Search results for "Protective"

showing 10 items of 512 documents

Physical activity as a preventive measure for coronary heart disease risk factors in early childhood.

2004

Physical activity causes acute physiological and long-term adaptive responses in the body. It is a protective factor for coronary heart disease (CHD) in adults. It has been assumed that children younger than 8 years of age may be in general active enough and there would be hardly any relationships between physical activity and CHD risk factors in early childhood. One hundred and fifty-five children (age 4-7 years) participated in this physical activity study during three consecutive years. Physical activity was examined twice a year with a special-purpose physical activity diary. CHD risk factors were measured during annual health care visits in the Specific Turku Coronary Risk-Factor Inter…

Malemedicine.medical_specialtyPediatricsProtective factorPhysical activityPhysical Therapy Sports Therapy and RehabilitationBlood PressureCoronary DiseaseGrowthRisk FactorsInternal medicinemedicineHumansOrthopedics and Sports MedicineEarly childhoodChildExerciseFinlandbusiness.industrySerum HDL cholesterolLipidsCoronary heart diseaseBlood pressureChild PreschoolFemalebusinessChd riskLipoproteinScandinavian journal of medicinescience in sports
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Improvement of the circulatory function partially accounts for the neuroprotective action of the phytoestrogen genistein in experimental ischemic str…

2012

Abstract We tested the hypothesis that the phytoestrogen genistein protects the brain against ischemic stroke by improving the circulatory function in terms of reduced production of thromboxane A2 and leukocyte–platelet aggregates, and of preserved vascular reactivity. Ischemia-reperfusion (90 min-3 days, intraluminal filament) was induced in male Wistar rats, and functional score and cerebral infarct volume were the end points examined. Genistein (10 mg/kg/day) or vehicle (β-cyclodextrin) was administered at 30 min after ischemia or sham-operation. Production of thromboxane A2 and leukocyte–platelet aggregates, as well as reactivity of carotid artery to U-46619 (thromboxane A2 analogue) an…

Malemedicine.medical_specialtyPlatelet AggregationIschemiaGenisteinPhytoestrogensNeuroprotectionBrain IschemiaThromboxane A2Thromboxane A2chemistry.chemical_compoundInternal medicinemedicineAnimalsPlateletRats WistarStrokePharmacologyChemistrymedicine.diseaseGenisteinRatsStrokeNeuroprotective AgentsEndocrinologyCirculatory systemIschemic strokeEuropean Journal of Pharmacology
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Postischemic application of lipid peroxidation inhibitor U-101033E reduces neuronal damage after global cerebral ischemia in rats.

1998

Background and Purpose —The lipid peroxidation inhibitor U-101033E was examined for effects on cerebral blood flow (CBF), cortical tissue hemoglobin oxygen saturation (HbS o 2 ), and neuronal damage. Methods —Fifteen minutes of global cerebral ischemia was induced by two-vessel occlusion and hypobaric hypotension. Wistar rats (n=25) were randomized to receive vehicle (n=9) or 40 mg/kg U-101033E (n=9) intraperitoneally during 2 hours of reperfusion. A sham group (n=7) had neither ischemia nor therapy. Histology was evaluated 7 days after ischemia. Results —During late hyperperfusion (at 17 minutes), vehicle-treated animals had a higher ( P =0.044) cortical tissue HbS o 2 (72.0±1.4%) than di…

Malemedicine.medical_specialtyPyrrolidinesVasodilator AgentsIschemiaCell CountHippocampal formationAntioxidantsCentral nervous system diseaseLipid peroxidationchemistry.chemical_compoundCortex (anatomy)Internal medicinemedicineLaser-Doppler FlowmetryAnimalsRats WistarAdvanced and Specialized NursingNeuronsbusiness.industryCarbon Dioxidemedicine.diseaseSurgeryRatsEndocrinologymedicine.anatomical_structureNeuroprotective AgentsPyrimidinesCerebral blood flowchemistryCerebral cortexIschemic Attack TransientCerebrovascular CirculationOxyhemoglobinsNeurology (clinical)Lipid PeroxidationHypotensionCardiology and Cardiovascular MedicinebusinessReperfusion injuryStroke
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[3H]-DA release evoked by low pH medium and internal H+ accumulation in rat hypothalamic synaptosomes: involvement of calcium ions

2003

The pH fluctuations have been often interpreted as an insufficient regulation or as a consequence of the onset of pathological events, such as ischemia, in which a significant decrease in pH levels occurs. Neurotransmitter release appears to be affected by pH drop significantly. In this study, we investigated the effect of an extracellular and an intracellular acidification on tritiated dopamine release ([3H]-DA release), from superfused rat hypothalamic synaptosomes. When compared to basal release, extracellular acidification, due to a reduction in the external pH of the nominally carbonic-free superfusion media, provoked a significant increase in [3H]-DA release that showed a sensitivenes…

