInternal medicinemedicineAnimalsRenal arteryPharmacologybusiness.industryTetraethylammoniummedicine.diseaseEpoprostenolPotassium channelVasodilationEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemEndothelium VascularRabbitsbusinesshormones hormone substitutes and hormone antagonistsAtrial Natriuretic Factorcirculatory and respiratory physiologymedicine.drugPharmacological research
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