Search results for "Protein Multimerization"

showing 10 items of 99 documents

Wild-type Cu/Zn superoxide dismutase stabilizes mutant variants by heterodimerization

2014

Mutations in the gene encoding Cu/Zn superoxide dismutase (SOD1) are responsible for a subset of amyotrophic lateral sclerosis cases presumably by the acquisition of as yet unknown toxic properties. Additional overexpression of wild-type SOD1 in mutant SOD1 transgenic mice did not improve but rather accelerated the disease course. Recently, it was documented that the presence of wild-type SOD1 (SOD(WT)) reduced the aggregation propensity of mutant SOD1 by the formation of heterodimers between mutant and SOD1(WT) and that these heterodimers displayed at least a similar toxicity in cellular and animal models. In this study we investigated the biochemical and biophysical properties of obligate…

Genetically modified mouseanimal diseasesMutantSOD1HeterodimerizationPeptideBiologyProtein aggregationlcsh:RC321-571Superoxide Dismutase-1Humanslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryGenechemistry.chemical_classificationMisfoldingSuperoxide DismutaseWild typenutritional and metabolic diseasesSOD1Molecular biologynervous system diseasesHEK293 Cellsnervous systemNeurologychemistryBiochemistryDismutase activityMutationDismutaseProtein aggregationProtein MultimerizationMutant homodimersNeurobiology of Disease
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Thermal aggregation of glycated bovine serum albumin

2010

International audience; Aggregation and glycation processes in proteins have a particular interest in medicine fields and in food technology. Serum albumins are model proteins which are able to self-assembly in aggregates and also sensitive to a non-enzymatic glycation in cases of diabetes. In this work, we firstly reported a study on the glycation and oxidation effects on the structure of bovine serum albumin (BSA). The experimental approach is based on the study of conformational changes of BSA at secondary and tertiary structures by FTIR absorption and fluorescence spectroscopy, respectively. Secondly, we analysed the thermal aggregation process on BSA glycated with different glucose con…

Glycation End Products AdvancedGlycosylationHot TemperatureGlycoxidation02 engineering and technologyProtein aggregationBiochemistryProtein Structure SecondaryAnalytical Chemistrychemistry.chemical_compoundProtein structureGlycationSpectroscopy Fourier Transform InfraredScattering RadiationGlycated Serum AlbuminBovine serum albuminGlycation0303 health sciencesbiologyChemistryTryptophanSerum Albumin Bovine021001 nanoscience & nanotechnology3. Good healthSpectrophotometryProtein aggregation0210 nano-technologyOxidation-ReductionGlycosylationBiophysicsSerum albuminIn Vitro Techniques03 medical and health sciencesAnimalsMolecular BiologySerum Albumin030304 developmental biologyChromatographyAlbuminAlbuminLight scatteringSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)Protein tertiary structureProtein Structure TertiaryKineticsFTIR spectroscopyGlucoseSpectrometry FluorescenceUnfolded Protein Responsebiology.proteinCattleProtein Multimerization[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyBiochimica et Biophysica Acta (BBA) - Proteins and Proteomics
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The GlpF residue Trp219 is part of an amino-acid cluster crucial for aquaglyceroporin oligomerization and function

2018

The vestibule loop regions of aquaglyceroporins are involved in accumulation of glycerol inside the channel pore. Even though most loop regions do not show high sequence similarity among aquaglyceroporins, loop E is highly conserved in aquaglyceroporins, but not in members of the homologous aquaporins. Specifically, a tryptophan residue is extremely conserved within this loop. We have investigated the role of this residue (Trp219) that deeply protrudes into the protein and potentially interacts with adjacent loops, using the E. coli aqualgyeroporin GlpF as a model. Replacement of Trp219 affects the activity of GlpF and impairs the stability of the tetrameric protein. Furthermore, we have id…