Malemedicine.medical_specialtySodium-Hydrogen ExchangersNigericinDopamineHypothalamusIonophoreIntraterminal acidificationchemistry.chemical_elementIn Vitro TechniquesCalciumCalcium in biologyPotassium ChlorideAmiloridehypothalamic synaptosomesCellular and Molecular Neurosciencechemistry.chemical_compoundDopamineInternal medicinemedicineExtracellularlow pHCalcium dependenceAnimalsChelationRats WistarNeurotransmitterIonophoresCell BiologyHydrogen-Ion ConcentrationRatsNeuroprotective AgentsEndocrinologychemistryNigericinSettore BIO/14 - Farmacologiadopamine releaseSuperfused synaptosome[3H]-DA outflowSettore MED/26 - NeurologiaCalciumProtonsExtracellular SpaceSynaptosomesmedicine.drugNeurochemistry International
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Dietary phytoestrogens improve stroke outcome after transient focal cerebral ischemia in rats.

2006

As phytoestrogens are postulated as being neuroprotectants, we assessed the hypothesis that dietary isoflavone-type phytoestrogens are neuroprotective against ischemic stroke. Transient focal cerebral ischemia (90 min) was induced by middle cerebral artery occlusion (MCAO) following the intraluminal thread technique, both in rats fed with soy-based diet and in rats fed with isoflavone-free diet. Cerebro-cortical laser-Doppler flow (cortical perfusion, CP), arterial blood pressure, core temperature, PaO2, PaCO2, pH and glycemia were measured before, during and after MCAO. Neurological examination and infarct volume measurements were carried out 3 days after the ischemic insult. Dietary isofl…

Malemedicine.medical_specialtyTime FactorsIschemiaNeurological examinationPhytoestrogensNeuroprotectionchemistry.chemical_compoundInternal medicineStroke outcomemedicineLaser-Doppler FlowmetryAnimalsRats WistarChromatography High Pressure LiquidNeurologic Examinationmedicine.diagnostic_testbusiness.industryGeneral NeuroscienceNeurological statusBody WeightCerebral InfarctionIsoflavonesmedicine.diseaseDietRatsStrokeDisease Models AnimalBlood pressureEndocrinologyNeuroprotective AgentschemistryIschemic Attack TransientAnesthesiaReperfusionPhytoestrogensbusinessThe European journal of neuroscience
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Pravastatin treatment causes a shift in the balance of hippocampal neurotransmitter binding densities towards inhibition

2009

Since pravastatin, a HMG-CoA reductase inhibitor, has recently been shown to reduce infarct volumes and glutamate release in a rat model of ischemic stroke, the aim of the present study was to investigate whether this neuroprotective effect may be due to a modulation of excitatory and inhibitory neurotransmitter receptors. Therefore, Wistar rats were treated six times in 4 days with pravastatin or saline and allowed to survive for 6 hours or 5 days (n=10 per time point and group), respectively. Using quantitative receptor autoradiography, ligand binding densities of [(3)H]MK-801, [(3)H]AMPA, and [(3)H]muscimol for labeling of NMDA, AMPA, and GABA(A) receptors were analyzed in sensorimotor c…

Malemedicine.medical_specialtyTime FactorsKainate receptorAMPA receptorBiologyPharmacologyHippocampusReceptors N-Methyl-D-AspartateNeurotransmitter bindingRandom Allocationchemistry.chemical_compoundInternal medicinemedicineAnimalsReceptors AMPARats WistarLong-term depressionMolecular Biology5-HT receptorPravastatinCerebral CortexNeurotransmitter AgentsGABAA receptorGeneral NeuroscienceGlutamate receptorReceptors GABA-ACorpus StriatumRatsNeuroprotective AgentsEndocrinologynervous systemMuscimolchemistryNeurology (clinical)Developmental BiologyBrain Research
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Early-onset tolerance in rat global cerebral ischemia induced by a mitochondrial inhibitor

2000

It was studied whether a subtoxic dose of the mitochondrial neurotoxin, 3-nitropropionic acid (3-NPA), can initiate early-onset tolerance induction for subsequent ischemic injury. Wistar rats were pretreated for 3 h by intraperitoneal 3-NPA (20 mg/kg body weight; n=13) or solvent (n=12). Fifteen minutes global cerebral ischemia was induced by bilateral carotid artery occlusion and hypobaric hypotension. rCBF and tissue hemoglobin oxygen saturation were measured by laser Doppler scanning and a microspectrophotometric method. Ischemic insult and brain temperature were identical in both groups. Body weight and neurological scores recovered in the pretreated group but further deteriorated in th…

Malemedicine.medical_specialtyTime FactorsNeurotoxinsIschemiaConvulsantsMotor ActivityHippocampal formationBrain IschemiaCentral nervous system diseaseBrain ischemiaProsencephalonInternal medicinemedicineAnimalsNeurotoxinRats WistarNeuronsNeocortexbusiness.industryGeneral NeuroscienceNitro Compoundsmedicine.diseaseMitochondriaRatsTolerance inductionNeuroprotective Agentsmedicine.anatomical_structureEndocrinologyReperfusion InjuryAnesthesiaPropionatesbusinessReperfusion injuryNeuroscience Letters
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Relationship between adherence to the Mediterranean Diet, intracerebral hemorrhage, and its location

