Search results for "Psychopharmacology"

showing 10 items of 24 documents

Is erythropoietin a worthy candidate for traumatic brain injury or are we heading the wrong way?

2016

Traumatic brain injury (TBI) is a leading cause of death and disability in the modern society. Although primary prevention is the only strategy that can counteract the primary brain damage, numerous preclinical studies have been accumulated in order to find therapeutic strategies against the secondary damage. In this scenario erythropoietin (EPO) has been shown to be a promising candidate as neuroprotective agent. A recent clinical trial, however, has shown that EPO has not an overall effect on outcomes following TBI thus renewing old concerns.  However, the results of a prespecified sensitivity analysis indicate that the effect of EPO on mortality remains still unclear. In the light of the…

0301 basic medicinemedicine.medical_specialtyMolecular PharmacologyNeuropharmacology & PsychopharmacologyTraumatic brain injurySolid baseBrain damageNeuroprotectionGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesTraumatic brain injury0302 clinical medicinePrimary preventionmedicineGeneral Pharmacology Toxicology and PharmaceuticsIntensive care medicineErythropoietin; Neuroprotection; Traumatic brain injuryErythropoietinCause of deathGeneral Immunology and Microbiologybusiness.industryArticlesGeneral MedicineOpinion Articlemedicine.diseaseNeuroprotectionClinical trial030104 developmental biologyErythropoietinmedicine.symptombusinessNeuroscience030217 neurology & neurosurgerymedicine.drugF1000Research
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A History of the Pharmacological Treatment of Bipolar Disorder.

2018

In this paper, the authors review the history of the pharmacological treatment of bipolar disorder, from the first nonspecific sedative agents introduced in the 19th and early 20th century, such as solanaceae alkaloids, bromides and barbiturates, to John Cade’s experiments with lithium and the beginning of the so-called “Psychopharmacological Revolution” in the 1950s. We also describe the clinical studies and development processes, enabling the therapeutic introduction of pharmacological agents currently available for the treatment of bipolar disorder in its different phases and manifestations. Those drugs include lithium salts, valproic acid, carbamazepine, new antiepilep…

Farmacología veterinariamedicine.drug_classPsychopharmacologyFarmacologíaantipsychotic drugsAtypical antipsychoticCariprazineReviewPharmacologyLamotrigineLithiumHistory 21st CenturyCatalysisTreatment of bipolar disorderInorganic Chemistrylcsh:Chemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicinepharmacological treatmentmedicineAsenapineAnimalsHumansZiprasidoneantiepileptic drugsPhysical and Theoretical ChemistryMolecular Biologylcsh:QH301-705.5SpectroscopyPsiquiatríaLurasidonebipolar disorderbusiness.industrymood stabilizer drugsOrganic ChemistryHistory 19th CenturyGeneral MedicineHistory 20th Century030227 psychiatryComputer Science ApplicationsTranquilizing Agentschemistrylcsh:Biology (General)lcsh:QD1-999Quetiapinebusiness030217 neurology & neurosurgerymedicine.drug
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The Hamilton Depression Scale and Its Alternatives: A Comparison of Their Reliability and Validity

1990

The efficacy of medical treatments is evidenced by a demonstrated reduction in the severity of the disorder being treated relative to a control condition. At the beginning of the era of psychopharmacology, in the late 1950s, neither a well-defined concept nor a well-defined measurement of the severity of depression, schizophrenia, or anxiety disorders were available. Consequently, it was difficult to compare groups of treated patients on the basis of treatment-specific rates of recovery. Hamilton was one of the first to recognize this lack and to create methods for standardizing the measurement of the effects of drugs across both patients and treatments. His idea was that a standardized mea…

Hamilton depression scalebusiness.industryMEDLINEmedicine.diseaseSchizophreniaEndogenous depressionmedicineAnxietyPsychopharmacologymedicine.symptombusinessDepression (differential diagnoses)Reliability (statistics)Clinical psychology
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CNTN6 mutations are risk factors for abnormal auditory sensory perception in autism spectrum disorders

2017

International audience; Contactin genes CNTN5 and CNTN6 code for neuronal cell adhesion molecules that promote neurite outgrowth in sensory-motor neuronal pathways. Mutations of CNTN5 and CNTN6 have previously been reported in individuals with autism spectrum disorders (ASDs), but very little is known on their prevalence and clinical impact. In this study, we identified CNTN5 and CNTN6 deleterious variants in individuals with ASD. Among the carriers, a girl with ASD and attention-deficit/hyperactivity disorder was carrying five copies of CNTN5. For CNTN6, both deletions (6/1534 ASD vs 1/8936 controls; P=0.00006) and private coding sequence variants (18/501 ASD vs 535/33480 controls; P=0.000…