GlycerolModels Molecular0301 basic medicineProtein ConformationTetrameric proteinBiophysicsAquaporinAquaporinsBiochemistry03 medical and health sciencesResidue (chemistry)TetramerEscherichia coliAmino Acidschemistry.chemical_classification030102 biochemistry & molecular biologyProtein StabilityChemistryEscherichia coli ProteinsTryptophanTryptophanCell BiologyAmino acid030104 developmental biologyAquaglyceroporinsBiochemistryMutationBiophysicsProtein foldingProtein MultimerizationAquaglyceroporinsBiochimica et Biophysica Acta (BBA) - Biomembranes
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Ligation Tunes Protein Reactivity in an Ancient Haemoglobin: Kinetic Evidence for an Allosteric Mechanism in Methanosarcina acetivorans Protoglobin

2012

Abstract: Protoglobin from Methanosarcina acetivorans (MaPgb) is a dimeric globin with peculiar structural properties such as a completely buried haem and two orthogonal tunnels connecting the distal cavity to the solvent. CO binding to and dissociation from MaPgb occur through a biphasic kinetics. We show that the heterogenous kinetics arises from binding to (and dissociation from) two tertiary conformations in ligation-dependent equilibrium. Ligation favours the species with high binding rate (and low dissociation rate). The equilibrium is shifted towards the species with low binding (and high dissociation) rates for the unliganded molecules. A quantitative model is proposed to describe t…

HEME ENVIRONMENTStereochemistrySILICA-GELSArchaeal ProteinsAllosteric regulationKineticsBiophysicslcsh:MedicinePlasma protein bindingBiochemistryDissociation (chemistry)HemoglobinsAllosteric RegulationBINDINGINTERNAL HYDROPHOBIC CAVITIESMoleculeGlobinFerrous CompoundsMethanosarcina acetivoransSettore BIO/10lcsh:ScienceBiologyT STATE HEMOGLOBINCarbon MonoxideMultidisciplinaryPhotolysisbiologyChemistryPhysicslcsh:RProteinsMethanosarcinabiology.organism_classificationRecombinant ProteinsEnzymesGlobinsKineticsOXYGEN-AFFINITYBiochemistryMethanosarcinaARABIDOPSIS-THALIANAlcsh:QGLOBIN-COUPLED SENSORSHuman medicineProtein MultimerizationLIGAND MIGRATIONNEUROGLOBINResearch ArticleProtein Binding
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Modification of the major tegument protein pp65 of human cytomegalovirus inhibits virus growth and leads to the enhancement of a protein complex with…

2010

The tegument protein pp65 of human cytomegalovirus (HCMV) is abundant in lytically infected human foreskin fibroblasts (HFF), as well as in virions and subviral dense bodies (DB). Despite this, we showed previously that pp65 is dispensable for growth in HFF. In the process of refining a DB-based vaccine candidate, different HCMV mutants were generated, expressing a dominant HLA-A2-presented peptide of the IE1 protein fused to pp65. One of the mutant viruses (RV-VM1) surprisingly showed marked impairment in virus release from HFF. We hypothesized that analysis of the phenotypic alterations of RV-VM1 would provide insight into the functions of pp65, poorly defined thus far. RV-VM1 infection r…

Human cytomegalovirusImmunoprecipitationvirusesMutantCytomegalovirusBiologyVirus ReplicationVirusInclusion bodiesViral Matrix ProteinsViral ProteinsVirologymedicineHumansImmunoprecipitationCells Culturedvirus diseasesRNAViral tegumentFibroblastsPhosphoproteinsmedicine.diseaseVirologyFusion proteinTrans-ActivatorsProtein MultimerizationProtein BindingJournal of General Virology
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The viral chemokine MCK-2 of murine cytomegalovirus promotes infection as part of a gH/gL/MCK-2 complex.