2018

INTRODUCTION: Although some authors evaluated the relationship between adherence to the Mediterranean Diet (MeDi) and both ischemic and hemorrhagic stroke, hemorrhagic stroke alone is not yet examined. AIMS: We conducted a retrospective study to evaluate the relationship between adherence to MeDi and intracerebral hemorrhage (ICH) and different locations of ICH (ganglionic/internal capsule, brainstem/cerebellum, or lobar). METHODS: We analyzed charts and collected data of all consecutive patients with ICH admitted to our Internal Medicine Ward from 2005 to 2014. A scale indicating the degree of adherence to the traditional MeDi Score was constructed. RESULTS: When compared with 100 subjects…

Malemedicine.medical_specialtyTime FactorsSettore MED/09 - Medicina InternaInternal capsuleMediterranean dietEndocrinology Diabetes and MetabolismMedicine (miscellaneous)030209 endocrinology & metabolism030204 cardiovascular system & hematologyDiet MediterraneanRisk Assessment03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicinePrevalenceHumansMedicineIn patientcardiovascular diseasesStrokeAgedCerebral HemorrhageRetrospective StudiesAged 80 and overIntracerebral hemorrhageNutrition and DieteticsProteinuriabusiness.industryRetrospective cohort studyMiddle AgedProtective FactorsPrognosismedicine.diseaseItalyMediterranean Diet ICHFemaleDiet Healthymedicine.symptomCardiology and Cardiovascular MedicinebusinessRisk Reduction BehaviorDyslipidemiaNutrition, Metabolism and Cardiovascular Diseases
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Pioglitazone Reduces Secondary Brain Damage after Experimental Brain Trauma by PPAR-γ-Independent Mechanisms

2011

Inflammatory and ischemic processes contribute to the development of secondary brain damage after mechanical brain injury. Recent data suggest that thiazolidinediones (TZDs), a class of drugs approved for the treatment of non-insulin-dependent diabetes mellitus, effectively reduces inflammation and brain lesion by stimulation of the peroxisome proliferator-activated receptor-γ (PPAR-γ). The present study investigates the influence of the TZD pioglitazone and rosiglitazone on inflammation and secondary brain damage after experimental traumatic brain injury (TBI). A controlled cortical impact (CCI) injury was induced in male C57BL/6 mice to investigate following endpoints: (1) mRNA expression…

Malemedicine.medical_specialtyTraumatic brain injuryPeroxisome proliferator-activated receptorInflammationStimulationBrain damageMiceDiabetes mellitusInternal medicinemedicineAnimalsHypoglycemic Agentschemistry.chemical_classificationPioglitazonebusiness.industrymedicine.diseaseMice Inbred C57BLPPAR gammaDisease Models AnimalNeuroprotective AgentsEndocrinologychemistryBrain InjuriesBrain Damage ChronicThiazolidinedionesNeurology (clinical)medicine.symptombusinessRosiglitazonePioglitazonemedicine.drugJournal of Neurotrauma
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Primary Radical Prostatectomy or Ablative Radiotherapy as Protective Factors for Patients With mCRPC Treated With Radium-223 Dichloride: An Italian M…

2020

Abstract Background We investigated, in a real-life setting, the prognostic relevance of previous primary treatment (radical prostatectomy [RP] or external beam radiotherapy [EBRT]) on overall survival for patients with metastatic castration-resistant prostate cancer (mCRPC) treated with radium-223 (223Ra). Materials and Methods In the present multicenter retrospective study, we enrolled 275 consecutive patients. The demographic and clinical data and mCRPC characteristics were recorded and evaluated at baseline and at the end of treatment or progression. 223Ra was administered according to the current label authorization until disease progression or unacceptable toxicity. We divided the who…

Malemedicine.medical_specialtyUrologymedicine.medical_treatmentoverall survivalBrachytherapy030232 urology & nephrologyUrology223-ra03 medical and health sciencesProstate cancer0302 clinical medicineAblative casemedicineHumansExternal beam radiotherapyradiotherapyAgedRetrospective StudiesAged 80 and overProstatectomyRadioisotopesbusiness.industryProstatectomyRetrospective cohort studymCRPCMiddle AgedProtective Factorsmedicine.diseasePrognosisCombined Modality TherapyRadiation therapySurvival RateProstatic Neoplasms Castration-ResistantOncologyMulticenter study223-ra; mCRPC; prostatectomy; radiotherapy; overall survivalItaly030220 oncology & carcinogenesisCohortDisease ProgressionbusinessSettore SECS-S/01Follow-Up StudiesRadium
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