Male0301 basic medicinegenetic structuresAutism Spectrum Disorder[ SDV.MHEP.PSM ] Life Sciences [q-bio]/Human health and pathology/Psychiatrics and mental health[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.MHEP.PSM] Life Sciences [q-bio]/Human health and pathology/Psychiatrics and mental healthmedicine.disease_causeChild[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsGeneticsMutationPsychiatry and Mental healthSchizophrenia[ SDV.NEU.NB ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.SP.PHARMA] Life Sciences [q-bio]/Pharmaceutical sciences/Pharmacology[ SCCO.NEUR ] Cognitive science/NeuroscienceAuditory PerceptionMedical geneticsOriginal ArticleFemalePsychopharmacologymedicine.symptomPsychologyAdultmedicine.medical_specialtyAdolescentDNA Copy Number Variations[SDV.GEN.GH] Life Sciences [q-bio]/Genetics/Human geneticsPolymorphism Single Nucleotidebehavioral disciplines and activities03 medical and health sciencesCellular and Molecular NeuroscienceContactinsmental disordersmedicineHumansDementiaGenetic Predisposition to DiseaseMolecular Biology[SCCO.NEUR]Cognitive science/Neuroscience[SCCO.NEUR] Cognitive science/NeuroscienceHyperacusis[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[ SDV.SP.PHARMA ] Life Sciences [q-bio]/Pharmaceutical sciences/Pharmacologymedicine.disease030104 developmental biology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsAttention Deficit Disorder with Hyperactivity[SDV.MHEP.PSM]Life Sciences [q-bio]/Human health and pathology/Psychiatrics and mental healthMutationBehavioral medicine[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyAutismNeuroscienceMolecular Psychiatry
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Hippocampal overexpression of Nos1ap promotes endophenotypes related to mental disorders

2021

Abstract Background Nitric oxide synthase 1 adaptor protein (NOS1AP; previously named CAPON) is linked to the glutamatergic postsynaptic density through interaction with neuronal nitric oxide synthase (nNOS). NOS1AP and its interaction with nNOS have been associated with several mental disorders. Despite the high levels of NOS1AP expression in the hippocampus and the relevance of this brain region in glutamatergic signalling as well as mental disorders, a potential role of hippocampal NOS1AP in the pathophysiology of these disorders has not been investigated yet. Methods To uncover the function of NOS1AP in hippocampus, we made use of recombinant adeno-associated viruses to overexpress muri…

MaleMedicine (General)Research paperDendritic spineEndophenotypesNOS1APGene ExpressionHippocampusnNOS610 Medicine & healthNitric Oxide Synthase Type IHippocampal formationBiologyHippocampusSpatial memoryGeneral Biochemistry Genetics and Molecular BiologyMiceGlutamatergicR5-920NOS1APnitric oxideCAPONAnimalsNOS-I610 Medicine & healthAdaptor Proteins Signal TransducingMental DisordersRGeneral MedicineGlutamatergic postsynaptic densityNeuropsychopharmacologyDisease Models Animalpsychiatric disordersGene Expression Regulationnervous systemMedicineDisease SusceptibilityDisks Large Homolog 4 ProteinNeuroscienceBiomarkersProtein BindingSignal Transduction
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What users think about the differences between caffeine and illicit/prescription stimulants for cognitive enhancement

2012

Pharmacological cognitive enhancement (CE) is a topic of increasing public awareness. In the scientific literature on student use of CE as a study aid for academic performance enhancement, there are high prevalence rates regarding the use of caffeinated substances (coffee, caffeinated drinks, caffeine tablets) but remarkably lower prevalence rates regarding the use of illicit/prescription stimulants such as amphetamines or methylphenidate. While the literature considers the reasons and mechanisms for these different prevalence rates from a theoretical standpoint, it lacks empirical data to account for healthy students who use both, caffeine and illicit/prescription stimulants, exclusively f…

MaleNon-Clinical MedicinePsychopharmacologymedicine.medical_treatment610 Medizinlcsh:MedicineScientific literatureMedical LawSocial and Behavioral SciencesDrug UsersCognition610 Medical sciencesMedical SociologyHuman PerformancePsychologylcsh:ScienceNootropic AgentsProblem Solvingmedia_commonPsychiatryMultidisciplinarySubstance AbuseQualitative StudiesSubstance abuseMental HealthNeurologyHealth Education and AwarenessMedicineFemalePublic HealthBehavioral and Social Aspects of HealthResearch ArticleAdultMedical Ethicsmedicine.medical_specialtyDrugs and DevicesPrescription DrugsUniversitiesSubstance-Related DisordersClinical Research DesignScience Policymedia_common.quotation_subjectCognitive NeuroscienceDecision MakingNeuropharmacologyNeuropsychologyCaffeinemedicineHumansMedical prescriptionStudentsPsychiatryBiologyBehaviorHealth Care Policybusiness.industryIllicit DrugsAddictionlcsh:RCognitive PsychologyBioethicsmedicine.diseaseStimulantScience Educationlcsh:QCentral Nervous System StimulantsCitationAttributionbusinessLawMedical ethicsNeuroscience
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Delineation of molecular pathway activities of the chronic antidepressant treatment response suggests important roles for glutamatergic and ubiquitin…