2013

Human cytomegalovirus (HCMV) forms two gH/gL glycoprotein complexes, gH/gL/gO and gH/gL/pUL(128,130,131A), which determine the tropism, the entry pathways and the mode of spread of the virus. For murine cytomegalovirus (MCMV), which serves as a model for HCMV, a gH/gL/gO complex functionally homologous to the HCMV gH/gL/gO complex has been described. Knock-out of MCMV gO does impair, but not abolish, virus spread indicating that also MCMV might form an alternative gH/gL complex. Here, we show that the MCMV CC chemokine MCK-2 forms a complex with the glycoprotein gH, a complex which is incorporated into the virion. We could additionally show that mutants lacking both, gO and MCK-2 are not ab…

Human cytomegalovirusViral DiseasesMuromegalovirusChemokinevirusesMurine Cytomegalovirus ; viral chemokine MCK-2 ; gH/gL/MCK-2 complexMiceViral Envelope ProteinsBiology (General)Cells Culturedchemistry.chemical_classificationMice Inbred BALB Cvirus diseasesHerpesviridae InfectionsRecombinant ProteinsSpecific Pathogen-Free OrganismsInfectious DiseasesLiverChemokines CCMedicineFemaleResearch ArticleQH301-705.5ImmunologyBiologyMicrobiologyVirusCell LineViral ProteinsMuromegalovirusGlycoprotein complexVirologyGeneticsmedicineAnimalsBiologyMolecular BiologyTropismMacrophagesVirionVirus InternalizationRC581-607medicine.diseasebiology.organism_classificationVirologyImmunity InnatechemistryCell cultureMutationMacrophages Peritonealbiology.proteinParasitologyProtein MultimerizationImmunologic diseases. AllergyGlycoprotein
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In situ structural analysis of SARS-CoV-2 spike reveals flexibility mediated by three hinges

2020

Flexible spikes The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spike protein enables viral entry into host cells by binding to the angiotensin-converting enzyme 2 (ACE2) receptor and is a major target for neutralizing antibodies. About 20 to 40 spikes decorate the surface of virions. Turoňová et al. now show that the spike is flexibly connected to the viral surface by three hinges that are well protected by glycosylation sites. The flexibility imparted by these hinges may explain how multiple spikes act in concert to engage onto the flat surface of a host cell. Science, this issue p. 203

In situElectron Microscope TomographyGlycanGlycosylationFlexibility (anatomy)virusesProtein domainPneumonia ViralHingeMolecular Dynamics SimulationBiologylaw.inventionBetacoronavirusProtein DomainslawTarget identificationmedicineHumansPandemicsResearch ArticlesHost cell surfaceMultidisciplinarySARS-CoV-2R-ArticlesCryoelectron MicroscopyBiochemCOVID-19MicrobioResearch HighlightCell biologymedicine.anatomical_structureSpike Glycoprotein Coronavirusbiology.proteinRecombinant DNASpike (software development)Protein MultimerizationStructural biologyCoronavirus InfectionsResearch ArticleScience (New York, N.y.)
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Study of the bacillus thuringiensis Cry1Ia protein oligomerization promoted by midgut brush border membrane vesicles of lepidopteran and coleopteran …

2020

Bacillus thuringiensis (Bt) produces insecticidal proteins that are either secreted during the vegetative growth phase or accumulated in the crystal inclusions (Cry proteins) in the stationary phase. Cry1I proteins share the three domain (3D) structure typical of crystal proteins but are secreted to the media early in the stationary growth phase. In the generally accepted mode of action of 3D Cry proteins (sequential binding model), the formation of an oligomer (tetramer) has been described as a major step, necessary for pore formation and subsequent toxicity. To know if this could be extended to Cry1I proteins, the formation of Cry1Ia oligomers was studied by Western blot, after the incuba…

