Search results for "Psychotomimetic"

showing 5 items of 5 documents

Association of extent of cannabis use and psychotic like intoxication experiences in a multi-national sample of first episode psychosis patients and …

2021

AbstractBackgroundFirst episode psychosis (FEP) patients who use cannabis experience more frequent psychotic and euphoric intoxication experiences compared to controls. It is not clear whether this is consequent to patients being more vulnerable to the effects of cannabis use or to their heavier pattern of use. We aimed to determine whether extent of use predicted psychotic-like and euphoric intoxication experiences in patients and controls and whether this differs between groups.MethodsWe analysed data on patients who had ever used cannabis (n = 655) and controls who had ever used cannabis (n = 654) across 15 sites from six countries in the EU-GEI study (2010–2015). We used multiple regres…

SYMPTOMSPoison control0302 clinical medicineSettore MED/48 -Scienze Infermierist. e Tecn. Neuro-Psichiatriche e Riabilitat.substance abuseApplied PsychologyPOPULATIONRISKeducation.field_of_studybiologyHuman factors and ergonomicsCannabis usePsychotomimeticSubstance abusePsychiatry and Mental healthSchizophreniaPsychotic-like experiencepsychotomimeticmedicine.drugmedicine.medical_specialtyDISORDERSPopulationFrequency of use03 medical and health sciencesFirst episode psychosisInjury preventionmedicineHumansAssociation (psychology)PsychiatryeducationSettore MED/25 - PsichiatriaDELUSIONAL IDEATIONEffects of cannabisMETAANALYSISCannabisCannabinoid Receptor Agonistsbusiness.industryPsychotic-like experiencesbiology.organism_classificationmedicine.disease030227 psychiatryschizophreniaMulti nationalPsychotic DisordersHallucinogensPOTENCYCannabisbusiness030217 neurology & neurosurgery
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Unconventional ligands and modulators of nicotinic receptors

2002

Evidence gathered from epidemiologic and behavioral studies have indicated that neuronal nicotinic receptors (nAChRs) are intimately involved in the pathogenesis of a number of neurologic disorders, including Alzheimer's disease, Parkinson's disease, and schizophrenia. In the mammalian brain, neuronal nAChRs, in addition to mediating fast synaptic transmission, modulate fast synaptic transmission mediated by the major excitatory and inhibitory neurotransmitters glutamate and GABA, respectively. Of major interest, however, is the fact that the activity of the different subtypes of neuronal nAChR is also subject to modulation by substances of endogenous origin such as choline, the tryptophan …

SerotoninNeuroactive steroidPsychotomimetic drugReceptors NicotinicNeurotransmissionPharmacologyBiologyKynurenic AcidLigandsInhibitory postsynaptic potentialCholineCellular and Molecular Neurosciencechemistry.chemical_compoundKynurenic acidmental disordersmedicineAnimalsHumansPhencyclidineAnestheticsAmyloid beta-PeptidesGalantamineGeneral NeuroscienceGlutamate receptorNicotinic agonistnervous systemchemistryHallucinogensSteroidsNeurosciencemedicine.drugJournal of Neurobiology
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Structural connectivity and subcellular changes after antidepressant doses of ketamine and Ro 25-6981 in the rat: an MRI and immuno-labeling study

2021

© The Author(s) 2021.

MaleHistologyDendritic spineInfralimbic cortexPrefrontal CortexNeuroimagingNeurofilamentRats Sprague-DawleyInfralimbic cortexWhite matterDorsal raphe nucleusPhenolsPiperidinesmedicineAnimalsPrefrontal cortexbiologyChemistryGeneral NeuroscienceDorsal raphe nucleusPsychotomimeticMagnetic Resonance ImagingAntidepressive AgentsRatsMyelin basic proteinMyelinizationmedicine.anatomical_structureFast-acting antidepressantbiology.proteinNMDA receptorOriginal ArticleKetamineAnatomyNeurosciencemedicine.drugBrain Structure and Function
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Vulnerability to psychotogenic effects of ketamine is associated with elevated D2/3-receptor availability.

2012

Previous positron emission tomography (PET) studies employing competition paradigms have shown either no change or substantial declines in striatal ( 11 C)-raclopride binding after challenge with psychotogenic doses of the N-methyl-D-aspartate antagonist ketamine. We sought to probe the relationship between the severity of ketamine-induced psychotic symptoms and altered dopamine D2/3 receptor availability throughout brain using the high affinity ligand ( 18 F)-fallypride (FP). PET recordings were obtained in a group of 10 healthy, young male volunteers, in a placebo condition, and in the course of an infusion with ketamine at a psychotomimetic dose. Administration of the Positive and Negati…

AdultMalemedicine.medical_specialtyPsychosisCaudate nucleusContext (language use)Genetics BehavioralYoung AdultInternal medicinemedicineHumansPharmacology (medical)Single-Blind MethodPharmacologyRaclopridePositive and Negative Syndrome ScaleReceptors Dopamine D2Receptors Dopamine D3Psychotomimeticmedicine.diseaseUp-RegulationPsychiatry and Mental healthEndocrinologyFallyprideSchizophreniaAnesthesiaPositron-Emission TomographyBenzamidesKetaminePsychologymedicine.drugProtein BindingThe international journal of neuropsychopharmacology
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Phencyclidine-induced disruption of oscillatory activity in prefrontal cortex: Effects of antipsychotic drugs and receptor ligands

2016

The non-competitive NMDA receptor (NMDA-R) antagonist phencyclidine (PCP) markedly disrupts thalamocortical activity, increasing excitatory neuron discharge and reducing low frequency oscillations (LFO, <4Hz) that temporarily group neuronal discharge. These actions are mainly driven by PCP interaction with NMDA-R in GABAergic neurons of the thalamic reticular nucleus and likely underlie PCP psychotomimetic activity. Here we report that classical (haloperidol, chlorpromazine, perphenazine) and atypical (clozapine, olanzapine, quetiapine, risperidone, ziprasidone, aripripazole) antipsychotic drugs - but not the antidepressant citalopram - countered PCP-evoked fall of LFO in the medial prefron…

Male0301 basic medicineOscillationsmedicine.drug_classDopamine AgentsAtypical antipsychoticPhencyclidineKainate receptorPharmacologyNeurotransmissionPrefrontal cortex03 medical and health scienceschemistry.chemical_compoundSerotonin Agents0302 clinical medicineHistamine AgentsmedicineAnimalsPharmacology (medical)NMDA receptor antagonistsAntipsychotic drugsRats WistarChlorpromazineEvoked PotentialsPhencyclidineBiological PsychiatryPharmacologyRacloprideAnalysis of VarianceDose-Response Relationship DrugFourier AnalysisChemistryElectroencephalographyPsychotomimeticRatsPsychiatry and Mental health030104 developmental biologyNeurologynervous systemSchizophreniaNBQXNeurology (clinical)Excitatory Amino Acid AntagonistsNeuroscience030217 neurology & neurosurgeryAntipsychotic Agentsmedicine.drug
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