2017

AbstractThe aim of this study was to identify molecular pathways related to antidepressant response. We administered paroxetine to the DBA/2J mice for 28 days. Following the treatment, the mice were grouped into responders or non-responders depending on the time they spent immobile in the forced swim test. Hippocampal metabolomics and proteomics analyses revealed that chronic paroxetine treatment affects glutamate-related metabolite and protein levels differentially in the two groups. We found significant differences in the expression of N-methyl-d-aspartate receptor and neuronal nitric oxide synthase proteins between the two groups, without any significant alterations in the respective tra…

MaleProteomics0301 basic medicineProteasome Endopeptidase ComplexGlutamic AcidNitric Oxide Synthase Type IPharmacologyHippocampusReceptors N-Methyl-D-AspartateMice03 medical and health sciencesCellular and Molecular NeuroscienceGlutamatergic0302 clinical medicineUbiquitinmedicineAnimalsHumansMetabolomicsReceptorSwimmingBiological PsychiatryDepressive Disorder MajorbiologyUbiquitinParoxetineAntidepressive AgentsParoxetinePsychiatry and Mental health030104 developmental biologyProteasomeMice Inbred DBALeukocytes Mononuclearbiology.proteinAntidepressantOriginal ArticlePsychopharmacologyPsychology030217 neurology & neurosurgerymedicine.drugBehavioural despair testTranslational Psychiatry
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Is erythropoietin a worthy candidate for traumatic brain injury or are we heading the wrong way? [version 1; referees: 2 approved]

2016

Traumatic brain injury (TBI) is a leading cause of death and disability in the modern society. Although primary prevention is the only strategy that can counteract the primary brain damage, numerous preclinical studies have been accumulated in order to find therapeutic strategies against the secondary damage. In this scenario erythropoietin (EPO) has been shown to be a promising candidate as neuroprotective agent. A recent clinical trial, however, has shown that EPO has not an overall effect on outcomes following TBI thus renewing old concerns.  However, the results of a prespecified sensitivity analysis indicate that the effect of EPO on mortality remains still unclear. In the light of the…

Molecular PharmacologyNeuropharmacology & Psychopharmacologylcsh:Rlcsh:Medicinelcsh:Qlcsh:ScienceF1000Research
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Nanoparticulate Systems for Drug Delivery and Targeting to the Central Nervous System

2010

Brain delivery is one of the major challenges for the neuropharmaceutical industry since an alarming increase in brain disease incidence is going on. Despite major advances in neuroscience, many potential therapeutic agents are denied access to the central nervous system (CNS) because of the existence of a physiological low permeable barrier, the blood-brain barrier (BBB). To obtain an improvement of drug CNS performance, sophisticated approaches such as nanoparticulate systems are rapidly developing. Many recent data demonstrate that drugs could be transported successfully into the brain using colloidal systems after i.v. injection by several mechanisms such as endocytosis or P-glycoprotei…

Movement disorders/Parkinson’s diseaseDrug CarriersPolymersSurface PropertiesReviewsBrainAlzheimer's diseaseMultiple sclerosisDrug Delivery SystemsMovement disorders/Parkinson's diseaseSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoLiposomesNeuropsychopharmacology.AnimalsHumansNanoparticlesMultiple sclerosiParticle SizeNeuropsychopharmacologyAlzheimer’s diseaseMicellesCentral Nervous System Agents
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Nanoparticulate Systems for Drug Delivery and Targeting to the Central Nervous System

2010

Brain delivery is one of the major challenges for the neuropharmaceutical industry since an alarming increase in brain disease incidence is going on. Despite major advances in neuroscience, many potential therapeutic agents are denied access to the central nervous system (CNS) because of the existence of a physiological low permeable barrier, the blood-brain barrier (BBB). To obtain an improvement of drug CNS performance, sophisticated approaches such as nanoparticulate systems are rapidly developing. Many recent data demonstrate that drugs could be transported successfully into the brain using colloidal systems after i.v. injection by several mechanisms such as endocytosis or P-glycoprotei…

PharmacologyDrugLiposomebusiness.industrymedia_common.quotation_subjectCentral nervous systemPharmacologyEndocytosisBrain diseaseNeuropsychopharmacologyPsychiatry and Mental healthmedicine.anatomical_structurePhysiology (medical)Drug deliveryMedicinePharmacology (medical)businessDrug carrierNeurosciencemedia_commonCNS Neuroscience & Therapeutics
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