Leptinotarsa decemlineataBrush borderHealth Toxicology and MutagenesisBacillus thuringiensislcsh:MedicineSf21 cell lineOstrinia nubilalisToxicologyOligomer formationHemolysin Proteins<i>leptinotarsa decemlineata</i>03 medical and health sciencesWestern blotBacillus thuringiensisLobesia botranaSf9 CellsmedicineAnimalsProtein oligomerizationCry1AbIncubation<i>ostrinia nubilalis</i>030304 developmental biology0303 health sciencesBinding SitesBacillus thuringiensis ToxinsMicrovillimedicine.diagnostic_testbiology030306 microbiologyChemistryCommunicationVesiclelcsh:RfungiMembrane ProteinsMidgut<i>lobesia botrana</i>Trypsinbiology.organism_classificationColeopteraEndotoxinsLepidopteraBiochemistryBioassayProtein MultimerizationProtein Bindingmedicine.drug
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The Hsc/Hsp70 Co-Chaperone Network Controls Antigen Aggregation and Presentation during Maturation of Professional Antigen Presenting Cells

2011

The maturation of mouse macrophages and dendritic cells involves the transient deposition of ubiquitylated proteins in the form of dendritic cell aggresome-like induced structures (DALIS). Transient DALIS formation was used here as a paradigm to study how mammalian cells influence the formation and disassembly of protein aggregates through alterations of their proteostasis machinery. Co-chaperones that modulate the interplay of Hsc70 and Hsp70 with the ubiquitin-proteasome system (UPS) and the autophagosome-lysosome pathway emerged as key regulators of this process. The chaperone-associated ubiquitin ligase CHIP and the ubiquitin-domain protein BAG-1 are essential for DALIS formation in mou…

Macromolecular AssembliesImmune CellsCellular differentiationImmunologyAntigen presentationAntigen-Presenting Cellslcsh:MedicineAntigen Processing and RecognitionMajor histocompatibility complexBiochemistryMiceMolecular Cell BiologyMHC class IAutophagyAnimalsHSP70 Heat-Shock ProteinsAntigensProtein Interactionslcsh:ScienceAntigen-presenting cellBiologyImmune ResponseCellular Stress ResponsesAntigen PresentationMultidisciplinarybiologylcsh:RHSC70 Heat-Shock ProteinsImmunityProteinsCell DifferentiationDendritic cellChaperone ProteinsUbiquitin ligaseCell biologyProteostasisbiology.proteinlcsh:QProtein MultimerizationResearch ArticlePLoS ONE
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Oxidation Enhances Human Serum Albumin Thermal Stability and Changes the Routes of Amyloid Fibril Formation

2014

Oxidative damages are linked to several aging-related diseases and are among the chemical pathways determining protein degradation. Specifically, interplay of oxidative stress and protein aggregation is recognized to have a link to the loss of cellular function in pathologies like Alzheimer's and Parkinson's diseases. Interaction between protein and reactive oxygen species may indeed induce small changes in protein structure and lead to the inhibition/modification of protein aggregation process, potentially determining the formation of species with different inherent toxicity. Understanding the temperate relationship between these events can be of utmost importance in unraveling the molecul…

Macromolecular AssembliesProtein Foldinglcsh:MedicineProtein aggregationBiochemistryPhysical Chemistry01 natural sciencesProtein Structure SecondaryProtein structurePathologylcsh:Sciencechemistry.chemical_classification0303 health sciencesMultidisciplinarybiologyProtein StabilityChemistryPhysicsNeurodegenerationTemperatureNeurodegenerative DiseasesHuman serum albuminChemistryNeurologyBiochemistryMedicineOxidation-ReductionMolecular PathologyResearch Articlemedicine.drugAmyloidBiophysicsSerum albuminProtein degradation010402 general chemistry03 medical and health sciencesDiagnostic MedicinemedicineHumansProtein InteractionsBiologySerum Albumin030304 developmental biologyAmyloid Fluorescence Oxidation Protein aggregation Spectoscopy Light Scattering Serum AlbuminReactive oxygen specieslcsh:RProteinsHydrogen Peroxidemedicine.diseaseProtein tertiary structure0104 chemical sciencesKineticsbiology.proteinlcsh:QProtein MultimerizationGeneral Pathology
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